Psychopathology Flashcards

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1
Q

The definitions for abnormality are:

A

Statistical infrequency

Deviation from social norms

Failure to function adequately

Deviation from ideal mental health

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2
Q

Statistical Infrequency

A

Statistical infrequency defines abnormality in terms of behaviours seen as statistically rare or which deviate from the mean average or norm.

Statistics that measure certain characteristics and behaviours are gathered with the aim of showing how they are distributed among the general population.

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3
Q

Normal Distribution Curve

A

A normal distribution curve can be generated from such data which demonstrates which behaviours people share in common. Most people will be on or near the mean average however individuals which fall outside this “normal distribution” and two standard deviation points away are defined as abnormal.

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4
Q

Where do the majority of normal behaviours cluster?

A

The majority of normal behaviours cluster in the middle of the distribution graph with abnormal characteristics around the edges or tails making them statistically rare and therefore a deviation from statistical norms.

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5
Q

Statistical Infrequency - Appropriate Measure

A

Defining abnormality using statistical criteria can be appropriate in many situations; for example in the definition of mental retardation or intellectual ability.

In such cases normal mental ability can be effectively measured with anyone whose IQ falling more than two standard deviation points than most the general population being judged as having some mental disorder.

When used in conjunction with other definitions such as the failure to function adequately, statistical infrequency provides an appropriate measure for abnormality.

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6
Q

Statistical Infrequency - Characteristics

A

A weakness, however, is in other occasions defining peoples characteristics on statistical rarity solely is unsuitable.

For example, people with exceptionally high IQ’s could, in theory, be diagnosed as having a mental disorder as their intelligence may be two deviations above the rest of the population and technically “abnormal”. This is why statistical infrequency is best used in conjunction with other tools to define abnormality.

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7
Q

Statistical Infrequency - Objective

A

Statistical infrequency provides an objective measure for abnormality.

Once a way of collecting data on behaviour/characteristics and a cut-off point is agreed, this provides an objective way of deciding who is abnormal.

However, a weakness here is that the cut-off point is subjectively determined as we need to decide where to separate normal behaviour from abnormal and again this blurs the line in some cases.

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8
Q

Statistical Infrequency - Imposed Etic

A

A major weakness is that statistical infrequency has an imposed etic of whatever culture is measuring the behaviour, usually western cultures and therefore suffers from cultural bias.

It does not consider cultural factors in determining abnormal behaviour and what is a normal behaviour in one culture may be seen as abnormal in another due to its infrequency.

Also, behaviours which were statistically rare many years ago may not be rare now (and vice versa).

Therefore this tool could run the risk of being era-dependent by adopting a statistical norm based on behaviours that may later become outdated.

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9
Q

Deviation from Social Norms

A

Deviation from social norms defines abnormality in terms of social norms and expected behaviours within society and certain situations.

Within society there are standards of acceptable behaviour which are set by the social group and everyone within this social group is expected to follow these behaviours.

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10
Q

Social Norms

A

Social norms can be explicit written rules or even laws and an example of this is the respect for human life and property which belongs to others.

These are norms enforced by a legal system within the UK however other social norms are unwritten but still generally accepted as normal behaviour.

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11
Q

Social Norms - Temporal Validity

A

One major issue with basing abnormal behaviour on a set of social norms is that they are subject to change over time. Behaviour that is socially acceptable now may suddenly be seen as socially deviant later and vice versa. Therefore this definition is very era-dependent.

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12
Q

Social Norms - Open to Abuse

A

Another issue is this form of diagnosis is open to abuse. In Russia, during the late 1950s, anyone who disagreed with the government ran the risk of being diagnosed as insane and placed in a mental institution.

Therefore defining people based on a deviation from socially acceptable behaviour allows people to be persecuted for being non-conformist

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13
Q

Social Norms - Context Dependent

A

There is no clear distinction on where this divide between normal and abnormal is as such behaviour may simply be eccentric but not due to any mental disorders.

Therefore social deviance on its own cannot offer a holistic definition of abnormal behaviour.

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14
Q

Social Norms - Provide Help

A

A strength of using deviations from social norms to define abnormality is it can if used correctly, help people as it gives society the right to intervene to improve the lives of people suffering from mental disorders who may not be able to help themselves.

This definition also helps protect members of society itself as a deviation from norms usually comes at the expense of others as social norms are usually designed to keep society functioning adequately.

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15
Q

Social Norms - Cultural Bias

A

Deviation from social norms is subject to cultural bias.

For example, western social norms reflect the majority of the white western population and ethnic groups which behave differently could be seen as “abnormal” simply because their customs or behaviours are based on eastern or European values

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16
Q

Failure to Function

A

Defining abnormality on the basis of failure to function adequately takes to account a persons ability to cope with the daily demands of life.

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17
Q

Functioning Affecting Lives

A

This inability to cope may cause the individual or others around them distress and this is factored into this definition as some people with mental disorders may themselves not be distressed but cause it to those around them.

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18
Q

Rosenhan’s Criteria for Abnormality

A

Observer Discomfort

Irrationality

Maladaptive Behaviours

Unpredictability

Personal Distress

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19
Q

FTF - Subjective

A

One weakness with this definition is it needs someone to judge whether the behaviour someone displays is abnormal or not and this may be subjective.

A patient experiencing personal distress through being unable to meet their bills or get to work may be judged as abnormal by one judge while another individual may see this as one of the many pitfalls of adult life.

This definition creates ideal expectations which many people may struggle to adhere to and risk being classed as abnormal.

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20
Q

FTF - Personal

A

A strength of this definition, however, is it does recognise the subjective experience of the individual themselves who may be struggling to function adequately and wish to seek intervention.

This definition takes a patient-centred view by allowing mental disorders to be regarded from the perception of sufferers.

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21
Q

FTF - Cultural Bias

A

Such a definition suffers from cultural bias as it will inevitably be related to how one culture believes an individual should live their lives.

Basing abnormality on the basis of failing to function is likely to lead to different diagnoses when applied to people from different cultures or even socio-economic classes.

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22
Q

FTF - Obvious or Not?

A

In other cases, the abnormality may not always be followed by observable dysfunctional traits.

For example, psychopaths and people with dangerous personality disorders can cause great harm to others yet still appear normal.

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23
Q

Deviation from Ideal Mental Health

A

Deviation from ideal mental health assesses abnormality by assessing mental health in the same way physical health would be assessed.

This definition looks for signs that suggest there is an absence of wellbeing and deviation away from normal functioning would be classed as abnormal.

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24
Q

Jahoda’s Criteria

A

Positive Self Attitude

Self-actualisation

Autonomy

Accurate Perception of Reality

Resisting Stress

Environmental Mastery

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25
Q

DFIMH - Unrealistic

A

The criteria for being classed as normal are over-demanding and unrealistic which is a major criticism of this definition.

By Jahoda’s standard, most people would be classed as abnormal as they fail to meet these requirements which means this diagnosis is more a set of ideals on how you would like to be rather than how you actually are.

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26
Q

DFIMH - Vague

A

The criteria Jahoda puts forth are subjective and difficult to measure due to being vague.

Measuring physical health is more objective through the use of equipment however mental health through these criteria is difficult to measure.

For example, measuring self-esteem, personal growth or environmental mastery would all be difficult and require a subjective opinion on where the cut-off point would be.

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27
Q

DFIMH - Western

A

This definition would be culturally biased as these set of ideals put forth by Jahoda are based on western ideals of what ideal health looks like within one particular culture.

If this was used to judge the behaviour of people from different cultures then this may provide an incorrect diagnosis of abnormality as they have different beliefs on what “ideal mental health” would look like.

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28
Q

Emotional Characteristics of Phobias

A

Anxiety

Unreasonable Emotional Response

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29
Q

Behavioural Characteristics of Phobias

A

Panic

Avoidance

Endurance

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30
Q

Cognitive Characteristics of Phobias

A

Selective Attention on Phobic Stimulus

Irrational Beliefs

Cognitive Distortions

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31
Q

Emotional Characteristics of Depression

A

Lowered Mood

Anger

Lowered Self-esteem

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32
Q

Behavioural Characteristics of Depression

A

Activity Levels

Disruption to Eat and Sleep Habits

Aggression and Self Harm

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33
Q

Emotional Characteristics of OCD

A

Anxiety and Distress

Accompanying Depression

Guilt and Disgust

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34
Q

Behavioural Characteristics of OCD

A

Compulsions

Avoidance

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35
Q

Cognitive Characteristics of OCD

A

Obsessive Thoughts

Cognitive Coping Strategies

Insight into Excessive Anxiety

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36
Q

Mowrer - 1947

A

Mowrer (1947) proposed the two-process model which attempts to explain phobias through the behaviourist explanation of either classical or operant conditioning.

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37
Q

Behaviourists

A

Behaviourists propose phobias are learned through experience and association and through classical conditioning phobias are acquired by a stimulus becoming associated with a negative outcome.

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38
Q

Watson and Rayner - Little Albert

A

A child was in introduced to a loud noise (unconditioned stimulus) which produced the fear response (unconditioned response).

A white rat (neutral stimulus) was introduced and paired with this loud noise which over time became paired with the fear response towards this white rat (conditioned response).

The rat then becomes a conditioned stimulus as it produces the conditioned response of fear.

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39
Q

Classical Conditioning and Operant Conditioning

A

Traumatic events that occur produce negative feelings which then become conditioned responses to such objects, animals or situations which are conditioned stimuli.

Phobias are then maintained by operant conditioning which explains why people continue to remain fearful or avoid the object or situation in question.

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40
Q

Positive Reinforcement

A

This proposes that behaviour is likely to be repeated if the outcome is rewarding in some way.

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41
Q

Negative Reinforcement

A

If the behaviour results in the avoidance of something unpleasant, this is known as negative reinforcement.

In the case of phobias and through negative reinforcement; the avoidance of the object/situation in question reduces anxiety or fear which the individual finds rewarding.

This then reinforces the avoidance behaviour further.

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42
Q

Further Behavioural Explanations

A

Another behavioural explanation is social learning theory and this explains phobias as having been acquired through modelling behaviours observed from others.

An individual may see a phobic response and emulate the reaction as it appears rewarding in some form i.e. attention.

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43
Q

Mowrer - Traumatic Event or Not?

A

The two-process model is generally supported through phobia sufferers being able to recall a traumatic or specific event which triggers it.

Not everyone who suffers a traumatic event then goes on to develop a phobia.

This suggests the two-process model is overly simplistic and not a holistic explanation as other factors must be at work.

However, a weakness is not everyone is able to link their phobia to a specific event they can recall.

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44
Q

Mowrer - Bagby Research Support

A

A case study by Bagby (1922) lends support for classical conditioning explaining her phobia of running water which caused her extreme distress.

The sound of running water had become associated with the fear and distress she experienced demonstrating how the two-process model has validity in some explanations of phobias.

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45
Q

Mowrer - Generalisability

A

However, with single case studies, we may not necessarily be able to generalise the findings to the wider population as the circumstances for that phobia developing may lack external validity to other peoples conditions.

In addition to this such case studies are time-consuming and almost impossible to replicate to test the reliability of findings to confirm they occurred as patients may describe.

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46
Q

Mowrer - Rachman’s Safety

A

Rachman 1984) offered an alternative view through the Safety signals hypothesis which undermines the two-process model.

This proposed that avoidance behaviour towards the object/animal in question is not motivated by negative reinforcement and the reduction in anxiety as the two-process model proposes but by the positive feelings the person associates with safety.

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47
Q

Mowrer - Biological Reasons

A

The diathesis-stress model may offer a better explanation which combines both psychological factors such as the two-process model and combines this with a genetic vulnerability.

This suggests that some people may inherit a genetic vulnerability for developing mental disorders such as phobias provided the right environmental stressors trigger this.

This would explain why phobias develop in some people but not necessarily others.

However again this would be incredibly difficult to validate for certain.

48
Q

Systematic Desensitisation

A

Systematic desensitisation is based on the assumption that if phobias are a learned response as classical and operant conditioning suggests, then they can be unlearnt.

Through a process of classical conditioning, systematic desensitisation teaches patients to replace their fearful feelings through a process of hierarchal stages which gradually introduces the person to their feared situation one step at a time.

49
Q

SD - Anxiety Hierarchy

A

The hierarchy is constructed prior to treatment starting from the least feared to most feared situation working towards contact and exposure.

Throughout these stages, patients are taught relaxation techniques that help manage their anxiety and distress levels to help them cope but also to associate these feelings of calmness towards the phobia.

50
Q

SD - Relaxation Techniques

A

Relaxation techniques taught may help the patient focus on their breathing and taking slower, deeper breaths as anxiety often results in faster, shallow breathing and this helps manage this.

Mindfulness techniques such as “here and now” skills may also be used which involves focusing on a particular object or visualising a relaxing scene.

Progressive muscle relaxation involves straining and relaxing muscle groups gently and this can help relax the body from tension too.

51
Q

SD - Counter Conditioning

A

Counter-conditioning involves classical condition and may also be used as part of systematic desensitisation by creating a new association which runs alongside the current phobic situation.

52
Q

SD - Appropriate

A

A weakness for systematic desensitisation is that it is not appropriate for all patients and only those who have the capacity to learn relaxation strategies and for those who have imaginations vivid enough to imagine the feared situations in question.

There is also no guarantee that learning to imagine and cope with phobic situations will actually translate into it working in the real world either.

53
Q

SD - Time Consuming

A

Another weakness is systematic desensitisation is time-consuming and costly for people to use which may make it inappropriate. Patients need to attend numerous appointments and to build trust with their practitioner who is a stranger which can in itself be difficult.

Also, the strategy is dependent on the skill-set of the practitioner themselves which can affect how long this treatment takes or if it works at all.

54
Q

SD - Research Support

A

A strength, however, is that there is strong evidence that suggests systematic desensitisation is effective with numerous research studies finding it a success.

McGrath et al (1990) reported 75% of patients responded positively with S and exposure to the feared stimulus (Vivo techniques) was believed to be one of the main reasons.

Vitro techniques which involve patients imagining the feared stimulus were less effective in comparison (Choy et al 2007).

55
Q

SD - Ethics

A

There are ethical issues which arise with SD as it deliberately exposes patients to their fears which can cause psychological harm as there is no guarantee they will cope with it well.

They may go on to have nightmares or their fear may even get worse to a point their life becomes dysfunctional.

With this in mind, it may not always be appropriate for all patients and a cost-benefit analysis may be needed to weigh the benefits and costs with both short-term and long-term in mind.

56
Q

Flooding

A

Flooding is an alternative approach to systematic desensitisation and either exposes the patient directly to their phobia or they are asked to imagine an extreme form of it.

57
Q

Flooding - Relaxation Techniques

A

The client is also taught and encouraged to use relaxation techniques prior to the exposure to the phobic situation which continues until the patient is able to fully relax.

58
Q

Flooding - Reciprocal Inhibition

A

In fear-based situations, the patient will release adrenaline however this will eventually cease with relaxation being associated with their feared stimulus as they are unable to use their normal avoidance methods.

59
Q

Flooding - Ethics

A

Flooding raises serious ethical issues as it deliberately exposes patients to their fears which can cause severe psychological harm as there is no guarantee they can eventually cope with the situation.

With this in mind, informed consent is always required before beginning treatment

60
Q

Flooding - Individual Differences

A

As not everyone may be able to cope with this form of treatment, its effectiveness may be down to individual differences.

Not everyone enjoys good physical health and subjecting such individuals to highly stressful situations through flooding may risk health problems i.e. heart attacks.

61
Q

Flooding - Cost Effective

A

A benefit to flooding, however, is the treatment is relatively quick to administer and effective with Choy et al (2007) reporting it to be more effective than SD.

62
Q

Beck

A

Beck developed a cognitive explanation of depression which has three components: a) cognitive bias; b) negative self-schemas; c) the negative triad.

63
Q

Cognitive Bias

A

Beck found that depressed people are more likely to focus on the negative aspects of a situation, while ignoring the positives. They are prone to distorting and misinterpreting information, a process known as cognitive bias.

64
Q

Types of Cognitive Bias

A

Catastrophising - exaggerate a minor setback and believe that it’s a complete disaster

Over-generalising - make a sweeping conclusion based on a single incident

65
Q

Schema

A

A schema is a ‘package’ of knowledge, which stores information and ideas about our self and the world around us.

66
Q

Negative Self-Schemas

A

These schemas are developed during childhood and according to Beck, depressed people possess negative self-schemas, which may come from negative experiences, for example criticism, from parents, peers or even teachers.

A person with a negative self-schema is likely to interpret information about themselves in a negative way, which could lead to cognitive biases, such as those outlined above.

67
Q

Negative Triad

A

Beck claimed that cognitive biases and negative self-schemas maintain the negative triad, a negative and irrational view of ourselves, our future and the world around us.

For sufferers of depression, these thoughts occur automatically and are symptomatic of depressed people.

68
Q

Ellis

A

Ellis took a different approach from Beck (cognitive triad) to explaining depression and started by explaining what is required for ‘good’ mental health.

ABC Model

According to Ellis, good mental health is the result of rational thinking which allows people to be happy and pain free, whereas depression is the result of irrational thinking, which prevents us from being happy and pain free.

69
Q

Activating Event

A

An event occurs that triggers the onset of emotions.

70
Q

Beliefs

A

Your belief is your interpretation of the event, which can either be rational or irrational.

71
Q

Consequences

A

According to Ellis, rational beliefs lead to healthy emotional outcomes, whereas irrational beliefs lead to unhealthy emotional outcomes

72
Q

Beck - Research Support from Boury

A

Boury et al. (2001) found that patients with depression were more likely to misinterpret information negatively (cognitive bias) and feel hopeless about their future (negative triad), which supports different components of Beck’s theory and the idea that cognitions are involved in depression.

73
Q

Cognitive Approach - Origins of Irrational Thoughts

A

One weakness of the cognitive approach is that it does not explain the origins of irrational thoughts and most of the research in this area is correlational.

Therefore, we are unable to determine if negative, irrational thoughts cause depression, or whether a person’s depression leads to a negative mindset.

Therefore, it is possible that other factors, for example genes and neurotransmitters, are the cause of depression and one of the side effects of depression are negative, irrational thoughts.

74
Q

Cognitive Approach - Practical Applications

A

One strength of the cognitive explanation for depression is its application to therapy.

The cognitive ideas have been used to develop effective treatments for depression, including Cognitive Behavioural Therapy (CBT) and Rational Emotive Behaviour Therapy (REBT), which was developed from Ellis’s ABC model.

These therapies attempt to identify and challenge negative, irrational thoughts and have been successfully used to treat people with depression, providing further support to the cognitive explanation of depression.

75
Q

Cognitive Behavioural Therapy

A

Cognitive Behavioural Therapy (CBT) involves both cognitive and behavioural elements.

76
Q

CBT - Cognitive Element

A

The cognitive element aims to identify irrational and negative thoughts, which lead to depression.

The aim is to replace these negative thoughts with more positive ones.

77
Q

CBT - Behavioural Element

A

The behavioural element of CBT encourages patients to test their beliefs through behavioural experiments and homework.

78
Q

Starting CBT

A

All CBT starts with an initial assessment, in which the patient and therapist identify the patient’s problems.

Thereafter, the patient and therapist agree on a set of goals, and plan of action to achieve these goals.

Both forms of CBT (Beck’s and Ellis’s) then aim to identify the negative and irrational thoughts, however their approaches are slightly different.

79
Q

Components of CBT

A

Initial Assessment

Goal Setting

Identifying Negative Thoughts

Homework

80
Q

Beck’s Cognitive Therapy

A

If a therapist is using Beck’s cognitive therapy, they will help the patient to identify negative thoughts in relation to themselves, their world and their future, using Beck’s negative triad.

The patient and therapist will then work together to challenge these irrational thoughts, by discussing evidence for and against them.

The patient will be encourage to test the validity of their negative thoughts and may be set homework, to challenge and test their negative thoughts.

81
Q

Ellis’ Rational Emotive Behavioural Therapy

A

Ellis developed his ABC model to include D (dispute) and E (effect or effective).

Like Beck, the main idea is to challenge irrational thoughts, however, with Ellis’s theory this is achieved through ‘dispute’ (argument).

82
Q

Components of REBT

A

The therapist will dispute the patient’s irrational beliefs, to replace their irrational beliefs with effective beliefs and attitudes.

Following a session, the therapist may set their patient homework. The idea is that the patient identifies their own irrational beliefs and then proves them wrong.

83
Q

Types of Dispute

A

Empirical - the therapists seeks evidence for a person’s thoughts

Logical - the therapist questions the logic of a person’s thoughts

84
Q

CBT - Research Support from March et al

A

Research by March et al. (2007) found that CBT was as effective as antidepressants, in treating depression.

The researchers examined 327 adolescents with a diagnosis of depression and looked at the effectiveness of CBT, antidepressants and a combination of CBT plus antidepressants.

After 36 weeks, 81% of the antidepressant group and 81% of the CBT group had significantly improved, demonstrating the effectiveness of CBT in treating depression.

However, 86% of the CBT plus antidepressant group had significantly improved, suggesting that a combination of both treatments may be more effective.

85
Q

CBT - Motivation

A

One issue with CBT is that it requires motivation.

Patients with severe depression may not engage with CBT, or even attend the sessions and therefore this treatment will be ineffective in treating these patients.

Alternate treatments, for example antidepressants, do not require the same level of motivation and maybe more effective in these cases.

This poses a problem for CBT, as CBT cannot be used as the sole treatment for severely depressed patients.

86
Q

CBT - Overemphasises Cognitions

A

Furthermore, cognitive behavioural therapy has been criticised for its overemphasis on the role of cognitions.

Some psychologists have criticised CBT, as it suggests that a person’s irrational thinking is the primary cause of their depression and CBT does not take into account other factors.

CBT therefore ignores other factors or circumstances that might contribute to a person’s depression.

87
Q

What does the genetic explanation believe about how you develop OCD?

A

You inherit a genetic predisposition to developing OCD

88
Q

How has the genetic explanation of OCD used twin and family studies?

A

Twin studies on genotype and phenotype are used to find concordance rates (measure of genetic similarity) using Mz (identical) and Dz (non-identical) twins.

Twin studies are used to find out if there is credibility in the idea of there being specific genes related to the onset of OCD, giving us a genetic vulnerability to the disorder, and so these studies look into what extent genes play their part over environment.

Mz twins are genetically similar and share the same environment, so if one twin has the disorder, so should the other theoretically.

89
Q

What did Nestadt find in 2000?

A

Found that first degree relatives of OCD sufferers, such as parents, siblings and children, had an 11.7% chance of developing the disorder compared to 2.7% of first degree relatives of non-OCD sufferers

90
Q

What did Miguel find in 2005?

A

Claims that when one Mz twin has OCD, the other has a 53-87% chance of developing it, compared with Dz twins having a 22-47% chance of both suffering.

The environment the twins grow up in will still be seen as the same, so therefore the difference in chance must be due to the greater genetic similarity in Mz twins over Dz.

91
Q

How can a malfunction of the COMT gene cause OCD according to the genetic explanation?

A

The COMT gene usually regulates dopamine activity, keeping normal levels, however, when the gene malfunctions, there is no regulation, producing lower activity.

Therefore, homeostatic balance is not maintained and therefore this causes higher levels of dopamine, which is found in many OCD sufferers.

92
Q

How can a SERT gene malfunction cause OCD according to the genetic explanation?

A

A malfunction of the SERT gene affects the normal reuptake of serotonin, leading to low serotonin levels, meaning that there is not enough to inhibit the worry associated with OCD.

This gene malfunction seems to run in families, and raising serotonin with anti-depressants alleviates symptoms, therefore validating the casual influence of serotonin.

93
Q

What did Ozaki find in 2003 about the SERT gene malfunction?

A

Found a mutation of the SERT gene in 2 unrelated families where 6 of the 7 family members with it had OCD.

94
Q

Give a strength of the genetic explanation of OCD

A

It is supported by research such as:

Lenane et al 1990 - found evidence for existence of heritable contributions to the onset of OCD

Stewart et al 2007 - performed gene mapping on OCD patients and their family members, finding that a variant of the OLIG-2 gene commonly occurred, suggesting a genetic link

95
Q

Genes - Concordance Rates

A

Concordance rates from twin studies were not 100%, meaning that there must be some environmental influence at play.

Therefore, genetics alone is an insufficient explanation

96
Q

Genes - Type of Genetic Link

A

There is a stronger genetic link between some types of OCD than others, particularly those associated with contamination/washing, meaning that genetics is not a complete explanation to OCD overall.

97
Q

Genes - Polygenic

A

No single gene causes the disorder, but many scattered across the genome, and these depend on environmental triggers to develop into full blown OCD

Taylor et al said that OCD is polygenic and may involve 230 genes - Twin and family studies show a link between genetics and OCD but do not explain the genetic mechanisms behind the disorder

98
Q

Genes - Pato’s Call for Further Research

A

Pato et al 2001 said that there is a huge amount of evidence for OCD being heritable, but few details are understood about the genetic mechanisms underpinning the disorder, indicating need for more focused research.

Family members often show different OCD symptoms

99
Q

Genes - Purely Inherited or Not?

A

If OCD was purely inherited, surely the symptoms would be the same

Grootheest et al 2005 - OCD originating in childhood is more genetic in nature than originating in adulthood - different types of OCD with different causes

100
Q

What is the diathesis-stress model in terms of genetics?

A

An interactionist approach to explaining the cause of OCD, believing that individuals inherit a genetic vulnerability” to developing OCD

101
Q

What is the diathesis-stress model in terms of neural explanation?

A

An interactionist approach to explaining the cause of OCD - you have an abnormally functioning brain mechanism, like a neurotransmitter imbalance, which has the potential to cause OCD (e.g. due to too low levels of serotonin), and catching a virus like flu or strep throat leads to an immune system attack and deficiency.

102
Q

What does the neural explanation believe about how you develop OCD?

A

Suggests that OCD results from abnormally functioning brain mechanisms.

This could be neurochemical (neurotransmitter imbalance) or neurophysical (referring to the brain area)

103
Q

What have PET scans on the brains of OCD sufferers revealed?

A

That OCD sufferers have low levels of serotonin - drugs have revealed that raising these levels reduces the symptoms of OCD.

That OCD sufferers have high activity in the orbitofrontal cortex, the area which is associated with initiating activity upon receiving an impulse.

104
Q

Which other brain area has been linked to OCD?

A

The basal ganglia has been identified as functioning abnormally in OCD sufferers - it is the area linked to compulsion production and is overactive in OCD sufferers

105
Q

What did Max et al discover about the basal ganglia during surgery?

A

Max et al. (1994) found that when the basal ganglia is disconnected from the frontal cortex during surgery, OCD-like symptoms are reduced, providing further support for the role of the basal ganglia in OCD.

106
Q

What type of treatments are used for OCD?

A
  • Low serotonin levels are believed to play a role in influencing OCD with the orbital frontal cortex (also known as the worry circuit) malfunctioning.
  • By using antidepressants, this is believed to bring levels back to normal but also help reduce anxiety which is associated with OCD too.
107
Q

What are the preferred drugs for treating OCD?

A

The preferred drugs are SSRI’s (selective serotonin reuptake inhibitors) with adults commonly prescribed Fluoxetine (Prozac).

Children aged 6 years are prescribed Sertraline while those aged 8 or over, are prescribed Fluvoxamine.

108
Q

How long does drug treatment for OCD last?

A

The treatment lasts between 12 to 16 weeks and SSRI’s work by inhibiting reuptake of serotonin which is released into the synapses from neurons by blocking receptor cells.

This increases serotonin levels and stimulation at the synapses alleviating OCD tendencies.

109
Q

Tricyclics - Exclusive for OCD

A

Tricyclics are also used exclusively for OCD and block the transportation mechanism which reabsorbs serotonin and noradrenaline into the pre-synaptic cell after firing.

As serotonin builds up in the synapse, this prolongs their activity and eases transmission of the next impulse.

110
Q

Anti-psychotic Drugs - lowers dopamine

A

Anti-psychotic drugs have also been used which aid in lowering dopamine levels as high levels have also been associated with the disorder.

These are normally given if SSRI’s do not prove effective due to their side effects.

111
Q

What do benzodiazepines do?

A

Drugs such as Benzodiazepines lower anxiety levels by slowing down activity in the central nervous system by enhancing GABA activity.

112
Q

Drug Therapy - Research Evidence

A

Research evidence shows drug therapies are effective compared to placebos in the treatment of OCD.

Pigott et al (1999) reviewed studies testing the effectiveness of drug therapies and concluded that SSRI’s have been consistently proven as effective in reducing OCD symptoms.

113
Q

Drug Therapy - Mask Symptoms

A

Drug therapies do not cure OCD which may make them inappropriate as they may simply mask the symptoms of a biological disorder rather than cure it.

Also, CBT has been shown to be effective in treating OCD for some patients and this treatment carries no risk of side-effects, unlike drug therapies.

Therefore CBT may be more appropriate to try first rather than drug therapies.

114
Q

Drug Therapy - Side Effects

A

Drug therapies such as SSRI’s carry side effects such as heightened levels of suicidal thinking, loss of sexual appetite, irritability, sleep disturbance, headaches and loss of appetite.

Dependent on the patient themselves, they may not always be appropriate, especially if sufferers of OCD have a history of depression or children who may struggle to cope.

115
Q

Drug Therapy - Quick and Convenient

A

Antidepressant drugs are cheap to manufacture, easy to administer and user-friendly when compared to psychological treatments such as CBT which can be time-consuming.

They are fast-acting and effective allowing individuals to manage their symptoms and lead normal lives. For these reasons of convenience alone they may be more appropriate for many patients who are unable to spare the time to try other more time-consuming options.