Sodium Flashcards

1
Q

Sodium?

A
  • Most abundant cation in ECF
  • Only small amt found in ICF
  • Normal serum value 135-145 mEq/L
  • Absorbed via GI & eliminated through urine
  • Most important electrolyte regulating osmolality
  • Important role in generation & transmission of impulses in nerves & muscles
  • Helps regulate acid-base balance
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2
Q

Sodium
-Regulation

A
  • Thirst
  • ADH
  • Aldosterone
  • ANP
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3
Q

Hyponatremia?

A
  • Most common electrolyte imbalance seen in hospitalized patients
  • Typically associated w/ ECF imbalances
  • Inadequate sodium intake
  • Increased excretion
  • Dilutional (water excess, usually assoc w/ hypervolemia)
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4
Q

Hyponatremia
-Risk factors

A
  • Sodium loss
  • Gain of water
  • SIADH
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5
Q

Hyponatremia
-Clinical manifestations

A
  • Largely result of intracellular shift of water
  • Dependent on rapidity & severity of hyponatremia
  • Most common sx r/t H20 shift from vascular space into cells and Na+ role in nerve impulse transmission & muscle ctx
  • Neurologic symptoms do not develop until Na+ value approx 120-125
  • Seizures occur when levels reach 115
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6
Q

Hyponatremia
-Lab values

A
  • Na+ less than 135 mEq/L
  • Serum osmolality less than 280 mOsm/kg
  • SG decreased (except w/ SIADH)
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7
Q

Hyponatremia
-Interventions

A
  • Assess & document LOC, orientation, neuro status w/ V/S
  • I&O
  • Daily wts
  • Monitor serum levels closely
  • Free fluid restriction
  • Encourage foods high in Na+
  • Medications (i.e. Demeclocycline, salt tabs )
  • Admin hypertonic 3% saline solution, only if dangerously low (at Least 118 or lower value)
  • Don’t correct too quickly to prevent neurologic damage secondary to lysis of myelin
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8
Q

Hypernatremia?

A
  • Greater than normal concentration of Na+ in ECF
  • Caused by excess water loss or overall sodium excess
  • May occur w/ water loss, water deprivation, or Na+ gain
  • Primary protection against development of hyperosmolality & hypernatremia is thirst
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9
Q

Hypernatremia
-Risk factors

A
  • Increased sensible & insensible H20 loss
  • Diarrhea
  • Water deprivation/sodium gain
  • Diabetes insipidus
  • Excess aldosterone secretion
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10
Q

Hypernatremia
-Clinical manifestations

A
  • R/t water shift from cells (cellular dehydration) into vascular space
  • Also r/t NA+ role in nerve impulse transmission & muscle contraction
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11
Q

Hypernatremia
-Diagnostic tests

A
  • Sodium greater than 145
  • Serum osmolality above 297
  • SG increased, except w/ diabetes insipidus
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12
Q

Hypernatremia
-Interventions

A
  • I&O
  • Daily wts
  • Assess & document loc, neuro status and orientation w/ v/s
  • IV or oral H20 replacement
  • Tx diabetes insipidus (Desmopressin acetate)
  • Reorient pt, as needed
  • Decrease dietary NA+ intake
  • Correct hypernatremia slowly, over 2 days
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