small groups 2 Flashcards

1
Q

Presence of rales indicate what?

A

Pt likely has fluid of some type in alveolar space (pus, blood, water)

ie: pneumonia (egophony can be heard too)

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2
Q

Does the presence of fever and hypotension with absence of heart sound suggest cardiac or non-cardiogenic edema?

A

noncardiogenic edema

- acute lung injury or ARDS

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3
Q

3 general physiologic reasons for hyperventilation

A
  1. compensation for metabolic acidosis
  2. hypoxemia
  3. CNS causes (trauma, stroke, drugs, voluntary)
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4
Q

if pt is severely hypotensive, what can you do?

A

intubate, give fluids, start on vasopressors

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5
Q

major classifications of pulmonary edema

A

1, cardiogenic

2. noncardiogenis

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6
Q

What is considered an normal PCWP?

A

15 mmhg

  • larger = cardiogenic P edema
  • normal or smaller = noncardiogenic P edema
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7
Q

what is a common way to measure compliance?

A

pt is on a ventilator and an inspiratory pause is performed

v/p

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8
Q

what would compliance be for pts with pneumonia and has developed ARDS? what does that mean?

A

decreased lung compliance (loss of surfactant –> increase in lung water and surface tension)

this means that it will take a higher pressure to deliver a similar volume on the ventilator

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9
Q

scleroderma

A

join pain -

scleroderma: indicative of collagen vascular disease

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10
Q

bilateral symmetric lower extremity edema usualy occurs due to?

A
  1. increase in venous P
    or
  2. decrease in oncotic P (starlings law)
    (ie: low serum albumin)
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11
Q

Post-capillary HTN is associated with which WHO group for pulm HTN?

A

WHO group 2: pulmonary venous HTN or Pulm HTN due to LH Disease

Kerly B lines and pulmonary edema on CXR will be seen on post cap HTN

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12
Q

Which WHO groups will demonstrate normal lung volumes, and spirometry, and a decreased DLCO

A

1: Pulm arterial HTN

4. Chronic thromboembolic Pulmonary HTN

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13
Q

How can you exclude WHO Group 4 HTN?

A

Chronic thromboembolic pulmonary HTN (4) can be excluded by looking for emboli using a ventilation-perfusion scan (preferred) or a CT with contrast

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14
Q

What neurologic disorder can also lead to respiratory muscle weakness and fatigue?

A

Guillan barre

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15
Q

hypercapnea will do what to respiration? Will giving O2 help dyspnea?

A

hypercapnea is a potent respiratory stimulus

  • problem is too much arterial CO2, giving O2 wont help dyspnea
  • ie: hypercapnea via CNS derrangement

if pt is also hypoxic, then you can give them supplemental P2
- ie: hypercapnea + hypoxemia via obesity

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16
Q

If pt has a normal A-a gradient, does this tell you anything about DLCO? What if there is a restrictive process going on?

A

Yes! after correction, DLCO will be normal

DLCO would be reduced in restrictive process due to low lung volumes

17
Q

Key signs pts have neuromuscular disease

A
  1. muscle forces PImax and Pemax will be ↓
  2. cannot voluntarily blow their PCO2 down to normal
  3. hypoventilation will cause dyspea (since central chemoreceptors are in tact)
18
Q

What does elevated hct do to exacerbate RHF?

A

Hypoxemia stimulates erythropoetin release from the kidney, which increases hemoglobin levels. The elevated hemoglobin and hematocrit levels increase the viscosity of the blood, which increases resistance to flow (it is easier to pump water than honey through a tube).

This increases the work of the right ventricle.

19
Q

Hypoventilation in fat people are called?

A

Obesity hypoventilation syndrome (Pickwickian syndrome)

- can be hypercapneic and hypoxemic 24 hours a day, not just sleep apnea at night