SI Acid/Base_Exam 4 Flashcards
____________ Imbalances
_______________
< 1.5 mEq/L
- Increased neuromuscular excitability
- Positive Chvostek’s (face twitches when masseter tapped) and Trousseau’s (hand tightens when blood pressure cuff kept above systolic pressure for long period of time)
- Insomnia
- Nystagmus
_______________
> 2.5 mEq/L
- Decreased neuromuscular excitability
- Flushing
- Diaphoresis - sweating
- Hypotension - low blood
- Bradycardia - low heart rate
Magnesium Imbalances
Hypomagnesemia (< 1.5 mEq/L)
Hypermagnesemia (> 2.5 mEq/L)
_________ imbalances
Neuronal Excitation/Bones
\_\_\_\_\_\_\_\_\_\_\_\_ < 8.5mg/dL 1. Increased neuromuscular excitability 2. Positive Chvostek’s (face twitches when masseter tapped) and Trousseau’s (hand tightens when blood pressure cuff kept above systolic pressure for long period of time) 3. Muscle cramps 4. Seizures
\_\_\_\_\_\_\_\_\_\_\_\_ > 10.5 mg/dL 1. Decreased neuromuscular excitability 2. Constipation 3. Muscle weakness 4. Diminished reflexes 5. ↓ LOC
Calcium Imbalances
Hypocalcemia
Hypercalcemia
Normal PaCO2 is 35 – 45 mmHg
Normal HCO3 is 22 – 26 mEq/L
Normal pH is 7.35 – 7.45
Study
• Occurs when PaCO2 is greater than 45 mmHg and pH is decreased
• Causes
o Alveolar hypoventilation
excess of CO2 in the blood (hypercapnia), which means excess carbonic acid
May be caused by depression of the respiratory center and disorders of the lung (pneumonia, PE)
• Signs/Symptoms
o Compensation by the kidneys: increased elimination of H+ ions
o Headache, blurred vision, breathlessness, restlessness, disorientation, muscle twitching, respiratory rate is rapid at first and gradually becomes depressed as the respiratory center adapts to increasing CO2
• Treatment – restoration of adequate alveolar ventilation removed excess CO2
Respiratory Acidosis
• Occurs when PaCO2 is less than 35 mmHg and pH is increased
• Causes
o Alveolar hyperventilation (deep, rapid respirations)
decreased CO2 (hypocapnia), which means decreased carbonic acid
May be triggered by hypoxemia, hypermetabolic states, salicylate intoxication, hysteria, cirrhosis, and gram negative sepsis
• Signs/Symptoms
o Compensation by kidneys: decrease H+ excretion and bicarbonate reabsorption
o Dizziness, confusion, tingling of extremities, convulsions, coma
• Treatment – treat the cause particularly hypoxemia
Respiratory Alkalosis
• Causes
o Diabetic ketoacidosis (occurs with type 1 diabetes when person has too little insulin)
o Renal failure
o Alcohol
o Aspirin overdose
o Prolonged diarrhea (losing bicarbonate)
• Signs/Symptoms
o Headache and lethargy
o Compensatory Kussmaul respirations
Metabolic Acidosis
• Causes:
o Hyperaldosteronim (decrease potassium)
o Diuretics (the ones that are not potassium sparing – aka loop diuretics)
o Antacids (increases the bicarbonate level)
o Vomiting
- S/S – weakness, tetany, muscle cramps, hyperactive reflexes, atrial tachycardia
- Treatment – treat the condition
Metabolic Alkalosis
Less concentrated to Higher concentrated
Osmosis
Water flows where _______ goes
sodium
_______ – equal concentration of solution
• D5W > when absorbed turns into a hypotonic
• Patients are given isotonic to ^ extracellular fluid:
- Blood Loss
- Dehydration
- Surgery
Isotonic
_______ – low concentration of solution
- Fluid moves intracellularly = cell swelling
- Cell swelling causes lyses = Rupture
- You can become hypovolemic
TX:
DKA
Hyperglycemia – hydrates the cells
Hyperosmolar
DON’T give _______ to a patient with ICP, burn patients, or trauma patients because they are hypervolemic.
Hypotonic
_______ – excessive concentration of solution
• Fluid wants to move extracellularly = shrink cell
If given IV can cause a fluid overload – pulmonary edma
TX
Cerebral Edema
Hyponatremia = Reduces swelling
Hypertonic
Potassium
Body it trying to DITCH K+
Diarrhea I nadequate intake – anorexia/lacking diet T oo much water intake C H igh fluid loss – vomiting
CM: Bilateral Ascending flaccid muscle weakness Cardiac Dysrhythmias – U waves Constipation Polyuria
At Risk:
Patients with n/v, diarrhea
Patients with excessive sweating
Hypokalemia
Potassium
Body CARED too much for K+
C ellular movement – massive transfusion A R enal failure – Oliguria E xcessive K+ intake – IV infusion D rugs - Use of K+ sparing diuretics
CM: Bilateral Ascending flaccid muscle weakness Cardiac Dysrhythmias – cardiac arrest N/V Oliguria
At Risk:
Patients with Chronic Kidney Disease
Patients with Chronic Diarrhea
Hyperkalemia
Sodium LOVES EXTRAcellular
Causes: Loss of Na+ Dilution of Na+ by water excess Vomiting Suctioning
CM: S eizures A pprehension L ethargy T otal Confusion
At Risk
Acute nausea and vomiting
Acute fluid loss
Hyponatremia