Test I (SECTION IV) Adaptions to Chronic Endurance Exercise Flashcards

1
Q

Resting
VO2 = HR x SV x AVO2diff
what happens to each and why?

A

NC VO2
decrease HR (time in diastole)
increase SV (increase preload, decrease afterload, increase ventricle size, increase blood volume)
NC AVO2diff

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2
Q

Submax Workload
VO2 = HR x SV x AVO2diff
what happens to each?

A

NC VO2
decrease HR
increase SV
NC AVO2diff

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3
Q

Max Workload
VO2 = HR x SV x AVO2diff
what happens to each?

A

Increase VO2
NC (sometimes decrease) HR
increase SV
Increase AVO2diff

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4
Q

MAP Training Adaption

A

small decrease at rest or during exercise

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5
Q

Blood pressure Training Adaption

A

small decrease at rest

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6
Q

Total Peripheral Resistance and Afterload Training Adaption

A

increase capillarization , decrease TPR/afterload

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7
Q

Respiratory Rate (rest, submax and max) Training Adaption

A

rest: NC
Submax: decrease slightly (air remains in lungs longer)
Max: increase

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8
Q

Tidal Volume (rest, submax and max) Training Adaption

A

rest: NC
Submax: increase significantly
Max: increase

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9
Q

Anaerobic Threshold or OBLA or Ventilatory threshold

A

occurs at a higher percentage of VO2 Max

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10
Q

Mitochondria Training Adaption

A

increase number, size and membrane surface area

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11
Q

Aerobic enzymes in exercising muscle Training Adaption

A

increase krebs cycle enzymes (succinate dehydrogenase)
increase beta oxidation enzymes (carnitine acyltransferase)
increase electron transport enzymes (cytochrome oxidase)

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12
Q

Fatty Acid & Glycogen Utilization Training Adaption

A

increased utilization of beta oxidation pathways to produce ATP
decrease RER for any given submax workload
increase muscle glycogen storage

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13
Q

Resting Metabolic Rate Training Adaption

A

No appreciable change except when training induced increase in lean muscle mass occurs

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14
Q

Platelet Aggregation
fibrinolytic Activity
circulating catecholamines
resistance to pathological events

A

Platelet Aggregation: decrease
fibrinolytic Activity: increase
circulating catecholamines: decrease
resistance to pathological events: increase

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15
Q

fibrin leads to

A

blood clot

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