CVA Flashcards

1
Q

Expected possible impairment with….

  • Anterior cerebral artery
A
  • Contralateral LE motor and sensory involvement
  • loss of bowel and bladder control
  • loss of behavioral inhibition
  • significant mental chnages
  • neglect
  • aphasia
  • apraxia and agraphia
  • perseveration
  • akinetic mutism with significant bilateral involvement
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2
Q

Expected possible impairment with….

  • middle cerebral artery
A
    • most COMMON site of CVA
  • wernicke’s aphasia in dominant hemisphere
  • homonymous hemianopsia
  • apraxia
  • flat affect with right hemisphere damage
  • contralateral weakness and sensory loss of face and upper extremity with lesser involvement in the LE
  • impaired spatial relations
  • anosognosia in non-dominant hemisphere
  • impaired body schema
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3
Q

What area of the brain does ACA serve?

A
  • anterior frontal lobe

- medial surface of frontal and parietal lobes

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4
Q

What area of the brain does MCA serve?

A
  • most of outer cerebrum
  • basal ganglia
  • posterior and anterior internal capsule
  • putamen
  • pallidum
  • lentiform nucleus
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5
Q

What area of the brain does PCA serve?

A
  • Portion of midbrain
  • subthalamic nucleus
  • basal nucleus
  • thalamus
  • inferior temporal lobe
  • occipital and occipitoparietal cortices
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6
Q

Expected possible impairment with….

PCA

A
  • contralateral pain and temp sensory loss
  • contralateral hemiplegia (central area), mild hemiparesis
  • ataxia, athetosis, or choreform movement
  • quality of movement is impaired
  • thalamic pain syndrome
  • anomia
  • prosopagnosia with occipital infarct
  • hemiballismus
  • visual agnosia
  • homonymous hemianopsia
  • memory impairment
  • alexia, dyslexia
  • cortical blindness from bilateral involvement
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7
Q

What are of the brain does the vertebral-basilar artery serve?

A
  • lateral aspects of pons and midbrain with superior surface of cerebellum
  • cerebellum: branches from the basilar artery (PICA, AICA, SCA
  • medulla: PICA
  • Pons: branches from basilar
  • midbrain and thalamus: PCA
  • occipital cortex: PCA and basilar artery
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8
Q

Expected possible impairment with….

vertebral- basilar artery

A
  • LOC
  • Hemiplegia or tetraplegia
  • comatose or vegetative state
  • inability to speak
  • locked-in syndrome
  • vertigo
  • nystagmus
  • dysphagia
  • dysarthria
  • syncope
  • ataxia
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9
Q

Locked- in syndrome

A

is a condition in which a patient is aware but cannot move or communicate verbally due to complete paralysis of nearly all voluntary muscles in the body except for vertical eye movements and blinking.

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10
Q

Wallenberg syndrome secondary to lateral medullary infarct

A
  • can develop when damage occurs in a part of the brainstem called the lateral medulla.
    -Hoarseness
    Dizziness, nausea, and vomiting
    Rapid involuntary eye movements
    Difficulty with balance and gait coordination
    Problems with body temperature sensation
    **Lack of pain and temperature sensation on one side of the face, or different symptoms on each side of the body
    Uncontrollable hiccups
    Loss of taste on one side of the tongue
    Decreased sweating
    Changes in heart rate and blood pressure
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11
Q

Primary risk for CVa

A
  • HTN
  • Cardiac disease or arrhythmias
  • DM
  • smoking
  • TIA
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12
Q

Secondary risk factors for CVA

A
  • Obesity
  • High cholesterol
  • stress, excessive salt intake
  • physical inactivity
  • incr alcohol consumption
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13
Q

Transient ischemic attack TIA

A
  • usually linked to an atherosclerotic thrombosis which causes a temporary interruption of blood supply to an area of the brain
  • similar to CVA but symptoms resolve quickly within 24 to 48 hours
  • often occurs in the carotid and vertobrobasilar arteries
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14
Q

stroke in evolution

A
  • CVa usually caused by a thrombus that gradually progresses

- total neurological deficts ar enot seen for one to two days after onset

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15
Q

ischemic stroke

- embolus

A
  • may be a solid, liquid or gas and can originate in any part of the body. Occurs rapidly with no warning
  • travels through body causing occlusion of a blood vessel and a resultant infarct
  • MCA is most commonly affected by an embolus from the ICA
  • tissues distal to infarct can sustain higher permanent damage than those of thrombotic infarct.
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16
Q

Ischemic stroke

- thrombus

A
  • atherosclerotic plaque develops in an artery and eventually occludes.
  • extremely variable where symptoms can occur in minutes or over several days
17
Q

Hemorrhage

A
  • (10-15% of CVA)

- abnormal bleeding in the brain due to rupture in blood supple

18
Q

Characteristics of a Left hemisphere CVA

A
  • weakness, paralysis of the R side
  • incr frustration
  • decr processing
  • possible aphasia
  • possible dysphagia
  • possible motor apraxia
  • decr discrimination between R and L
  • R hemianopsia
19
Q

Characteristics of a Right hemisphere CVA

A
  • weakness, paralysis of L side
  • decr attention span
  • left hemianopsia
  • decr awareness and judgement
  • memory deficits
  • left inattention
  • decr abstract reasoning
  • emotional lability
  • impulsive behaviors
  • decr spatial orientation
20
Q

Characteristics of a Brainstem CVA

A
  • unstable vital signs
  • decr consciousness
  • decr ability to swallow
  • weakness on both sides of the body
  • paralysis on both sides of the body
21
Q

Characteristics of a cerebellum CVA

A
  • decr balance
  • ataxia
  • decr coordination
  • nausea
  • decr ability for postural adjustment
  • nystagmus
22
Q

UE flexor synergy pattern

A
  • scapula elevation and retraction
  • shoulder abduction and ER
  • elbow flexion
  • forearm supination
  • wrist flexion
  • fingers flexion
23
Q

UE extensor synergy pattern

A
  • scapula depression and protraction
  • shoulder IR and adduction
  • elbow extension
  • forearm pronation
  • wrist extension
  • fingers flexion
24
Q

LE flexor synergy pattern

A
  • hip abduction and ER
  • knee flexion
  • ankle DF and supination
  • toes extension
25
Q

LE extensor synergy pattern

A

Hip extension, IR and ADD

  • knee extension
  • ankle PF with inversion
  • toes flexion and adduction
26
Q

Ramiste’s phenomenon

A
  • the involved LE will abduct or adduct with applied resistance to the uninvolved LE in the same direction
27
Q

Souques phenomenon

A
  • raising the involved UE above 100 deg with elbow extension will produce extension and abduction of the fingers
28
Q

Seven stages of recovery- Brunnstrom

A
  1. no volitional movement initiated
  2. appearance of basic limb synergies. beginning of spasticity
  3. synergies are performed voluntarily, spasticity incr
  4. spasticity begins to decr. movement patterns are not dictated solely by limb synergies
  5. a further dec in spasticity is noted with independence from limb synergy patterns
  6. isolated joint movements are performed with coordination
  7. normal motor function is restored