Blocked bladders Flashcards

1
Q

What are uroliths made of? Why do they form?

A
  • Magnesium ammonium phosphate complex – STRUVITES
  • Normal function – phosphorus is removed in salvia and faeces
  • Incorrect diet - high grain, phosphorus and magnesium
    ◦ low roughage diet
  • High grain/low roughage = decrease saliva production
  • Phosphorus now removed by kidneys in urine
  • Increase in urine phosphorus levels
  • Phosphorus consolidates into stones that can not pass – increase in UTI’s and blockages
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2
Q

What are the early clinical signs?

A
  • Colic/abdominal pain -Lying down, Stretching, Standing, Kicking
  • Straining to urinate
  • Crystals on preputial hair
  • Blood in urine
  • Decreased urine production
  • Dribbling urine
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3
Q

What are the late clinical signs?

A
  • Loss of appetite
  • Lethargy
  • Depression
  • Abdominal swelling – bladder rupture
  • Swelling around prepuce
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4
Q

What diagnostics are available?

A
  • Clinical exam – increased HR, RR, large bladder, positive on ballotment if rupture
  • Common sites for uroliths – urethral process/veriform appendage – felt of clinical exam
    ◦ Distal sigmoid flexure
  • Blood work – increased blood urea nitrogen BUN, creatinine, potassium – normally excreted in urine. Increased muscle enzymes CK, decreased sodium and chloride, acidosis
  • US – distended bladder
  • Contrast studies – ruptured bladder
  • Xray – may be able to visualise some stones
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5
Q

How are blockages treated?

A
  • Emergency surgery
  • Milked out under sedation
  • Pain relief
  • Spasmolytics
  • Cystocentesis
  • IV fluids – uraemic, high creatatine, hyponatraemic, hypochloraemic, hyperkalaemic
  • Abx – broad spectrum – neutrophilia
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6
Q

How can you prevent blockages from ocurring?

A
  • Adequate water intake – keep water clean and change regularly
  • Increase fodder feed intake
  • Increase grass feed
  • Decrease coarse mix feed
  • Increase salt intake 2 – 5% to increase urine formation
  • Add ammonium chloride to diet 0.5 – 1% to acidify urine
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