Review Questions Exam 3- Final Exam Flashcards

1
Q

Name the two parasites (only genus) that parasitize the stomach of horses. What are their specific predilection sites?

A

◦ Drascheia – in tumor-like swellings in the stomach (nodules with creamy pus)
◦ Habronema – on the mucosal surface (does not penetrate the intestinal wall.

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2
Q

How are stomach worms of horses transmitted? Direct or indirect lifecycle? Intermediate hosts?

A

◦ Indirect lifecycle - The housefly, Stable fly intermediate host
◦ Fly larva develops into adult fly, and then deposits nematode around mouths and lips and are then swallowed.

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3
Q

Which nematode causes summer sores in horses? Where are the larvae found on the horse?

A

◦ Cutaneous habronemiasis- Infective larvae (L3) deposited on skin wounds

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4
Q

What is the key clinical manifestation of Habronemiasis in horses? How can you control the infection?

A

◦ “Summer sores” Cutaneous habronemiasis – easy to diagnose based on c/s and L3 morphology( Non-healing skin wounds, Intense pruritus, Larvae in wound scrapings)
◦ Control: Ivermectin (ML) is effective, Control by proper manure management – break the lifecycle, reduce fly population, Fly repellents

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5
Q

Name the most common ascarid of horses? What age group of horse sheds the greatest number of ascarid eggs?

A

◦ Parascaris equorum
◦ Worm counts high at around 5 months of age!

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6
Q

Know the typical egg morphology of equine ascarid, Parascaris equorum.

A

◦ Sticky, Thick, Rough Shell, resistant

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7
Q

Which parasite undergoes hepatic-tracheal migration in horses? What is the clinical importance of this migration?

A

◦ Parascaris equorum -> Physical damage/scarring of liver, lungs, and intestine

◦ Strongyloides westeri-> ?

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8
Q

What is the most significant transmission route of equine threadworm?

A

◦ Trans-mammary (lactogenic) – most significant route of transmission in foals. Larvae shed in milk for up to 2 months

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9
Q

Which intestinal helminth can cause diarrhea in a 2-week-old foal?

A

◦ Strongyloides westeri (Equine threadworm)

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10
Q

What is the shed stage of Strongyloides westeri?

A

◦ Larvated eggs (L1)

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11
Q

Which large strongyle of horse is commonly referred to as “blood worm”?

A

◦ Strongylus vulgaris (Blood worm)

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12
Q

What is the key pathogenicity of Strongylus vulgaris?

A

◦ Verminous arteritis, Thrombus – may lead to necrosis

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13
Q

What is the common name of small strongyles of horses?

A

◦ Cyathostomins

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14
Q

Do small strongyles migrate to visceral organs?

A

◦ Do not migrate beyond mucous membrane

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15
Q

How does larval cyasthostominosis occur? Which horses are most likely to suffer from this? What are key clinical symptoms?

A

◦ Occurs in late winter/early spring, when enormous number of larvae emerge from into the gut lumen simultaneously!
◦ Young horses are more susceptible, but any ages can be infected during any season
◦ Diarrhoea, Serious colic and mortality up to 50% , Sometimes may result to granulomatous colitis

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16
Q

Is fecal flotation useful in the diagnosis of larval cyasthostominosis? Why?

A

◦ Fecal flotation and egg counts not useful – Condition caused by larval stages (not egg-producing adults)

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17
Q

What are common clinical signs associated with pinworm infection in horses? How can you diagnose pinworms? Approach and general morphology?

A

◦ c./s - Tailrubbing, automutilation
◦ Usually swab or tape mount from perianal region . Fecal float ( less sensitive- eggs not released in feces)
◦ Eggs morphologically distinct from other helminths : asymmetric (flat on one side of oval) with plug on one end. Larval development within egg – very tough!
◦ Morphology (adult): Pointed tail 3/4 length of entire body, may see adult females passed in feces or tail sticking out of perianal region. Classically greyish/white.

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18
Q

What is the most common species of tapeworm in horses? How does the transmission occur?

A

◦ Anoplocephala perfoliata- ileum and Caecum. Transmitted via free living mites (oribatid mites) Horse must ingest mite that had ingested eggs with hexecanth larvae/ pyriform apparatus.

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19
Q

Remember the characteristic morphology of an equine tapeworm egg.

A

◦ Eggs with hexacanth larvae/ pyriform apparatus

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20
Q

Where, on the body of the horse, can you find the adults of Onchocerca cervicalis? How is Onchocerca transmitted among horses?

A

◦ Filarial nematode – Adults in connective tissues - ligamentum nuchae in Equines
◦ Vector-borne – Flies (Biting midges) - Microfilaria in the dermis, rarely in the blood

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21
Q

Which parasite can cause severe coughing in horses, which is often difficult to diagnose from another disease? What do you recommend (approach) for diagnosing this parasite?

A

◦ Dictyocaulus arnfieldi – Lungworm
◦ Baermann method – Larvae in feces, Clinical signs – Respiratory disturbances, Examination of airways, x-rays, Bronchoscopy – BAL may contain larvae, egg

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22
Q

Which species of Eimeria is common in horses? Is this pathogenic?

A

◦ Eimeria leukarti
◦ Nonpathogenic

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23
Q

Which species of Sarcocystis is pathogenic in horses? What is the disease caused by this organism? What is the role of horses in the epidemiology of this species? What species maintain this parasite in the wild? Pathophysiology of EPM? The most common symptoms?

A

◦ Sarcocystis neurona
◦ Equine Protozoal Myeloencephalitis
◦ HORSES are Aberrant IH
◦ Opossum maintains this parasite in the wild.
◦ Pathophysiology of EPM:Zoites cause direct destruction of host neural cells, Acute immune response/inflammation.
◦ Clinical signs: 3A’s - Asymmetry, Ataxia, and Atrophy
‣ Weakness, Muscle atrophy, Ataxia, Seizures, Lameness, Head tilt, facial paralysis, Loss of reflexes

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24
Q

Which parasites are considered as foreign animal diseases (FAD) in horses?

A

◦ Babesia caballi,
◦ Theileria (Babesia) equi

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25
Q

What is the key clinical sign of Babesiosis in horses?

A

◦ Acute hemolytic anemia
◦ Fever > 40 C ! Dark urine (hemoglobinuria) Dx: Blood smear

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26
Q

Which mange can lead to “mud fever” in horses?

A

◦ Chorioptes equi

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27
Q

Which mite species of horses are most contagious and which species is least contagious?

A

◦ most contagious: Chorioptes equi , Psoroptes equi, Scarcoptes scabiei var. equi,
◦ least contagious: Demodex equi

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28
Q

Name the sucking and biting/chewing lice of horses?

A

◦ Chewing louse: Dorsolateral trunk - Damalinia (Werneckiella) equi
◦ Sucking louse: Mane, tailhead and fetlock- Haematopinus asini

29
Q

Which species of flies can cause stomach bots in horses? Know the general lifecycle of horse botflies.

A

◦ Gasterophilus intestinalis:
‣ eggs on forelimbs and shoulders
‣ L3 in the cardiac region of the stomach
Lifecycle:
◦ Adult flies lay eggs on hair of legs-> larvae (L1) ingested by horse -> L1 in gingiva -> L2 and L3 in GI tract-> horses shed Larvae (L3) in feces -> Pupae in environment -> become adult flies

30
Q

Name the stomach worm of pigs? How does the transmission occur?

A

◦ Hyostrongylus rubidus- Red stomach worms of pig. Pasture transmitted through ingestion of L3.

31
Q

What is the pathophysiology of stomach worms in pigs?

A

◦ In heavy infections – Pathogenesis like Ostertagiasis
‣ Damage to parietal cells à Increase in Gastric pH
‣ Catarrhal gastritis (characteristic)

32
Q

Which parasite is most common in indoor/intensively raised pigs?

A

◦ Ascaris suum – Swine roundworm

33
Q

What is the pathogenesis of Ascaris suum? What is the characteristic lesion formed by the migrating larvae?

A

◦ Adult worms may significantly reduce the growth rate of young pigs
◦ Rarely mechanical obstruction of intestine
◦ MIGRATING LARVAE causes hemorrhage, fibrosis and accumulation of lymphocytes in the liver – “MILK SPOTS”
◦ Liver – first infection - physical damage
◦ Following infections - hypersensitivity
◦ Allergic inflammation
◦ Eosinophilic infiltration
◦ “Milk-spots”
◦ Very rarely severe respiratory signs – Abdominal breathing –“THUMPS” or “HEAVES”

34
Q

Which parasite is more common in nursing piglets?

A

◦ Strongyloides ransomi / S. suis

35
Q

What is the shed stage of Strongyloides ransomi?

A

◦ Shed stage is embryonated egg excreted in pig feces or laid by free living females.

36
Q

How can you control Strongyloidiasis in pigs?

A

◦ Treat sows 14 days before farrowing and/or piglets <14 days (benzimidazoles, ML)
◦ Hygiene – clean & disinfect farrowing pens, keep floors dry

37
Q

Which parasites are maintained by older sows in a pig breeding herd?

A

◦ Oesophagostomum spp. (nodular worm of large intestine)

38
Q

Know the general morphology of Trichuris.

A

◦ Morphology: typical whipworm, “lemon-shaped” egg (hardy!)

39
Q

What is the predilection site of Metastrongylus spp. in pigs? How does the transmission occur?

A

◦ Adults in bronchi and bronchioli
◦ Transmission: Ingestion of earthworm with L3

40
Q

Which parasites of swine use earthworms as intermediate/paratenic hosts?

A

◦ Metastrongylus spp. (swine lungworm)- intermediate hosts
◦ Stephanurus dentatus (Swine kidney worm)- paratenic hosts

41
Q

What is the causative agent of verminous bronchopneumonia in young pigs housed outdoors?

A

◦ Metastrongylus spp. (Swine Lungworm)

42
Q

What is the kidney worm of swine? How do you diagnose this parasite?

A

◦ Stephanurus dentatus (Swine kidney worm)
◦ Eggs may be recovered in urine - Confirmatory diagnosis with worms and lesions during necropsy

43
Q

How can you control swine kidney worms?

A

◦ Confinement
◦ Sanitation
◦ Earthworm control
◦ Gilts only breeding program

44
Q

What is “gilts-only” breeding program? Why can this be effective in controlling swine kidney worms?

A

◦ Breeding gilts and then selling them after weaning, as well as replacing older boars replaced with young boars from clean heards.
◦ This will prevent patent infection from developing

45
Q

What parasite has its larval stage in the muscle tissues of hosts? How does the transmission occur? Is Trichinella zoonotic? How do people get Trichinellosis?

A

◦ Trichinella spp.
◦ Transmitted through consumption of infected meat – Foodborne zoonosis
◦ Yes it is zoonotic
◦ People can get it by eating infected meat that is not cooked thoroughly, infected meat that is cured, drying, smoking, or microwaving the meat.

46
Q

How do you diagnose trichinella infection in animals?

A

◦ You would have to look at tissue digestion. Typically tongue or diaphragm is used. Blend/ chop muscle tissue then mix with pepsin, hcl for one hour. Then do sequential sedimentation. Examine under microscope

47
Q

How can we prevent trichinellosis in humans and in animals?

A

◦ Do not let pigs eat meat scraps, each other, or infected rodents/other wildlife
◦ Cook meat thoroughly prior to human consumption

48
Q

Which mite species is also called Hog mange mite? How can we diagnose this species?

A

◦ Sarcoptes scabiei var. suis
◦ Dx: deep skin scraping

49
Q

Which species of coccidia causes clinical coccidiosis in pigs? What is the common clinical signs?

A

◦ Cystoisospora suis
◦ Clinical Signs: Yellow-gray, pasty diarrhea – often no blood

50
Q

Which ciliate protozoan cause moderate to severe diarrhea in pigs? Is this protozoon zoonotic?

A

◦ Balantidium coli: Causes balantidiasis – Zoonotic disease

51
Q

How do pigs get Toxoplasma infection? Do pigs shed toxoplasma oocyst?

A

◦ consume oocysts in feed contaminated with cat feces, Or consume tissue cysts in undercooked meat or rodents
◦ No - they are intermediate hosts if anything, cats shed the oocyst.

52
Q

What is the scientific name of the large roundworm of poultry? How is it different from the ascarids of other animals?

A

◦ Large roundworms – Ascarids - Ascaridida galli
◦ No migration, develop in intestinal mucosa, return to lumen and develop into adult

53
Q

Which nematode of poultry can end up in the eggs?

A

◦ Ascaridia galli

54
Q

Which parasite of poultry infects the crop and esophagus? How do you diagnose this parasite? High- level egg morphology?

A

◦ Capillaria spp. Threadworm/Capillarid worm of poultry
◦ Dx: Fecal Float
◦ high level egg morphology : Eggs bipolar, golden brown, thick wall.

55
Q

Which parasite can be found in cecal blind pouches of poultry? What is the clinical importance of this nematode?

A

◦ Heterakis gallinarum
◦ Worms donot affect chicken typically – worms carry Histomonas melegradis – protozoan
‣ Causes Histomoniasis (blackhead) in turkeys

56
Q

*** Which parasite causes respiratory distress in poultry? Common name? Where are these parasites found typically? Which protozoal parasite of poultry can cause respiratory signs?

A

◦ Syngamus trachea
◦ Common Name: Gapeworm
◦ Adults in trachea blood-feed (anemia)
◦ Cryptosporidium spp?

57
Q

Name the burrowing and surface mites of poultry.

A

◦ Burrowing mites- C nemidocoptes (also Knemidocoptes)
‣ C. mutans – Scaly Leg
‣ C. gallinae – De-pluming Itch
‣ C. pilae – “Beak Rot”
◦ Surface mites
‣ Dermanyssus gallinae ( Red Mite, Roost Mite)
‣ Ornithonyssus sylvarum ( Northern Fowl Mite)

58
Q

*** Which parasite can cause scaly leg lesions and beak rot in poultry?

A

◦ Scaly leg: C. mutans
◦ Beak rot: C. pilae

59
Q

What is the main difference between red mite and northern fowl mite? (Morphology and behavior) How do you control mite problems in poultry?

A

◦ Dermanyssus - On birds at night, environment during the day, anus posterior in anal plate
◦ Ornithonyssus - On birds all the time, obligate blood feeder anus anterior in anal plate
◦ Clean environment, heat > 45 degrees celcius between batches, mite traps, diatomaceous earth (SiO2), sulfer dust. ( W/ ornithonyssus- extralabel ivermectin, 2 tx a week apart) Acaricides.

60
Q

Do poultry get lice infestation? The clinical sign of lice infestation in poultry?

A

◦ Chicken body louse- Menacanthus stramineus
◦ yes, Itching, feather damage (live on feathers and may suck blood out of quill feathers, killing feather), skin lesions

61
Q

Which insect is more commonly found in poultry breeder flocks? What is the clinical importance?

A

◦ Cimex lectularius (Bed bugs)
◦ C/S: excessive feather loss & irritation, lesions on the breasts and legs, and possibly anemia in severe cases
◦ Can cause production loss: fewer egg numbers and increased feed intake

62
Q

Which protozoan parasite causes the blackhead of turkeys?

A

◦ Histomonas meleagridis

63
Q

General lifecycle of Histomonas melagridis? How is this protozoan transmitted within poultry? Main lesion associated with Histomoniasis in poultry?

A

◦ Egg of Heterakis gallinaru is shed in feces, egg is infected with Histamonis melagridis. When cecal worm larvae hatch, Histomonas is released into the intestinal lumen of poultry!
◦ Transmitted directly: flagellate shed in fresh feces or Transmitted indirectly by nematode PH: flagellate inside the larvated eggs of nematode Heterakis gallinaru.
◦ Main Lesion: Postmortem – “bulls-eye” lesions on liver, caseous cores in caecum

64
Q

Which nematode carries Histomonas melagridis?

A

◦ Heterakis gallinaru

65
Q

Which protozoan is more prevalent among domestic pigeons and wild doves?
◦ Trichomonas gallinae/ T. stableri

A

◦ Trichomonas gallinae/ T. stableri

66
Q

If you see caseous necrosis around the oral cavity, orbitals, and brain, which protozoa do you suspect? How does this protozoa transmit between hosts?

A

◦ Trichomonas gallinae T. stableri
◦ Transmission:
‣ From parent bird to their young ( feeding young, adults may carry the pathogen asymptomatically for years)
‣ Through infected saliva/ poop ( contaminated drinking water or feed, trophozioite short lived in environmnent (i.e minutes outside of the host)

67
Q

What are the risk factors for avian coccidiosis?

A

◦ Immunocompromised, stressed – develop coccidiosis

68
Q

How do you diagnose Eimeria infection in poultry?

A

◦ Clinical Signs
◦ Fecal test – Eimeria oocysts
‣ Let to sporulate
‣ 4 sporocysts with 2 sporozoites each!
◦ PM findings – GI lesions

69
Q

What is the key pathology associated with Eimeria infection in poultry? How can you control coccidiosis in poultry?

A

◦ Pathology
‣ Damage to bird’s intestinal lining
‣ Protein leakage into the bowel
‣ Disruption of digestion and nutrient absorption
‣ Dehydration, anemia
‣ Reduced feed and water consumption
‣ increased susceptibility to secondary infections
‣ Runny, off-color droppings (often blood tinged) - Key
◦ Control:
‣ Chemoprophylaxis (anticoccidials in feed/ water) (antibiotics, to control secondaty bacterial growth)
‣ Vaccines in feed, water, sprays or gels; injected into eggs or layers
‣ Supportive care: Important to keep birds hydrated
‣ Environmental/Managemental changes - Hygiene and good husbandry
• Slow down oocyst sporulation
• Keep bedding dry
• No overcrowding
• Rotate birds to different areas