The Child with Fluid & Electrolyte Alterations Flashcards

1
Q

ECF Total birth weight at birth

A

45-50%

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2
Q

ECF Total birth weight at 2 years

A

30%

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3
Q

ECF Total birth weight at maturity

A

20%

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4
Q

Cerebral changes in hypertonic dehydration

A

Disturbances of consciousness
poor ability to focus attention
lethargy
increased muscle tone with hyperreflexia
hyperirritability to stimuli (tactile, auditory, bright light)
Decorticate posturting
unable to talk
Unresponsive to pain

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5
Q

Treatment of hypernatremia

A

treat by IV hypotonic fluids -> dilutes body fluids back to normal
If dehydrated, may give isotonic first to replenish volume then hypotonic to correct osmolality. Careful monitoring

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6
Q

S&S of hypernatremia

A

will be thirst, decreased urine output
decreased LOC -> confusion, lethargy, coma from shrinking of brain cells; seizures if rapid or severe; can be fatal
Increased osmolality

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7
Q

Special consideration with hypertonic solutions

A

Interstitial IV burns

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8
Q

Manifestations of Dehydration

A

Thready, rapid pulse
Dry skin & mucous membranes
Sunken fontanel
Coolness & mottling of extremities
Decreased skin turgor
Delayed capillary refill
Increased small vein filling time
Dizziness, syncope
Oliguria
Weight loss
Postural BP drop (older children)

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9
Q

Clinical manifestations are a result of

A

Decreased fluid
Body’s response to decreased fluid

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10
Q

Earliest detectable sign of dehydration

A

Tachycardia

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11
Q

how is magnitude of fluid loss described?

A

described as a % (5,10,15) & ascertained by comparison of pre-illness weight & current weight.
However, water is only 60-70% of infant weight, & adipose tissue, which contains very little water is highly variable.
More accurate to reflect acute loss in mL/kg of body weight

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12
Q

Clinical signs:

A

provide clues to extent of dehydration. Earliest detectable sign is usually tachycardia, followed by dry skin & mucous membranes, sunken fontanels, signs of circulatory failure (coolness & mottling of extremities), loss of skin elasticity, & delayed capillary filling time.

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13
Q

Dehydration caused by loss of Na+ containing fluid form body

A
  • vomiting
  • diarrhea
  • NG suction
  • hemorrhage
  • burns
  • LBW infants under radiant warmers, adrenal insufficiency
  • overuse of diuretics
  • third space accumulation
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14
Q

Etiology ECF deficit: decreased fluid volume

A

Manifestation: weight loss, sunken fontanel

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15
Q

Etiology ECF deficit: inadequate circulating blood volume to offset

A

Manifestation: postural BP drop, dizziness

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16
Q

Etiology ECF deficit: decreased intravascular volume

A

delayed cap refill, flat neck veins when supine (older children)

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17
Q

Etiology ECF deficit: Inadequate circulation to brain

A

dizziness, syncope

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18
Q

Etiology ECF deficit: Inadequate circulation to kidneys

A

oliguria

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19
Q

Etiology ECF deficit: Cardiac reflex response to decreased intravascular volume

A

Thready, rapid pulse

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20
Q

Decreased interstitial fluid volume

A

decreased skin turgor

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21
Q

Early Compensation

A

Interstitial fluid moves to vascular compartment; vasoconstriction maintains pumping pressure.
When BP is involved it is usually a late sign and more significant

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22
Q

Compensatory mechanisms

A

attempt to maintain fluid volume by adjusting to these losses. Interstitial fluid moves into vascular compartment to maintain blood volume in response to hemoconcentration & hypovolemia, & vasoconstriction of peripheral arterioles helps maintain pumping pressure.

23
Q

Late Compensation

A

BP falls - tissue hypoxia & metabolic acidosis (accumulation of lactic acid, pyruvate)
Renal compensation impaired due to reduced blood flow; urine output decreased
- ADH to conserve fluid
- Renin-angiotensin -> vasoconstriction
- Aldosterone -> Na retention & water conservation.

24
Q

Shock

A

Tachycarida, poor perfusion (skin cool & mottled, decreased cap refill), oliguria & azotemia (nitrigenous waste esp. urea), low BP (late sign & may herald onset of cardiovascular collapse)

25
Degrees of dehydration: Mild
Up to 5% weight loss Irritable (infants); thirsty
26
Degrees of dehydration: Moderate
6-9% weight loss - lethargic & sleepy; restless & irritable - decreased skin turgor; dry mucous membranes; urine dark & decreased - pulse increased & BP normal or decreased
27
Degrees of dehydration: Severe
> 10% weight loss lethargy or non-responsive, decreased BP, rapid pulse, poor skin turgor, dry mucous membranes, decrease or absent urine output
28
ECF Deficit (Dehydration)
occurs when total output of fluid exceeds total intake, regardless of underlying cause. may result from lack of intake but more often result of abnormal losses (vomiting, diarrhea), when oral intake only partially compensates for the abnormal losses. Other significant causes of dehydration are DKA & excessive burns. Dehydration - not enough fluid in ECF compartment (vascular & interstitial). Na+ generally lose with water, therefore may be hyponatremia Signs vary with severity or degree of body water deficit. Signs are result of: decreased fluid (turgor, dry mucous membranes), body's response to fluid deficit (increased HR, decreased BP) Mild -> hard to detect
29
Increased Fluid Requirements
Fever (add 12% per rise of 1 degree C) Vomiting Diarrhea High-output kidney failure Diabetes insipidus Diabetic ketoacidosis Burns Shock Tachypnea Radiant warmer Phototherapy Post-op bowel surgery
30
Normal Urine Output: Infant
2 ml/kg/h
31
Normal Urine Output: Child (>1 yr)
1 ml/kg/h
32
Normal Urine Output: Adolescent (10-19)
0.5 ml/kg/h
33
Diarrhea
Caused by abnormal intestinal water & electrolyte transport which results - dehydration - electrolyte imbalance - metabolic acidosis Intestinal mucosa of young infant is more permeable to water than that of older child, therefore, more fluids & electrolytes are lost in infant with diarrhea
34
Dehydration caused by diarrhea: patho
voluminous losses of fluid in frequent, watery stools losses with vomiting reduced fluid intake from nausea or anorexia increased insensible water losses from fever, hyperpnea continued (although diminished) renal losses
35
Electrolyte imbalance cause by diarrhea
losses of Na, Cl, K & sometimes bicarb inadequate replacement of electrolytes when hypotonic or hypertonic solutions used
36
metabolic acidosis caused by diarrhea
increased absorption of short-chain fatty acids produced in colon from bacterial fermentation of unabsorbed dietary CHO lactic acid from tissue hypoxia secondary to hypovolemia loss of bicarb in stools ketosis from fat metabolism when glycogen stores are depleted in untreated diarrhea dehydration or inadeqaute CHO intake; may result in malnutrition
37
Acute Diarrhea
Leading cause of illness in children < 5 years often caused by infectious agent (rotavirus, norwalk-like organisms, E. coli, Salmonella) may be associated with URTI or UTI, antibiotic therapy, or laxative use Usually self-limited (< 14 days) - no specific treatment occurs usually required unless dehydration occurs Spread through fecal-oral route, through contaminated water or close contact Major risk factors: lack of clean water, crowding, poor hygiene, nutritional deficiency, poor sanitation
38
Chronic Diarrhea
> 14 days Often caused by malabsorption causes, IBD, immunodeficiency, food allergy, lactose intolerance, radiation, motility disorders, endocrine causes, parasitic infestations Increase in stool frequency & increased water content Malabsorption: celiac, short bowel, lactose intolerance Motility: hirschusprung disease Parasites: Giardia
39
Factors that Predispose to Diarrhea
Age: the younger the child, the greater the susceptibility & the more severe the diarrhea Impaired health: malnourished or immunocompromised child Environment: crowding, substandard sanitation, poor facilities for preparation & storage of food, inadequate health care education
40
Therapeutic Management Goals of Acute Diarrhea
Assessment of fluid & electrolyte imbalance Rehydration Maintenance fluid therapy Reintroduction of adequate diet First-line treatment is oral rehydration therapy (ORT) - more effective, safer, less costly, & less painful than IV rehydration
41
Therapeutic Management
Oral rehydration solutions (ORS) - enhance & promote reabsorption of sodium & water - reduce vomiting, volume loss from diarrhea & duration of illness Continued feeding or reintroduction of normal diet (when child is dehydrated we want to promote eating normally and whatever they can tolerate) - lessens severity & duration of illness - Continue with BF & use ORS for replacement - lactose-free formula if usual formula not tolerated IV therapy for severe dehydration & shock (usually use saline solution with 5% dextrose. may add bicarb. Antibiotics - maybe
42
ORS
ORT is first line treatment. After rehydration, use ORS during maintenance fluid therapy by alternating ORS with water, breastmilk, lactose-free formula, or ½ strength lactose-containing formula. In older children, continue regular diet with ORS Replace ongoing stool losses 1:1 with ORS If vomiting, give small amounts of ORS frequently (5-10 mL q 1-5 mins.); may give with NGT or GT If no clinical signs of dehydration in infants, they do not need ORT
43
Diarrhea Treatment "no-nos"
- Do not encourage po clear fluids, such as fruit juices, carbonated soft drinks & jello (high in CHO & low electrolyte, high osmolality - Do not drink caffeinated pop or other drinks (caffeine is mild diuretic) - Do not give chicken or beef broth (excessive sodium & inadequate CHO) - Do not use BRAT diet (bananas, rice, applesauce, & toast or tea) - little nutritional value (low in energy & protein), high in CHO, low in electrolytes
44
What should you do when you have diarrhea
eat to what is tolerated with oral rehydration therapy
45
Nursing Care Management of Diarrhea
Education re: treatment & prevention Accurate weight Accurate intake & output Monitor IV therapy Specimen collection (sometimes they are not making urine it just looks like it) Skin care
46
Hypovolemic Shock
Complex clinical syndrome Circulatory failure (shock) - tissue perfusion that is inadequate to meet metabolic demands of body - results in cellular dysfunction & eventual organ failure
47
Physiological consequences of hypovolemic shock
1. hypotension (later sign) 2. tissue hypoxia 3. metabolic acidosis
48
Pathophysiology of Hypovolemic Shock
If hemorrhage reduces the circulating blood volume sufficiently then the compensatory mechanisms support blood circulation by increasing the heart rate and constricting the peripheral blood vessels. This response shifts the remaining blood to larger blood vessels so that the vital organs continue to be perfused. When the blood loss exceeds 20% to 25%, the child's body can no longer compensate; blood pressure falls and circulatory collapse is imminent. Very fatal in little kids
49
Hypovolemic shock: etiology
Most common type in children Reduction in circulating blood volume beyond child's physiological ability to compensate related to: - blood loss (trauma, major bleeding) - plasma losses (burns) - ECF losses (diarrhea, dehydration)
50
Hypovolemic Shock Clinical Manifestation - Early Compensated Cardiac Respiratory Neurologic Skin Renal
mild tachycardia, weak distal pulses, strong central pulses, normal BP Mild tachypnea Normal, anxious, irritable Mottled; cap refill > 2 sec; cool, clammy extremities Renal: decreased urine output; increased SG
51
Hypovolemic Shock Clinical Manifestation - Moderate Uncompensated Cardiac Respiratory Neurologic Skin Renal
Mod tachycardia; thready distal pulses, weak central pulses, decreased systolic BP mod tachypnea confusion, agitation, combative behaviour, lethargy, decreased pain response pallor, cap refill > 3 sec, dry extremities, sunken eyes Oliguria, increased SG
52
Hypovolemic Shock Clinical Manifestation - Severe Uncompensated shock Cardiac Respiratory Neurologic Skin Renal
Extreme tachycardia, decreased BP, absent distal pulses, thready central pulses Severe tachypnea Comatose state pale, cold skin, cyanosis, cap refill > 5 sec no urine output
53
Principal differentiating signs at all stages of shock are
degree of tachycardia & perfusion to extremities level of consciousness blood pressure Compensated: vital organ function maintained by intrinsic mechanisms & child's ability to compensate is effective Subtle signs initially In decompensated, signs become more obvious
54
Therapeutic Management of Hypovolemic Shock
1. Ventilation 2. Fluid administration 3. Improvement of pumping action of heart (vasopressor support) - first priority is to establish airway & administer oxygen - place one or more multi-lumen central lines or cutdown or intraosseous needle - preferably place central line above diaphragm to deliver drugs closer to heart & limit tissue injury from caustic meds - lines needed for rapid volume replacement, administration of drugs, and hemodynamic monitoring.