Exam 2 Review Flashcards
Bradycardia can be caused by
hypoxia
what medication can we give to fix bradyacardia
atropine
what is our first goal for Vtach
pulse
agonal rhythm is called
dying heart
what do we do in agonal rhythm
CPR
what med do we use for agonal
epi
what do we do in Vfib
check pulse
torsades is treated with
mag
failure to capture we need to increase
milliamp
SVT med
adenosine
SVT treatment
cardioversion
A fib treatment and some contraindications
cardioversion but this depends on the time because Afib are more at risk for mural thrombi and we would not want to release a clot
PR interval normal
0.12-0.20
how to measure PR
beginning of P to start of Q
QRS normal
0.10 or less
how to measure QRS
start of Q to end of S
QT interval time
0.38 or less
how to measure QT
start of Q to end of T
chronotropic drugs
affects the rate
inotropic drugs
contractility
epi chrono and ino
increase for both
dig chrono and ino
increase ino
decrease chrono
dopmine chrono and ino
increase both
beta blockers chrono and ino
decrease both
low does of dopamine does what
dilates renal and mesentery arteries
increase of dopamine does what
beta and alpha stimulation
what should we monitor with dig
K
doubutamine ino
increase
when is doubtamine used
heart failure cardiogenic shock
epi stimulates what
a and b
norepinephrine low does impacts
beta
norepi high dose impacts
a
class I
blocks Na channels
how do class 1 drugs work
prolongs the absolute refractory period and decreases the risk of premature impulses, decreses automaticity
examples of class 1 drugs
quindine
procaniamide
lidocaine
class 2
beta blockers
how do class 2 work
inhibits dysrhythmias mediated by SNS
competes with endogenous catecholamines for receptor sites
example of class 2
propanolol
metaprlol
esmolol
how do class 3
slows repolarization and prolongs action potential
examples of class 3
amiodarone
dofetilide
ibutilide
class 4
calcium channel blockers
how do class 4 drugs work
blocks calcium channel primaryilu in atria, sinus and AV node; depresses sinus AV condition and terminates tachys
proarrythmic drugs
quindine
procaniamide
dofetilide
ibutilide
cardiac output equation
heart rate x stroke volume
stroke volume 3 components
preload
afterload
contractility
preload
volume
afterload
resistance
what can make afterload higher
vasoconstriction
what drugs do we use in asthma
beta agonist
ARDS common side effect
refractory hypoxemia
- increase O2 but sats still low
chest tube does what
restore the negative pressure in the pleural space
what side do we turn a patient with diffuse disease throughout all left lung fields
right side
- good side down
ischemic stroke
blood vessel supplying the brain is occluded
raccoons eyes and battles sign is found in
basilar fractures
anisocoria
unequal pupil size
classic presentation of epidural hematoma
unconsiousness
lucid
rapid deterioration
normal ICP
0-15
cerebral perfusion pressure normal
70-100
how to calculate CPP (cerebral perfusion pressure)
MAP-ICP=CPP
treatment of increase ICP is
manitol
what do we need to know about
use a filter
cushings triad
increase systolic BP
decrease pulse
decrease respiration
what does Cushing triads indicate
increase in ICP
what are some things that can increase ICP
suctioning
straining
increase CO2
cardiac glycosides
a fib CHF
asthma
beta agonist
ARDS Xray
at first normal
then white out
how to give phenoytoin (dilantin)
undiluted
syringe pump thought large bore needle into large forearm vein