Quick recall Flashcards
Features of serotonin syndrome
3As
- Altered mental status
- Neuromuscular abnormalities
- Autonomic hyperactivity
Plummer-vinison syndrome characterised by triad of what:
Plummers DIE
D- dysphagia
I- iron deficiency anaemia
E- eosophageal webs
A 5-year-old boy presents with recurrent episodes of sinusitis. The casualty staff are surprised to find his liver lying in the left upper quadrant of the abdomen
Kartagener’s syndrome
- immotile cilia syndrome
- assocaited with sinus inversus
What is vestibular neuronitis?
–> cause of vertigo
–> develops following viral infection
Features:
1. recurrent vertigo attacks lasting hours or days
2. nausea and vomiting may be present
3. horizontal nystagmus is usually present
4. no hearing loss or tinnitus
Differentiating vestibular neuronitis from posterior circulation stroke
HiNTs exam
Management of vestibular neuronitis:
- buccal or intramuscular prochlorperazine : rapid relief for sever cases
- vestibular rehab exercises for patients with chronic symptoms
mydriasis
dilated eye
miosis
smaller pupil
3rd nerve palsy characterised by:
- ptosis
- down and out eye
- mydriasis
Status epilepticus management
Management
1. ABC
2. BENZOs
–> Prehospital –> PR diazepam or buccal midazolam
–> Hospital IV lorazepam –> repeated once after 5-10 mins
3. Established status
–> leve, phenytoin, sodium valproate
4. No response w/i 45 mins?
–> call the anaesthetist!–> RSI
Mx Bells palsy
- PRED, w/i 72 hrs onset of bells palsy
- Eyecare
–> artificial tears
–> eye lubricants - refer to ENT if paralysis no improvement 3/52
51 y/o female
PC: facial flushing + diarrhoea
HPC: 6/12 facial flushing, worse on exertion, associated palpitations. 3/12 diarrhoea.
OE: peripheral oedema, soft mid-diastolic murmur
characteristic of:
CARCINOID SYNDROME
–> secretion of serotonin, kinins and amines leads to flushing
Murmur –> tricuspid stenosis, R heart failure
Investigation for carcinoid syndrome
24 hour collection of urinary 5-HIAA
Management of carcinoid syndrome
- Somatostatin analogues e.g. octreotide
- diarrhoea: cyproheptadine
What occurs in primary hyperparathyroidism?
Cause –> solitary parathyroid adenoma –> increase release of PTH
Result:
–> high calcium and low phosphate
–> increase bone turnover by OC activation –> more ALP
Presentation: hypercalcaemia symptoms: constipation and mood changes
Describe secondary hyperparathyroidism:
Chronically low serum [Ca2+] –> parathyroid hyperplasia
Increased PTH secretion will not be able to reverse chronically low calcium and high phosphate caused by condition.
PTH –> excessive bone turnover –> high ALP
Presentation: bone pain, tenderness, proximal myopathy and recurrent fractures
Tertiary hyperparathyroidism is caused by:
ongoing hyperplasia of parathyroid glands after correction of long standing renal disorder.
Presentation of tertiary hyperparathyroidism
- high calcium
- elevated PTH
- decreased phosphate
- elevated ALP
management of endometrial hyperplasia
Simple w/o atypia –> high dose progestogens with repeat sampling in 2-4 months. maybe IUS.
If Atypia: HYSTERECTOMY w/ bilateral salpingectomy
what sound be suspected in a patient who develops post operative shortness of breath and hypoxaemia
Atelectasis
management of atelectasis
- position patient upright
- chest physio: breathing exercises
What rules are used to minimise use of X-rays in patients presenting with ankle injuries
OTTAWA ANKLE RULES
- inability to weight bear for 4 steps
- tenderness over distal tibia
- bone tenderness over distal fibula
Weber classification
A - below syndesmosis (CAM + weight bearing)
B- tibial plafond may extend proximally to involve syndesmosis
C - above syndesmosis (ORIF)
What is Wolff-Parkinson-White syndrome?
- accessory pathway that bypasses the AVN
- causing early depolarisation of the ventricles
- short PR interval (<120ms)
Possible ECG features
- short PR
- widened QRS
Management of WPW syndrome
Definitive treatment –> radiofrequency ablation of accessory pathway
Medical therapy: amiodarone, flecainide