Respiratory Flashcards

1
Q

Hypoxemic Respiratory Failure Causes

A
  • Ventilation/Perfusion (Q) mismatch:
    • ↓ Ventilation
    • ↓ Perfusion
  • Shunt:
    • Anatomic
    • Intrapulmonary
  • Diffusion limitation
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2
Q

V/Q stands for

A

Ventilation/Perfusion

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3
Q

Hypercapnic Respiratory Failure Causes

A
  • Airways and alveoli
  • Central nervous system (decreased drive to breath: OD/stroke)
  • Chest wall (decreased chest wall expansion: flail chest)
  • Neuromuscular conditions that cause muscle weakness, impairing resp muscle use
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4
Q

Acute Respiratory Failure Clinical Manifestations - Hypercapnic Failure?

A
  • Neuro changes
  • Dyspnea and HA
  • ↑ ↓ RR
  • ↑ HR
  • ↑BP
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5
Q

Acute Respiratory Failure Clinical Manifestations - Hypoxemic Failure?

A
  • Neuro changes
  • Dyspnea
  • ↑ RR
  • ↑ HR
  • ↑BP
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6
Q

How what diffentiates hypoxemic failure from hypercapnic failure?

A

retention of CO2 in hypercapnic failure causes a HEADACHE

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7
Q

What diagnostic tests are run in a pt in Acute Respiratory Failure?

A
  • Arterial Blood Gas (ABG) analysis
  • Chest x-ray (CXR)
  • CBC,
  • Complete Metabolic Panel (e-lytes)
  • Urinalysis
  • Sputum/blood cultures (infection)
  • V/Q lung scan/CT chest (PE)
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8
Q

What is the difference between BiPAP and CPAP?

A
  • BiPAP: different pressure for inspiration and experation

- CPAP: constant pressure for both

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9
Q

Acute Respiratory Failure Nursing/Collaborative Management

A
  • Prevention-early recognition

- Respiratory therapy

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10
Q

Types of respiratory therapy for patient with acute respiratory failure?

A
  • Oxygen therapy
  • Mobilization of secretions (pat back with cupped hands)
  • Positive pressure ventilation (PPV)
    • Mask (BiPAP, CPAP)
    • Intubation
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11
Q

Drug Therapy for acute respiratory failure?

A
  • Bronchodilators (albuterol)
  • Corticosteroids (methylprednisolone)
  • Diuretics
  • Nitrates
  • IV antibiotics (vancomycin/ceftriazone)
  • Sedatives, Benzodiazepines, Narcotics (ativan/versed)
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12
Q

Physiologic aging results in what respiratory changes?

A
  • ↓ Ventilatory capacity
  • Alveolar dilation
  • Larger air spaces
  • Loss of surface area
  • Diminished elastic recoil
  • Decreased respiratory muscle strength
  • ↓ Chest wall compliance
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13
Q

Acute Respiratory Failure Gerontologic Considerations

A
  • Physiologic aging
  • Lifelong smoking
  • Poor nutritional status
  • Less available physiologic reserve
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14
Q

Sudden progressive form of acute respiratory failure?

A

Acute Respiratory Distress Syndrome (ARDS)

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15
Q

Acute Respiratory Distress Syndrome (ARDS)

A
  • Sudden progressive form of acute respiratory failure
  • Alveolar capillary membrane damaged -↑ permeability to intravascular fluid
  • 50% mortality rate
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16
Q

Acute Respiratory Distress Syndrome (ARDS) starts with?

A

Alveolar capillary membrane damaged, which ↑ permeability to intravascular fluid

17
Q

Direct causes of ARDS?

A

aspiration, pneumonia, inhalation

18
Q

Indirect causes of ARDS?

A

secondary result of a massive systemic insult

19
Q

Pathophysiology of ARDS?

A

thought to be due to stimulation of the inflammatory and immune systems

20
Q

ARDS causes?

A
  • Severe dyspnea
  • Hypoxia
  • Decreased lung compliance
  • Diffuse pulmonary infiltrates (white out lung)
21
Q

3 phases of ARDS?

A

1) Injury or Exudative
2) Reparative or Proliferative
3) Fibrotic

22
Q

Injury or Exudative phase of ARDS occurs?

A

1 - 7 days after initial injury

23
Q

Summary of processes of the Injury or Exudative phase of ARDS?

A
  • Pulmonary edema and atelectasis
  • V/Q mismatch-severe
  • Shunting of pulmonary capillary blood
  • Refractory hypoxemia
24
Q

Occurs when oxygenation continues to decrease regardless of the amount of O2 given?

A

refractory hypoxemia

25
Reparative or Proliferative phase of ARDS occurs?
1 to 2 weeks after initial injury
26
Summary of processes of the Reparative or Proliferative phase of ARDS?
- Influx or neutrophils, monocytes, and lymphocytes - Lung becomes dense and fibrous – ↓ compliance - ↑ Pulmonary vascular resistance, pulmonary HTN
27
Fibrotic or chronic/late phase of ARDS occurs?
2 to 3 weeks after initial injury
28
Summary of processes of the Fibrotic or chronic/late phase of ARDS?
- Lung completely remodeled - ↓ Lung compliance - ↓ surface area for gas exchange - Pulmonary hypertension
29
Early symptoms of ARDS?
- May not exhibit respiratory symptoms or - Dyspnea, tachypnea, cough, restlessness - Normal or scattered crackles
30
Interpretation of Arterial Blood Gas results/values of pt in the early stage of ARDS will show?
Mild hypoxemia and respiratory alkalosis
31
Early chest xrays of ARDS patient?
May be normal or have minimal scattered interstitial infiltrates
32
ARDS clinical manifestations: Progressive
- Symptoms worsen - evident discomfort, ↑ WOB - ↑ fluid accumulation, - ↓ lung compliance - Pulmonary Function Tests (PFTs) show ↓ compliance and lung volume
33
ARDS clinical manifestations: Late
- Suprasternal retractions - Tachycardia, diaphoresis - ↓ mentation - Cyanosis, pallor - Hypoxemia despite ↑ FIO2 - Hypercapnia causing muscle fatigue, hypoventilation
34
ARDS Complications
- MO Dysfunction - Sepsis - Ventilator - Ventilator Associated - Pneumonia (VAP) - Ventilator complications - Stress ulcers - Renal failure
35
Major causes of death in ARDS patient?
- MODS | - Sepsis