Renal Disorders 2 Flashcards

1
Q

UTO

A

interference with flow of urine
causes urine to accumulate behind obstruction leading to damage to organs

backs up into kidneys

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2
Q

where can UTO’s occur

A

anywhere in UT

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3
Q

kidney stones

A

masses of crystal and protein

crystals made of stone forming substances

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4
Q

what is the most common cause of UTO

A

kidney stones

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5
Q

kidneys stones in men

A

distal- see slower streak, maybe scarring
no significant damage
above prostate, bladder affected

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6
Q

patho of kidney stones

A

crystals made up of stone forming substances trapped within urinary tract and accumulate other crystals –> stones

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7
Q

contributing factors of kidney stones

A

pH of urine- affects solubility

high urinary conc. of stone forming substance

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8
Q

types of kidney stones

A

calcium oxalate or phosphate
struvite
uric acid

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9
Q

calcium oxalate is most common in

A

men

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10
Q

etiology of calcium oxalate

A

idiopathic
inc leves of Ca causes formation
people who are immobile- Ca buildup

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11
Q

struvite is most common in

A

women

precipitated by inf

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12
Q

etiology of uric acid

A

inc urine acid production in body by diet

high purine diet, high meat, fish, poultry, gout

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13
Q

CM for kidney stones

A

pain
N/V
hematuria
s&s of UTI

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14
Q

complication of kidney stones

A

can travel, cause irritation to binary tract. jagged crystals are painful

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15
Q

diagnostic tests for kidney stones

A

urine pH
xRay of abd., IVP, ultrasound
stone analysis- determines cause

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16
Q

treatment for kidney stones

A
pain meds
anx if inf
fluids
dec dietary intake of comp. in stone
ultrasonography or laser lithotripsy
surgery
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17
Q

neurogenic bladder

A

functional obstructive condition caused by dysfunction in nerve supply to bladder. problem can occur at CNS or at level of spinal cord.

not physical obstruction

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18
Q

patho of neurogenic bladder

A

paralysis of bladder interferes with normal flow of urine and emptying of bladder

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19
Q

CM of neurogenic bladder

A

urgency/frequency
incontinence
dribbling of urine
urine retention

20
Q

diagnostic tests for neurogenic bladder

A

ultrasound
cystoscopy
urodynamic studies

21
Q

treatment for neurogenic bladder

A
bladder training
kegel exercises
catheters
manual compressions
surgery
22
Q

acute renal failure

A

rapid decline in kidney “f” leading to azotemia
when azotemia symptomatic= uremia

dec UO < 400 cc/24hrs. (oliguria)

23
Q

Bun, Cr levels for acute renal failure

A

high

24
Q

etiology of acute renal failure

A

surgery

  1. prerenal
  2. intrarenal
  3. postrenal
25
Q

prerenal

A

conditions outside kidneys that impair renal bld flow and cause dec glomerularr perfusion or dec GFR

26
Q

causes of prerenal

A

dec VV
dec CO
intravascular pooling of bld/ peripheral vasodilation
inc renal vas. resistance or an obstruction

27
Q

intrarenal

A

actual damage to renal tissue

28
Q

causes of intrarenal

A
prolonged prerenal problem
infection
nephrotoxins
intravascular hemolysis
myoglobinuria
29
Q

postrenal

A

mechanical obstruction of urinary outflow

anuria < 200ml/ day

30
Q

causes of postrenal

A

calculi formation
prostatic hypertrophy
trauma

31
Q

patho of acute renal failure

A
  1. renal vasoconstriction
  2. cellular edema
  3. dec glomerular capillary perm.
  4. intratubular capillary permeability
  5. leakage of glomerular filtrate
32
Q

stages of CM of acute renal failure

A

oliguric phase
diuretic phase
recovery phase

33
Q

oliguric phase

A
UO < 400 ml/24hrs
urinary changes
fluid vol excess
metab. acidosis
elyte imbalance
34
Q

elyte levels in acute renal failure

A

dec Na
inc K
dec Ca
inc phosphate

35
Q

diuretic phase

A

gradual inc output to 1-3 l/day

36
Q

recovery phase

A

GFR inc

37
Q

diagnostic tests for acute renal failure

A

urinalysis

renal ultrasound

38
Q

therapeutic management of acute renal failure

A

treat precipitating cause
correct fluid/elyte disturbances
control symptoms and prevent complications

39
Q

chronic renal failure

A

progressive, irreversible destruction of kidneys

nephrons destroyed, replaced with scar tissue

40
Q

phases of chronic renal failure

A
  1. diminished renal reserve: often undiagnosed
  2. renal insufficiency: GFR < 25% of normal
  3. end stage renal disease (ESRD) or uremia: GFR < 5-10 % of normal
41
Q

etiology of CRF

A

most common:
HTN
diabetes

42
Q

patho of CRF

A

adaptive response: depends on location of kidney damage

intact nephron hypothesis: remaining nephrons compensate. leads to hypertrophy of nephrons

43
Q

CM of CRF

A
8 types;
urinary system
metabolic disturbances
respiratory
cardiovascular
gastrointestinal
neuromuscular
integumentary
reproductive
44
Q

diagnostic tests for CRF

A

serum BUN, Cr- inc

ultrasound, xRay- shrinkage, nothing bu scar tissue

45
Q

treatment for CRF

A

pharmacological: elyte management, HTN, anemia
nutritional: protein restrictions, carb and fats, fluid restrictions, Na restictions, K restrictions
DIALYSIS

46
Q

indications for dialysis

A
fluid overload
hyperkalemia
severe acidosis
progressive uremia
altered CNS "f"
pericarditis
47
Q

goals of dialysis

A

remove waste products
maintain safe conc of elytes
correct acidosis
remove excess fluid