Respiratory syndromes I Flashcards

1
Q

Which side of the heart’s cardiac output affect perfusion?

A

Right side of the heart

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2
Q

Emphysema

A

Abnormal permanent enlargement of air spaces distal to terminal bronchioles and destruction of alveolar septal walls.

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3
Q

Why is emphysema a problem?

A

Because smaller alveoli have more contact surface with capillaries per volume than larger alveoli

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4
Q

Why emphysema happens?

A

There is obstruction in the airways, expiration does not get air out and there is too much air inside the alveoli and they rupture.

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5
Q

Emphysema types

A

Alveolar

Interstitial (air in the connective tissue)

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6
Q

Emphysema causes

A
Trauma
Parasites
Tumor/mass
Chronic obstructive lung disease
Congenital bronchial hypoplasia
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7
Q

Emphysema physiopathology

A

Decreased alveolar surface -> diffusion rf
Decreased pulmonary compliance -> restrictive rf
Pulmonary hypertension -> right-sided cardiac failure

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8
Q

Emphysema clinical signs

A
Respiratory dyspnea
Dry cough
Tachycardia
Tympanic sound
Cyanosis in advanced cases
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9
Q

Pleural effusion

A

Excess fluid accumulates inside the pleural cavity

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10
Q

Pleural effusion aetiology

A
Increased hydrostatic pressure
Reduced plasma oncotic pressure
Increased vascular permeability
Lymphatic drainage obstruction
Bleeding
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11
Q

Increased hydrostatic pressure and reduced plasma oncotic pressure cause

A

transudate (clear) pleural effusion

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12
Q

Inflammation and alteration in protein metabolism cause

A

exsudate (opaque with clots) pleural effusion

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13
Q

lymphatic drainage obstruction and lymphoma produce

A

chylothorax (lymph in pleura)

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14
Q

Pleural effusion signs

A
mixed dyspnea
superficial and rapid breathing movement
overinflated chest
dull sound on percussion
reduced lung sounds on auscultation
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15
Q

What type of rf does pleural effusion produce`?

A

restrictive rf

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16
Q

Fibrosis

A

Replacement of lungs’ parenchyma by fibrous tissue/progressive scarring of the lungs.

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17
Q

Aetiology of fibrosis

A

Inhalation of toxin (asbestos, saw dust, gas)
Drugs
Autoimmune(sle)
Idiopathic

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18
Q

Physiopathology of fibrosis

A

Thickened alveolo-capillary barrier -> diffusion rf
Decreased pulmonary distensibility -> restrictive rf
Pulmonary hypertension -> right-sided cardiac failure

19
Q

Signs of fibrosis

A

Dry cough
crackle sounds
tachypnea
shallow respiratory movements

20
Q

How to tell apart fibrosis and emphysema by clinical signs?

A

Emphysema -> tympanic sound

Fibrosis -> crackles

21
Q

Mediastinal syndrome

A

Compression of mediastinal structures by any mass causing signs.

22
Q

Mediastinal syndrome aetiology

A

Mass:
tumour
hematoma
abscess

23
Q

Mass in the mediastinum can lead to

A

Cranial v cava compression
airway obstruction
sympathetic trunk, vagal or laryngeal nerve compression
oesophagus compression

24
Q

cranial v cava compression leads to

A

cr v cava syndrome

25
Q

airway obstruction leads to

A

obstructive rf

26
Q

oesophagus compression leads to

A

regurgitation

27
Q

nerve compressions lead to

A

neurological signs

28
Q

signs of cr v cava syndrome

A

edema in neck head and forelimbs

29
Q

SYMPATHETIC TRUNK COMPRESSION

A

HORNER´S SYNDROME
– Ptosis (upper eyelid drop)
– Miosis (pupil constriction)
– Enophtalmos (3rd eyelid)

30
Q

Vagal nerve compression

A

cardiac activity disturbances

31
Q

Laryngeal nerve

A

laryngeal paralysis

32
Q

Pneumonic syndrome

A

lung parenchyma consolidates with fluid or solid

33
Q

pneumonic syndrome etiology

A

pneumonia, tumor, hemorrhage

34
Q

pneumonic syndrome physiopathology

A

increased acb thickness leads to diffussion rf
decreased pulmonary distensibility leads to restrictive rf
mismatch in ventilation/perfusion ratio (v/q) leads to increase in death space

35
Q

clinical signs of pneumonic syndrome

A

signs linked to pulmonary consolidation

  • cough
  • decreased thorax motility
  • dull percussion
  • decreased vesicular murmur

signs linked to infection

  • fever
  • lethargy
36
Q

atelectasis

A

a decrease in pulmonary volume due to complete or partial lung collapse

37
Q

atelectasis types

A
contraction atelectasis (lack of surfactant)
resorption (obstruction) atelectasis (airway obstruction)
compressive atelectasis (tumors, pleural effussion compress lung tissue)
38
Q

atelectasis clinical signs

A
inspiratory dyspnea
tachypnea
reduced size and movement of the affected hemithorax
percussion (dull sound)
auscultation (no sound)
39
Q

pneumothorax

A

abnormal accumulation of air pleural cavity

40
Q

pneumothorax types

A

traumatic, spontaneous or iatrogenic

41
Q

traumatic pneumothorax aetiology

A

open:

  • bite
  • gunshot

closed:
-due to internal breakage of trachea, bronchi or lung parenchyma

42
Q

spontaneous pneumothorax aetiology

A

primary:
-idipathic
secondary:
-due to previous pulmonary disease

43
Q

iatrogenic pneumothorax aetiology

A

surgical procedures or in felines trachea breakage due to et tube

44
Q

clinical signs of pneumothorax

A
  • inspiratory dyspnea
  • tachypnea with superficial breathing
  • cyanosis
  • increased volume of affected hemothorax
  • decreased movement of affected hemothorax
  • tympanic percussion
  • increased auscultation sounds