Stress & Anxiety Disorders Flashcards

1
Q

HPA axis

A

one of the primary systems involved in the fight or flight system comprising the hypothalamus, pituitary gland which secretes ACTH, and adrenal glands which secrete cortisol, epinephrine, and norepinephrine

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2
Q

What are the effects of extreme or prolonged stress?

A

extensive physical and psychological problems like increased reactivity in sympathetic nervous system, decreased efficacy of immune system and psychological self-efficacy, personality deterioration, and death

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3
Q

2 types of psychosocial contributing factors

A

external and internal (in terms of the stressor, crisis, and resources available)

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4
Q

External vs Internal stressor

A

the nature of the stressor (e.g. accidental or intentional) vs the perception of the stressor (e.g. having a sense of control) affects how we process it and cope with it

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5
Q

External vs Internal crisis

A

life changes that occur due to crisis (the aftermath) vs the experience of the crisis (the meaning or what you make of it; may lead to posttraumatic growth)

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6
Q

External vs Internal resources

A

social and financial support vs stress tolerance (biological and psychological resources)

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7
Q

Posttraumatic stress disorder (PTSD)

A

exposure to an event that threatened death, serious injury, or sexual violence through direct experience, witnessing others’ experience, learning about it happen to close friends/family, or repeated/extreme exposure to aversive details of the event (e.g. in the news)

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8
Q

4 basic categories of PTSD symptoms

A

intrusion (nightmares and flashbacks), avoidance of things associated with trauma, negative cognitions and mood, arousal and reactivity

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9
Q

Negative cognitions and mood that occur with PTSD

A

detachment, anger, shame, distorted blame with self/others

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10
Q

How does heightened arousal and reactivity occur in PTSD?

A

insomnia, difficulty concentrating, hypervigilance, heightened startle response

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11
Q

4 most common triggering events of PTSD

A

combat, physical or sexual assault, natural disaster, and torture

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12
Q

What are the binary gender differences in rates of PTSD?

A

men are more exposed to traumatic events but women are 2x more likely to experience to PTSD and have more severe symptoms (typically due to sexual assault)

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13
Q

3 factors that affect the gravity of psychological problems caused by sexual assault

A

past coping skills, current psychological functioning, ability to disclose experience of assault to others

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14
Q

What biological mechanism causes transgenerational trauma?

A

epigenetics

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15
Q

How do genes affect the occurence of PTSD?

A

genes account for 33% of the variance in symptom severity

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16
Q

Neurobiological factors of PTSD

A

a hyperactive limbic system with increased norepinephrine, decreased serotonin and endogenous opioids (decreased ability to tolerate pain), higher cortisol levels (for women)

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17
Q

Psychological factors of PTSD

A

threat-related psychological processes (e.g. hypervigilance and increased vulnerability), high neuroticism, negative attributions or maladaptive appraisal, low cognitive ability

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18
Q

Social factors of PTSD

A

previous experience of trauma and severity of current trauma, early experience of uncontrollable/unpredictable events, membership in a minoritized group, low social support, lack of education, engaging in combat or war

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19
Q

How does being involved in combat influence the experience of PTSD?

A

acceptability of war goals and one’s identification with their unit lessens likelihood while returning to an unaccepting social environment after war increases likelihood

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20
Q

Neurobiological symptoms of PTSD

A

fear learning in the amygdala (i.e. perceiving threats and responding physiologically), cell death in and reduced size of hippocampus due to cortisol, hyperactive limbic system and sustained release of cortisol

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21
Q

Psychological symptoms of PTSD from diagnosis

A

persistent re-experience (nightmares and flashbacks), avoidance of things associated with trauma and emotional numbing, increased arousal (insomnia and difficulty concentrating)

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22
Q

Social symptoms of PTSD from diagnosis

A

avoidance of things associated to trauma and emotional numbing (e.g. detachment from others and restricted range of affect)

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23
Q

Biological treatment of PTSD

A

beta-blockers (e.g. propranolol) and SSRIs

24
Q

When are beta-blockers taken for PTSD?

A

taken when knowingly going into a traumatic situation (e.g. combat) to decrease the likelihood of developing PTSD because it calms the limbic system

25
Effects of taking SSRIs
decreases depression, intrusive thoughts and avoidance
26
Cons of taking beta-blockers
suppression of natural warning signs and future reliance on medication
27
5 psychological treatments for PTSD
cognitive-behavioral therapy (53% effective), prolonged exposure, eye movement desensitization and reprocessing (EMDR), cognitive processing therapy (CPT), building positive coping skills
28
Prolonged exposure therapy
repeatedly processing a traumatic experience that increases anger (due to blame) and decreases shame
29
Cognitive processing therapy
processing the consequences and beliefs attached to the traumatic experience to help see it in a new light
30
How can you prevent PTSD?
stress-inoculation training
31
Stress-inoculation training
advanced preparation to increase resistance to the effects of stressors (e.g. combat)
32
Social treatment of PTSD
having social support and being able to disclose trauma
33
5 main anxiety disorders
specific phobias, social anxiety disorder or social phobia, panic disorder, agoraphobia, general anxiety disorder (GAD)
34
Etiology of anxiety disorders
biological and psychological causes include genes, neuroticism, classical conditioning, lack of perceived control; social factors depend on culture
35
Presentation of anxiety disorders
unrealistic, irrational fears or anxieties at a disabling intensity
36
What is the most effective treatment for anxiety disorders?
exposure
37
2 factors that decrease anxiety
perceived controllability and predictability
38
How does perceived threat influence anxiety?
a perceived threat leads to fear and anxiety, which then leads to an interpretive bias toward threat and an increase in perception of threat
39
Specific phobia
strong fears of specific kind of situation or object that are unreasonable or out of proportion to the actual danger and and avoidance of that situation/object that becomes disruptive to daily life
40
Examples of specific phobias
animal, natural environment, blood-injection-injury (3-4% of population, usually genetic), situational, choking, vomiting, etc.
41
Rate of concurrence of specific phobias
75% of people with a specific phobia have at least one other specific fear
42
Biological factors of specific phobias
increase likelihood with increased speed and strength of fear conditioning, having a first degree relative (particularly BII phobia), being behaviorally inhibited
43
Psychological factors of specific phobias
prepared learning, traumatic conditioning of fear, high neuroticism (e.g. worrying a lot and poor handling of stress)
44
Prepared learning
objects or situations that have an evolutionary reason to be feared (e.g. spiders) are more likely to be conditioned as things to be feared
45
Social factors of specific phobias
modeling, vicarious learning, fear immunization, environment, parenting (inadvertent reinforcement increases fear)
46
Fear immunization
gradually exposing a person to what they fear to immunize them against later fear development
47
Biological symptoms of specific phobias
autonomic arousal pre-fight/flight in response to the presence or thought of the feared object/situation; decreased GABA and serotonin; higher norepinephrine
48
Psychological symptoms of specific phobias
heightened vigilance to the feared object/situation, negative mood, worry about potential danger, self-preoccupation, have a lower sense of efficacy, diminished internal locus of control
49
Social symptoms of specific phobias
avoidance of situations eliciting anxiety
50
Biological treatments for specific phobias
though not often treated biologically: SSRIs and benzodiazepines (cause a short-term decrease in anxiety and increases avoidance instead of habituating or building tolerance)
51
Psychological treatment of specific phobias
behavior therapy or exposure (most effective), modified exposure (e.g. virtual reality, combining cognitive techniques)
52
Extinction
no longer feeling afraid of the object/situation
53
Habituation
getting more used to the feared object/situation over time
54
Systematic desensitization
technique used for habituation (gradual exposure)
55
Flooding
sudden exposure to feared object/situation (but most people leave before habituation occurs)
56
Social treatment of specific phobias
modeling (learning by imitating others' behavior)