Ruminant Ophthalmology Flashcards

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1
Q

ruminant fundi - arteries

A

Cows and sheep
* 3-4 major arterioles with venules
* come off at right angles
* Kidney bean-shaped optic disc
<><>
Goat
* More major arterioles
than cow/sheep
* 15-30 minor arterioles
* Round optic disc
(cow > sheep)
<><>
Camelids
* 3-4 major arterioles with venules
* 15-30 minor arterioles
* Right angles
* Atapetal

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2
Q

Ocular examination - dilation method

A

Ruminants: use atropine 1%, Camelids: use tropicamide.

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3
Q

Local Blocks
1. Auriculopalpebral nerve block

A
  1. Auriculopalpebral nerve block
    * Motor only to upper eyelid
    * Zygomatic arch
    > Where the a-p nerve
    crosses the zygomatic
    arch.
    > 6 mg/kg. 5-6 ml 2% lidocaine hydrochloride
    * WDT: mean and milk = 24 hrs
    <><><><>
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4
Q

Peterson block

A
  1. Peterson block
    - Analgesia to the orbit and globe but not eyelids
    (CN III, IV, V1,2,3, VI)
    - Results in: mydriasis without PLR
    - Slightly curved 10 cm 18 ga needle
    - 15-20 ml lidocaine
    - Risk of death if injected into blood vessel/dural sheath therefore aspirate before injection.
    - Directed from the notch between the zygomatic arch and the supraorbital bone towards the back of the eye.
    - Direct the needle straight in until the mandibular ramus is touched, then redirect the needle forward toward the back of the globe
    (pterygopalatine fossa). Often you can see the eye move in response.
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5
Q

Four-point block

A
  • Analgesia to the orbit and globe (CN III, IV, V1,2,3, VI) but not eyelids.
  • 6 cm” needle inserted between orbit and globe (trans conjunctival/trans palpebral) 12, 3, 6, 9 o’clock position: 5-10 mls lidocaine at each site.
  • Safer than Peterson Block
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6
Q

Eyelid Line block

A
  • local infiltration of 2% lidocaine above the upper eyelid incision and below the lower eyelid incision.
  • avoid large volumes as it will distort the eyelid tissues.
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7
Q

Strabismus - etiologies, C/S

A
  • Abnormal ocular globe position
    <><>
  • Hereditary (Holstein, Jersey, Ayshire, German Brown, Swiss, Shorthorn)
    <><>
    Acquired
  • Orbital neoplasia (LSA)
  • CN dysfunction from listeriosis,
  • Polioencephalomalacia
    <><>
    C/S:
  • Divergent (exotropia)
  • Convergent (esotropia)
    Strabismus + exophthalmos
  • Shorthorns can be
    progressive
  • If severe: visual impairment
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8
Q

Nystagmus
etiolgies

A

Constant oscillating horizontal movement of the ocular globe
<><>
Hereditary congenital:
* Ayrshires, Guernsey’s, and Holsteins
<><>
Acquired:
* Calves that develop dysfunction of their
peripheral vestibular system or cerebellum
following an ear infection or systemic disease
* Can be seen with listeriosis, MCF, PEM, rabies
<><>
Treatment: none

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9
Q

Microphthalmia
etiologies, clinical signs

A
  • Congenitally small eyes
    <><>
    Inherited or acquired
  • BVD - unilateral or bilateral
    <><>
    Clinical Signs:
  • small ocular globe
  • protrusion of third eyelid
  • small palpebral opening
  • iris anomalies
  • cataract
  • retinal detachment/dysplasia
    Treatment: enucleation (to reduce
    infection)
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10
Q

Entropion
- etiology
- clinical signs, treatment

A
  • The inversion of eyelids (usually lower)
    <><>
    Neonatal cattle
    Secondary
  • systemic diseases (dehydration, wt
    loss)
  • corneal ulcers
    <><>
    Clinical signs (uni/bi):
  • lacrimation from irritation
  • corneal ulceration
  • conjunctivitis
  • vision deficits (if bilateral)
  • reduced growth/weight gain
    <><>
    Treatment:
  • tacking sutures (temporary)
  • Hotz-Celsus (permanent)
  • Temporary correction with placing vertical mattress sutures (tacking sutures)
  • Permanent correction with removal of a skin crescent with wound closure (Hotz-Celsus)
  • Topical antibiotics and atropine if corneal ulcers present
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11
Q

Conjunctiva and Cornea
Dermoid
- what is it?
etiology, clinical signs, tx

A

Ectopic skin cells
* Hair follicles, skin glands
<><>
Third eyelid, conjunctiva, cornea
<><>
Herefords
* Genetically predisposed
<><>
C/S: Epiphora
<><>
Treatment:
* Surgical excision
(superficial keratectomy)

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12
Q

Infectious Bovine Keratoconjunctivitis (IBK) = “pink eye”
- causes, transmission

A

Calves: predominately
Moraxella bovis
* Gm-negative bacillus
<><>
Secondary pathogens
* Mycoplasma and IBR
<><>
Transmission: flies
> Summer outbreaks, indoor housing winter
> Herefords, Hereford crosses, Murray Grays
* Highest susceptibility

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13
Q

Infectious Bovine Keratoconjunctivitis (IBK, “pink eye”) cont’d
clinical signs, progression

A

Initial stages:
Hyperemic conjunctiva
Blepharospasm
Serous discharge
<><>
Progression:
Mucopurulent discharge
Corneal edema, ulceration, neovascularization
Corneal perforation
Secondary uveitis is also commonly seen

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14
Q

Infectious Bovine Keratoconjunctivitis (IBK, “pink eye”) treatments, prevention

A

Control the fly population
Isolate affected cattle
<><>
Antibiotics:
* IM long-lasting tetracycline (q3d)
* Topical tetracycline (TID)
* Topical atropine (BID)
<><>
Corneal rupture:
* Conjunctival flaps or enucleation
<><>
Vaccination: debatable efficacy

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15
Q

Infectious Bovine Rhinotracheitis (IBR)
- clinical signs, dx, tx

A

Herpes virus
<><>
C/S: Ocular and systemic (respiratory) disease
Mimics IBK ocular signs (serous-m/p discharge, conjunctivitis, corneal bv’s)
BUT no ulceration
<><>
Dx: Viral culture or paired serum samples for titres
<><>
Tx: Controlling secondary bacterial infections with topical antibiotic
ointments TID (tetracycline, BNP)

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16
Q

Uveitis
Bacterial
- etiology, pathogens
- c/s, tx, prevention

A

Bacteria entering blood stream → uvea
> Navel Ill (septicemia - Omphalitis/Omphaloplebitis)
> Salmonella, Streptococcus, E.coli, Klebsiella, Rhodococcus
<><>
C/S: conjunctival and episcleral hyperemia miosis, aqueous flare, hypopyon, vitritis epiphora → mucopurulent sticky discharge
<><>
Tx: anti-inflammatory medication – topical and systemic
atropine – topical
antibiotics – systemic
<><>
Prevention:
clean calving area
adequate navel treatment
adequate passive antibody transfer
(3 litres of good quality colostrum during the first 2 hour).

17
Q

Listeria monocytogenes (“Silage eye”)
- c/s, tx, prevention

A

Secondary to feeding of big-bale silage
<><>
C/S:
* same as Navel Ill
* Bluish-white corneal opacity
* Pain (esp in bright light)
* Blindness (if inflammation of visual cortex)
<><>
Tx:
* procaine penicillin G (early stages)
* atropine (topical)
* anti-inflammatory medications (steroid and NSAID)
<><>
Prevention:
* monitor silage quality
* minimize eye contact while feeding

17
Q

Histophilus somni (prev Haemophilus somnus)
- what is it? who affected and when
- systemic and ocular signs

A

Gram-negative bacterium
All ages, all months but mostly feeder calves (fall-winter) W. Canada
=one of the four major pathogens of the bovine respiratory disease (BRD) complex
(with Mannheimia haemolytica, Pasteurella multocida, and Mycoplasma bovis).
<><><><>
Systemic signs: thrombotic-meningoencephalitis (“sleeper sickness”), pneumonia, abortion and other reproductive diseases, mastitis, polyarthritis, pericarditis, myocarditis, and septicemia
<><>
Ocular signs: conjunctival and episcleral hyperemia , miosis, aqueous flare, hypopyon, vitritis, retinal hemorrhages, chorioretinal scars (when healed)
<><>
Treatment:
* procaine penicillin G (early stages)
* atropine (topical), anti-inflammatory medications
(steroid and NSAID)
<><>
Prevention:
* vaccines – questionable efficacy
* prophylactic antibiotics in starter rations

18
Q

Malignant Catarrhal Fever
- who gets it?
- C/s
- tx

A

Cattle, llamas, deer, bison - systemic
Ovine herpes virus-2 infection
<><>
C/S:
* OU: lacrimation, serious to mucopurulent ocular
discharge, episcleral and conjunctival hyperemia, severe corneal edema, miosis, hypopyon, and iritis
* not choroid
<><>
Histopathology
<><>
Tx:
* supportive care: flunixin, fluoroquinolone, top prednisolone acetate 1%, top atropine
* high mortality rate

19
Q

Cataract
- etiology
- tx

A

Congenital, genetic or acquired
<><>
Genetic:
* Hereford, Holstein, and Jersey
<><>
Acquired:
* BVD can be transmitted form the mother in the early to mid-stages of the pregnancy
Anterior uveitis
<><>
Treatment
* Monitor
* Cataract surgery