Test 1 misc 2 Flashcards

1
Q

submersion injury timeline to poor outcome in normal water temp

A

4-5 min

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2
Q

severe resp complication of submersion injury

A

ARDS

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3
Q

Post Submersion Neuro Class

Alert, fully conscious

A

A

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4
Q

Post Submersion Neuro Class

Obtunded, stuporous but arousable; purposeful response to pain; normal respiration

A

B

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5
Q

Post Submersion Neuro Class

Comatose; not arousable; abnormal response to pain; abnormal resp

A

C

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6
Q

Post Submersion Neuro Class

flexor to pain, cheyne stokes

A

C1

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7
Q

Post Submersion Neuro Class

Extensor response to pain; central hyperventilation

A

C2

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8
Q

Post Submersion Neuro Class

Flaccid; apneic

A

C3

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9
Q

initial chest x ray expectation for submersion injury

A

Initial Chest x-ray can be normal, show patchy infiltrates (frequently in the periphery or bibasilar regions) or show pulmonary edema.

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10
Q

what pH imbalance do you expect in submersion injury

A

resp acidosis that leads to metabolic acidosis due to lactic acid buildup

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11
Q

Treating submersion injury

A

Some patients require positive pressure for 48-72 hours,
§ Antibiotics and steroids are not indicated unless the pt becomes febrile
§ Hypovolemia should be treated with isotonic crystalloids (10-20 mL/kg). inotropic support with dopamine, dobutamine or epi can be required if continued hypertension or poor perfusion are ongoing.
Abnormal CT= most likely death
§ Is the patient is symptomatic evaluation via arterial BG, and chest x-ray should be done. Also consider a CBC, coag studies, and electrolyte panel.

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12
Q

submersion injury obs time and necessary orders if well appearing

A

prolonged time frame after submersion incident because even mild hypoxia can increase permeability of pulm capillaries with alveolar fluid leak and surfactant damage

Pulse oximetry monitoring
ABG if resp distress
Chest x ray (all submersion victims)

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13
Q

symptoms commonly seen with submersion injury

A

anxiety, vomiting, cough, wheezing, hypothermia, altered mental status, metabolic acidosis, respiratory failure, and finally respiratory/cardiac arrest.

Kline, Andrea M.; Haut, Catherine. Lippincott Certification Review: Pediatric Acute Care Nurse Practitioner (p. 1031). Wolters Kluwer Health. Kindle Edition.

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14
Q

______ should be used to prevent atelectasis and overcome intrapulmonary shunting

A

Positive end expiratory pressure

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15
Q

CN impairment initially in Botulism

A
Bulbar Palsy
IX
X
XI
XII
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16
Q

Treatment in Infant Botulism

A

mostly supportive by assisting in breathing

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17
Q

Presentation of Infant Botulism

A

§ Usually starts from the head down, with oculobulbar muscle weakness, blurring of vision, dipoplia, ptosis, ophthalmoplegia, dysarthria, and dysphagia.

§ Infants usually present listless, feeding poorly, have a weak cry, and are constipated. This is followed by a descending pattern of weakness with the upper limbs, lower limbs, and ultimately the respiratory muscles.

§ Occurs more frequently in infants less than 6 months of age but can occur up to 1 year of age.

§ S&S- constipation, lethargy, poor feeding, excessive drooling, and increasing weakness.

§ On physical exam, general hypotonia and symmetrical cranial nerve palsies are present. Infants appear to have expressionless face. CRANIAL NERVE PALSIES ARE ALWAYS PRESENT, and part of the definitive diagnosis.

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18
Q

management for infant botulism

A

§ There is an infant botulism antitoxin (BIG-IV) -Administration of human botulism immune globulin intravenous (BIG-IV).

available and if used early markedly shortens the intensive care course. Abx are not usually recommended because they will lyse bacteria and release more toxin into the circulation.
§ Feed via NG tube or NJ tube.

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19
Q

bacteria that causes botulism

A

bacteria,

Kline, Andrea M.; Haut, Catherine. Lippincott Certification Review: Pediatric Acute Care Nurse Practitioner (p. 572). Wolters Kluwer Health. Kindle Edition.

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20
Q

•Infants have weak cry, expressionless face, ptosis, and sluggish pupillary responses. Gag, suck, and swallow reflexes are diminished or may be absent.

A

Botulism

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21
Q

causes of meningitis in Neonates

A

Group B Strep,
E. Coli
Listeria monocytogenes
Klebsiella species

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22
Q

causes of meningitis in infants 3 months-9mos

A
Strep Pneumoniae
Nesseria meningitidis (group B strep is responsible for small amounts of meningitis
H. influenzae type B
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23
Q

causes of meningitis in children 10-18 yrs

A

N. meningitidis is responsible for the majority of bacterial meningitis cases.

streptococcus pneumonia

H. influenzae

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24
Q

meningitis in unvaccinated children, what etiology to consider

A

IF CHILDREN ARE UNVACCINATED!!! Think possibly H. influenzae type B, also if in an underdeveloped countries.

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25
most common cause of viral meningoencephalitis
Enterovirus
26
Bacterial meningitis usually involves the fluid in the ______ space
subarachnoid
27
causes of Meningitis in 5 yr old
N. Meningitis | Strep pneumoniae
28
Meningeal signs of meningitis
Severe throbbing headache Photophobia nuchal rigidity Kernig sign: Passive extension of the knee in supine position, resulting in back pain and resistance. • Brudzinski sign: • Passive flexion of the neck, resulting in involuntary flexion of the knees and hips.
29
csf changes associated with bacterial meningitis
``` Increased WBC increased protein decreased glucose increase pressure increased lactate (>25 is indicative of bacterial meningitis ```
30
abx for meningitis with recent neurosurgery or ventricular shunt
vancomycin + cefepime; vancomycin + ceftazidime; or vancomycin + meropenem. •
31
abx for meningitis with basilar skull fx
vancomycin + 3rd-generation cephalosporin.
32
abx for meningitis older than 1 mos
empirical broad-spectrum antimicrobial coverage with VANCOMYCIN, and either cefotaxime or ceftriaxone should be started. § ACYCLOVIR should be initiated for all infants and children with suspected HSV encephalitis.
33
most common cause of meningitis in children
viral meningitis - specific agent not usually specified but (Enterovirus >80% of cases) other echovirus, coxsackievirus, arbovirus, mumps, EBV, Cytomegalovirus, Varicella, adenovirus, HSV
34
csf changes in viral meningitis
glucose normal, slightly decreased Protein slightly increased WBC - mildly increased
35
what is the gold standard for diagnosis of HSV meningitis
CSF PCR for HSV
36
management of viral meningitis
self limiting - resolves in 7-10 days control seizures acyclovir if HSV meningitis is suspected
37
differences/additional symptoms in HSV meningitis
function: ataxia, focal neurologic signs, acute encephalopathy (high suspicion). Kline, Andrea M.; Haut, Catherine. Lippincott Certification Review: Pediatric Acute Care Nurse Practitioner (p. 604). Wolters Kluwer Health. Kindle Edition.
38
GBS has what shape bacteria (ie: gram neg, gram pos...)
gram positive cocci
39
what is the gold standard test for determining if a case of meningitis is bacterial in etiology
CSF bacterial culture
40
steroid injections make you more at risk for what kind of meningitis
fungal infection
41
what csf test tests for fungal meningitis
beta-D-glucan
42
suspect viral meningitis over bacterial when you see what in csf
normal glucose and protein levels along with a lower WBC with a monocytic predominance
43
if pathogens make it into the brain parenchyma itself, the response is
Encephalitis
44
most common cause of pure encephalitis
enterovirus
45
temporal lobe enhancement on CT is a symptom of
HSV encephalitis
46
presence of RBCs in CSF is suggestive of
Herpes Simplex encephalitis
47
Ampicillin covers what type of meningitis
Listeria monocytogenes
48
Medications to avoid with eating disorders
TCAs - cardiotoxicity in malnourished individuals Bupropion - increased risk of seizures in individuals with eating disorders
49
complications of Bulimia nervosa
erotion of enamil sialadenosis (swelling of parotid gland) bad breath Russels sign - calleouses on knuckles from making yourself vomit Tears in distal esophagus or stomach itself (Mallory Weiss syndrome - abd pain and hematemesis) Amenorrhea type 2 DM
50
Russels sign
sign of Bulimia nervosa - callous on knuckles
51
Mallory weiss syndrome
associated with Bulimia nervosa - tears in distal esophagus or stomach resulting in abd pain and hematemesis
52
``` Bulimia Nervosa: What does BP HR: Electrolytes: Ph: ```
Dehydration causes Hypotension (<90/50) Tachycardia HR >100 ``` Electrolytes low NA Cl Mg phosphorous K+ ``` General Metabolic Alkalosis
53
Lab tests to order in Bulimia nervosa
``` Serum electrolytes serum creatinine BUN CBC with diff LFTs Urinalysis ```
54
Pharm for Bulimia Nervosa
SSRI choice is Fluoxetine If they dont respond, try diff SSRI - Sertraline ``` If dont respond or tolerate -> Either tricyclic antidepressant Trazodone MAOIs Topiramate ```
55
Med you cant describe for a pt with Bulimia nervosa
Bupropion - increased risk of seizures in individuals with eating disorders
56
Binge Eating disorder treatment
Psychotherapy is first line second line is SSRI s third line is Topiramate Mod severe cases may benefit from Lisdexamfetamine
57
what is the unintentional Food regurgitation disorder
called: Rumination Disorder nor due to a medical disorder not purposeful associated with mood disorders
58
What diagnostics rule out other differentials for Rumination disorder
High resolution impedance manometry testing upper GI endoscopy
59
initial management for rumination disorder
Education and treatment of mood disorder Diaphragmatic abd breathing immediately after a meal and for 10-15 min after
60
if initial mgmt of rumination disorder doesnt work, what meds can you try
Baclofen increases LES tone decreases LES relaxation
61
diagnostic criterion for PICA
strong cravings to eat non-food items >= month has to be culturally inappropriate should not happen in the context of another health condition such as schizophrenia, autism or another eating disorder
62
what is PICA associated with
anemia (esp iron deficiency) emotional trauma malnutrition comorbid psychiatric disorders
63
complications with PICA
``` nutritional deprivation anemia toxicity constipation intestinal obstruction parasitic infections jaw or tooth injury weight changes ```
64
treatment for PICA
1) Nutritional rehab and psychotherapy Identify substance -> decrease exposure -> healthier alt to decrease cravings If comorbid psych disorders -> SSRIs
65
significant food restrictions at least once a week for 3 months with fear of gaining weight and BMI below 18.5
Anorexia
66
cycles of binging and purging once a week for 3 months with normal weight or over weight (BMI >18.5)
Bulimia
67
large amounts of food that causes lots of insulin to be released which leads to cellular intake of electrolytes and produces arrhythmias
Refeeding syndrome
68
expected weight gain in anorexia in hospitalized vs outpatient patients
2-3 lbs/week (hospitalized) | 0.5-1lb/week (outpatients)
69
how many kcal/kg/day in anorexia treatment
30-40 kcal/kg/day
70
in bulimia, what acid base disorder is seen
metabolic alkalosis
71
``` Salicylate intoxication Hyperventilation Hyperthyroidism Interstitial pulmonary disease CNS diseases ``` causes what acid base imbalance
Resp alkalosis High pH low CO2
72
diarrhea will cause what acid base imbalance
metabolic acidosis low pH low HCO3
73
``` Methanol Uremia DKA Paraldehyde Isoniazid and iron lactic acid ethanol glycol ethanol induced ketoacidosis salicylates ``` All do what do your anion gap and give you what kind of a acid base imbalance
increased anion gap metabolic acidosis low pH low HCO3
74
``` Loss of gastric acid due to vomiting NG suctioning Diuretic use Alterations to parenteral nutrition Excessive bicarb administration ``` all put you at risk for what kind of acid base balance
metabolic alkalosis high pH High bicarb