Review IV Flashcards

1
Q

SOAP PMH for OMT

A

birth Hx
sports Hx
trauma Hx
braces

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2
Q

SOAP OMT procedure

A

need consent

pt and dr perception of improvement

CPT code

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3
Q

SOAP assessment for OMT

A

DDx - not specific diagnosis but area of SD

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4
Q

estrogen

A

after menopause - HTN increases in women

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5
Q

african american

A

more likely to have HTN

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6
Q

causes of essential HTN

A
white coat
metabolic syndrome
genetics
salt intake
obstructive sleep apnea
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7
Q

causes of secondary HTN

A
kidney
adrenal gland
congenital vessel defect
meds
illegal drugs - coke/amphetamines
thyroid disorders
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8
Q

age 60 and older

A

initiate pharmacy for BP > 150/90

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9
Q

age 30-59

A

initiate pharmacy for BP > 140/90

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10
Q

age less than 30

A

initiate pharmacy for BP > 140/90

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11
Q

cardiovascular sympathetics

A

T1-5,6

-sympathetic

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12
Q

renal sympathetics

A

T6-L2

-sympathetic to adrenal gland and kidney

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13
Q

tight psoas major

A

altered motion and function in lower thoracic/upper lumbar region

catecholamines from adrenal - increased CO and TPR

activated RAAS - vasoconstriction and sodium/fluid retention via aldosterone

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14
Q

sympathetics on kidney

A

vasoconstriction of afferent arterioles

  • decreased GFR
  • increased renin
  • decreased urine
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15
Q

T1-6,5

A

sympathetics to heart

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16
Q

T6-L2

A

sympathetics to adrenals and kidney

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17
Q

clavicles

A

affecting anterior cervical fascia - and carotid artery baroreceptors

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18
Q

cranium SD

A

SBS compression
occipitomastoid compression
affecting jugular formane

occiput, atlas and c spine

alter carotid receptor function
-contribute to alterations in blood pressure

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19
Q

kidney chapmans

A

1 “ lateral and 1 “ superior from umbilicus

intertransverse region T12-L1

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20
Q

myocardium chapmans

A

anterior between ribs 2 and 3 near sternal border

posterior between T2-3 near spinous processes

21
Q

adrenal chapmans

A

1” lateral and 2 “ superior from umbilicus

intertransverse region T11-12

22
Q

JNC8 meds

A

for HTN

thiazide diuretics, ACE inhibitors, ARBs, CCBs

titrate at monthly intervals

23
Q

chapmans for adrenal reseaerch

A

serum aldosterone dropped as well as systolic and diastolic BP

24
Q

kidney

A

retroperitoneal T12-L2
-surrounded perirenal fat

fascia - funnel - apex on top

fascia unites with its pair at level of T12-L1 in front of vertebrae

tethering one kidney affects the other

25
Q

tethered kidney

A

traction on renal a
-changing laminar to turbulent flow

decrease in BP in afferent arteriole across JG apparatus
-renin release

also increased neural input because of stretch on arterial supply

26
Q

sympathetics to heart

A

T1-5,6
-upper thoracic and cervical chain ganglia synapses

right sided - right deep cardiac plexus - right heart and SA node

left sided - left deep cadiac plexus - left heart and AV node

27
Q

SA node

A

right sided sympathetic fibers

28
Q

AV node

A

left sided sympathetic fibers

29
Q

increased sympathetics to heart

A

increased workload
increased O2 demand
decreased O2 supply
increased cardiac tissue irritability

30
Q

parasympathetics to heart

A

decreasead workload
decreased irritability and arrhythmias
decreased morbidity and mortality

31
Q

sympathetic ganglia

A

attached to rib heads

32
Q

hemorrhagic stroke

A

linked to HTN

happened to FDR

33
Q

louisa burns

A

did experiment to increase SD at atlas, T3 and rib 3

increased HR, weak pulse, irregular rhythm, atlas lesions - variable rhythm

resulted in:

  • cross striations in myocardium
  • hemorrhages in myocardium
  • increased edema between muscle fibers
  • overfilling of blood vessels
  • loss of visible plasma layer within the vessels
34
Q

OMT after CABG

A

increased O2 sat 1-2 hours post surgery

35
Q

cardiac index

A

CO normalized to body surface area

with OMT after CABG
-increased - more CO

36
Q

sleep apnea

A

predictor for CAD

  • also in heart failure patients
  • associated with HTN
  • hypoxia and hypercapnia - increase sympathetics

CV4 - can improve sleep in normal subjects

37
Q

cardiorenal model for HF

A

excessive salt or water retention or renal dysfunction

-systolic heart failure

38
Q

hemodynamic model for HF

A

reduced CO and excessive TPR

increased EF doesn’t always correlate with improved outcome

decreased peripheral vasoconstriction did not improve EF

39
Q

muscle hypothesis model for HF

A

abnormal skeletal m results in activation of muscle ergoreceptors which leads to enhanced signals to resp ventilation and sympathetic activation

fix with exercise, OMT, biofeedback, yoga, cardio-selective beta blockers

40
Q

constraint on diastole

A

internal contour of lower thoracic cage

-kyphosis, scoliosis, pectus excavatum

41
Q

ANG II

A

activates collagen synthesis

42
Q

aldosterone

A

stimulates collagen deposition

stimulates sodium retention

43
Q

rigid thoracic cage and diaphragm restriction

A

decreased area for heart during diastole

44
Q

exercise

A

release of endothelial nitric oxide

beneficial effect

45
Q

course of vagus nerve

A

originates on medulla

exits skull at jugular foramen
-between occipital and temporal bones

connections with first 2 cervical somatic nerves

enters chest via thoracic inlet

46
Q

occipitomastoid compression on jugular foramen

A

parasympathetics

47
Q

lymph from heart and lungs

A

right lymphatic duct

-through thoracic inlet

48
Q

OMT for HF

A

reduction of SNS to upper left thoracic

cranial - CV4

rib raising - decrease sympathetics