Trauma Flashcards
What’s the approach to assessment (& simultaneous management) of a trauma patient?
Ax:
Airway & C-spine stabilisation
Breathing & ventilation assessment + oxygenation (target SpO2 >92% & PaO2 >65mmHg): utilise lung-protective ventilation
Circulation (>=2x lg-bore PIVC, art line +/- CVC (shouldn’t delay urgent surgical intervention), control haemorrhage (including limit coagulopathy, electrolyte & acid-base derangements) & maintain end-organ perfusion, FAST scan, damage control resus principles until haemorrhage arrested, consider cell salvage for OT)
Disability (neurological examination, analgesia/sedation/anaesthesia)
Exposure (hypothermia, smoke inhalation, intoxicants- undress to search for possible injury while preventing hypothermia- aim temp >=35.5degc
What’s the PaO2 & SpO2 target for trauma pts? why?
PaO2 >65mmHg
SpO2 >92% & <98%
hyperoxaemia associated with increased mortality in acutely ill patients with TBI, recent cardiac arrest or stroke
No evidence supporting high FiO2 in trauma patients
How does IO work?
there are veins draining medullary sinuses of long bones which don’t collapse in shock or hypovolemia
What are the sites & venous drainage of the various IO sites?
proximal tibia (popliteal vein)
femur (branches of femoral vein)
distal tibia/medial malleolus (great saphenous vein)
proximal humerus (axillary vein)
manubrium/upper sternum (internal mammary & azygous vein)
Which IV drugs or routine resus fluids can be administered safely via IO route? How to give a drug?
All, with same dosing as for IV administration, but the IO route may not be as effective as UL peripheral IV access for adenosine in Rx of SVT in young infants
for rapid fluid administration & viscous solutions, administer under pressure (infusion pump, pressure bag). monitor site frequently for infiltration. Infusion rates similar to a 21g PIVC are generally achieved (approx 160mL/min)
Onset of action & serum drug [] after IO infusion during CPR are comparable to IV
Flush the IO cannula before U& after each medication
what are absolute & relative contraindications to IO?
absolute:
fracture or previously penetrated bone
extremity with vascular interruption
ideally avoid if: cellulitis burns osteomyelitis osteogenesis imperfecta or osteoporosis R)- to L)- intracardiac shunts as they may be @ higher risk of cerebral fat or bone-marrow emboli
Where should be the first attempt site (unless a contraindication) for IO placement?
proximal tibial
where is IO inserted in infants/children? adults?
1-2cm (1 finger) below tibial tuberosity & up to 1cm medial on the tibial plateau
adults 3cm below inferior tip of patella & 2cm medially
prep skin
angle 10-15 degrees caudal from vertical
drill until get give (no further)
What labs are not accurate from an IO? how much waste before sample
blood oxygenation, WCC, plt, potassium, AST, ALT & ionised calcium
plt likely to be lower
bcc higher
waste of 1mL
Can marrow aspirates be used for a group & screen?
yes
can also be used for glucose, Hb, pH, pCO2, serum bicarb, Na+, Cl-, BUN, Cr, serum drug levels & cultures
What elements are assessed on the FAST scan?
pericardium (looking for hemoperricardium & tamponade)
R) & L) flank, pelvis to look for intraperitoneal free fluid
pneumothorax (in extended FAST (E-FAST))
What are the damage control resus BP goals? and after haemostasis achieved?
SBP approx 90mmHg & <=110mmHg for older adults
once haemostasis, >=90mmHg & MAP >=65mmHg
What are some dynamic parameters for guiding intraoperative fluid therapy?
systolic pressure variations- easy to manually calculate but depends on diastolic pressure & on changes in pleural pressure & hasn’t been studied in prone
pulse pressure variations- directly related to stroke volume variations. not easy to manually calculate & needs a specific device for continuous display
stroke volume variations- accurate analysis despite multiple extrasystoles, needs specific device (on transgastric LV short-axis view with TEE, hypovolaemia vs normovolaemia shows decreased LV end-diastolic area and diameter.
How to interpret respiratory variations in the arterial waveform?
if high variation, on the steep part of the Frank-Starling curve. goal= to increase cardiac output until it reaches the plateau of the Frank-Starling relationship. Lower volume status= higher systolic pressure variation (reduction in VR & CO with mechanical ventilation)
According to ATLS, what’s the maximum amount of warm 0.9% saline that could be given before blood?
1L