Substance Abuse Flashcards

1
Q

substance abuse

A

habitual use outside medical necessity
purpose to alter mood, emotion, LOC
results in adverse effects

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2
Q

tolerance

A

when person has to take more of the drug to “stay normal” and prevent withdrawal

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3
Q

addiction

A

tolerance
control over substance lost
can lead to OD and be fatal

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4
Q

4 Cs of addiction

A

Craving
Continued use
Control is lost
Cognitive Impairment

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5
Q

examples of CNS stimulants

A
Crack
Cocaine
Meth
Caffeine
Nicotine
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6
Q

common signs of CNS stimulant abuse

A

dilated pupils
dry oronasal cavity
excessive motor activity

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7
Q

withdrawal sx of CNS stimulants

A
depression
paranoia
anger
lethargy
anxiety
insomnia
N/V
sweating
chills
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8
Q

examples of opiates

A
Morphine
Heroin
Codeine
Fentanyl
Methadone
Meperidine
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9
Q

triad of sx for opioid toxicity

A

pinpoint pupils
depressed respirations
coma

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10
Q

opiate intoxication

A
constricted pupils
decreased RR, BP
slurred speech
drowsiness
psychomotor retardation
impaired concentration, judgment, memory
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11
Q

withdrawal effects from opiates

A
yawning
insomnia
irritability
rhinorrhea
panic
diaphoresis
cramps
N/V
muscle aches
chills and fever
lacrimation
diarrhea
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12
Q

opiate overdose

A
anoxia
pupil dilation
respiratory arrest
cardiac arrest
coma
shock
convulsions
death
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13
Q

Meds for opioid addiction

A
methadone (Dolophine)
l-a-acetylmethadol (LAAM)
naltrexone (ReVia)
clonidine (Catapres)
buprenorphine (Subutex/ Subaxone) combined with naltrexone
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14
Q

methadone (Dolophine)

A

used for opioid addiction

blocks craving for and effects of heroin

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15
Q

naltrexone (ReVia)

A

used for opioid addiction

blocks euphoric effects of opioids

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16
Q

clonidine (Catapres)

A

used for opioid addiction

somatic treatment if combined with naltrexone

17
Q

buprenorphine (Subutex/Subaxone)

A

blocks s/sx of opioid withdrawal

18
Q

Alcohol/CNS depressants

A

ETOH
Barbiturates
Benzos

19
Q

ETOH and CNS depressant intoxication

A
slurred speech
ataxia
drowsiness
decreased BP
sexual or aggressive behavior
impaired judgment, cognition
irritability
20
Q

ETOH withdrawal

A

early signs in a few hours
peaks at 24-48 hrs
rapidly and dramatically disappears unless it progresses to delirium
irriability and “shaking inside”
grand mal seizures possible 7-49 hrs after cessation
illusions

21
Q

ETOH delirium

A
medical emergency
possible death
peaks 2-3 days after cessation/reduction
autonomic hyperactivity
sensorial/perceptual disturbances
fluctuating loss of consciousness
paranoid delusions
agitated
temp 100F and higher
22
Q

early ETOH withdrawal s/sx, onset, duration

A

tremors, anxiety, palpitations, nausea, anorexia

onset: 6-8 hrs
duration: 1-2 days

23
Q

ETOH withdrawal seizures onset, duration

A

onset: 6-48 hrs
duration: 2-3 days

24
Q

ETOH hallucinations: types, onset, duration

A

visual, tactile, auditory

onset: 12-48hrs
duration: 1-2 days

25
ETOH delirium sx, onset, duration
tachycardia, HTN, low-grade fever, diaphoresis, delirium, agitation onset: 48--96 hrs duration: 1-5 days
26
meds to tx alcoholism
naltrexone (ReVia) acamprosate (Campral) topiramate (Topamax) disulfiram (Antabuse)
27
naltrexone (ReVia) for ETOH
reduce or eliminate ETOH and opioid craving
28
acamprosate (Campral)
helps pt abstain from ETOH
29
topiramate (Topamax)
decrease ETOH cravings
30
disulfiram (Antabuse)
causes unpleasant physical effects (N/V)
31
Marijuana s/e
``` mixed depressant and hallucinogenic detachment relaxation euphoria apathy intensified perceptions impaired judgment/memory slowed perception of time heightened sensitivity to stimuli ```
32
marijuana dependence
lethargy, anhedonia, difficulty concentrating, memory impairment
33
conventional tx for people with addictions
``` psychotherapy group therapy CBT motivational incentives motivational interviewing ```
34
recovery models for people with addictions
``` 12 step program SMART residential intensive outpatient outpatient drug-free employee assistance ```