Spinal Cord Injury Flashcards
Complete lesions
- a lesion to the SC where there is no preserved motor or sensory function below the level of the lesion
incomplete lesions
a lesion to the SC with incomplete damage to the cord. there may be scattered motor function, sensory function or both below the level of the lesion
Autonomic Dysreflexia
- most dangerous complication of SCI and can occur in patients with lesions at or above T6.
- a noxious stimulus below the level of teh lesion triggers the ANS causing a sudden elevation in BP,
- common causes include distended or full bladder, kink or blockage in catheter, bladder infection, ulcers, temp change, tight clothing or even ingrown toenail
- if not treated, condition can lead to convulsions, hemorrhage and death
Autonomic Dysreflexia Symptoms
- high BP, severe HA, blurred vision, stuffy nose, profuse sweating
- goosebumps below the level f the lesion, and vasodilation above the level of injury
Autonomic dysreflexia treatment
- immediately check catheter for blockage while having the patient assume or remain in a sitting position.
- lying a patient down is contraindicated and will only assist to further elevate BP.
- the pt shoulde be examined for any irritating stimuli and potentially checked for bowel impact
Ectopic Bone/ Heterotopic Ossification
-spont formation of bone in the soft tissue. it typically occurs adjacent to larger joints such as knees or the hips. theories regarding etiology range from tissue hypoxia to abnormal calcium metabolism
Ectopic Bone/ Heterotopic Ossification symptoms
- early symptoms include edema, decreased range of motion, and increased temperature of the involved joint
Ectopic Bone/ Heterotopic Ossification treatment
- medicinelike diphosphates that inhibit ectopic bone formation. physical therapy and surgery are often incorporated into tx. PT should focus on maintaing functional ROM and allowing the patient the most independent functional outcome possible
spasticity
- can occasionally be useful to a pt with SCI, however, more often serve to interfere with functional activities.
- spasticity can be enhanced by both internal and external sources such as stress, decubiti, UTI,bowel or bladder obstruction, temp changes or touch
Anterior Cord Syndrome
-incomplete lesion that results from compression and damage to the anterior part of the SC or anterior spinal artery. the mechanism of injury is usually cervical flexion. there is loss or motor function and pain and temp sense below the lesion due to damage of teh corticospinal and spinothalamic tracts
Brown-sequard’s syndrome
- incomplete lesion usually caused by a stab wound, which produces hemisection of the SC. there is paralysis and loss of vibratory and position sense on teh same side as teh lesion sue to the damage to the corticospinal tract and dorsal columns,
- there is a loss of pain and temp sense on the opposite side of the lesion from damage to the lateral spinothalamic tract.
- pure brown sequard syndrome is rare since most spinal cord lesions are typical
cauda Equina injuries
- injury below the L1 spinal level where the long nerve roots transcend. Cauda equina injuries can be complete , however they are frequently incomplete due to the large number of nerve roots in the area. a cauda equina injury is considered a peripheral nerve injury.
- characteristics include flaccidity, areflexia, and impairment of bowel and bladder function. full recovery is not typical due to the distance needed for axonal regeneration
Central cord Syndrome
- incomplete lesion that results from compression and damage to the central portion of the SC. the mechanism of injury is usually cervical hyperextension that damages the spinothlamic tract, corticospinal tract and dorsalcolumns. the upper extremities present with greater involvements than the LE and greater motor deficits exist as compared to sensory deficits
Posterior cord syndrome
-relatively rare syndrome that is caused bu compression of the posterior spinal artery and is characterized by loss of proprioception, two point discrimination and stereognosis. motor function is preserved
SCI sequential recovery stages
- Stage 1: Initial flaccidity, no voluntary movement
- Stage 2: emergence of spasticity, hyoerreflexia, synergies
- Stage 3: voluntary movement possible, but only in synergies, spasticity strong
- Stage 4: voluntary control in isolated joint movements emerging, corresponding decline of spasticity and synergies
- Stage 5: incre voluntary control out of synergy; coordination deficits present
- Stage 6: control and coordination near normal