Review posters 16/05/2016 Flashcards

1
Q

What is IBS?

A

Characterised by chronic relapsing abdominal pain and discomfort. Associated with bloating.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Why do people get IBS?

A

Genetics and the environment.

Also 20% of people who have IBS had an infection of infectious gastroenteritis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Characteristics and symptoms of IBS

A
Disturbed gut motility- increase in duodenum but decreased in the stomach
Exaggerated by food
Bloating
Diarrhoea/constipation
Nocturia
Mucus per rectum
Aggravated by stress
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Rome III criteria

A

Over the period of a month having 3 or more days of abdominal pain or discomfort along with 2 of the following:
Improvement by defecation
Onset associated with a change in stool consistency
Difference in stool appearence

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Pathogenesis of IBS

A

Peripheral sensitisation- inflammatory mediators up regulate the sensitivity of nocireceptors
Central sensitisation- spinal nerves become more sensitive

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Treatment of IBS (non pharmacological)

A

Diet- decrease fibre intake
Try to stop tea and coffee
Reduce intake of resistent starch
FODMAP diet

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Treatment of IBS pharmacological

A

Antispasmodics e.g. laxatives.
They will stop constipation. However senna is not could for long term use and lactulose promotes flatulence
Antidiarrhoeals- loperamide
Opiates- relax gut and decrease constipation. However not good for pain
Use PRN or prophylactically
Anti-depressents e.g. tricyclics e.g. amitryptiline
Help to regulate sleep pattern
Reduce diarrhoea

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Haemorrhoids

A

Enlargement of vascular cushion in the lower rectum and anal canal

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Symptoms of haemorrhoids

A

Bleeding (painless)- bright red, fresh and not mixed with stool
Perianal itchiness
May be visible
Examination may be normal

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Investigations into haemorrhoids

A

PR exam normal- haemorrhoids cannot be felt

Rigid sigmoidoscopy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Where do haemorrhoids normally present?

A

when patient is in lithotomy position they are at 3, 7 and 11 o clock due to this being where the branches of the superior haemorrhoidal artery are.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Treatment of haemorrhoids

A

Rubber band ligation
Sclerotherapy (involves inserting phenol and almond oil into the vessels)
Open haemorrhoidectomy
HALO- haemorrhoidal artery ligation- blood vessels tied off- supposedly painless due to dentate line position

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Rectal prolapse

A

Could be complete or incomplete. Incomplete only involves the mucosal layer.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Symptoms of rectal prolapse

A

Protruding mass especially on defecation. May be reducible.
Bleeding and mucus per rectum
Examination always shows poor anal tone.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Management of complete rectal prolapse

A

Most patients too frail to undergo surgery so advice is given on self reducing and diet. Also given a bulking agent
Operations include perineal rectopexy or an abdominal rectopexy.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Anal fissure

A

Tear in the anal margin due to constipation. Described as passing glass.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Symptoms of anal fissures

A

Bleeding per rectum
Acute presentation
PAIN

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Treatment

A

Botox injections
Dietary advice and stool softeners
Sphyncterectomy (pharmacological- 6 week treatment of GTN and diltiazem ointments)
Lateral sphyncterectomy- surgery

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Fistula in ano

A

Abnormal communication between epithelial surfaces.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

Presentation of fistula in ano

A

One or more external holes and an internal opening in the anus. Generally due to inadequately treated or delayed treatment of anorectal abscesses.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

Treatment of fistula in ano

A

Two step process of an operation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

Investigations of fistula in ano

A

Rectal exam under anaestetic
Rigid sigmoidoscopy
Flexible sigmoidoscopy
MRI

23
Q

Which nerve is the fibrous pericardium supplied by?

A

The phrenic nerve

24
Q

Which nerve is the diaphragm supplied by?

A

The phrenic nerve

25
Q

What makes up the phrenic nerve?

A

Combined anterior rami of spinal nerves C3,4 and 5

26
Q

Where is the cardiac plexus located?

A

On the pulmonary trunk

27
Q

What does the cardiac plexus contain?

A

Sympathetic axons
Parasympathetic axons
Visceral afferent fibres

28
Q

Parasympathetic supply to the heart

A

Vagus nerve

29
Q

How do cardiac visceral afferents travel?

A

Associated with sympathetic fibres

30
Q

Describe the path of the phrenic nerve

A

Travels anteriorly to the scalenus anterior and into the chest descending over the lateral aspect of your fibrous pericardium.

31
Q

Recurrent laryngeal nerve

A

Supplies laryngeal muscles

32
Q

Innervation of the mouth down to the trachae

A

First part of nasal cavity- cranial nerve 5 branch 1
Second part of nasal cavity- cranial nerve 5 branch 2
Nasal pharynx and oral pharynx- cranial nerve 9 (glossopharyngeal)
Laryngopharynx- cranial nerve X (vagus)

33
Q

Where is the pulmonary plexus?

A

The bifurcation of the trachae (carina)

34
Q

What does the pulmonary plexus contain?

A

Sympathetic, parasympathetic and visceral afferents

35
Q

Where do the sympathetic nerves supplying the lung, heart and oesophagus arise from?

A

T4-T6

36
Q

Nervous supply of the ribs

A

Intercostal nerves

37
Q

Where do the intercostal nerves arise from?

A

Anterior rami of spinal nerves T1-T11

38
Q

What do branches of the intercostal nerves go on to supply and what are their names?

A

They go on to supply the abdominal muscles. There is the subcostal nerve, then the iliohypogastric nerve and then the ilioinguinal nerve.

39
Q

Organs within the abdomen are supplied by

A

enteric nervous system
Parasympathetic
Sympathetic
Visceral afferents

40
Q

Sympathetic supply to the gut

A

Leave by T5-L2 ( oesophagus T4-T6)
Liver, stomach, small intestine, pancreas T7-T9
Colon and appendix T10-T11
Rectum T12

41
Q

What do they become when they leave the sympathetic chain?

A

Abdominosphlancic nerves

42
Q

Where are the abdominal plexuses

A

Termed periarterial plexuses- they lie on the anterior of the descending aorta. Named according to the artery they are associated with e.g. coeliac plexus.

43
Q

Adrenal gland nervous supply

A

T10-L1
Enter abdominopelvic sphlancic nerves
Carried with periarterial plexus to the adrenal gland where they synapse straight onto it.

44
Q

Parasympathetic supply

A

Vagus nerve supplied whole GI system up to the splenic flexure. Nerves travel to periarterial plexuses and then synapse at the organ.
Then it is supplied by pelvic splanchnic nerves.

45
Q

Causes of bowel obstruction

A

Volvulus- twisting of mobile bits of bowel causing obstruction at its head.
Hernias
Adhesions or bands- could be congenital or could be from previous surgeries
Tumours
Inflammatory strictures e.g. Crohns and UC
Bolus obstruction- food, impacted faeces, gallstones
Intususecpetion- occurs in children quite commonly- part of the bowel is swallowed by the bowel distal to it (telescoped)
Bowel strangulation- URGENT

46
Q

Symptoms of bowel obstruction

A

Vomiting
Constipation
Abdominal distention
Pain

47
Q

How would vomiting differ depending on where the obstruction is?

A

Gastric outlet obstruction- semi-digested food that had been eaten a day or two previously
Upper small bowel obstruction- copious, bile stained fluid
Lower small bowel/colonic obstruction- thickened, brown, feaculent vomitus

48
Q

The more proximal the obstruction occurs:

A

The earlier vomiting develops

49
Q

Large bowel obstruction

A

Symptoms develop later due to massive capacity of the colon and the caecum.

50
Q

Ileocaecal valve and large bowel obstruction

A

If the ileocaecal valve is competent- symptoms will arise sooner due to backward flow of accumalated bowel content being prevented.
The thin walled caecum distends with swallowed air and eventually may rupture.
If the ileocaecal valve is incompetent- the small bowel will distend delaying symptoms

51
Q

Incomplete obstruction

A

Clinical features may be less clear
Vomiting may be intermittent and bowel habit may be erratic.
Chronic obstruction leads to gradual hypertrophy of the muscle wall.

52
Q

Investigations into bowel obstruction

A

AXR supine- bowel proximal to obstruction is distended with gas
CT
In a distended small bowel you can see plicae circularis
In a distended large bowel you can see haustra coli.

53
Q

Management of bowel obstruction

A

Nil by mouth
IV cannula and send blood
Resusitate with IV fluids
Nasogastric tube to decompress stomach

54
Q

Adynamic bowel obstruction

A

Paralytic ileus- failure of peristalsis- symptoms and signs similar to bowel obstruction. Risk factors are recent GI surgery, peritonitis and diabetic keto acidosis.
Treat ‘drip and suck’ while awaiting peristalsis to resume
Psuedo obstruction- acute dilation of the colon in the abscense of obstruction in acutely unwell patients