SDH Flashcards

1
Q

def

A

bleeding or a collection of blood between the dura mater and arachnoid mater

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2
Q

aetiology

A

commonly trauma which occurred long ago or was so minor it is forgotten
trauma often causes a rapid acceleration-deceleration injury which results in shearing of the ‘bridging veins’

non-trauma causes include reduced ICP and dural metastases

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3
Q

epi

A

acute - more common in younger patients with head trauma

chronic - more common in elderly

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4
Q

history

A

characteristic is a headache, fluctuating level of consciousness with or without an insidious physical or mental slowing, and unsteadiness

acute
-history of head trauma and reduced consciousness
subacute
-worsening headache 1-2wks post head trauma
chronic
-headaches, confusion
-gait deterioration + focal weakness

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5
Q

what are the risk factors or associations of SDH

A

age
alcoholism (falls)
anticoagulation

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6
Q

examination

A

acute
-reduced GCS
-raised ICP (worse on coughing + leaning forward)
-large haematomas cause
chronic
-focal neurological signs (unequal pupils, hemiparesis)

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7
Q

investigations

A

CT head

  • crescent-shaped mass over brain surface, acute subdurals are hyperdense and appear white, subacute are isodense and appear a light grey colour, chronic are hypodense and appear black
  • may also see midline shift
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8
Q

management

A

acute
-ABCDEFG
-if signs of raised ICP, elevate the head and consider IV mannitol +/ hyperventilation
-once stable, obtain CT-head
conservative treatment for asymptomatic (small) SDHs
surgical approach for symptomatic (large) SDHs
-wash out or remove the blood using a burr twist drill and burr hole craniostomy, this is considered first line
-craniotomy is second line

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9
Q

complications

A

raised ICP + cerebral oedema pre-dispose to secondary ischaemic damage or tentorial herniation and coning

seizures + meningitis are common post surgery

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10
Q

prognosis

A

chronic SDH has better prognosis than acute SDH

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