RADIOLOGY - other pathologies Flashcards

1
Q

what factors contribute to lesions appearing radiopaque on radiographs?

A

increased thickness of bone
osteosclerosis of bone
presence of abnormal tissues
mineralisation of normally non-mineralised tissues

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2
Q

what are the 3 questions you ask yourself when you see an abnormality on a radiograph?

A

is it anatomical?
is it artefactual?
is it pathological?

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3
Q

when describing radiopaque and mixed lesions, what is the main deviation when comparing to radiolucencies?

A

its ‘internal structure’

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4
Q

what are the main categories when describing the internal structure of radiopaque and mixed lesions on imaging?

A

entirely radiopaque vs mixed (homogenous or heterogenous)
organised vs haphazard

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5
Q

what is an odontoma?

A

a dental hamartoma - benign tumour composed of dental tissues (enamel, dentine, cementum and pulp)

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6
Q

what similarities are there between normal teeth and odontomas seen on imaging?

A

surrounded by dental follicle
mature to a certain stage (i.e., do not grow indefinitely)

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7
Q

what is the incidence of odontomas?

A

1st or 2nd most common odontogenic tumour
most common in 2nd decade (correlates with development of normal dentition)
F=M

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8
Q

what are the sub types of odontomas?

A
  1. compound (ordered dental structures)
  2. complex (disorganised mass of dental tissue)
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9
Q

describe compound odontomas? and where would you find them?

A

may present as multiple ‘mini teeth’ (denticles)
more common in anterior maxilla

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10
Q

describe complex odontomas? and where would you find them?

A

may have a ‘clump of cotton’ appearance
more common in posterior body of mandible

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11
Q

what are radiographic features of odontomas?

A
  • well defined radiopacity/ radiopacities of varying radiodensity
  • areas with radiodensity of enamel
  • thin radiolucent margin i.e., follicle
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12
Q

what clinical issues do odontomas present?

A
  • impaction of adjacent teeth
  • external root resorption of adjacent teeth
  • development of dentigerous cyst
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13
Q

what is the management of odontomas?

A
  • excision if impeding eruption
  • no risk of recurrence
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14
Q

what is idiopathic osteosclerosis?

A

localised area of increased bone density of unknown cause
- no association with inflammatory, neoplastic or dysplastic processes

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15
Q

what area of dentistry is idiopathic osteosclerosis relevant to?

A

ortho

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16
Q

what are names for areas of idiopathic osteosclerosis?

A

‘dense bone island’
‘enostosis’

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17
Q

what is the incidence of idiopathic osteosclerosis?

A

up to 6% of the population
typically presents in adolescence and stops growing by adulthood
most common in premolar-molar region of mandible

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18
Q

describe the radiographic presentation of idiopathic osteosclerosis?

A
  • well-defined radiopacity
  • often homogenous but can have slightly radiolucent internal areas
  • no radiolucent margin
  • variable shape: round, elliptical, irregular
  • size usually <2cm
  • not associated with teeth but can appear next to them due to circumstance: teeth not displaced and no affect on the PDL spaces of teeth
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19
Q

what is sclerosing osteitis?

A

localised area of increased bone density in response to inflammation
- inflammation often low-grade and chronic

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20
Q

does sclerosing osteitis have symptoms?

A

may have concurrent symptoms due to source of inflammation
no expansion or displacement of adjacent structures

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21
Q

what is another name for sclerosing osteitis?

A

condensing osteitis

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22
Q

describe the radiographic presentation of sclerosing osteitis?

A

well defined or poorly defined radiopacity
directly associated with source of inflammation - apex of necrotic tooth, infected cyst etc

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23
Q

how can you differentiate sclerosing osteitis and idiopathic osteosclerosis?

A

if radiographic features inconclusive then look for a source of inflammation e.g., check for signs and symptoms, sensibility test teeth

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24
Q

what is hypercementosis?

A

excessive deposition of cementum around root

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25
does hypercementosis have symptoms?
non-neoplastic and asymptomatic tooth vital (unless necrotic due to another reason
26
what causes hypercementosis?
cause unknown but more common in certain conditions i.e., pagets disease of bone and acromegaly
27
what is the clinical relevance of hypercementosis?
makes xla more difficult
28
describe the radiographic presentation of hypercementosis?
- single or multiple teeth involved - involves either entire root or just a section - homogenous radiopacity continuous with root surface - radiodensity subtly different to dentine of root - PDL space of tooth extends around periphery - margins often smooth but can be irregular
29
what is a cementoblastoma?
benign odontogenic tumour of cementum
30
where may you find cementoblastomas?
around root of a tooth (which remains vital)
31
what are the symptoms associated with cementoblastoma?
often painful can displace adjacent teeth and cortical bone
32
what is the incidence of cementoblastoma?
rare wide age range but often in 2nd and 3rd decades typically affects mandibular premolars or 1st molars
33
describe the radiographic presentation of cementoblastoma?
attached to a tooth root - root outline may become indistinct thin radiolucent margin continuous with PDL space of root * no radiolucent margin separating tumour from root surface well-defined and radiopaque - typically homogenous and round, can be mixed radiodensity and irregularly shaped
34
what are tori?
bony protuberances or normal bone at characteristic sites
35
where may you find tori?
middle of hard palate - torus palatinus lingual to mandibular premolars - torus mandibularis
36
have tori got associated symptoms?
asymptomatic may slowly increase in size
37
what is the cause of tori?
potentially related to genetic factors and masticatory stresses
38
what is the clinical relevance of tori?
can hamper denture wear potentially traumatised during eating
39
what is the incidence of tori?
varies between populations torus palatinus - 20% of population: often arise before age 30 torus mandibularis - 8% of population: often arise in middle age
40
describe the radiographic presentation of tori?
solitary of multiple (torus mandibularis often bilateral) cortical bone or a mix or cortical and trabecular bone sessile or pedunculated variable in size
41
what is an osteoma?
benign tumour of bone they can occur anywhere in body but have a predilection for craniofacial skeleton
42
clinical presentation of osteoma?
hard, asymptomatic, slow-growing lump single or multiple
43
osteoma incidence?
rare wide age range posterior mandible is most common jaw site
44
describe the radiographic presentation of osteoma?
entirely cortical bone or a mix of cortical and trabecullar sessile or pedunculated rounded, smooth margins
45
what is the clinical relevance of osteoma?
no malignant potential cosmetic or function issues (excise) multiple osteomas may indicate gardeners syndrome
46
what is gardener syndrome?
a rare variant of familial adenomatous polyposis
47
what is gardener syndrome characterised by?
colorectal polyposis osteomas (esp of mandible) soft tissue tumours e.g., epidermoid cyst of skin also tend to have supernumeraries, impacted teeth and multiple areas of idiopathic osteosclerosis
48
why is it important for early diagnosis of gardener syndrome?
colorectal polyps inevitably become malignant - mean age of cancer diagnosis (if not removed) if 39 years old
49
what is the name of a rare genetic condition with various skeletal defects?
cleidocranial dysplasia
50
what effect does cleidocranial dysplasia have on the teeth and jaws?
- delayed eruption - multiple supernumerary teeth, leading to impaction of other teeth - multiple unerupted secondary teeth, due to multiple retained primary teeth - hypoplastic maxilla with high arched palate - increased prevalence of cleft palate - coarse trabecular pattern
51
what effect does cleidocranial dysplasia have on the sinuses and rest of the body?
- small maxillary sinuses - absent or partially formed clavicles - bossing (bulging) of the skull - hypertelorism (increased distance between orbits)
52
what is osteomyelitis?
inflammation of bone and bone marrow due to bacterial infection
53
what is osteoradionecrosis?
bone death resulting from irradiation requires high energies of radiation (radiotherapy)
54
what is MRONJ?
bone death associated with anti-resorptive or anti-angiogenic drugs
55
what are radiographic features of osteomyelitis and osteonecrosis?
osteolysis + osteosclerosis: variable mixture of radiolucent and radiopaque areas irregularities on inner/ outer aspects of cortical bone sequestration of bone periosteal bone reaction (primarily in osteomyelitis) loss of lamina dura around teeth pathological # of bone
56
describe osteolysis?
breakdown of bone
57
describe osteosclerosis?
thickening of bone
58
name a reactive lesion with benign tumour-like behaviour?
central giant cell granuloma
59
describe central giant cell granuloma?
slow growing lesion causing expansion of bone and displacement of teeth (minority of cases more aggressive and grow rapidly)
60
central giant cell granuloma symptoms?
often asymptomatic but may be tender to palpation may invade overlying soft tissue
61
what is the incidence of central giant cell granuloma?
wide age range but majority before age 20 F>M most commonly affects mandible anterior to molars
62
what is the typical radiographic presentation of central giant cell granuloma?
site - mandible anterior to molars size - any size shape - unilocular or multilocular (when large) sometimes with internal thin septae margins - well defined, poorly corticated, scalloped internal structure - radiolucent tooth involvement - no effects - displacement of cortices; displacement of teeth; occasional external root resorption number - single
63
name the group of rare, benign, non-inheritable conditions where normal bone is replaced by connective tissue and abnormal bone?
fibro-osseous lesions
64
what are the main types of fibro-osseous lesions?
cemento-osseous dysplasia (COD) fibrous dysplasia (FD) ossifying fibroma (OF)
65
what type of fibro-osseous lesions can affect any part of the skeleton but have a predilection for the jaws?
fibrous dysplasia ossifying fibroma
66
what type of fibro-osseous lesions only affect the jaws?
cemento-osseous dysplasia
67
how are fibro-osseous lesions diagnosed?
radiology *histopathology can be unable to distinguish between different types
68
why is accurate diagnosis of fibro-osseous lesions important?
prognosis and tx options vary greatly inappropriate management increases the pt morbidity
69
what are radiographic features to consider when diagnosing different types of fibro osseous lesions?
anatomical location shape size pattern of matrix margin (including zone of transition)
70
what are the different forms of cemento-osseous dysplasia?
focal COD: single/ few localised lesions periapical COD: lesions associated with apices of anterior mandibular teeth florid COD: extensive lesion or many lesions
71
what is the incidence of cemento-osseous dysplasia?
typically pts age 30-50 years F>>M most common in Black ethnicities mandible> maxilla
72
clinical signs/ symptoms of cemento-osseous dysplasia?
often no signs/ symptoms may be expansile (esp florid type) rarely painful can become infected: pain, suppuration
73
radiological appearance of cemento-osseous dysplasia?
mixed-radiodensity lesions located at apices of vital teeth - well defined radiolucency with varying amounts of well defined radiopaque material - lamina dura lost - PDL unaffected - tooth displacement/ ERR is rare
74
what does the appearance of cemento-osseous dysplasia depend on?
stage of the lesion maturation (fully mature lesions can appear entirely radiopaque)
75
what is the mx of cemento-osseous dysplasia?
usually no mx needed removal if exposed by xla, mandibular atrophy or trauma
76
what is the risk associated with intervening cemento-osseous dysplasia?
secondary infection *avoid biopsy and xla of teeth involved
77
why may you want to consider periodic radiographic review for cemento-osseous dysplasia?
to check for development of secondary solitary bone cysts
78
what are the forms of fibrous dysplasia?
monostotic: single bone affected (most common) polyostotic: multiple lesions affecting multiple bones craniofacial: typically single lesions affecting multiple (fused) bones
79
what is the incidence of fibrous dysplasia?
1:30000 mean age 25 years F=M favours posterior maxilla
80
what is the clinical presentation of fibrous dysplasia in the jaw?
facial swelling due to bony expansion may displace teeth typically painless
81
what is the radiological presentation of fibrous dysplasia in the jaw?
altered bone pattern - highly variable: granular/ orange peel/ swirling/ wispy/ amorphous - radiodensity increases as lesions matures bone enlarges but maintains rough anatomical shape margins indistinct and blend into adjacent bone - broad zone of transition
82
what is the mx of fibrous dysplasia?
no mx required if not causing functional/ aesthetic issues if required; recontouring or radical resection
83
explain the lifespan of fibrous dysplasia lesions?
they normally stop growing but may reactivate, typically after a precipitating event like pregnancy or jaw surgery
84
name a fibro-osseous neoplasm occurring most often in tooth bearing areas?
ossifying fibroma
85
where may you find ossifying fibroma?
majority occur in the mandible rare cases in other craniofacial bones
86
clinical presentation of ossifying fibroma?
slow growing bony swelling (however juvenile sub type can grow rapid) often painless
87
incidence of ossifying fibroma?
occurs at any age - mean age 31 years F>M
88
Radiographic presentation of ossifying fibroma?
rounded, expansile lesions - affected teeth displaced and may be resorbed ranges from entirely radiolucent to completely radiopaque - radiodensity depends on stage of the lesion maturation margins usually well defined surrounding bone may be sclerotic
89
what is the mx of ossifying fibroma?
removal indicated due to progressive growth surgical enucleation or resection (usually enucleates in one piece) 12% recurrence rate
90
name a chronic condition causing disordered remodeling of bone?
Pagets disease of bone
91
what does pagets disease affect?
multiple bones at the same time - resulting in enlargement of bones, malocclusion, nerve impingement, and brittle bones majority asymptomatic
92
Pagets disease incidence?
up to 5% of patients >55 years rare <40 years M>F more common in UK than other parts of the world
93
what are radiographic features of Pagets disease of bone?
general enlargement of bones abnormal bone pattern (cotton wool appearance) osteolytic/ osteosclerotic patches of bone radiodensity of altered areas linked to stage of disease dental issues: migration. hypercementosis. loss of lamina dura
94
what are the stages of Pagets disease of bone?
early/ osteolytic (more radiolucent) intermediate/ mixed late/ osteosclerotic
95
what is osteoporosis?
decreased bone mass
96
what causes osteoporosis?
age secondary to nutritional deficiency, medications etc
97
what are radiographic features of osteoporosis?
thinned cortices e.g., inferior border of mandible sparse trabecular bone pattern - general radiolucent appearance thinned lamina dura around teeth