Blocked bladders Flashcards
1
Q
What are uroliths made of? Why do they form?
A
- Magnesium ammonium phosphate complex – STRUVITES
- Normal function – phosphorus is removed in salvia and faeces
- Incorrect diet - high grain, phosphorus and magnesium
◦ low roughage diet - High grain/low roughage = decrease saliva production
- Phosphorus now removed by kidneys in urine
- Increase in urine phosphorus levels
- Phosphorus consolidates into stones that can not pass – increase in UTI’s and blockages
2
Q
What are the early clinical signs?
A
- Colic/abdominal pain -Lying down, Stretching, Standing, Kicking
- Straining to urinate
- Crystals on preputial hair
- Blood in urine
- Decreased urine production
- Dribbling urine
3
Q
What are the late clinical signs?
A
- Loss of appetite
- Lethargy
- Depression
- Abdominal swelling – bladder rupture
- Swelling around prepuce
4
Q
What diagnostics are available?
A
- Clinical exam – increased HR, RR, large bladder, positive on ballotment if rupture
- Common sites for uroliths – urethral process/veriform appendage – felt of clinical exam
◦ Distal sigmoid flexure - Blood work – increased blood urea nitrogen BUN, creatinine, potassium – normally excreted in urine. Increased muscle enzymes CK, decreased sodium and chloride, acidosis
- US – distended bladder
- Contrast studies – ruptured bladder
- Xray – may be able to visualise some stones
5
Q
How are blockages treated?
A
- Emergency surgery
- Milked out under sedation
- Pain relief
- Spasmolytics
- Cystocentesis
- IV fluids – uraemic, high creatatine, hyponatraemic, hypochloraemic, hyperkalaemic
- Abx – broad spectrum – neutrophilia
6
Q
How can you prevent blockages from ocurring?
A
- Adequate water intake – keep water clean and change regularly
- Increase fodder feed intake
- Increase grass feed
- Decrease coarse mix feed
- Increase salt intake 2 – 5% to increase urine formation
- Add ammonium chloride to diet 0.5 – 1% to acidify urine