Psychopathology: Biological approach to explaining and treating OCD Flashcards
What are the key assumptions of the biological approach?
3
-all behaviour has a biological cause
-behaviour is influenced by genes , neurotransmitters and brain structures
-psychological disorders are best treated with biological methods
Are obsessions the internal or external component of OCD , why ?
Are compulsions the internal or external component , why?
they are the internal component as they are thoughts which are intrusive
they are the external component as they are behaviours which are repetitive
What are the behavioural characteristics of OCD?
There are 2
Compulsions-these are repetitive and the purpose of them is to reduce anxiety
Avoidance-keep away from situations that trigger anxiety
What are the cognitive characteristics of OCD?
There are 3
Obsessive thoughts-recurrent intrusive thoughts
Awareness that the behaviour is irrational
Attentional bias and maintaining alertness
What is an additional cognitive characteristic of OCD?
Cognitive strategies to deal with obsessions-this helps manage anxiety but can lead to failing to function adequately
What are the emotional characteristics of OCD?
There are 2
Anxiety and distress-the thoughts are frightening and anxiety can be overwhelming , the urge to repeat behaviour also causes anxiety
Guilt and disgust-irrational guilt over minor issues and disgust with external environment or self
Describe the genetic explanation for OCD…
make 2 points
Candidate genes-there are specific gene markers that make people vulnerable to developing OCD eg the SERT gene which is linked with low serotonin levels
OCD is polygenic-there are over 230 different genes that may be involved in OCD
Evaluate the genetic explanation for OCD
1 of each
+there is good supporting evidence from twin studies
Nestadt et al 2010-reviewed previous twin studies and found 68% of MZ twins shared OCD compared to 31% of DZ twins
strongly suggests a genetic influence on OCD
-there are also environmental factors
Cromer et al 2007 found over half the OCD patients from his sample had experienced a traumatic event and OCD was more sever when they had more than one trauma
suggests OCD isn’t entirely caused by genetics
Describe the neural explanation for OCD…
make 2 points
role of serotonin-OCD linked with low levels of serotonin which prevents normal transmission of mood relevant info between neurons
this impacts mood and other mental processes
brain structure-basal ganglia system and other systems are implicated , the communication within these areas are disrupted and may account for repetitive behaviours
Evaluate the neural explanation for OCD…
1 of each
+there is supporting evidence for both
drugs which increase serotonin levels are effective in reducing symptoms and DBS that targets the loops in the basal ganglia has been effective at reducing symptoms
this suggests that both systems are involved in OCD
-neural mechanisms may not be the cause
people with OCD show abnormalities in brain structure and neurotransmitter levels but these could be a result of OCD not a cause
means the explanation is incomplete as can’t est cause and effect
State the three main points for synaptic transmission
-when a neuron is activated by a stimulus it generates an action potential that travels down the axon of the neuron
-the gap between 2 neurons is known as the synaptic cleft
-for the action potential to reach the next neuron it must travel across the synapse as a chemical signal using neurotransmitters
What is an action potential?
an electrical impulse
What happens at the beginning of synaptic transmission?
2 points
-action potential travels down the axon of the pre-synaptic neuron
-action potential triggers release of neurotransmitters from vesicles
What happens in the middle of synaptic transmission?
2 points
-neurotransmitters diffuse across the synapse
-they bind to receptors on the post-synaptic membrane
What happens at the end of synaptic transmission?
2 points
-stimulation of post-synaptic receptors by neurotransmitters results in either excitation or inhibition of the post-synaptic membrane
-neurotransmitters are reabsorbed into the pre-synaptic neuron