Heart Failure Flashcards
how come heart failure patients might die
ventricular arrythmia or a fib
twin epidemics due to rising of both issues
heart failure and a fib
decrease output causes decrease EJ which leads to
decrease blood flow to organs
things that affect preload
volume
things that affect afterload
blood pressure/vasoconstricition
what dose increase afterload do to the heart
increase in muscle size because heart has to work harder
if EF is low what does that do to the volume
increase ECF
if EF is low and there is not a lot of perfusion to kidneys what happens
increase in renin and angiotensin and then an increase in salt and water which leads to fluid overload
is the fluid overload in HF always in the vessels
no so it doesn’t necessarily increase preload
what type of relationship is associated with afterload and cardiac performance
inverse
names for HF with EF less than 40%
Reduced, systolic, HFrEF
names for HF with EF greater than 40%
persevered, diastolic, HFpEF
left sided HF will have what symptoms
LUNGS
- crackles
what symptoms with right sided HF have
peripheral edema
REST OF BODY
causes of depressed ejection fraction
myocardial infarction
hypertension
volume overload
causes of preserved EF
pathology hypertropy
aging
decreased CO will lead to what in the vessels
vasoconstriction (ultimately leading to increase afterload)
what effect will angiotensin 2 have on preload and aferload
increase in both
RAAS happens becasue
the body is attempting to compensate
what gender experiences what type of HF
men is reduced
women is preserved
blood pressure in reduced
normal to low
blood pressure in preserved
hypertension
edema in reduced
pitting
edema in preserved
no edema
EJ in reduced
low
EJ in preserved
normal or increased
JVD in reduced
yes
