Quiz #3 (hepatic) prep Flashcards

1
Q

If a patient has hepatic disease, what skin changes would you expect?

A
Jaundice
Bruising
Spider angioma
Palmar erythema
Purpura (rash and purple spots)
Caput medusa (epigastric viens radiate from umbilicus)
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2
Q

If a patient has hepatic disease, what neurological changes would you expects?

A

confusion
sleep disturbances
muscle tremors
asterixis (flapping tremor)

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3
Q

What common sign/symptom could you see in a hepatic disease patients urine and stools?

A

dark urine

light or clay-colored stools

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4
Q

What common sign/symptom could you see in a hepatic disease patients abdomen?

A

Edema/ascites

Right upper quadrant abdominal pain

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5
Q

What common sign/symptom or condition could you expect a hepatic disease patient to have in their bones?

A

Hepatic osteodystrophy

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6
Q

What, why and when would a person with liver disease become jaundice (icterus)?

A

what - defects in bilirubin metabolism, liver disease or obstruction of bile flow.
why - due to liver damage (AST/ALT), decreases albumin
when - normal bilirubin 0.3-1.0 mg/dL; seen in eyes at 3.0 mg/dL and seen in the skin at 5-6 mg/dL.

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7
Q

What are some special implications for the PT when working with patients with viral hepatitis?

A
  • use universal precautions
  • don’t wear clothes out of building
  • 1/3 of patients c/o joint pain
  • teach energy conservation for ADL’s
  • Watch for signs of “fluid” shifting
  • Watch for adverse drug reactions
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8
Q

Prehepatic hyperbilirubinemia caused by excess bilirubin formation secondary to hemolysis is called?

A

unconjugated

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9
Q

Hyperbilirubinemia caused by liver cell necrosis is called?

A

mixed or unconjugated

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10
Q

Hyperbilirubinemia caused by an obstruction of bile flow is called?

A

conjugated

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11
Q

What body system become involved when a patient had cirrhosis of the liver?

A

all systems become involved including cardiopulmonary, renal, endocrine and hematopoietic

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12
Q

What are 3 clinical features of cirrhosis?

A
  • large increase in serum liver enzyme ALT and AST
  • loss of liver functions
  • consequences of portal hypertension
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13
Q

If liver inflammation leads to necrosis what clinical manifestations will result?

A
  • decreased bilirubin metabolism (jaundice)
  • decreased bile in GI tract (light-color stools)
  • decreased vitamin K absobption (bleeding)
  • decreased metabolism of proteins/carbs/fats (hypoglycemia)
  • decreased plasma proteins (ascites & edema)
  • decreased hormone metabolism
  • increased urobilinogen (dark urine)
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14
Q

What 3 things are associated with hepatic disorders and portal hypertension?

A
  • Ascites (d/t portal hypertension & hypoproteninemia
  • Splenomegaly
  • Anastomoses between portal and systemic circulation (varies and is very serious)
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15
Q

Special implications for working with patients with ascites?

A
  • Fowler’s position (head of bed raised 18-20in above level with knees elevated)
  • Breathing techniques (prevent atelectasis and pneumonia)
  • Monitor vital signs (incr oxygen saturation)
  • Monitor for bacterial peritonitis (fever, chills, abdomen pain & tenderness)
  • Monitor fluid intake/outake (daily weights, 5-10 lbs/day)
  • decreased SERUM ALBUMIN (incr edema, decr healing/clotting factors, drugs attach to albumin)
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16
Q

What do you suspect when your patient has shape abdomen pain and they vomit after eating high fat foods?

A

an obstructed bile duct or cystic.

17
Q

What treatments are available for patients with obstructed bile ducts (cystic)?

A
  • surgery (laparoscopy)
  • extracorporeal shock waves (lithotripsy)
  • drugs