SCI Flashcards

1
Q

how’d reeve die

A

pressure sore ischial tuberosity - cardiac arrest

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

3 concerns of SCI

A

walking bowel sex

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

SCI in injuries

A

7%

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

paraplegia

A

motor and or sensory legs

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

tetraplegia

A

motor /sensory legs n arms

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

another name tetraplegia

A

quadriplegia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

para or tetra more common

A

tetra

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

after 45 most common cause SCI

A

falls

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

concussion

A

from blow, temporary loss function

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

contusion

A

gill and cord are intact

loss of central grey /white matter

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

laceration

A

glial disrupted, cord torn

transection possible

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

mechanism of SCI

A

excessive flexion / extension

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

excessive flexion who, extension who

A

flexion younger

extension older ppl

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

general patho of SCI

A

first 18 hours necrotic death of axons damaged
lesion program up n down (ie. 4 sets)
apotosis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

primary injury

A

structure damage instantly after event

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

secondary injury

A

extensive injuries if not immobilized

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

what trigger necrosis

A

excitotoxic changes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

how long can necrosis last

A

weeks

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

how much of cord supports locomotion

A

10% so save everything

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

T/F preventing edema helps SC injuries

A

true

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

whats demyelination do

A

reduce firing rate

degenerates

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

how much grey matter loss in SCI

A

1-1.5 spinal segments about the lesion

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

what happens to neural function below lesion

A

accelerated aging n loss of function

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

diaschesis

A

atrophic changes from loss of signals in brain

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Q

phases of spinal shock

A

1-4

26
Q

phase 1 spinal shock

A

loss reflexes below injury
one day
hyperpolarized

27
Q

phase 2 spinal shock

A

return of some reflexes

28
Q

phase 3 spinal shock

A

monosynaptic (deep tendon) reflexes restored

29
Q

phase 4 spinal shock

A

hyperreflexia

internureron and LMN attempt to reestablish

30
Q

A asia

A

complete. no motor or sensor in s4/5

31
Q

B asia

A

incomplete

sensory preserved below neuro level and s4/5

32
Q

C asia

A

incomplete

motor preserved below neuro level + half mm’s below neuro have grade 3

33
Q

D asia

A

incomplete

motor preserved below neuro level + grade 3 or more

34
Q

E asia

A

motor n sensory are normal

35
Q

Complete injury

A

absence of sensory n motor function in segment below the injury

36
Q

incomplete injury

A

partial motor n sensory below neuro level

37
Q

sacral sparring

A

voluntary anal spinster contraction or sensory function

38
Q

why sacral sparring important

A

some white matter long tracts (corticospinal and spinothalamic tracts intact)

39
Q

why don’t CNS regenerate as PNS

A

CNS has oligos
myelin debris removed
CNS myelin has inhibitory factors
CNS astro make scars

40
Q

brown squared syndrome

A

Hemi section of cord
ipsilateral
contralateral
dorsal column tract

41
Q

brown squared ipliateral feature

A

mm wekaness
abnormal reflex
clonus / spastic

cortico / reticospinal damage

42
Q

brown squared contralateral features

A

pain n temp loss belo lesion

lateral spinothalmic

43
Q

contralateral spinothalamic tract lesion

A

loss of pain temp light ouch below dermatome

44
Q

genearl brown squared

A

loss all modality at one or more dermatomes

loss of pain / temp below specific dermatome

loss proprio, disc touch , limb weakness at similar level

45
Q

partial lesion also known as

A

brown

46
Q

complete cord lesion

A

bilateral loss all modalities

bilateral leg weakness

47
Q

central cord lesion

A

bilateral loss of pain n temp

has proprio and discrimination sensations

48
Q

syringomyelia

A

cyst in cord
spontaneous or after injury
blocks CSF flow

49
Q

symptoms stringomyelia

A

pain spasm phantom

reflex changes

50
Q

wheres autonomic dysreflexia

A

above t5

51
Q

autonomic dysreflexia

A

irratating stimulus below injury
reflex activated
results in vasospasm (inc BP)

52
Q

signs of autonomic dysrefelxia

A
pounding headache
goose bumps
sweating above injury
nasal congestion 
cold clammy skin below injury
53
Q

associated changes after SCI

A

spastic
thermal regulation
skin
mm

54
Q

spasticity

A

inc sensory responsiveness

55
Q

spasticity from

A

distended bladder, infection , smoking, fracture, stressors

56
Q

treat spasticity

A

meds, positioning, stretching

57
Q

thermal regulation

A

loss of tract from hypothalamus

Shivers n sweats

58
Q

most common cause of death SCI

A

resp - pneumonia

59
Q

bowel and bladder controlled by

A

s2-4

60
Q

at conus what happens to bladder

A

flaccid

61
Q

above s2 what happens bladder

A

reflex

62
Q

mm issues for SCI

A

fatigue
ulcers
stress injuries
osteoporosis