Surgical Trauma And Emergency Flashcards

1
Q

Site for needle insertion and chest drain in tension pneumothorax

A

Adult 5th intercoastal space in mid axillary line
Children 2nd intercoastal space mid clavicular line

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Size of chest tube for tension pneumothorax or hemorrhoids

A

28-32Fr gauze

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Burn fluid

A

For thermal
2ml/kg×surface area
For electrical
4 no/kg×surface area

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Type of chest drain in traumatic pneumothorax

A

Under water seal

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Beck’s triad is for what and components

A

Pulsus paradoxus, also known as paradoxical pulse, is a condition where a person’s systolic blood pressure, stroke volume, and pulse wave amplitude drop abnormally during inhalation. It’s a sign of heart or lung disease and is the opposite of what would normally happen, as blood pressure usually increases when someone inhales.

For cardiac tamponade
Hypotension
Raised JVP
Muffled heart sound
And
There will be a pulsus paradoxus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Boerhaave’s syndrome causes and sign symptoms

A

Complete rupture at lower thoracic esophagus
Hamman’s sign -
Crunching sound upon auscultation of heart due to pneumomediastinum
Chest pain
Shock
Subcutaneous emphysema

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Mallory Weiss syndrome causes and sign symptoms

A

Incomplete tear only affecting muchos and submucosa
Tear on the gastric side of gastroesophageal junction which may extend to distal esophagus
Repeated vomiting then
Hematemesis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

What to do if a patient of perforated peptic ulcers came with sepsis

A

Approach as ATLS protocol

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

DD for epigastric pain

A

Peptic ulcers
Perforated peptic ulcer
Pancreatitis
Cholecystitis
Myocardial infarction
diabetes may cause of DKA

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Primary treatment of MI

A

Antiplatet
Thrombolysis
Angioplasty

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Sign symptoms of pulmonary embolism and ECG changes and confirmatory investigation

A

Hemoptysis
Hypoxia
Small pleural effusion
ECG
S wave in lead 1
Q wave in lead 3
T inversion in lead 3
Investigation
CTPA

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Collapsing signs of pulmonary embolism
And management

A

Chestpain
Hypotension
Tachycardia
Breathlessness
Desaturation
Mx
Patient is in peri arrest state so make thrombolysis with alteplase
Heparin administration upto achieving INR of 2-3. Then stop heparin and start warfarin 4-6 weeks with temporary risk factors
Oral anticoagulant only after confirmation of venous thrombosis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

When to suspect fat embolism and
Management

A

After long bone fractures
Liposuction
Mx
Fat embolism syndrome (FES) is generally treated with supportive care in the hospital, often in the intensive care unit. Treatment focuses on maintaining intravascular volume and ensuring good arterial oxygenation. Some treatments include:
Oxygen
One of the fastest and easiest treatments for respiratory problems. You may be given oxygen or need help breathing with mechanical ventilation.
Intravenous fluids
Helps remove damaging free fatty acids from the body. Human albumin is recommended to restore blood volume and bind to fatty acids to reduce lung injuries.
Drugs
Your doctor may prescribe steroids or the blood thinner heparin, but these drugs have not been proven to be highly effective.
Surgical management
This includes early stabilization of long-bone fractures, rigid fixation within 24 hours, and appropriate surgical technique.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Massive pulmonary embolism Mx
Vs
Small embolism

A

British thoracic society guideline
Massive
3mg bolus of alteplase
Heparin
Thrombus fragmentation
IVC filter insertion
Small
Heparin if high probability of embolism before imagine like unfractionated heparin in massive pulmonary embolism where rapid reversal is needed. If not then LMWH cause it has the same efficacy and is easier to administer upto reaching INR 2-3
Oral anticoagulant should be used only after confirmation of embolism upto 4-6 weeks with temporary risk factors,3 months for idiopathic,6 months for any other reason

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

GCS components

A

Eye 4
Spontaneous
Response to verbal command
Response to pain
No eye opening
Verbal 5
Oriented(knows who and where he is)
Confused(answers,disoriented)
Inappropriate words(no conversation)
Incompetent sounds
No verbal response
Motor 6
Obeys command
Localizes response to pain
Withdrawal response to pain
Flexion to pain(decorticate-flex, int.Rotat)
Extremely to pain(decerebrate-ex,ext.rot)
No motor response
#below 8 intubate
*Inapplicable have
Macroglossia
Laryngeal edema
Puffy eyes
Hematoma eye

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Organized hematoma in splenic injury

A

Don’t disrupt it

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Infectious organism after splenectomy
And measures to be taken

A

Encapsulated
Streptococcus pneumoniae
Hemophylus influenzae
Neisseria meningitidis
Vaccine and penicillin
Hib
Pneumoniae
Meningitis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Blood film post splenectomy

A

Reticulocyte
Howell jolly bodies
Heinz bodies

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Must common cause subarachnoid hemorrhage

A

Ruptured berry aneurysm
Looks like a berry in a stalk
Along the circle of Willis
Associated with autosomal dominant polycystic kidney disease

20
Q

Lethal triad

A

*Injury>exposure>hypothermia >coagulopathy>acidosis
*Injury>haemorrhage>hypoperfusion
>Acidosis>coagulopathy>hypothermia
*Injury>hemorrhage>coagulopathy
>Acidosis>hypothermia

21
Q

Stages of hypothermia

A

Mild- conscious, shivering(32-35°C)
Moderate-drowsy, not shivering(28-32)
Severe-uncooked, not shivering (20-28)
Profound-no vital signs (<20)

22
Q

Response stages of hypothermia
Response stages of hyperthermia

A

Hypo
Vasoconstriction(aw37:an35.5)>non shivering metabolic thermogenesis(aw36:an35)>shivering thermogenesis(aw35.5:an34)
*N B.aw=awake,an=anesthetized
Hyper
Sweating(aw37:an37.5)>vasodilation (aw37.5:an38)

23
Q

Rx of hypothermia
ECG changes

A

Rewarming
Mild- external
Serve -active core : peritoneal lavage, hemostasis, cardiac bypass
ECG
J wave
Arrhythmia

24
Q

Antidote of prilicaine

A

IV methylene blue

25
Q

Antidote of lignocaine

A

Intralipid

26
Q

Repeating technique of vessels and type of suture

A

Figure of 8 with 5-0 prolene

27
Q

Rate of air change in laminar floor OT

A

300 times per hour

28
Q

What to use facial reconstruction

A

Local flap
Or
Full thickness graft

29
Q

Adverse effect of central line on lungs

A

May cause pneumothorax

30
Q

Which coronary artery may be associated with abdominal aortic aneurysm and ECG finding

A

Right coronary artery
So
Inferior MI in ECG

31
Q

Chest x-ray changes in ruptured thoracic aorta confirmatory investigation and Mx

A

Widening mediastinum
Trachea and esophagus shifted to right
Obliteration of space between aorta and pulmonary artery
May be rib fractures and hemothorax

CT, angiography, aortogram
Tx is emergency endovascular repair

32
Q

Dx and Tx of achalasia cardiac

A

Investigation
Ph
Manometry
Barium swallow x-ray
Endoscopy
Tx
Balloon dilatation
Botulinum toxin
Cardiomyotomy

33
Q

If suspecting intracranial or intracerebral gardenia what to do

A

First surgery
Then
Antiplatet or anticoagulant

34
Q

Beck’s triad and cause

A

Elevated venous pressure
Reduced arterial pressure
Reduced heart sound
And maybe associated with
pulsus paradoxus
Cause
Cardiac tamponade even with 100 ml blood

35
Q

Le Fort’s classification of mid face fractures

A

1-no speak
2-no see
3-be hear

36
Q

Criteria of immediate CT

A

Loss of consciousness for more than 5 minutes
Amnesia for more than 5 minutes
Abnormal drowsiness
3 or more episodes of discrete vomiting
Clinical suspicious of non accidental inquiry
Part traumas seizure nutty no history of epilepsy
GCS<14 in adults
GCS<15 in children below 1 year old or with bruise/swelling/laceration >5cm
Sign of base of skull fractures -hemotympanum
-CSF otorrhea or rhinorrhea
-battle sign
-halo sign
Focal neurological deficit
Fall from height>3 meter
High speed injury like RTA

37
Q

Tx of boerhaave’s syndrome

A

Surgery of within 12 hours
Beyond that controlled fistula

38
Q

Antidote of opioid toxicity

A

Naloxone
But risk of rebound pain

*Rebound release of catecholamines may cause hypertension, tachycardia, and ventricular arrhythmias. Pulmonary edema has also been observed after naloxone administration. Small doses of naloxone can precipitate a withdrawal syndrome in subjects that are opioid-dependent.

39
Q

Compartment syndrome

A

Compartment syndrome can occur in any muscle, but it’s most common in the forearms and lower legs. Symptoms can be difficult to detect, and can become severe within a few hours of an acute injury. Some early signs of compartment syndrome include:
Pain
Pain that’s more severe than expected from the injury, and that doesn’t go away after taking pain medication or raising the affected area. The pain may feel like a deep ache or burning sensation, and it can get worse when you move the affected body part.
Swelling
The muscle may swell or bulge, or you may have difficulty moving the affected body part.
Other sensations
You may experience numbness, tingling, or a burning feeling under your skin (paresthesia). The skin may also appear pale.
Other signs of compartment syndrome include: tightness, weakness, and pins and needles.
Numbness and paralysis are later signs of compartment syndrome, and usually indicate permanent tissue injury.
Compartment syndrome can be caused by fractures, severe contusions, crush injuries, or reperfusion injury after vascular injury and repair.

40
Q

Virtual and disarthia suggest what type of vascular injury and if sudden deterioration of consciousness what may be main cause

A

Posterior circulation
Basilar artery occlusion

41
Q

Signs of occlusion of anterior circulation

A

Hemiparesis
Hemisensory loss
Homonymous hemianopia
Higher cognitive dysfunction
Dysphagia

42
Q

Confirm CSF

A

Transferrin test
Halo sign on gauze
*Fluid containing CSF is classically described to make a “halo” or “double-ring” pattern on gauze or linen.

43
Q

Allergic/anaphylactic shock Mx

A

Remove allergen
ABC survey
Adrenaline 1:1000 IM repeat every 5 minutes if no response
Chlorpheniramine
Hydrocortisone

44
Q

Causes of Addisonian crisis

A

Sepsis or surgery causing acute exacerbation of chronic deficiency like Addison’s or hypopituitarism
Adrenal haemorrhage in Waterhouse Fredricksen syndrome
Fulminant meningococcemia
Steroid sudden withdrawal

45
Q

Mx of Addisonian crisis

A

Hydrocortisone 100mg IM or IV
Normal saline 1L over 30 to 60 minutes or dextrose if hypoglycemia
Oral replacement after 24 hours with maintainance dose over 3-4 days

46
Q

ECG in pulmonary embolism

A

s1 q3 t3 ,tall r in v1,t inversion in v123, peak p pulmonale in inferior leads,RBB, right axis deviation, right ventricular strain, atrial arrhythmia

47
Q

Cushing triad

A

Hypertension—due to sympathetic activation after raised ICP
Bradycardia
Bradypnea