Special cardiac conditions Flashcards

1
Q

This complication is defined as the inability of the heart to pump enough CO to meet the tissue’s O2 demands, which results in inadequate peripheral tissue perfusion

A

Heart Failure

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2
Q

Name 5 underlying conditions that cause HF

A

ischemia heart disease, MI, cardiomyopathy, valvular heart disease, and hypertension

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3
Q

of people with HF in US

A

5.8 million

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4
Q

HF is accountable for how many hospitalizations a year and how many hospital days

A

1 million, 6.5 million days

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5
Q

True or False, HF is a disease

A

False, HF is a complication of cardiac and non cardiac abnormalities

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6
Q

What is hospitalized heart failure (HHF)

A

new onset or worsening signs and symptoms that require urgent therapy and hospitalization

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7
Q

Non cardiac precipitation factors of AHF include

A

COPD and pneumonia, non adherence to therapeutic regimen, infection, ETOH, diabetes, surgery

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8
Q

Two dysfunction types of HF include

A

systolic and diastolic failure

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9
Q

Systolic HF

A

leads to problems with contraction and ejection of blood resulting in increased system vascular resistance. Increased SVR causes increase in afterload.Also reduced EF

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10
Q

Diastolic HF

A

stiff myocardium doesn’t allow left ventricle to fill up with blood which leads to increased pressure in left atrium and pulmonary vasculature. Preserved EF

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11
Q

Left ventricle can’t pump enough blood out to system circulation, blood backs up into pulmonary circulation and pressure increases in pulmonary venous system, CO falls and weakness and fatigue occur

A

systolic dysfunction

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12
Q

ability of left ventricle to relax and fill during diastole is reduced and stroke volume falls, high volumes needed in ventricles to maintain CO

A

diastolic dysfunction

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13
Q

Signs of left-sided heart failure

A

pulmonary congestion, dyspnea, tachypnea, crackles, dry cough, paroxysmal nocturnal dyspnea, increase BP form fluid excess or decreased BP from pump failure

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14
Q

LEFT sided heart failure FORWARD failure) description

A

Results in PULMONARY congestion due to the inability of the LEFT ventricle to pump blood to the periphery.

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15
Q

Symptoms of left sided failure include

A

SOB, dyspnea on exertion, paroxysmal noctural dyspnea, orthopnea, pulmonary edema, dry & unproductive cough that occurs mostly at night, tachycardia, fatigue, anxiety, restlessness, confusion, tachycardia S3 ventricular gallop

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16
Q

RIGHT sided failure ( BACKWARD failure) description

A

results in PERIPHERAL congestion due to the inability of the RIGHT ventricle to pump blood out to the lungs; often results due from left-sided failure or pulmonary disease

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17
Q

Symptoms of RIGHT sided failure

A

edema in ankles, unexplained weight gain, abdominal pain assoc. with liver congestion, JVD, abnormal fluid in body cavities (pleural space, abd. Cavity), anorexia and nausea assoc. w/ hepatic and visceral engorgement, nocturia, weakness

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18
Q

How many pounds must you gain to have pitting edema?

A

10 lbs or 4.5kg of fluid

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19
Q

Pneumonic to remember the difference between left and right sided HF

A

Right= Rest of the body. Left = Lungs

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20
Q

Ejection fraction

A

Measurement of volume of blood pushed out by ventricles seen on an echo. Normal 55-60%. < 40 =HF or cardiomyopathy. > 75= hypertrophic cardiomyopathy

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21
Q

Treatment for HF

A

treat reversible cause, heart transplant, drugs, cardiac resynchronization therapy

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22
Q

Drugs used to treat HF include

A

digitalis, diuretics, ace inhibitors of beta blockers

23
Q

2 common digitalis

A

digitoxin and digoxin

24
Q

Indications and actions of digitalis

A

HF. Increases the contractility of cardiac muscle and slows HR and conduction by blocking Na/K atpase converting to Ca++.

25
Digitalis neurohormonal control decreases what 3 things and increases what 1 thing.
decreases plasma noradrenaline, peripheral nervous system activity, and RAAS activity, and increases vagal tone
26
Nursing implications for digitalis
check apical pulse prior to administration. Call provider if HR< 60 or > 120 bpm or has a markedly changed rhythm
27
What increases risk for digitalis side effects and toxicity?
hypokalemia
28
5 signs of digitalis toxicity
bradycardia, tachycardia, dysrhythmias, n & v, headache
29
Indication and actions of ACE inhibitors
decreases BP by suppressing renin-angiotensin aldosterone system and inhibiting conversion of angiotensin 1 into angiotensin 2
30
Drugs ending in -prol belong to what class of drugs?
beta blockers
31
Drugs ending in -pril belong to what class of drugs?
Ace Inhibitors
32
Indication and actions of Beta Blockers
Increase density of B1 receptors, inhibits cardiotoxicity of catecholamines, decrease neurohormonal activation and decrease HR
33
Why are Beta Blockers not good for diabetics?
may mask symptoms of hypoglycemia or may prolong hypoglycemic reaction
34
Nursing implications for Beta Blockers
check apical or radial pulse daily, monitor for GI distress, watch for SOB
35
Cardiac Resynchronization Therapy (CRT)
Useful for dilated cardiomyopathy. It is a cardiac pacing that stimulates both ventricles simultaneously. Standard pacemakers only stimulate the right ventricle.
36
Adverse effects of ACE-Inhibitors
hypotension ( 1st dose), worsening renal function, hyperkalemia, cough, angioedema, rash, aguesia (taste), neutropenia
37
There are 5 anti-______ associated with beta blockers, what are they?
antiischemic, antihypertensive, antiarrhythmic, antioxidant, antiproliferative
38
Hypotension, fluid retention/worsening HF, fatigue, bradycardia/heartblock are all adverse effects of what class of drugs?
beta blockers
39
If you notice your patient is having Hypotension, worsening HF, and bradycardic heartblocks, what might be possible interventions you might bring up with the MD
review tx (+/- diuretics or other meds), reduce dose, consider cardiac pacing, discontinue beta blockers in severe cases only
40
What are some common non-pharmacological tx of CHF?
restrict Na to 2-3 g/day, ETOH to 1 drink a day, avoid excess fluid intake, exercise, and treat underlying cause (HTN, arrhythmia)
41
What do you assess in a patient with acute HF?
Respiratory function- SOB, RR, Crackles. Cardiac- heart sounds, vss, color, peripheral pulses, fluid evaluation, enzymes, daily wts. Renal- U.O., BUN, and creatine. GI- bruits, distention. Other - activity intolerance, med hx, IV mgmt. and review of cardiac tests
42
Interventions for pt. with CHF.
monitor vs q 4; monitor apical hr for dysrhytmias, S3, or S4; assess for hypoxia (restlessness, tachycardia, angina); auscultate lungs (wet sounds or crackles), admin o2 prn., edema, Limit Na intake, elevate lower extremities when sitting, rest, diuretics in AM
43
How do you observe for edema?
check daily weights, monitor I & O, measure abdominal girth, observe ankles and fingers
44
What is the indication for a heart transplant?
end-stage heart disease not remediable by more conservative measures
45
What types of meds might a person be on after a heart transplant?
anti-rejection, anti-infective
46
Tacrolimus (prograd), cyclosporine, azathiprine (imuran), steroid (prednisone) are all examples of what type of drug?
Anti-rejection
47
4 types of anti-infectives
ganciclovir, spetra, acyclovir, nystatin
48
After the patient's heart is removed, why are there 2 p waves?
a remnant of the client's atria remains
49
What are symptoms of heart rejection?
hypotension, dysrhythmias, weakness, fatigue, dizziness
50
What is the triple therapy anti-rejection treatment most transplant patient receive?
cyclosporine, azathioprine and prednisone
51
After transplant, when is the patient likely to have a rejection?
6 to 12 weeks.
52
true or false, a patient that receives a heart transplant will require lifelong immunosuppresants?
TRUE
53
Lack of or gain of independence / dependence, marital /partner stresses and donor guilt are all what?
psychological issues with transplant patients
54
What are common electrolyte problems associated with transplant patients?
Mg, K, Phosphate