thalamus, limbics, and cerebellum Flashcards
VPL of thalamus: input, info, and destination
input: spinothalamic and dorsal columns/medial lemniscus.
info: pain and temp, pressure, touch, vibration, and proprioception
goes to the primary somatosensory cortex
VPM of thalamus: input, info, and destination
input: tigeminal and gustatory pathways
info: face sensation and taste
goes to the primary somatosensory cortex
LGN: input, info and destination
input: CN II. input: vision
destination: calcarine sulcus. lateral = light
MGN: input, info, and destination
input: superior olive and inferior colliculus of the tectum
gives info about hearing
goes to the auditory cortex of the temporal lobe
(medial = music)
nucleus VL of the thalamus: input, info, and destination
input: basal ganglia, cerebellum
gives motor info
goes to the motor cortex
What are the structures of the limbic system?
hippocampus, amygdala, fornix, mammillary bodies, and cingulate gyrus.
What does the limbic system do?
feeding, fleeing, fighting, feeling, sex
What is the input to the cerebellum?
contralateral cortex via the middle cerebellar peduncle
ipsilateral proprioceptive info via inferior cerebellar peduncle from the spinal cord. these are climbing and mossy fibers
What is the output of the cerebellum?
sends info to the contralateral cortex to modulate movement.
output nerves: purkinje cells to the deep nuclei of the cerebelum to the contralateral cortex via the superior cerebellar peducnle.`
What are the deep nuclei of the cerebellum?
dentate, emboliform, globose, fastigial ( lateral to medial)
Manifestations of lateral lesions to the cerebellum? What side will a patient fall to?
affects the voluntary movements of the extremities. When the cerebellum is injured, the patient is likely to fall toward the injured side
Manifestations of medial lesions to the cerebellum
vermis and fastigial nuclei lesions cause truncal ataxia
floculonodular lobe causes nystagmus, head tilting
patients may have a wide-based gait and deficits in truncal coordination. midline lesions result in bilateral motor deficits of the axial and proximal limb musculature.
