Shock, Sepsis, and Multiple Organ Dysfunction Syndrome Flashcards

1
Q

3 types of distributive shock

A

septic
anaphylactic
neurogenic

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2
Q

refractory phase of shock

A

unresponsive to therapy and irrevresible

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3
Q

causes of hypovolemic shock

A

bleeding
dehydration
burns
N/V
diarrhea

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4
Q

causes of cardiogenic

A

MI
tamponade
drugs
cardiac
surgery

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5
Q

obstructive shock cause

A

PE
tamponade
congentical
penuo

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6
Q

MAP less than

A

60

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7
Q

hypovolemia preload

A

decreased

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8
Q

if preload is decreased in hypovolemia what happens to tissue perfusion

A

decreases

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9
Q

hypovolemic shock nursing management

A

intake and output
weights

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10
Q

cariogenic causes

A

failure of heart to pump effectively

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11
Q

cardio what happens to SV and CO and this leads to

A

decrease SV and CO and this leads to impaired tissue perfusion

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12
Q

cardio s/s
- resp

A

resp alk (imprve oxygen

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13
Q

cardio
- heart

A

tachy

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14
Q

cardio
- left ventricle failing

A

pulmonary edema
- crackles
- rhonci
- CVP/PCWP >15

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15
Q

cardio
- severe symptoms

A

met acid
renal failure
cerebral hypo perfusion

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16
Q

obstruction causes*****

A

cardiac tamponade
massive PE
tension pneumo

17
Q

s/s tamponade

A

muffled heart sounds
JVD
paradoxical pulse (decrease in BP with inhalation)

18
Q

s/s PE

A

right ventricular failure

19
Q

s/s tension

A

decrease or absent breath sounds on affected side
hypertesonant
tracheal deviation to unaffected side

20
Q

anaphylactic vessel

21
Q

anaphylactic s/s

A

vasodilation
increased capillary membrane permeability
decrease venous return

22
Q

anaphylactic CO

23
Q

treatment of anaphylactic

24
Q

neurogenic s/s

A

profound hypotension (vasopressor)
BRADYCARDIA
poikilothermic (no sweating, inability to control temp)

25
difference between sepsis and shock
organ involved = shock
26
sepsis/shock s/s
hypotension hypoxemia change in LOC met acid (increase lactic) increase temp decrease urine output
27
Q sofa
altered mental status fast rr low bp
28
what should we do before antibiotics
culture
29
primary MODS
well defined insulin
30
secondary MODS
widespread sustained systemic inflammation
31
DIC will they bleed
yes
32
DIC increased labs
PT PTT fibrinogen split products
33
DIC decreased labs
platelets fibrinogen
34
cause of DIC
massive transfussion hypothermia trauma
35
DIC treatment
platelet FFP heparin
36
SIRS what is it
produce similar response to sepsis without an organism
37
SIRS criteria
fever/hypotehrm achy and hypo tachypnea elevate or decreased WBC
38