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Physiology 210 > Special Senses Physiology > Flashcards

Special Senses Physiology Flashcards

1
Q

Sensory System

A

Sensory receptors receive stimuli from the external or internal environment which is then carried by neural pathways to the brain or spinal cord

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2
Q

Somatosensory System

A

Part of the sensory system that is concerned with the perception of touch, pressure, pain, temperature, position, movement, vibration

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3
Q

Somatic Sensation

A

Defined as sensation from the skin, muscles, bones, tendons, and joints initiated by somatic receptors

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4
Q

Sensory Receptor

A

Specialized cells that generate graded potentials called receptor potentials in response to a stimulus

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5
Q

Somatosensation

A

The process that conveys information regarding the body surface and its interaction with the environment
-submodalities: touch, pressure, temperature, pain

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6
Q

Proprioception

A

Sense of posture and moment; a sensation of the position of your different body parts and muscle contraction in space
-different from somatosensation

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7
Q

Modality

A

A particular form of sensory perception

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8
Q

Meissner’s Corpuscles

A

Mechanoreceptors that respond to touch and pressure, rapidly adapting

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9
Q

Merkel’s Corpuscles

A

Mechanoreceptors that respond to touch and pressure, slowly adapting

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10
Q

Free Neuron Ending

A

Close to the surface of the skin

Include nociceptors, thermoceptors, mechanoreceptors,

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11
Q

Pacinian Corpuscle

A

Responds to vibration and deep pressure, rapidly adapting mechanoreceptor

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12
Q

Ruffini Corpuscle

A

Responds to skin stretch, slowly adapting mechnoreceptor

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13
Q

How are Afferents Activated?

A

When mechanoreceptors are activated, sodium channels open and sodium flows down its concentration gradient into the afferent neuron, resulting in a graded depolarization of the sensory receptor

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14
Q

2 Types of Sensory Receptor

A
  1. The sensory receptor is located directly on the afferent fiber
  2. The sensory receptors is located on a specialized receptor cell - releases a neurotransmitter that binds to the receptors of the afferent neuron
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15
Q

APs or EPSPS for Sensory Receptor Activation?

A

EPSP - activation of a sensory receptor generates a grade potential

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16
Q

Receptor Potential

A

The greater the stimulus, the more action potentials that are fired

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17
Q

Stimulus Intensity

A

Most receptors have multiple sensory endings

As more sensory endings are depolarized, more action potentials fire in the afferent neuron

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18
Q

Slow Adapting Receptors

A

Action potentials are generated the entire time that the stimulus is on
e.g. holding your arms out in front of you

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19
Q

Rapidly Adapting Receptors

A

Immediately generates a receptor potential with the initial stimulus; the receptor potential then quickly decays back to baseline
Another receptor potential is generated when the stimulus turns off
e.g. putting on and taking a shirt off

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20
Q

Stimulus Localization

A

Different mechanisms are responsible for stimulus localization so that we have the ability to localize where a stimulus is coming from

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21
Q

3 Factors for Stimulus Localization

A
  1. Receptive field size - the extent of the body which senses the poke
  2. Density of innervations - the number of sensory receptors within a certain area of the skin
  3. Multiple receptive fields exist and some overlap
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22
Q

Receptive Field

A

Different sensory neurons have different receptive field sizes
Each sensory neuron takes information back to the CNS

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23
Q

What Allows for a Better Localization of the Specific Site of Stimulation?

A

Smaller receptive fields

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24
Q

Density of Innervations

A

The more densely packed the sensory receptors are, the greater the ability to localize the stimulus

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25
Q

Overlapping Receptive Fields

A

Helps localize the site of a stimulus
Overlapping receptive fields allows the brain to compute the specific site of the stimulus based on the relative activation of different sensory neurons with the overlapping receptive fields

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26
Q

Lateral Inhibition

A

Occurs when there are overlapping receptive fields
Only in somatosensation and vision
Involves inhibitory interneurons

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27
Q

How Does Lateral Inhibition Work?

A

Information from afferent neurons whose receptors are at the edge of a stimulus is strongly inhibited compared to information from the stimulus’ center
Lateral inhibition enhances the contrast between the center and periphery of a stimulated region, thereby increasing the brains ability to localize a sensory input
Lateral inhibition removes the information from peripheral regions

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28
Q

Center Control of Somatosensory Information

A

Sensory signals are subject to extensive modification before they reach higher levels of the central nervous system

Modification comes from inhibition from collaterals from other ascending neurons, pathways descending from higher centers in the brain, by synapses on the axon terminals of the primary afferent neurons, or indirectly via interneurons that affect other neurons in the sensory pathways

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29
Q

2 Neural Pathways of the Somatosensory System

A
  1. Anterolateral system - pathway which carries pain, or hot/cold information up to the somatosensory cortex
  2. Dorsal column system - pathway which carries information on fine touch mechanoreception to the somatosensory cortex
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30
Q

Anterolateral System

A

Exposure to a painful stimulus activates free neuron endings

  • first synapse is located in the dorsal horn of the grey matter of spinal cord on same side of body which was stimulated
  • secondary neuron crosses over to the other side of the CNS at the level of the spinal cord
  • secondary neuron synapses with projection neuron in the thalamus which travels to somatosensory cortex
  • painful information crosses immediately and travels up the contralateral, or opposite, side of the body
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31
Q

Dorsal Column System

A

A tap should activate mechanoreceptors

  • first synapse between the sensory neuron and the secondary neuron is in the brainstem
  • secondary neuron crosses over to the other side of the CNS at the level of the brainstem
  • secondary neuron synapses with projection neuron in the thalamus which travels to somatosensory cortex
  • touch information travels up the spinal cord on the same side of the body as the stimulation
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32
Q

The Somatosensory Cortex

A

All sensory information goes from the thalamus to the somatosensory cortex
Located behind the motor cortex and the central sulcus
Activates motor cortex neurons
Each region in the body maps to a very specific region in the somatosensory cortex
-the smaller and more densely packed the sensory receptors are, the larger the region in the somatosensory cortex

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33
Q

Vision

A

Photoreceptors in the eye are depolarized at rest and hyperpolarized when activated
-contains an optical component and a neural component

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34
Q

Optical Component

A

responsible for focusing the visual image on the receptor cells, the front part of the eye

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35
Q

Neural Component

A

the back part of the eye where the photoreceptors are located, transforms the visual image into a pattern or graded potentials and action potentials

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36
Q

Sclera

A

white part of the eye, the membrane surrounding the eyeball

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37
Q

Extraocular Muscle

A

attached to the sclera, responsible for eye movements

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38
Q

Cornea

A

responsible for refracting light waves

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39
Q

Pupil

A

the hole that allows light to pass through into the back of the eye

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40
Q

Iris

A

regulates the size of the pupil and amount of light entering the eyeball, gives your eyes colour
-innervated by the ANS

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41
Q

Lens

A

behind the iris; works with the cornea to focus the visual image on the retina; the shape and size of the lens can change

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42
Q

Zonular Fibers

A

attached to the lens; attached to ciliary muscles

43
Q

Ciliary Muscles

A

can contract/relax; change the shape of the lens

44
Q

Retina

A

located behind the lens, back of the eye where the photoreceptors are found

45
Q

Rods

A

activated in very light light conditions and are monochromatic

46
Q

Cones

A

activated when there is more light present and are responsible for colour vision

47
Q

Retinal Ganglion Cells

A

activated by the rods and cones; take information back towards the brain

48
Q

Optic Nerve

A

leaves through the back of the eyeball towards the thalamus and the cortex; made of axons and retinal ganglion cells

49
Q

Aqueous Humour

A

a gelatinous fluid that fills the space between the lens and the cornea

50
Q

Vitreous Humour

A

a gelatinous fluid found behind the lens

51
Q

Refraction

A

Light travels towards the eyeball and bends once they hit the cornea
Structure that is primarily responsible for refraction is the cornea
Lens focuses the visual image on the retina

52
Q

Ciliary Muscles and Refraction

A

When an image comes very close to the eye, the ciliary muscle contracts which causes the lens to get fatter and shorter and increases the amount of refraction; allowing the visual image to focus on the back of the retina

53
Q

Accomdation

A

The process of using ciliary muscles in order to focus on objects that are very close, lose this ability around 45 due to the breakdown of ciliary muscles

54
Q

Prebyopia

A

loss of elasticity of the lens resulting in the inability to accommodate for near-vision; age-related

55
Q

Myopia

A

Can focus on objects close-up but not far away
The eyeball is too long and too much refraction occurs
Corrected by wearing glasses or contact lenses with a concave shape to reduce the refraction

56
Q

Hyperopia

A

Can focus on objects far away but not close up
The eyeball is too short and the visual image is reconstructed behind the retina as there is not enough refraction
Corrected by wearing classes or contact lenses with a convex shape to increase refraction

57
Q

Astigmatism

A

Oblong shape of the eyeball

58
Q

Glaucoma

A

Damage to the photoreceptors due to increased intraocular pressure
-buildup of aqueous humour which pushes on lens, lens then pushes back on the vitreous humour, which pushes back on the retina and photoreceptors causing damage

59
Q

Cataracts

A

Clouding of the lens

Age-related

60
Q

Interneurons in the Eye

A

horizontal
bipolar
amacrine

61
Q

Bipolar Cells

A

interneurons which take information from the photoreceptors to the retinal ganglion cells

62
Q

How are Cones Activated in the Dark?

A
  • guanylyl cyclase converts GTP into cyclic GMP
  • cyclic GMP-gated cation channels are in the membrane of the photoreceptor; the ligand which activates it is cGMP
  • cGMP binds to its receptor on the cation channel; the cation channel opens, allowing sodium and calcium to flow into the cell
  • the photoreceptor depolarizes as the positively charged ions enter the cell
  • relatively depolarized when light is absent
63
Q

How are Cones Activated when it is Light?

A
  • the disk of the cones contains a photopigment which contains a chromophore called retinal
  • when light hits the photopigment, retinal changes conformation from cis to trans conformation; this change in conformation activates a molecule called cyclic GMP phosphodiesterase
  • cyclic GMP phosphodiesterase breaks down cGMP and into GMP
  • cGMP is removed from the ion channel and the ion channel then closes
  • sodium and calcium can no longer enter the cell and the photoreceptor is relatively hyperpolarized when light is present
64
Q

OFF Pathway

A
  • “no light is hitting me”
  • relative depolarization of the photoreceptor
  • graded potentials are generated in the photoreceptors and the result is glutamate release from this photoreceptor
  • OFF bipolar cell is activated by glutamate and ON bipolar cell is inhibited by glutamate
  • when no light is present: action potentials generated in the OFF pathway but not in the ON pathway
65
Q

ON Pathway

A
  • “light is hitting me”

- light is present: ON pathway is activated due to the release of the inhibition on the ON bipolar cell by glutamate

66
Q

Effect of Light on ON/OFF Pathways

A

In both pathways, the photoreceptors is depolarized in the absence of light
When light strikes the photoreceptor cell, the photoreceptor hyperpolarizes
-cGMP in the photoreceptor is broken down by cGMP-dependent phosphodiesterase, decreasing cytoplasmic concentrations of cGMP and allowing cation channels to close -> cell hyperpolarizes -> when photoreceptor cell is hyperpolarized, the cell decreases its release of glutamate

67
Q

Effect of Light on ON Pathway

A

When glutamate release from the photoreceptor cell is decreased, the glutamate inhibition onto the bipolar cell is decreased -> bipolar cell depolarizes and releases more glutamate -> ganglion cell depolarizes and generated action potentials

68
Q

Effect of Light on OFF Pathway

A

There is reduced excitation of the OFF bipolar cell, as glutamate release from the photoreceptor has decreased -> bipolar cell hyperpolarizes and releases less glutamate -> the ganglion cell hyperpolarizes and generates fewer action potentials

69
Q

Neural Pathways of Vision

A

The light signals are converted into action potentials through the interaction of the photoreceptors with bipolar cells and ganglion cells

  • photoreceptor cells and bipolar cells undergo graded responses and lack voltage-gated sodium channels needed to generate an action potential
  • ganglion cells have voltage-gated sodium channels and are the first cells in the pathway where action potentials can be initiated
70
Q

Key Differences Between the ON and OFF Pathway

A
  • Bipolar cells of the ON pathway spontaneously depolarize in the absence of input while bipolar cells of the OFF pathway hyperpolarize in the absence of input
  • Glutamate receptors of ON pathway bipolar cells are inhibitory, while glutamate receptors of OFF pathway bipolar cells are excitatory
71
Q

Neural Pathways of Vision: ON Pathway

A
  • When glutamate is released onto ON bipolar cells, it binds to metabotrophic receptors that cause the breakdown of cGMP which hyperpolarizes the bipolar cell and prevents the release of glutamate onto the ganglion cells
  • no light = no action potentials fired
  • light = action potentials fired because ganglion cells are depolarized
72
Q

Neural Pathways of Vision: OFF Pathway

A
  • OFF pathway bipolar cells have ionotropic glutamate receptors that are non-selective cation channels, which depolarize the bipolar cells when glutamate binds
  • depolarization of bipolar cells stimulates them to release excitatory neurotransmitters onto ganglion cells, firing action potentials
  • OFF pathway generates action potentials in the absence of light (none in the presence of light)
73
Q

What Does the Coexistence of ON and OFF pathways in Each Region Do?

A

improves image resolution by increasing the brain’s ability to perceive contrast at edges or borders

74
Q

Where is the Visual Cortex

A

Occipital Lobe

75
Q

Visual Pathways

A

Information from the lateral field - goes to the opposite side of the brain
Information from the medial field - goes to the same side of the brain

76
Q

Pinna

A

The external ear on the side of the head

77
Q

Where is the Auditory Cortex

A

Temporal Lobe

78
Q

How is Sound Transmitted?

A

air is the most common medium in which we hear sound energy

79
Q

Zones of Compression

A

Regions where air molecules are tightly packed or close together

80
Q

Zones of Rarefaction

A

Regions where there are relatively few air molecules

81
Q

How do Zones of Compression and Rarefaction Move?

A

Ripple outward, transmitting the sound wave over distance

82
Q

Amplitude

A

The volume or loudness of sound
Determined by how many air molecules are located within one of the zones of compression or the difference between the pressure of molecules in the zones of compression and rarefaction

83
Q

Frequency (Pitch)

A

Determined by the distance between the zones of compression, or the number of zones of compression or rarefaction in a given time

84
Q

Tympanic Membrane

A

Ear drum

Vibrates in and out as molecules push against it

85
Q

Cochlea

A

Inner ear

Contains the scale vestibuli, scala tympani, and the cochlear duct

86
Q

Malleus, Incus, Stapes

A

The malleus is connected to the tympanic membrane
Bones act as levers and amplify sound
Skeletal muscles attached to the malleus and stapes contract to dampen movement of bones for loud sounds

87
Q

Muscle Connected to Malleus

A

Tensor tympani muscle

88
Q

Muscle Connected to Stapes

A

Stapedius muscle

89
Q

Why do Muscles in the Ear Contract

A

Muscles contract to protect the ear from consistent, ongoing loud sounds
Muscles would not protect the ear from a loud sudden bang as they would not be contracted when the sudden loud noise occurred

90
Q

Oval Window

A

The stapes pushes against the oval window and the cochlea is full of fluid, the stapes pushes fluid forward within the ear

91
Q

Scala Vestibuli

A

has fluid called perilymph

92
Q

Scala Tympani

A

has fluid called perilymph

93
Q

Cochlear Duct

A

has fluid called endolymph; region of the inner ear where the sensory receptors for the auditory system are located

94
Q

Sound Transmission in the Ear

A

Movement of fluid down from scale vestibuli to scala tympani results in the activation of the sensory receptors for the auditory system

95
Q

Sensory Receptors in the Ear?

A

Hair cells

  • located in the organ of Corti
  • have stereocilia protruding from them at their tips
96
Q

Organ of Corti

A

A specialized epithelium that allows for the transduction of sound vibration into neural signals

97
Q

Stereocilia of the Single Row of Inner Hair Cells

A

extend into the endolymph and transduce pressure waves caused by fluid movement in the cochlear duct into receptor potentials

98
Q

Stereocilia of the 3 Rows of Outer Hair Cells

A

At the bottom, each outer hair cell is attached to the basilar membrane
Different regions of the basilar membrane vibrate maximally at different frequencies

99
Q

Vestibulocochlear Nerve

A

Takes auditory information from the ear towards the brain

100
Q

Hair Cells of the Organ of Corti

A

The hair cells move back and forth when the basilar membrane moves
When stereocilia move, a mechanically-gated potassium channel opens
-how the auditory receptors are activated and how the depolarize
-when the basilar membrane moves, the hair cells move back and forth and the stereocilia bend

101
Q

Neural Pathways in Hearing

A

Cochlear nerve fibers synapse with interneurons in the brainstem
-from the brainstem information is transmitted by a multineuron pathway to the thalamus and hten to the auditory cortex in the temporal lobe

102
Q

Hearing Aids

A

An amplifier which is placed in the auditory canal which activates the existing auditory machinery
Amplifies the existing sounds
Restricted to the outer ear component and can be turned up and down in volume

103
Q

Cochlear Implants

A

Machinery of the ear doesn’t work
A speaker on the outside of the head picks up noises and transduces them into electrical impulses
Electrodes go from the speaker to the vestibulocochlear nerve
The receiver takes auditory information, bypases the outer middle and inner ear, and directly stimulates the nerve

Physiology 210 (12 decks)

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