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NUR5925 > SHOCK > Flashcards

SHOCK Flashcards

1
Q

Define shock

A
  • Shock is a life threatening condition of inadequate circulatory functioning
  • Inadequate tissue perfusion resulting in impaired cellular metabolism and functioning
  • potentially life-threatening
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2
Q

Discuss the pathophysiology relating to the four stages of shock

A

Initial stage:

  • hypo-perfusion begins
  • imbalance between supply and demmand
  • anaerobic metabolism begins
  • cellular acidosis is developing

Compensatory stage:

  • Compensatory mechanisms activated (decreased CO stimulates baroreceptors and chemoreceptors –> adrenaline and noadrenaline released)
  • Blood flow to the heart lungs and brain is maintained (decreased blood flow to the kidneys, RAAS activated, ADH released)

Progressive stage:

  • compensatory mechanisms fail
  • Decreased ATP production
  • Hypoxia of vital organs
  • decreased cellular perfusion and tissue ischaemia
  • Failure of sodium-potassium pump
  • Altered cell membrane permeability
  • Metabolic acidosis
  • Decreased CO
  • Myocardial Ischaemia
  • Increased gastric ulcers and GI bleeding
  • Increased risk of disseminated intravascular coagulation
  • Acute renal failure
  • Aggressive management required to prevent MODS

Irreversible/Refractory stage:

  • Compensatory mechanisms overwhelmed
  • severe tissue hypoxia with ischaemia, necrosis and death of cell
  • build up of toxins
  • MODS
  • recovery unlikely
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3
Q

Identify the clinical manifestations of the four stages of shock

A
  • hypotension
  • hypoxia
  • tachy/bradycardia
  • decreased UO
  • diaphoresis
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4
Q

Identify the four classifications of shock

A
  • Cardiogenic
  • Hypovolaemic
  • Distributive
  • Obstructive
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5
Q

Identify the three types of distributive shock

A
  • Septic shock
  • Anaphylactic shock
  • Neurogenic shock
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6
Q

List the causes of the types of distributive shock

A
  • Septic shock (infection - inflamm)
  • Anaphylactic shock (allergic reaction - allergen)
  • Neurogenic shock (lesions or pressure on the spinal cord)
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7
Q

Discuss ‘cold’ shock

A
  • Usually a late and ominous stage of septic shock
  • Generally irreversible and indistinguishable from terminal hypovolaemic shock
  • Characterised by a decreased cardiac output, low temperature, and low WCC
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8
Q

Discuss ‘warm’ shock

A
  • Usually 1st phase of septic shock preceding cold shock
  • characterised by high cardiac output and low peripheral vascular resistance
  • fluid begins to shift into third spaces
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9
Q

Identify the three primary aims in the management of shock

A
  • fluid replacement
  • reperfuse
  • identify and treat cause
  • prevent MODS
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10
Q

Outline the aims of ED management for Hypovolaemic shock

A 
Airway - assess and secure
Breathing - Maximise O2 carrying capacity
 - Assess RR/WOB/SpO2/ABG’s (PaO2>80mmHg)
 - apply O2T
 - appropriate ventilation
Circulation - 
Assess:
– Source of fluid loss,
– HR, BP, Cap refill, Peripheral pulses, U/O, ECG/CCM
Interventions:
– Control external bleeding
– Two large bore IVC
– Fluid resuscitation
– CVC
– Artline
– IDC
– Where indicated adrenaline NOT NORADRENALINE
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11
Q

Compare the administration of crystalloid and colloid for fluid resuscitation

A

Crystalloids:

  • salts and electrolytes
  • commonly used for intravascular expansion
  • low molecular weight
  • inexpensive
  • readily available
  • short half-life
  • extravascular space expander

Colloids:

  • large molecules
  • provide oncotic pressure as well as intravascular volume
  • longer half life
  • risk of anaphylaxis
  • not readily available
  • intravascular space expander
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12
Q

Discuss the administration of blood products to a patient in a shock state

A
  • Blood products are most suitable in a hypovolaemic shock from an origin of blood loss
  • blood products should be used cautiously as it is a colloid solution and cannot pass through the membrane. Therefore could cause fluid overload.
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13
Q

Define systemic inflammatory response syndrome (SIRS)

A

Systemic inflammatory response syndrome (SIRS) is an inflammatory state affecting the whole body. It is the body’s response to an infectious or noninfectious insult. Although the definition of SIRS refers to it as an “inflammatory” response, it actually has pro- and anti-inflammatory components.

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14
Q

Define multiple organ dysfunction syndrome (MODS)

A

Medical Definition of multiple organ dysfunction syndrome.: progressive dysfunction of two or more major organ systems in a critically ill patient that makes it impossible to maintain homeostasis without medical intervention and that is typically a complication of sepsis and is a major factor in predicting mortality

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15
Q

List the causes / predisposing factors for MODS

A 
Sepsis and septic shock are most common causes
Other triggers:
– Burns,
– Acute pancreatitis
– Major surgery
– Severe trauma
– Circulatory shock
– ARDS
– Necrotic tissue
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16
Q

Discuss pro-inflammatory and anti-inflammatory mediators

A

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17
Q

Discuss the mechanism of tissue injury in MODS

A

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18
Q

Discuss the ED management of a patient with MODS

A

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19
Q

Define the terms positive and negative inotrope

A

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20
Q

Define the terms ‘positive chronotrope’ and ‘negative chronotrope’

A

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21
Q

Define cholinergic transmission

A

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22
Q

List the two types of adrenergic receptors and their location

A

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23
Q

Describe the effects when adrenergic receptors are stimulated

A

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24
Q

List the location and action of dopaminergic receptors

A

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25
Q

Discuss the use of Adrenaline in the critically unwell patient

A

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26
Q

Discuss the use of Noradrenaline in the critically unwell patient

A

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27
Q

Discuss the use of Isoprenaline in the critically unwell patient

A

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28
Q

Discuss the use of Dopamine in the critically unwell patient

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29
Q

Discuss the use of Dobutamine in the critically unwell patient

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30
Q

Discuss the use of Metaramimimol in the critically unwell patient

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31
Q

Outline the risk factors that would increase your index of suspicion for a patient presenting with signs of shock

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32
Q

Describe the three main aims of shock management

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33
Q

Explain the interventions that are available in your ED to increase arterial oxygenation

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34
Q

Identify the interventions that are available to you in the ED to improve tissue perfusion

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35
Q

Provide an example of a colloid solution found in your department

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36
Q

Discuss fluid resuscitation, including type of fluid, for a patient in cardiogenic shock

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37
Q

Outline the benefits for using packed cells, plasma and platelets

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38
Q

Differentiate between an inotrope and a chronotrope

A

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39
Q

Specify where α-adrenergic receptors are located and when stimulated explain the effect

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40
Q

Specify where β-adrenergic receptors are located and when stimulated explain the effect

A

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41
Q

Specify where dopeminergic receptors are located and when stimulated explain the effect

A

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42
Q

Which receptor is stimulated when adrenaline is administered

A

– Catecholamine
– Direct acting sympathomimetic – α and β agonist
– Low doses β effects – e.g. increase HR
– Higher doses – α effects – e.g. vasoconstriction
– Maintains coronary and cerebral blood flow

43
Q

Which receptor is stimulated when noradrenaline is administered

A

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44
Q

Which receptor is stimulated when isoprenaline is administered

A

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45
Q

Which receptor is stimulated when dopamine is administered

A

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46
Q

Which receptor is stimulated when dobutamine is administered

A

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47
Q

Which receptor is stimulated when metaraminimol is administered

A

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48
Q

List the adverse reactions of adrenaline

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49
Q

List the adverse reactions of noradrenaline

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50
Q

List the adverse reactions of isoprenaline

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51
Q

List the adverse reactions of dopamine

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52
Q

List the adverse reactions of dobutamine

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53
Q

List the adverse reactions of metaraminimol

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54
Q

For each shock state identify which inotrope would be most appropriate and rationalise why

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55
Q

Discuss the nursing considerations when administering inotropes to a patient in a shock state

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56
Q

Explain the ED management of a patient presenting with anaphylactic shock

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57
Q

Discuss the ED management of a patient presenting in septic shock

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58
Q

Describe the complications of a persistent hypothermic state and how are they treated

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59
Q

Explain the mechanism of tissue injury in MODS

A

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60
Q

Discuss the supportive care of a patient with MODS

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61
Q

A 56 year old male presents to the ED complaining of spontaneous onset of severe back pain whilst watching TV. The patient has a past history of hypertension and obesity.

On arrival the patient is diaphoretic, anxious and complaining of 6/10 back pain. His abdomen is hard and distended. An abdominal aortic aneurysm is suspected.

On returning from an ultrasound the patient becomes difficult to rouse, responds only to verbal stimuli and BP is 80 systolic.

Discuss your assessment management and rationale for this case

A

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62
Q

Outline the aims of ED management for Cardiogenic shock

A

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NUR5925 (27 decks)

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