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Epi Pq Final Viro CORRECTED > Retrovirus > Flashcards

Retrovirus Flashcards

1
Q

Retroviruses are frequently carried lifelong

A

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2
Q

Retroviruses carry an integrase enzyme

A

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3
Q

Malignant transformation of host cells is a typical effect of several retroviruses

A

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4
Q

Retroviruses are enveloped, their resistance is low

A

T

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5
Q

The reverse transcriptase transforms DNA of the retroviruses to mRNA

A

F

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6
Q

Retroviruses are stable viruses; genetic changes are rare

A

F

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7
Q

Retroviruses are euryxemic agents

A

F

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8
Q

Retroviruses are frequently carried lifelong

A

T

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9
Q

Retroviruses are generally host specific viruses

A

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10
Q

Mutation of retroviruses is very rare

A

F

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11
Q

Immunosuppression is a typical effect of several retroviruses

A

T

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12
Q

Retroviruses can integrate into the genome of host cells

A

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13
Q

Reverse transcriptase is an important enzyme of retroviruses.

A

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14
Q

Retroviruses results in lifelong infection.

A

T

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15
Q

Retroviruses replicate mainly in the endothelial cells.

A

F

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16
Q

Several retroviruses can cause malignant transformation in the hosts

A

T

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17
Q

Retroviruses are generally species specific

A

T

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18
Q

Retroviruses are generally resistant, they can survive in the environment for several weeks

A

F

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19
Q

Retroviruses frequently cause permanent infection

A

T

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20
Q

Retroviruses are generally stable viruses, mutations are very rare

A

F

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21
Q

Retrovirus has weak resistance

A

T

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22
Q

Retrovirus has a wide host spectrum

A

F

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23
Q

Retrovirus has a good immunogenicity

A

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24
Q

Retrovirus infection is long-lasting

A

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25
Q

Retroviruses show high host specificity

A

T

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26
Q

Retroviruses are generally not carried for more than a month

A

F

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27
Q

Retroviruses generally cannot survive in the environment for a long time

A

T

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28
Q

Retroviruses are enveloped viruses

A

T

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29
Q

Retroviruses transcribe their nucleic acid to DNA

A

T

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30
Q

Frequent genetic changes of retroviruses are common

A

T

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31
Q

Retroviruses carry reverse transcriptase enzyme

A

T

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32
Q

Retroviruses generally cause long, frequently life-long infection

A

T

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33
Q

Retroviruses are generally genetically very stable.

A

F

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34
Q

The resistance of retroviruses is generally good, they survive in the environment well.

A

F

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35
Q

Reverse transcriptase is produced by retroviruses

A

T

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36
Q

The nucleic acid of retroviruses can be integrated into the genome of the host cell.

A

T

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37
Q

Retroviruses frequently cause immune suppression

A

T

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38
Q

Retroviruses are enveloped viruses

A

T

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39
Q

The host range of retroviruses is generally narrow

A

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40
Q

Reverse transcriptase converts RNA of retroviruses into DNA

A

T

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41
Q

Retroviruses are generally very stable viruses, mutations are exceptional in them.

A

F

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42
Q

Retroviruses are generally shed in infected lymphoid cells

A

T

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43
Q

Certain retroviruses can cause proliferation of the lymphoid cells

A

T

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44
Q

Retroviruses spread with infected lymphocytes

A

T

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45
Q

Retroviruses have a tegument or rind

A

F

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46
Q

You cannot multiply retrovirus artificially

A

F

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47
Q

Retroviruses can incorporate into the genome

A

T

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48
Q

Retroviruses cannot spread from animal to animal.

A

F

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49
Q

Retroviruses are widely distributed in Hungary

A

T

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50
Q

Retroviruses replicate mainly in endothelium cell

A

F

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51
Q

Retrovirus can replicate without helper retroviruses.

A

T

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52
Q

Retroviruses can integrate the cellular genome

A

T

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53
Q

The resistance of retroviruses is low, they cannot survive in the environment for a long time.

A

T

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54
Q

Retroviruses are generally good antigens.

A

T

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55
Q

Retroviruses have own metabolic enzymes

A

F

would have thought else thats why then integrate into the genome and use host cell

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56
Q

Antibodies against enzootic bovine leukosis virus can be detected 1-4 months after infection.

A

T

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57
Q

Antibodies against enzootic bovine leukosis virus can be detected only for 1-2 months after
infection

A

F

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58
Q

Maternal Antibodies against enzootic bovine leukosis virus can be detected only for 1-2
months

A

F

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59
Q

Lymphosarcoma can be seen postmortem in the case of enzootic bovine leukosis

A

T

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60
Q

Generation shift is the only way of eradication of enzootic bovine leukosis

A

F

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61
Q

Enzootic bovine leukosis virus does not spread from animal to animal.

A

F

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62
Q

Mild clinical signs can be seen in the incubation phase of enzootic bovine leukosis

A

F

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63
Q

Enzootic bovine leukosis virus is not shed in the colostrum

A

F

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64
Q

Enzootic bovine leukosis virus can be transmitted with blood

A

T

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65
Q

Enzootic bovine leukosis virus can spread from cattle to sheep, goats , and other ruminants

A

F

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66
Q

Enzootic bovine leukosis virus has uniform antigenic structure

A

T BC ONE SEROTYPE????

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67
Q

In the case of Enzootic bovine leukosis the clinical signs appear at the age of 6-8 month

A

F

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68
Q

Enzootic bovine leukosis is carried lifelong

A

T

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69
Q

Enzootic bovine leukosis virus can be transmitted in tracheal discharge

A

T

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70
Q

Enzootic bovine leukosis occurs only in Holstein Friesian cattles

A

F

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71
Q

Enzootic bovine leukosis virus can infect foetuses of pregnant animals

A

T

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72
Q

Enzootic bovine leukosis virus has several serotypes and subtypes.

A

F

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73
Q

Enzootic bovine leukosis can spread by air within the herd

A

T

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74
Q

Enzootic bovine leukosis can spread by the veterinarian

A

T

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75
Q

Enzootic bovine leukosis virus cannot result tumour formation

A

F

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76
Q

Serological examinations cannot be used to the diagnosis of enzootic bovine leukosis

A

F

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77
Q

Immune tolerance can happen in the case of enzootic bovine leukosis

A

T

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78
Q

Selection cannot be used for eradication of enzootic bovine

A

F

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79
Q

Bovine enzootic leukosis infect only bovine

A

F

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80
Q

Bovine enzootic leukosis does not spread with excretion

A

F

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81
Q

Bovine enzootic leukosis spreads slow in the herd.

A

T

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82
Q

Bovine enzootic leukosis can be transmitted by blood

A

T

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83
Q

Bovine leukosis virus can give lifelong carriers.

A

T

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84
Q

Bovine leukosis virus causes seropositivity in latency period.

A

T

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85
Q

Enzootic bovine leukosis the pre-tumour phase usually in 6-10 months old animals

A

F

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86
Q

Enzootic bovine leukosis during pre-tumour phase causes lymphocytosis

A

T

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87
Q

Bovine enzootic leukosis virus can be transmitted with lymphoid cells

A

T

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88
Q

Iatrogenic infection is frequent in the epidemiology of bovine enzootic leukosis

A

T

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89
Q

The target cells of the bovine enzootic leukosis virus are the T-lymphocytes

A

F

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90
Q

The typical signs of bovine enzootic leukosis can be seen in cattle under 1 year of age

A

F

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91
Q

Antibodies against enzootic bovine leukosis virus can be detected in the ELISA test

A

T

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92
Q

Antibodies against enzootic bovine leukosis virus can be detected in the milk

A

T

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93
Q

Selection (test and slaughter) method cannot be used to eradicate enzootic bovine leukosis
virus

A

F

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94
Q

Generation shift method cannot be used to eradicate enzootic bovine leukosis virus

A

F

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95
Q

Enzootic bovine leukosis virus is spreading horizontally in a cattle herd

A

T

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96
Q

Enzootic bovine leukosis virus cannot infect foetuses

A

F

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97
Q

Enzootic bovine leukosis virus is passed to newborn calves mainly with colostrum in
endemically infected herds.

A

F

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98
Q

By the end of the incubation phase the animals become seropositive leukosis virus

A

T

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99
Q

Tumours can be seen in about 90% of the animals infected with enzootic bovine leukosis
virus

A

F

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100
Q

Antibodies in the milk against enzootic bovine leukosis virus can be detected with ELISA

A

T

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101
Q

Tumours caused by enzootic leukosis virus generally appear at the age of 6 months

A

F

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102
Q

The infection with enzootic leukosis virus is detected by AGP and ELISA

A

T

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103
Q

Enzootic bovine leukosis virus is zoonotic

A

F

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104
Q

Enzootic bovine leukosis virus is shed in lymphoid cells

A

T

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105
Q

Enzootic bovine leukosis virus cannot cause intrauterine infection

A

F

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106
Q

Enzootic bovine leukosis virus is spreading slowly in the herd.

A

T

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107
Q

The target cells of enzootic bovine leukosis virus are the B lymphocytes

A

T

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108
Q

Enzootic bovine leukosis virus is not shed by the infected animals

A

F

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109
Q

Enzootic bovine leukosis virus can be transmitted with organic infection.

A

T

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110
Q

Enzootic bovine leukosis virus can be transmitted with per os infection.

A

T

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111
Q

Clinical signs of enzootic bovine leukosis are seen mainly in 6-8-month-old calves

A

F

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112
Q

Enzootic bovine leukosis virus has several serotypes and subtypes

A

F

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113
Q

Enzootic bovine leukosis can spread by air within the herd

A

T

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114
Q

Enzootic bovine leukosis virus can not result in tumour formation.

A

F

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115
Q

Immune tolerance can happen in the case of enzootic bovine leucosis

A

T

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116
Q

During incubation phase of bovine enzootic leucosis the animal become seropositive.

A

T

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117
Q

The tumours in the case of bovine enzootic leucosis can be seen from the age of 6 months.

A

F

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118
Q

PCR is used for the detection of bovine enzootic leucosis in immunotolerant calves

A

T

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119
Q

Bovine enzootic leucosis can be eradicated with selection

A

T

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120
Q

Bovine enzootic leucosis virus has several serotypes

A

F

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121
Q

There is no horizontal spread in the case of bovine enzootic leucosis.

A

F

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122
Q

There is genetic predisposition in the case of bovine enzootic leucosis

A

T

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123
Q

Enzootic bovine leucosis occurs in all ruminant species.

A

F

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124
Q

Enzootic bovine leukosis virus can infect cattle, pigs and horses.

A

F

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125
Q

Iatrogenic infection can be important in the transmission of enzootic bovine leukosis virus.

A

T

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126
Q

Aerogenic infection occurs in the case of enzootic bovine leukosis virus

A

T

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127
Q

Enzootic bovine leucosis is spreading very fast in infected herd

A

F

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128
Q

Enzootic bovine leucosis virus can infect the foetus

A

T

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129
Q

Enzootic bovine leucosis only infects cattle

A

F

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130
Q

Enzootic bovine leukosis occurs only in Holstein-Frisian cattle, other cattle races are resistant

A

F

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131
Q

Enzootic bovine leukosis has low resistance; it cannot retain its infectivity for a long time in
environment

A

T

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132
Q

The most severe clinical signs of ovine pulmonary adenomatosis can be seen in lambs
younger than 6 months.

A

F

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133
Q

Antibodies of animals infected with ovine pulmonary adenomatosis virus can be detected
with ELISA.

A

F

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134
Q

Adenocarcinoma can be seen postmortem in the case of ovine pulmonary adenomatosis

A

T

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135
Q

Ovine pulmonary adenomatosis virus is transmitted with tracheal discharge

A

T

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136
Q

Ovine pulmonary adenomatosis virus can be transmitted with contaminated objects to other farms

A

F

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137
Q

Ovine pulmonary adenomatosis virus can infect sheep, goats, and cattle

A

F

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138
Q

The most severe clinical signs of ovine pulmonary adenomatosis can be seen in lambs
younger than 6 months

A

F

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139
Q

Shedding large amount of nasal discharge is a typical clinical sign of ovine pulmonary
adenomatosis

A

T

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140
Q

Ovine pulmonary adenomatosis virus replicates in lymphoid cells and causes viraemia

A

F

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141
Q

Metastasis are rare in the case of Ovine Pulmonary Adenomatosis

A

T

142
Q

Ovine pulmonary adenomatosis occurs only in South Africa

A

F

143
Q

Ovine pulmonary adenomatosis virus has no onc-gen

A

T

144
Q

Ovine pulmonary adenomatosis is prevented with inactivated vaccines

A

F

145
Q

Ovine pulmonary adenomatosis spreads with nasal discharge

A

T

146
Q

The primary replication site of OPA is in the mucosal cells of the intestines.

A

F!!!!!!

147
Q

Metastasis are frequently seen in parenchymal in the case of OPA

A

F

148
Q

Ovine pulmonary adenomatosis virus is found in 2-4 months old lambs.

A

F

149
Q

Ovine pulmonary adenomatosis virus is replicating in the epithelium of the airways.

A

T

150
Q

Tumours can frequently be seen in the liver and the spleen in the case of ovine pulmonary
adenomatosis

A

F

151
Q

Ovine pulmonary adenomatosis can be complicated by Pasteurella and Mannheimia strains.

A

T

152
Q

In the case of ovine pulmonary adenomatosis lesions are common in the liver

A

F

153
Q

Ovine pulmonary andenomatosis can be diagnosed by detecting antibodies with ELISA

A

F

154
Q

Ovine pulmonary adenomatosis virus causes interstitial pneumonia

A

F

155
Q

Lung adenomatosis causes usually dry cough

A

F

156
Q

Lung adenomatosis causes a lot of metastasis.

A

F

157
Q

Metastasis are rare in the case of ovine pulmonary adenomatosis

A

T

158
Q

Ovine pulmonary adenomatosis has no antibody production

A

T

159
Q

Ovine pulmonary adenomatosis virus results in malignant transformation of macrophages

A

F

160
Q

Europe is free from ovine pulmonary adenomatosis

A

F

161
Q

No antibodies to ovine pulmonary adenomatosis virus can be detected in infected animals

A

T

162
Q

Faces of infected animals contain large amount of ovine pulmonary adenomatosis virus

A

F

163
Q

Tumour transformation of the epithelial cells happens in the case of ovine pulmonary
adenomatosis

A

T

164
Q

Jaagsiekte affects lambs of 3-6 months

A

F

165
Q

Jaagsiekte virus can transform human cells.

A

F

FeLV!!!

166
Q

Jaagsiekte is only present in Africa

A

F

167
Q

Jaagsiekte causes metastatic abscess formation all over the body

A

F

168
Q

Feline leukosis virus will be shed lifelong by infected cats

A

F

169
Q

There are no vaccines for the prevention of feline leukosis

A

F

170
Q

FOCMA antigen is a typical surface antigen of feline leukosis viruses.

A

T

171
Q

Feline leukosis virus can only be transmitted with saliva

A

F

172
Q

Cats remain infected with feline leukosis virus lifelong

A

F

173
Q

Feline leukosis is maintained by persistently infected cats

A

T

174
Q

Feline leukosis virus can be transmitted by direct contact.

A

T

175
Q

Feline leukosis virus can cause horizontal and vertical infection

A

T

176
Q

Vaccination of only seronegative cats is responsible against feline leukosis

A

T

177
Q

Immunotolerant kittens can be born in the case of feline leukosis

A

T

178
Q

Feline Leukosis can be eliminated in some cats

A

T

179
Q

There are several subgroups of feline leukosis virus

A

T

180
Q

Feline leukosis virus can cause immune tolerance

A

T

181
Q

Feline sarcomatosis virus is a recombinant virus from feline leukosis virus and host DNA

A

T

182
Q

Feline leukosis virus is uniform

A

F

183
Q

In the saliva of cats is a high-titer of FeLV

A

T

184
Q

FeLV spreads horizontally and vertically

A

T

185
Q

Persistent infected cats maintain the infection

A

T

186
Q

Feline leukosis virus can cause protective immunity.

A

T

187
Q

In Feline leukosis, anaemia is an important sign

A

T

188
Q

Cat leukosis virus can be diagnosed with PCR

A

T

189
Q

There is no vaccine against Feline leukosis virus

A

F

190
Q

Feline leukosis can infect dogs and cats

A

F

191
Q

Asymptomatic infection cannot happen in the case of Feline leukosis

A

F

192
Q

Feline leukosis virus is immunosuppressive

A

T

193
Q

Feline leukosis virus is frequently spread with saliva

A

T

194
Q

Feline leukosis virus can infect dogs, cats and wild living carnivorous animals

A

F

195
Q

Feline leukosis is a very rare disease

A

F

196
Q

Feline sarcomatosis is a defect virus

A

T

197
Q

Feline leukosis can cross the placenta

A

T

198
Q

FeLV is a uniform virus

A

F

199
Q

Infection with feline leukosis virus always appears in clinical signs

A

F

200
Q

Feline leukosis virus is spreading by discharge of the infected animal

A

T

201
Q

Persistently infected cats can shed the feline leukosis virus in high titres

A

T

202
Q

Saliva of the animal contains large amount of the feline leukosis virus

A

T

203
Q

Feline leucosis spreads by direct contact

A

T

204
Q

Feline immunodeficiency virus causes persistent infection

A

T

205
Q

Feline immunodeficiency virus is widespread

A

T

206
Q

Feline immunodeficiency virus in cat could be asymptomatic

A

T

207
Q

Feline immunodeficiency virus is spread by excretes

A

T

208
Q

Feline immunodeficiency virus develops in 3 phases

A

F

has 3 phases possible depending on immunity. EBL developed in 3 phases

209
Q

Inactivated vaccines are used for the prevention of avian leukosis

A

F

210
Q

Attenuated vaccines are used for the prevention of avian leukosis

A

F

211
Q

Avian leukosis viruses have several subgroups

A

T

212
Q

Tumours in the liver can be seen in the case of avian leukosis

A

T

213
Q

Avian leukosis viruses cause horizontal infection.

A

T

214
Q

J subtype of avian leukosis virus is more virulent than the other ones

A

T

215
Q

Avian leukosis can be diagnosed by detecting COFAL antigen

A

T

216
Q

Lymphoid leukosis is the most frequent clinical form of avian leukosis

A

T

217
Q

Avian leukosis viruses cause germinative infection

A

T

218
Q

All avian leukosis viruses are oncogenic

A

T

219
Q

Avian leukosis viruses have several subgroups

A

T

220
Q

Avian leukosis viruses a resistant, they can survive in the bedding for several weeks

A

F

221
Q

Avian leukosis causes the malignant transformation of B lymphocytes

A

T

222
Q

Avian leukosis virus occurs only in tropical and subtropical countries

A

F

223
Q

Avian leuKosis virus can cause only lymphoid leukosis

A

F

224
Q

Avian leukosis viruses can cause malignant transformation in different tissues.

A

T

225
Q

Germinative infection is an important way of transmission of avian leukosis virus

A

T

226
Q

Clinical signs of avian leukosis can be seen typically in broiler chicken

A

T

227
Q

Osteopetrosis can be a clinical form of avian leukosis.

A

T

228
Q

Avian Leukosis virus is uniform

A

F

229
Q

Every avian leukosis viruses is oncogenic

A

T

230
Q

Avian leukosis virus cannot infect by germinative way

A

F

231
Q

Avian leukosis virus infects B lymphocytes

A

T

232
Q

Infection of poultry herds with avian leukosis virus is widespread

A

T

233
Q

Clinical signs of avian leukosis generally appear in day old chicken

A

F

234
Q

Lymphoid leukosis is the most frequent form of avian leukosis.

A

T

235
Q

The main way of prevention of avian leukosis is vaccination using attenuated strains

A

F

236
Q

Avian leukosis viruses are shed in the faeces.

A

T

237
Q

There is no germinative infection in the case of avian leukosis viruses.

A

F

238
Q

The target cells of the avian leukosis viruses are the B lymphocytes

A

T

239
Q

There are several subgroups of avian leukosis viruses.

A

T

240
Q

All avian leukosis viruses cause malignant transformation of the host cells

A

T

241
Q

In a flock infected with avian leukosis virus generally 50-60% of the animals have tumours

A

F

242
Q

Detection of COFAL antigen is a frequent way of diagnosis of avian leukosis

A

T

243
Q

Avian leukosis is seen during the first week of life in chicken

A

F

244
Q

There are resistant lines to avian leukosis

A

T

245
Q

Proportion of the animals with tumours is low, 1-4% in the case of avian leukosis

A

T

246
Q

Inactivated vaccines are widely used in order to prevent avian leukosis

A

F

247
Q

Tumours can be seen in different parenchymal organs in the case of avian leukosis.

A

T

248
Q

T-lymphocytes are the target cell of the avian leukosis virus.

A

F

249
Q

Avian leucosis and sarcoma infections are very common

A

T

250
Q

Avian leucosis can be caused by different retroviruses

A

T???? check maybe just different subgroups

251
Q

Congenital transmission of avian leucosis results in immune tolerance

A

T

252
Q

Reticuloendotheliosis is caused by J type of avian leukosis virus.

A

F

253
Q

Germinative infection can happen in the case of Reticuloendotheliosis

A

T

254
Q

Reticuloendotheliosis virus is shed in the faces

A

T

255
Q

Reticuloendotheliosis virus can cause germinative infection

A

T

256
Q

Stunted growth is a clinical sign of reticuloendotheliosis

A

T

257
Q

Reticuloendotheliosis is prevented by vaccination of the parent animals.

A

F

258
Q

Immunosuppression is common in the case of Reticuloendotheliosis

A

T

259
Q

Retardation is a clinical sign of Reticuloendotheliosis

A

T

260
Q

Wide vaccination is used to prevent Reticuloendotheliosis

A

F

261
Q

In the case of reticuloendotheliosis immunotolerant chicken can be hatched.

A

T

262
Q

Pneumonia is a typical lesion of reticuloendotheliosis

A

F

263
Q

In the case of reticuloendotheliosis tumors can be found in the parenchymal organs

A

T

264
Q

Avian reticuloendotheliosis may be similar in appearance to Marek ́s disease.

A

T

265
Q

Clinical signs of maedi visna are more severe in young animals than in adults

A

F

266
Q

Heavy nasal discharge is a clinical sign of maedi

A

F

267
Q

The maedi virus and the visna virus are related but they can be differentiated with PCR

A

F

268
Q

Interstitial pneumonia is the main postmortem lesion of visna

A

F

269
Q

Interstitial pneumonia is the main postmortem lesion of Maedi

A

T

270
Q

Maedi and visna are caused by the same virus.

A

T

271
Q

Maedi-visna virus is shed in tracheal discharge and milk

A

T

272
Q

Maedi-visna is maintained by persistently infected sheep

A

T

273
Q

Weakness of the hinder legs is a clinical sign of visna

A

T

274
Q

Inactivated and attenuated vaccines are widely used for the prevention of maedi-visna

A

F

275
Q

Clinical signs of maedi can be seen in sheep above 3-4 years of age

A

T

276
Q

Maedi-visna can occur in sheep, goats, and cattle

A

F

277
Q

Europe is already free from maedi-visna

A

F

278
Q

Maedi/visna is spreading slowly in the flock.

A

T

279
Q

Maedi/visna virus is shed in the milk.

A

T

280
Q

Maedi is seen in 3-4 years old sheep

A

T

281
Q

Large amount of mucoid nasal discharge is typical in the case of maedi.

A

F

282
Q

Maedi/visna spreads from sheep to other animals

A

T

283
Q

Maedi/visna virus is shed in nasal discharge, respiratory secretions and milk.

A

T

284
Q

Clinical sign of maedi/visna appear from the age of 6-8 months

A

F

285
Q

Meadi/visna most important clinical sign is profuse diarrhoea

A

F

286
Q

Maedi/visna virus is shed only in tracheal discharge

A

F

287
Q

Clinical signs of maedi are mainly seen in lambs below half a year of age

A

F

288
Q

Maedi/visna virus causes interstitial pneumonia in sheep

A

T

289
Q

Clinical signs of the central nervous system can be seen in the case of visna

A

T

290
Q

Maedi/visna virus causes viraemia

A

T

291
Q

Wet cough and intensive nasal discharge are typical signs of maedi.

A

F

292
Q

De-myelinization is the reason for the clinical signs of visna

A

T

293
Q

Attenuated vaccines are widely used to prevent maedi/visn

A

F

294
Q

Maedi appears in Hungary

A

T

295
Q

Maedi is spreading fast

A

F

296
Q

In order to eradicate maedi/visna infected ewes have to be culled with their lambs

A

T

297
Q

Clinical sign of maedi/visna appear from the age of 6 months

A

F

298
Q

Maedi/visna is spreading fast in the flock

A

F

299
Q

Maedi/visna virus is shed only in the tracheal discharge

A

F

300
Q

Clinical signs of maedi are generally seen above 3-4 years of age

A

T

301
Q

Caprine arthritis-encephalitis virus can cause persistent infection

A

T

302
Q

Sheep are resistant against caprine arthritis encephalitis virus

A

F

303
Q

Arthritis caused by caprine arthritis encephalitis virus is mainly seen in lambs

A

F

304
Q

Encephalitis caused by caprine arthritis encephalitis virus is generally seen in 2-4 month old
kids

A

T

305
Q

Caprine arthritis encephalitis virus can be transmitted by milk

A

T

306
Q

There is intensive vaccination against Caprine arthritis encephalitis in endemic countries

A

F

307
Q

Caprine arthritis encephalitis virus is more frequent in dairy goats than in rural breeds

A

T

308
Q

Caprine arthritis encephalitis virus is shed in the milk

A

T

309
Q

In the case of caprine arthritis encephalitis the signs of encephalitis can be seen in 2-4
months old kids

A

T

310
Q

In the case of caprine arthritis encephalitis, arthritis is less frequent than encephalitis.

A

F

311
Q

Kids are recommended to be isolated in a herd where caprine arthritis encephalitis is
present

A

T

312
Q

Kids can be infected with caprine arthritis encephalitis virus through the milk

A

T

313
Q

Arthritis caused by caprine arthritis-encephalitis virus is typically seen in kids below half a
year of age

A

F

314
Q

Caprine arthritis encephalitis is characterized by CNS signs in young goats

A

T

315
Q

Caprine arthritis encephalitis virus replicates in the intestinal tract

A

T!!!!!!!

316
Q

Equine infectious anaemia virus is transmitted with blood of the infected animals

A

T

317
Q

Blood sucking arthropods can transmit Equine infectious anaemia virus

A

T

318
Q

Mosquitoes are the main vectors of equine infectious anaemia virus; the virus can replicate
in them

A

F

319
Q

Agar gel diffusion test is used to detect antibodies against equine infectious anaemia

A

T

320
Q

Equine infectious anaemia is an acute disease; it does not have a chronic form

A

F

321
Q

Iatrogenic transmission of Equine infectious anaemia can happen

A

T

322
Q

Equine infectious anaemia virus can damage the bone marrow

A

T

323
Q

There are no vaccines for the prevention of equine infectious anaemia

A

T

324
Q

Equine infectious anaemia virus is more resistant than other retroviruses.

A

T

325
Q

Equine infectious anaemia virus disappears from animals after the viraemia

A

F

326
Q

Fever is a major clinical sign of equine infectious anaemia

A

T

327
Q

Equine infectious anaemia virus is resistant, it can survive several months in the environment

A

T

328
Q

Only Equidae are susceptible to equine infectious anaemia virus

A

T

329
Q

Ticks are vectors of equine infectious anaemia virus

A

F

330
Q

Febrile waves can be seen in equine infectious anaemias

A

T

331
Q

Equine infectious anaemia is a vector borne disease

A

T

332
Q

Equine infectious anaemia virus causes persistent infection.

A

T

333
Q

There is no immune reaction in the case of equine infectious anaemia

A

F

334
Q

Equine infectious anaemia is zoonotic

A

F

335
Q

Equine infectious anaemia is caused by a lentivirus

A

T

336
Q

Blood sucking arthropods are mechanical vectors of equine infectious anaemia virus

A

T

337
Q

There is no immune reaction in horses against equine infectious anaemia virus

A

F

338
Q

Agar gel precipitation test can be used to the detection of antibodies against equine
infectious anaemia virus

A

T

339
Q

Equine infectious anaemia can damage the medulla of the bone.

A

T

340
Q

Equine infectious anaemia has a weak resistance

A

F

341
Q

You cannot diagnose Equine infectious anaemia with serology

A

F

342
Q

The resistance of equine infectious anaemia is very low

A

F

343
Q

Horses and cattle are susceptible to equine infectious anaemia virus

A

F

344
Q

In the case of equine infectious anaemia, haemorrhages cannot be seen

A

F

345
Q

Animals infected with equine infectious anaemia virus are lifelong carriers

A

T

346
Q

Equine infectious anaemia virus is mainly transmitted with tracheal discharge

A

F

347
Q

Attenuated vaccines are used for the prevention of equine infectious anaemia

A

F

348
Q

Equine infectious anaemia can be asymptomatic

A

T

349
Q

Equine infectious anaemia may cause recurrent fever in horses.

A

T

350
Q

Horseflies are mechanical vectors equine anaemia virus.

A

T

351
Q

Equine anaemia is a notifiable disease

A

T

Epi Pq Final Viro CORRECTED (123 decks)

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