Psychology, Psychiatry and the Mind Flashcards
What factors contribute to us making a ‘free’ choice?
Genetic Previous experience Social experience Culture Neuroanatomy
What are agnosia?
Disorders which are usually the effect of a stroke.
You lose the ability to know that you’re damaged.
Individual knows they’ve had a stroke but not that they are hemiplegic and this can lead to accidents.
If you ask them about their disorders with will give inaccurate answers and believe that they can do everything fine, however if asked HOW they would do something they give very strange answers - they have lost some component of mind
How much information can a human store at any one time?
7 pieces - MILLER
This is very low, but there are things going on that we aren’t aware of - e.g. cocktail party effect. brain is receiving sensory information but can’t relay everything
What was the Wegner and Erskine experiment on free will?
asked people to do simple actions such as answer phone or pick up brick and ask if they did that or if it just happened.
Discovered that if you tell people to do something and then make them do it again when thinking very strongly about something else and feels less like you did it when thinking about the other thing
Why can it not possibly be us making the choice to do something?
In order for you to have a thought the brain has to change - but in order for the brain to change you have to have already changed the brain. So therefore cant possibly be you doing it, as the brain has changed already in order t have the thought.
What was the Libbet experiment on free will?
Was a simple experiment on this where got people to try and move one finger. Sitting in chair which minimised movement in front of a clock which made movements not every second but went round whole clock once a second. Scanned the brains of these people seeing which area was active in making the movement happen.
As there is the clock in front of them they can record the position of the clock when they have the intention to move the finger. Said to move the finger randomly when feel like it, record time on the clock and say whether it is right or lef.t data showed that in terms of the clock position and the intention to move the finger, the intention incurred after the brain area had already caused the movement.
Brain has done the action before you become aware of the fact that you’re going to do it. This is problem as tend to equate ourselves with consciousness, and although its still us doing it, it is just our unconscious mind. This makes its less intentional.
Humans aren’t vey comfortable with this idea. The experiment also found that after the brain area responsible for the movement occurs first, then become aware of the intention, and then the movement. May not have free will but said that you may be able to stop it as you become aware of it just before it happens.
The problem with this is that when he told people to try and do this in front of the clock (stop the movement) found that the inhibition area of the brain fires after the area of intention still before the area of awareness and then movement. This is hard for people to believe because none of us are aware of the area of the brain with the intention, only of the awareness and the movement.
Also showed that the brain messes around with the timings and puts some experiences before other experiences.
Brain only allows certain information form processing through to consciousness - know this because of things like the cocktail party effect.
What is an example of not having free will through the media?
‘Bad is stronger than good’ - why we are sensitised to notice bad things. Media is a good example of this - negativity catches our attention so hardly any stories in papers are positive. Why is it the case that we are programmed to notice the bad things - evolutionarily it is more important for us to notice these things as they are potential threats. This has developed to enable us to survive. The problem with the brain deciding what it will pay attention to is that this is clearly not free will.
About feelings accompanying actions (free will/agency)…
- Intentions, goals and plans are often experiences yet actions can still be unwilled as long as the individual reports they are
- Consciously willing an action requires that one feels they were consciously willing it
- Many and possibly most actions are accompanied by this feeling
- But not all…
Some that may not feel willed could include spending more or drinking more than you has anticipated. These things can be done without awareness.
What is alien hand syndrome?
- Often results following damage to middle of the frontal lobe or Corpus Callosum.
- Patients report one hand operates as if it has a mind of its own
- The ‘alien’ hand operates autonomously
- Eg a patient reports their right hand ‘automatically’ reaching out to pinch an attractive nurses bum while their left hand restrains it
- Another reports their left hand unbuttoning a shirt they are trying to put on
Corpus callosum gives 80-90% of the hemispheric communication. Can be severed to try and lessen the effects of epilepsy because means that the electrical storm occurs only in one hemisphere as it cant spread across.
Alien hand syndrome is when one hand does things you don’t want it to do, and the other will try and stop it - eg buttoning shirt and unbuttoning with the other, trying to hurt self and the other hand trying to restrain it, etc.
This tells us that if you sever the corpus callosum you get two centres of human agency which can act against each other. Its almost like you have two brains which can want two different things and can act independently. Some animals can do this and shut down half their brain - dolphins and wales can do this allowing it to sleep half the brain and carry on swimming without getting air and then sleeps the other one - otherwise will drown. Can function normally with half its brain shut down.
What is frontal syndrome?
Frontal syndrome - can lose 30% of front portion of your brain and still be fine. Phineas gage for example who got the railroad spike through his brain. He was fine but had personality change with lack of inhibition. Whatever stops you from doing things in every day life, they don’t have this. Any attractive stimulus they will take it up and not be able to stop themselves.
What are some examples of involuntariness?
- The alien hand appears to act involuntarily
- Other examples:
- Hypnosis - things are happening but ‘you’ are not doing them - Lynn et al 1990, roughly 15% of people are responsive to this with subgroups within this. Lift study is a more simple version of this where people turn around to go along with the crowd, these people are more likely to be hypnotisable
- Everyday examples - Dostoyevsky’s protagonists, wrote about characters who constantly acted against their own best interest.
- Experiment - try another persons hands
Not your hand experiment - put someone elses arms through so looks like your arms and make them do simple actions for a few minutes and then at the end pull elastic band on their wrist and you feel the pain.
Phantom itch syndrome and phantom limb.
Shows that something even as radically as personal as pain isn’t necessarily your own. If we had an MRI scanner would see your brain acting exactly as if it was happening to you.
What are critical distinctions of agency?
Feeling, not feelings as doing
Doing, voluntary action, automatism
Not doing, illusion of control, normalnaction
How is will also a force?
- Will refers not only to an experience but can also be conceptualised as a force
- For example when e restrain ourselves we experience a perception of out consciousness as ‘causing’ the control of action
- While a dog may be brown or black we seem to have a power within that ‘causes’ our actions, but does it?
What is causality?
Hume - causality is not a property of object or things we cannot see causality we infer it
It is a judgement and a decision. we like causality because it allows us to explain the world around us.
How do we infer causality?
- Priority - causal events precede their effects in time (eg turning on the lights - Michotte 1963)
- Priority helps humans perceive causality as thoughts usually precede actions
- Consistency - the resulting action should be consistent with the prior motion or action-in the human terms the prior thought is consistent with the result
- Exclusivity -we discount causal inference if other potential causes are apparent (Kelly 1972) -
Usually don’t give people the benefit of the doubt when they do something odd -infer that is the characteristics of that person not the situation (fundamental attribution error?) which is useful to us because if say it’s the situation then cant say much about the person, but if say it’s the person we are able to predict and base our behaviour on what they are likely to be like.Milgram experiment with the electric shocks in other room and ~68% of normal people went right up to ‘chance of death’ due to command, the situation, power. The situation is a very powerful determinant of behaviour.
Situation is actually a better predictor of behaviour, but human prefer to belief it is more based on personality because we want to know ourselves and not feel that we change with situations.
What is human causal agency?
Causal agents ar goal seeking - we often feel a desire to describe phenomena in goal directed terms (Heider and Simmel video of shapes moving).
We understand the world around us by goal based terms - where things are headed. We look for goals within things.
How do babies see causality?
Neonates have nothing innate to help them with causal ascriptions. Good evidence now that causal ascription is a learned behaviour and occurs between 2 and 3 years of age. Found this out by getting a baby with a screen in front of them and roll a tennis ball seen, behind the screen, then seen again. An adult would look to where the ball would come out because they understand that it will continue on its trajectory, but a baby wont look for it because they don’t understand that the rolling causes it to continue its motion and it isn’t just gone behind the screen. There is a moment of shock from the baby when they see it the other side of the screen. This is how they learn about causality.
What are the two explanatory systems that humans have?
- one for minds
- one for everything else
What did Spinoza say about agency and volition?
“men are mistaken in thinking themselves free; their opinion is made up of consciousness of their own actions, ignorance of the causes by which they are determined. Their idea of freedom, therefore, is simply their ignorance of any cause for their actions” (Spinoza 1677)
What is modularity of mind?
there are a lot of perceptual/visual illusions that if shown you see it. If you then show the visual illusion again and know it is wrong then it doesn’t change how you see it. That is why the mind is said by Fodor to be impenetrable (eg blind spots and brain filling in the gaps)
What did Wegner and Wheatley say about free will (1999)?
“people experience conscious will when they interpret their own thought as the cause of their action”
We think of ourselves as causal when:
- We experience relevant thoughts of the act in advance of the act and infer our mental processes have 'caused' the act - Third variable problem?
What are quantitative methods?
Measurements (numeric, quantification)
Using scales (e.g. for pain but can be problematic in humans as subjective)
induces PREDICTABILITY - this is often the goal of science (allows survival)
makes science apolitical, valueless and unbiased
What are qualitative methods?
Describing, understanding a phenomena.
Understanding what it’s like to be or to have.
The EXPERIENCE
This is also SUBJECTIVE but qualitative takes much more account of individual perceptions than quantitative
What are the levels of measurement?
(LOW/MINIMUM/DISCRETE) 1. Nominal 2. Ordinal 3. Interval 4. Ratio (HIGH/MAXIMUM/CONTINUOUS)
Only higher levels allow prediction
What is NOMINAL measurement?
1 (lowest)
categorical, but sometimes no categorical/quantitative difference between categories. Only allows for descriptive stats
What is ORDINAL measurement?
2
still categorical but starting to be able to say one is bigger than another eg political voting behaviour. There may not be units between points.
What is INTERVAL measurement?
3
no zero eg age or height which we can measure and find infinite divisions and we know there is always the same difference between intervals (in a cm for example)
What is RATIO measurement?
4 (highest)
a continuous scale where there is an absolute zero e.g. velocity.
About qualitative types of procedure
Historically, qualitative information has been missed.
Observation - ask them to describe their experience eg with colorectal cancer (very common, 3rd most
Likely cause of death with ~10,000 deaths in UK per year).
–> If we can understand what it’s like to have something then their care can be improved - what they would have liked to happen.
Want as big n as possible in quantitative BUT in qualitative we want a representative but not too huge as analysis takes a very long time –> 6-20 subjects average for qualitative
Science protects objectivity. Science sometimes finds answers which aren’t pleasant or beautiful (eg evolution) - people don’t always like it.
Science often wants truth which can be unpleasant - eg a diagnosis (colorectal cancer!) - but this is a truth.
Humanity has sought to protect this method/science –> apolitical, valueless, unbiased.
For qualitative research:
- Need 6-20 subjects - Can conduct interviews - Input is always verbal or textual (interview is verbal [recordings, transcription, thematic analysis], diaries or facebook is textual)
What are types of qualitative methods?
- discourse analysis
- thematic content analysis
- grounded theory
What is discourse analysis?
Qualitative method
Much more attentive to the type of language they used than the meaning of it. How they use language is very important and it is thought to say something about their experiences and beliefs - eg swearwords in languages and cultures (fewest in Japanese and most in Russian).
WHY does this vary culturally? Believed that language reflects the culture - needed to develop a language to reflect their experience - “language encodes culture”
What is thematic content analysis?
Qualitative method
This is the process of extracting themes and immersing oneself in the data trying to understand as if it were yourself whilst trying not to bring bias and also you are not them so this is very hard. They are a different person with different subjective views, which can never be overcome. No matter what you do no one will ever fully understand you. Psychologists have said because of this that you are truly alone from other subjects and objects. One day we may be able to upload our self and our experiences to some kind of machine so it wouldn’t be lost when we die and could be explored by others. We can do this yet so instead we do qualitative research.
If 200-300 themes are extracted from data then we look for the common recurring ones until we have 5-10 overarching themes which encompass all of the 300 themes. Then a short paragraph would be constructed encapsulating what each theme is/means, with as much of what they actually said as possible. We can quantify the ones which are the most mentioned - what’s not there is as important as what is there.
What is grounded theory?
Qualitative method
Start with theoretical premise which is dangerous in qualitative because you may interpret their subjectivity through your theoretical framework eg holding doors - kind or sexist? A priori theory is where you make a theory before hand and this takes certain stand points eg what it’s like to be a woman in the 21st century.
What is scientific tenure?
Tenure was originally developed to allow scientists to ask these questions without consequences in order to protect science.
What are the types of statistics?
- descriptive stats
- non-parametric stats
- parametric stats
What are examples of descriptive stats?
graphs, bar charts, mean, mode, median
Why are non-parametric stats?
can be used if data can be ranked but is less accurate (e.g. chi squared)
What are parametric stats?
T-tests, ANOVE (analysis of variance - amount of variability within your data)
Eg mean IQ in males vs females. Mean IQ globally for both makes and females is 1–, but really doesn’t stand world wide as female education is rare in many places. If it is measured in the right way in the right places then you can get the same value.
What does a T-test show?
- Nothing to do with relationship
- To do with variance within 2 groups can not do it with more than 2 groups
- Tells you if there’s a significant different between the two values
- Continuous data
- Looking for a difference in the score on the two groups
- Eg statistically comparing males and females IQ
P value = 0.05 –> means that if the value is below 0.05 there is a 95% certainty that the results are not due to chance. If the value is set at 0.01 then there is a 99% certainty.
What are experiments?
Experiment = GOLD STANDARD!
1. Manipulation of one variable = independent variable 2. Look for effect on dependent variable - continuous 3. Random allocation to groups
Finds CAUSALITY which is the only way can establish predictability. Psychiatrically we cant give someone a disorder so can’t really do experiments, so cant really establish causation. To get around this we use correlational data. This can’t establish causality. Instead finds a linear relationship between two variables - cant establish causality with this.
When there is a linear relationship between 2 variables we like to say that one causes the other.
What are the problems with correlation?
- We like to infer causation because this allows predictability (previously would help survival)
- 3rd variable problem - when another variable that you’re not measuring causes both fo the other things - eg hot weather on ice cream sales and drowning
- Partial correlation = correlation between 2 variables while controlling for another variable. If you take out the third variable then the one youre controlling for then the correlation drops to ZERO and this shows the relationship between the two isnt real and was caused entirely by the third variable - a spurious relationship.
- 3rd variable problem - when another variable that you’re not measuring causes both fo the other things - eg hot weather on ice cream sales and drowning
Measurements at one time point = cross sectional
Measurements at several time points = longitudinal or cohort
How might experiments be done in a drug trial?
Three groups (control, old drug, new drug) given IQ test and measure how long they take to solve it to find speed of processing which is the dependent variable. One month after the drug is given you would retest and compare the mean of the two set of results. We would predict that the control group stays the same, the rug currently used would have some effect and the new proposed drug would have more of an effect than that.
To test for significance we use analysis of variants = for more than two groups
If three groups, for example, then the results of analysis of variance gives an f value which states whether there is significance at p=0.05. The problem is that it doesn’t show WHICH groups are significant from which others.
We then do post-hoc tests.
What are post-hoc tests?
These are only done if significance is found - otherwise it isn’t worth it. These are types of t-tests between the groups, between each possible pairing of the three groups.
In total at this point you would have done 3 tests –> CAPITALISATION ON CHANCE which says that the more tests you do, the more likely you are to find something significant by chance. This has to be corrected for statistically by setting the p value lower making it harder for something to be accepted as significant.
What is the Bonferroni correction?
- Work out how many tests you’re doing (eg = 4), and you want the p value to be 0.05
- Divide the p vale by the number of tests
- Eg 0.05/4 = 0.0125 0.013 - which is now your new p value
This is how you account for capitalisation on chance. The problem with this however is that it will approach zero (0.001) with the more tests you do, eventually making it near impossible to find anything significant.
What is multiple regression?
If we want to predict a variable fro 2 other variables we do this by multiple regression. We predict one variable from several others - needs interval or ratio level of measurement. Gives three axes with a 3D plane on the graph. (A line of best fit on a scatter grpah shows correlation aka simple regression).
N dimensional space - can be 5 places.
Multiple regression allows us to make predictions on things such as likelihood of getting certain diseases.
What is parsimony?
Aparsimoniousmodel is a model that accomplishes a desired level of explanation or prediction with as few predictor variables as possible. For model evaluation there are different methods depending on what you want to know.
–> The simplest explanation is the best.
How are clinical interviews used in psychological investigations?
Another form of test used in psychiatry (less so in psychology) is clinical interviews and also get a score for diagnosis.
Clinical interviews are not completely subjective. Questions are asked which map on to a specific diagnosis. We can also do single case studies when you find someone with a conditions and describe it in detail - especially done when the field/condition is new.
What are the demographics of a psychological history?
- Name
- Age
- Gender
- Employment
- Relationship status
- Children
- [Summary of diagnoses]
What are the presenting circumstances of a psychological history?
- Circumstances of presentation
- Context of presentation
- Timeline of problems
- Precipitating events
- Perception of problems
Under what circumstances could someone come to a psychiatrist?
- under a section 136 - when someone is brought in from the public place by a police officer for example, someone who was acting in a manner suggesting they may be suffering from a mental disorder and in need of care and control
- Referred by their GP
- Present to A+E in crisis with suicidal feelings or following self harm or suicide attempts
- Referral from other specialists
Timeline of problems can be years apart.
What are the essential areas of affective symptoms on a psychological history?
- core symptoms
- somatic
- psychology
What are the core symptoms in history taking?
- Low mood - feeling unhappy, sad, down.
- Poor energy/fatigue
- Anhedonia - inability to feel pleasure
What are the somatic symptoms in a psychological history?
- Poor appetite (or increased)
- Weight loss (or increased)
- Insomnia (or hypersomnia) - typically wake up tired, unrefreshed, but unable to return to sleep
- Loss of libido - women is mainly loss of interest, in men sexual dysfunction can be caused by depression or by antidepressants, but in general the effect on libido of depression is mainly loss of interest rather than erectile dysfunction/vaginal dryness.
- Poor concentration/short-term memory
What are the psychological symptoms in a psychological history?
- Helplessness
- Hopelessness
- Worthlessness
- Guilt
- Suicidal thoughts - have to ask about it. Direct approach often appreciated by patients, talking about it openly rather than making it a taboo subject to encourage people to give honest answers.
- [psychotic]
How can we split symptoms?
Can split symptoms into affective and psychotic. Always want to ask about related symptoms, not just the initially obvious ones.
Affective means to do with mood - affect is like saying the weather specifically today, but mood is saying that it is January so what it is like normally (in context)
A normal mood should fluctuate throughout the day/week and with events.
What is needed to diagnose depression?
For depression has to be consistent low mood, most of the day, most of the days, for two weeks or more. Typically lowest in the mornings and evenings, more severe more the time.
By ICD-10 need 2 core and 2 associated for mild depression.
Moderate: 2 core and 4 associated
Severe: 3 core and 6 associated
Psychotic: must be severe + psychosis
What needs to be asked about suicide in a history?
- Nature and duration of any suicidal thoughts
- Intent
- Plans
- Previous suicidal behaviour
- Deliberate self harm
Intent - many people have suicidal fantasies but wouldn’t ever act on it
What is asked about psychosis in history taking?
- Open questioning - have you had any other unusual experiences recently, like seeing or hearing things that other people cant that shouldn’t be there. Any other worries that we haven’t talked about, like people trying to harm you or send you special messages?
- Delusions
- Hallucinations
What other areas are asked about in a history?
- Anxiety - becomes pathological when it is of frequency or intensity that it interferes with your ability to function (socially, occupationally, etc)
- Gender identity
- ADHD - hyperactivity symptoms and inattention symptoms
- Eating disorders
- Paraphilias - a condition characterized by abnormal sexual desires, typically involving extreme or dangerous activities
- PTSD
- Memory
- Substance use
Past psychiatric history
- Previous diagnoses
- Previous hospitalisations
- Previous treatments
Past medical history
- Comorbid medical problems
- Particularly metabolic disorders
- Recent blood tests
- Head injuries/CNS surgery
Social history
- Current home circumstances
- People at home, relations and ages
- PoC - package of care
- Employment/income
- Financial situation
Family history
- history of illnesses in family
- include relation and age
Drug history
- psychoactive medication
- others
- allergies
Substance history
- alcohol
- tobacco
- recreational drugs
- OTC
- herbal treatments
Personal history
- life story!
- psychosexual history - current relationships, first relationship, pattern of relationships, gender preferences, previous marriages, etc
Mental state examination
- Appearance
- Behaviour
- Speech
- Mood
- Thought
- Perception
- Cognition
- Insight
About appearance in the mental state examination
- Dress
- Self-care
- Age
- Ethnicity
- Gender
Tells you a bit about how long the problems may have been there for.
Manic can also be seen through dress - bright colours etc
About behaviour in the mental stat examination
- Appropriateness of behaviour
- Eye contact
- Rapport
- Engagement
- Any particular abnormal movements, stereotyped movements, mannerisms, tics etc
About speech in the mental state examination
- Rate
- Rhythm
- Tone
- Volume
- [content] - eg frequent swearing
- Other abnormalities
About mood in the mental state examination
- Subjective - how they describe their mood
- Objective - how we objectively assess someone’s mood
- Affect - reactive affect, depending on what you’re talking about how they react
About thought in the mental state examination
- form
- content
- remember suicide!
About perception in the mental state examination
- Hallucinations
- Other abnormalities of perception
- Reported or apparent from observation
About cognition in the mental state examination
- Not always needed to formally assess
- [Orientation]
- Other formalised
- AMTS
- MMSE
- ACE
About insight in the mental state examination
- Insight into condition
- Extent of condition
- Attribution of symptoms
What is diagnosis?
The identification of the nature of an illness or other problem by examination of the symptoms.
DIFFERENTIAL DIAGNOSIS: a possible list of diagnoses pertaining to a specific symptom/set of symptoms
DIAGNOSTIC CRITERIA: a list of criteria or algorithm required to demonstrate a specific diagnosis
Why do we need diagnosis?
- To enable following treatment protocol and rehabilitation strategies including national guidelines eg NICE guidelines
- To enable patient education
- To access services
- To aid in focusing research questions
To be able to pertain to treatment protocol, patient education so they can look up what they have and come back with questions, research
How do we go about making a diagnosis?
• Shortness of breath: differential diagnosis: - Asthma - COPD - Pneumonia - PE - Pneumothorax - Malignancy - Interstitial lung disease - Pulmonary oedema • Patient history • Examination • Investigations - Peak flow - Lung function tests - ABG - Chest x-ray - Chest CT - VQ scan - Bronchoscopy - Lung biopsy
Making a psychiatric diagnosis…
• History • Mental state examination • Physical examination • Investigations - Blood tests - Imaging - Psychological questionnaires - Other investigations eg EEG, LP • Collateral history, MDT input • Longitudinal documentation
What is meant by the mental state examination providing longitudinal documentation?
Provides longitudinal documentation, like a photograph which can be lined up to see if the patient is getting better or worse. Only thi
What are the possible differential diagnoses in psychiatry e.g. from hearing voices?
- Side effects of L-Dopa
- Psychosis associated with tumour
- Psychosis associated with infection (eg HIV)
- Psychosis associated with autoimmune disease
- Drug-induced psychosis (substances)
- Schizophrenia
- Mania
- Psychotic depression
- Post-partum psychosis
- Emotionally Unstable Borderline Personality disorder
How can we cut down the differential diagnosis of hearing voices with investigations?
- Side effects of L-Dopa - medication review
- Psychosis associated with tumour - CT/MRI head
- Psychosis associated with infection (eg HIV) - blood test, CSF test
- Psychosis associated with autoimmune disease - blood test
- Drug-induced psychosis (substances) - urine drug screen
- Schizophrenia
- Mania
- Psychotic depression
- Post-partum psychosis - has the patient given brith recently?
- Emotionally Unstable Borderline Personality disorder
We don’t do all of these tests on everyone - needs to be some kind of indication
What are the different methods of diagnosis?
ICD 10
DSM V
Formulation
About ICD 10 diagnosis
• 10th revision of the international statistical classification of diseases and related health problems listed by WHO
• >14,400 codes for symptoms, disease, social circumstances
• Work on ICD-10 began in 1983, completed in 1992, though it is now on it’s 5th edition
• Awaiting ICD-11…
• Used in 27 countries for resource allocation in healthcare
• 110 countries use it for statistical purposes
• Notable change/inclusion:
- Homosexuality removed from ICD-10 after 1992
- Gender identity disorder - F64.0
- Fetishism still in the ICD-10 (F65) (F56.1: transvestitism, F65.51: masochism, F65.52: sadism)
About DSM V diagnosis
- Diagnostic and statistical manual of mental disorders
- Published by American Psychiatric Association
- Classification of mental disorders based on standard criteria
- DSM V - 2013
- Used in the US
- On-going issues around credibility, possible cultural bias - in 1973 5854 for removing homosexuality vs 3810 against!
What is Axis I of DSM V diagnosis?
Principle disorder that needs immediate attention e.g. major depressive episode, acute psychotic episode in schizophrenia
What is Axis II of DSM V diagnosis?
Personality disorders; developmental disorders, learning disability that may shape Axis I
What is Axis III of DSM V diagnosis?
Medical or neurological problems relevant to psychiatric history e.g. asthma may be confused with acute panic attacks
What is Axis IV of DSM V diagnosis?
Major psychosocial stressors e.g. family bereavement, divorce, loss of employment
Wha tis Axis V of DSM V diagnosis?
level of function; global assessment of function, out of 100
What is the GAF scale
move up the scale with better levels of functioning - 0-100
What is formulation?
combination of predisposing, precipitating and perpetuating self, family and environmental factors
Formulation is the process of making sense of a person’s difficulties in the context of their relationships, social circumstances, life events, and the sense that they have made of them. It is a bit like a personal story or narrative that a psychologist or other professional draws up with an individual and, in some cases, their family and carers.
When does something become a psychiatric disease?
Disease:
i. Discovered the causes ii. Understood the pathogenesis iii. Comprehensively described the clinical picture and the reaction to treatment iv. Measured the natural history
None of the illnesses with which psychiatrists deal satisfies these criteria –> would it then become neurology
What are the positives to diagnosis?
- Education and health care plan (EHCP)
- Social care package
- Access to community activities
- DLA
- Freedom pass
- Free prescription
- Housing aid
- Help with CV/job-seeking
Means that the patient gets access to these things. EHCP - teaching assistant for example.
What is behavioural consistency?
Eg dress code (a casual dress sense causes people to take a person less seriously and less professionally).
Characteristics within behavioural consistency can reflect character or context, and we should ask which of these is more likely to determine the factor, such as dress sense. We ask ourselves this both consciously and subconsciously.
Dress code is mostly due to the context of the situation. It is hard for us to believe that this is the case for many factors because it means we aren’t in charge of the way we are - it is due to something external.
Science wants to predict, and so do humans. People acting dependently on context makes prediction hard as people may change depending on the environment.
What is personality?
Traits and characteristics that don’t vary across time and situation.
Eg introversion and extraversion are known to be more biological as can be measured from birth - crying baby or quiet?
What is personality disorder?
Behaviour that consistently transcends social norms.
- ICD10 and DSMIV/V give 9-11 forms - Psychopath is an example (aka 'dangerous and severe PD' or 'antisocial PD' - 4-10% (~8%) of people are affected by a form of PD - This is the only psychiatric disorder diagnosed on the basis of behaviour - 1% of the worlds population is schizophrenic therefore much more likely to encounter PD
What is the critical period for human brain plasticity?
In humans the critical period is 0-12 years of age of maximal brain plasticity - personality sort of solidifies at aged 12. Biological maturity Is reached around 21-23 years old and it is a decline from then on. This is shown in increasing defective reproductive rates as age increased. The current social norms are jut under 30 for marriage and 31 for having children. >35 is when a real risk of birth defect begins to occur.
We are always looking for what is ‘normal’ in society, culture, those around you from the day you are born, and looking at how to behave.
- Eg if both parents smoke then ~80% likelihood a child will too, but I the whole country smoking rates are ~20% - Eg twin studies
What are the 5 stable characteristics of personality? The Big Five.
- Openness to experience
- Conscientiousness
- Extraversion
- Agreeableness
- Neuroticism (propensity to experience negative feelings on a regular basis, but useful in spotting threat)
how does an abusive childhood impact personality?
An abusive childhood heavily impacts personality, with 92% of people with a PD having suffered abuse. Society diagnosis and treatment of disorders such as schizophrenia do vary culturally - eg might be made leader of a tribe in some places as might be thought to be communicating with the spirit world.
If someone suffers from a PD and never acts within norms then there are two options –> they don’t know, or they don’t care.
What are some factors that contribute to personality that aren’t included in the big five?
- Time keeping
- Spending behaviour
- Humour
- Consistent negativity
- Proclivity to opposite/same sex (obsession)
- Stubbornness
- Aggression/violence
What are contributing factors to personality?
Genetics IQ alcohol Appearance Sex Childhood Morality Society Religion gender sex attitudes personality peer group diet
How do genetics contribute to personality?
- 50-54% of personality is inherited from parents.
- Means we are not as unique as we might like to think.
- Intelligence is >50% inherited.
How does IQ contribute to personality?
• Data shows that as IQ increases, length of life increases too
• Less likely to engage in risky behaviour
• Not just for that reason however, the main reason is nothing to do with human behaviour
The latest theory is that IQ is a very rough measure of how well you are put together as a biological machine (this doesn’t necessarily mean better though!
How does alcohol contribute to personality?
30% of all UK hospital admissions every year (mostly on Friday and Saturday night). Society and culture affects this with difference age limits and rules.
How does appearance contribute to personality?
better and younger looking get shorter sentences, more success, more jobs etc.
We tend to judge attractive people are more intelligent, which there may be a link with. Similarly to IQ, attractiveness may also be a rough measure of how well the biological machine is put together.
How does childhood contribute to personality?
experiences, nutrition, oxygen, in the womb etc
how does society contribute to personality?
- Laws
- Convention (subjective)
- No objective reason for things being right or wrong, if was for an objective reason then go to either religion or morality
How does religion contribute to personality?
religions make us quite predictable as they often enforce a code of conduct
How does our peer group contribute to personality?
social norms - behaving in socially acceptable ways
How might diet contribute to personality?
different foods affecting health?
What is personality?
- Traits that are stable across time and situations
- But beware: most often the situation one finds oneself in is a better determinant of eventual behaviour
- Fundamental attribution error = individuals tendency to overemphasise internal characteristics in explaining behaviour rather than external factors. Converse = actor-observer bias which people overestimate role of situation and underemphasise personality
- Milgram experiments 1960’s and 1970’s
Milgram showed that if you take normal people and put them in a situation where you demand they kill someone else, 70% of the did (electric shock experiments) - and most almost did.
This is an example of how you can take a normal individual and make them do something which society says is horrific, quite easily.
People are very good at doing bad things when they have been told to do it and so therefore they can shift the blame to someone else. This makes them a lot more likely to do it. This is known as the fundamental attribution error.
Actor observer bias is when people put more on the situation. Real world examples of this include bystander apathy - when you see something bad happening but don’t do anything about it, eg there was a rape in Central Park New York and there was over 150 witnesses yet no one did anything about it, they just watched. There is a phenomenon that the more people that watch something, the less likely any one person is to do something about it. Everyone diffuses the responsibility, and doesn’t feel individually responsible for what they are witnessing. Assume that someone else will do about it, and when all assume the same thing, no one does anything about it at all.
Who said that 50% of variations in the 5 dimensions of personality are determined by genetics?
Any, Lively and Vermon 1996
the five were derived through factor analysis. Started off with 150-160 criteria that were stable over time and didn’t vary between individuals. Then did factor analysis which tells you if they are all statistically different, or if there are categories of higher order things which could explain a number of other things and these five were found to be able to explain all of the others.
• But beware what is inherited are more likely to be limits around set points rather than absolute characteristics
The environment clearly plays a role
What are the 5 dimensions of personality?
John and Srivastava 1999
OCEAN
Openness Conscientiousness Extraversion Agreeableness Neuroticism
What is Openness?
willingness to try new things, ‘openness to experience’, like novelty, get bored easily
What is conscientiousness?
Hardworking, committed, plan, ensure goals
What is extraversion?
Sociability, more likely to be happier on the whole, less likely to get psychologically sick, lots of social contact which is the most protective and beneficial ting humans can do (introverts risk psychopathologies of certain forms but also more likely to come up with new ideas through focus etc)
What is agreeableness?
The extent to how agreeable you are, being a person that people want to be around, helpfulness, if you help other people lessen your own likelihood of survival in evolutionary terms (eg giving food away), makes sense in kin but not in non-kin, in modern day agreeableness is useful as need to get on with other people in order to get by (non-agreeableness has advantages in someone who you need to go and get something serious from someone else eg politicians)
What is neuroticism?
Propensity to feel negative, not necessarily pathological, means you regularly feel quite off, don’t feel happy very often, but there is nothing wrong with you. Could be useful in terms of recognising threat, may be able to better understand other people’s pain and unhappiness, in evolutionary terms might have been made a doctor/healer/spiritual teacher/watchman
What is meant by the 5 dimensions of personality being orthogonal?
Modern research is showing they are ‘orthogonal’, particularly neuroticism - can be extremely positive and extremely negative at the same time. One does not preclude the other - one makes the other less likely but there are still instances where you can have both.
What is meant by the 5 dimensions of personality being bipolar factors?
These are all known as bipolar factors, eg agreeable, disagreeable and a midpoint where you are neither. There is a scale from one end to the other.
How do we test personality?
- Almost invariably assessed by questionnaire
- Based on research some more narrow traits have been identified but factor analysis suggests the big 5 encompass most fo the information
- Big 5 inventory where do you fall?
- Alternative models - Jung
Does personality predict important outcomes?
What are important outcomes?
- Individual outcomes - that can be manifested by an individual outside of a social context. Eg. Physical health, psychopathology, happiness - Interpersonal outcomes - involve other individuals and it generally matters who the other is eg forming and maintaining quality relationships - Social/institutional - more impersonal, organisational, societal=level processes involving interactions with more generalised others eg work satisfaction and performance
If we ask what does personality predict, what do we find? We separate outcomes into three (individual, interpersonal and social).
Extraverts live slightly longer
More conscientious live longer
Neuroticism more likely to become pathological so might get some form of mental health issue
Highly disagreeable more likely to die
Mental illness has advantages as evolution hasn’t got rid of it, 25% of people have some sort of mental health problem, in very influential/dynamic/world changing people = more than 50% of them had something. Mental illness may influence genius/creativity etc.
How does personality affect happiness and subjective well being?
- Happiness and subjective wellbeing
- SWB includes a cognitive component/judgement of one’s life satisfaction (Diener et al. 1985), and an affective component that includes
- And an emotional component comprising of the experience of positive and absence of the negative emotions (Larsen 2000)
- Importantly personality dispositions are strong predictors of most components of Subjective Well Being (SWB) (see Diener and Lucas 1999)
- Furthermore, demographic factors, including age, sex, marital status, employment, social class, and culture, are only weakly to moderately related SWB (Diener et al. 1999, Ryan and deci 2001)
- In detail - people high in extraversion and low in neuroticism tend to see events and situations in a more positive light and tend to discount opportunities that are not available to them
- Differences in conscientiousness, agreeableness and openness to experience are less strongly and consistently associated with SWB (Diener and Lucas 1999)
- Personality traits have a stable effect on health and longevity (Caspi et al 2005)
- Longevity studies show - positive emotionality (extraversion) and conscientiousness predict longer lives (Danner et al 2001, Friedman et al 1995), and hostility (low agreeableness) predicts poorer physical health (eg cardiovascular health(
- Neuroticism and health and longevity is more complex, some studies suggest an association between neuroticism and increased disease risk, whereas other show associations with illness behaviour only (Smith and Spiro 2002)
- Whether personality has a causal role remains unclear (Caspi et al 2005)
- Goodwin and Friedman (2006)
- Examined 5 factor personality and health in 3032 representative North Americans
- Conscientiousness is associated with reduced physical and mental health risk
- Neuroticism is associated with increased physical and mental health risk
- Inconsistent effects of other personality characteristics
Personality more affects how happy you are than any factors such as employment, marriage, age, social class, sex. This is interesting as society teaches us that these things make you happy, but actually your character which we don’t choose is what makes you happy.
Hedonic Treadmill - seeking pleasure, but this is bad news in humans as it means that (illustrated by human temperature regualtion) human temp is 37*, if we get any hotter we start sweating caused by homeostasis, and this also occurs for happiness. Which is why happiness is set largely by the factors making up your personality.
The other factors do make a difference and can help you to move towards a different state, but you are fighting against homeostasis. This is why unhappy lottery winners are happier for a while, but after 6 months to a year their happiness levels falls back down as that is what their personality is telling them to do.
Neuroticism is related to mental health illnesses - but the relationship is messy
The problem with all of this is that the data is correlational so we cant confer causality. Does your personality cause you to have happiness or does happiness cause you to have a certain personality? Longitudinally we have seen that it is more that personality later maps onto your happiness. We know this because a lot of these things operate from birth and we can track people over time.
Self selection of environment - when we reach biological maturity we select our own environment partially on the basis of personality. For example might choose a job which suits you - selecting an environment which agrees with your personality. This can be a problem because this might mean that although we said that personality doesn’t change over time and situation, maybe its because we are choosing for it not to. This means that we won’t change.
Studies have been done on this, eg conscientiousness associated with reduced mental health risk.
What are the interpersonal effects of personality?
The strongest personality links shown for empathy, ie a combination of extraversion and agreeableness, and emotional regulation, best predicted by low neuroticism
Romantic relationships - neuroticism and low agreeableness consistently shown to be predictors of negative relationship outcomes eg relationship dissatisfaction (Karney and Bradbury 1995)
Certain pathologies make this not possible (understanding how other people see the world) eg psychopaths because they don’t have the mental models. About 10-15% of people can be ‘naughty’ and it’s useful because boundaries are pushed and things advance when things are questioned.
As psychopaths struggle with empathy, they struggle also with guilt, remorse and regret - this significantly affects their behaviour.
Extroversion and agreeableness are linked to emotional regulation.
What are the social effects of personality?
- Barrick et al 2003, larson et al 2002
- Examined using meta analysis relations between personality traits and occupational types
- Extraversion was related to social and enterprising occupational
- Agreeableness to social interests
- Openness to investigative and artistic interests
- Neuroticism not related to any occupational interest
- Barrick et al 2001
- Conscientiousness predicts work performance across occupations
- Ways, in all included occupations. Smaller, though nearly as broad, effects were found for
- Extraversion and emotional stability - smaller effects but are important for some, though not all occupational groups
- Only weak and narrow effects for agreeableness and openness were identified
- Agreeableness related to job performance when a teamwork is important
- Best known occupation-specific measure of job performance is grade point average (GPA) in the US. But in the UK school leaving exams are related to later work outcomes and- there is a positive relation between GPA and conscientiousness (Paunonen 2003)
- Years o education, is related to intellect, or openness (Goldberg et al 1998)
- Thoresen et al 2003 extraversion and emotional stability associated with jo satisfaction and organisational commitment. Furthermore, they are negatively related to a wish to change jobs and burnout.
- Conscientiousness best predicts how well one performs at work
- But extraversion and emotional stability are more important for understanding how one feels about work
- Roberts et al 2003
- Emotional stability (negative emotion) is strongly related to financial security
- Agreeableness (positive emotion-communion) related to occupational attainment
- Resource based power and work involvement predicted by extraversion (positive emotion-agency)
- Low conscientiousness consistently associated with various aspects of criminal/antisocial actions. And is related to adolescent behaviour problems (Shiner et al 2002), deviance and suicide attempts (Verona et al 2001)
- Low agreeableness, and low conscientiousness associated with substance abuse (Walton and Roberts 2004)
This says that your personality leads you to certain environments.
Dichotomy between affect and … - how good you are at your job and how you feel about it
There has been seen to be combinations/patterns of personality traits which lead to things such as spending habits.
Occupational attainment is linked to being more agreeable than actually being any better at your job.
What is the relationship between personality and age?
- Stability or change?
- Roberts, walton and viechtbauer 2006
- With age conscientiousness and emotional stability increase especially between 20 and 40
- Openness decreases across the lifespan
- Agreeableness rises
- These changes are independent of sex
- Carstensen - individuals change their social networks - also positivity effect
- Diary studies
Personality kid of sets the limit on how happy you can be, which is basically what we all want. Maybe chasing after happiness is just down to convention.
Also sets limits around the quality of engagement you might have with relationships with people etc
Rates of various disorders (eg PTSD) go down with age. Age is protective because your social network is adjusted, a lot of factors which make sense when young don’t make sense when you’re old, such as chasing things which pay off in the future as opposed to immediate results. Older people don’t chase after future satisfaction as they have to wait, staying unhappy in the moment so that you can have more pleasure in the future. An older person doesn’t do that as they might not be there in 6 months time, they do whatver they want to do in order to make them happy right now.
What are extreme stressors?
Extreme stressors are events that are potentially traumatic of that involve severe loss.
After exposure, most people will experience distress but will not develop a condition that needs clinical management
This might lead to PTSD
Examples - abuse, bereavement, car accidents - things that are out of the ordinary.
Most people have coping mechanisms but experience some distress.
Palpitations were almost a universal stress response to extreme situations such as earthquakes - heart very fast as though going to have a heart attack and very scared.
Cortisol firing, increasing HR, fight or flight. Following disasters etc this keeps occurring because baseline stress level is so high. This isn’t PTSD, it is normal stress for someone who has been in that situation - these people need to know that this is normal and they are doing well - positive coping mechanisms.
What are two types of conditions following exposure to extreme stressors?
Problems and disorders that are more likely to occur after exposure to extreme stressors but that also occur in the absence of such exposure:
- Depressive disorder - Psychosis - Behavioural disorders - Alcohol use and drug disorder - Self-harm/suicide - Other significant emotional or medically unexplained complaints
Problems and disorders that require exposure to extreme stressors:
- Significant symptoms of acute stress
- Post-traumatic stress disorder (PTSD)
- Grief and prolonged grief disorder
These often occur in combination with other conditions.
Grief is normal but prolonged grief isn’t eg a partner or a child
Some people never recover from it
Explain that conditions following exposure to extreme stressors fall into two categories.
Explain that the biggest mistake many clinicians make when treating people after trauma exposure is to only think of conditions specifically related to stress. Explain that many clinicians forget that many mental disorders (e.g. all those listed on the slide) are more common after extreme stress.
Emphasize the importance of assessing other possible mhGAP conditions as appropriate.
Prevalence of mental illness after humanitarian disaster
Prevalence almost doubles after a disaster and normal distress is everywhere
Symptoms of acute stress
- After recent exposure to potentially traumatic events, people’s reactions tend to be diverse.
- symptoms of acute stress (within one month of the traumatic event) cover a wide range of symptoms occurring in both adults and children, such as:
- feeling tearful, frightened, angry or guilty
- jumpiness or difficulty sleeping, nightmares or continually replaying the event in one’s mind
- physical reactions (eg hyperventilation)
- These symptoms can indicate mental disorder, but often are transient and not part of a disorder. If they impair day-to-day functioning or if people seek help for them, then they are significant symptoms of acute stress.
Emphasize that most symptoms of acute stress are normal and transient. People tend to recover from them naturally. However, sometimes there is a need to intervene when they impair day-to-day functioning or if people seek help for them
What are the three key symptom clusters following trauma?
• Three clusters of traumatic stress symptoms require attention
1. Re-experiencing symptoms
2. Avoidance symptoms
3. Symptoms related to a sense of heightened current threat
• In people with PTSD, the event occurred more than approximately one month ago, at least one symptom from all 3 clusters is present, and the symptoms cause difficulties in day-to-day functioning.
Avoidance
Re-experiencing
Heightened sense of recurring threat - jittery
What is re-experiencing symptoms after trauma?
- These are repeated and unwanted recollections of the traumatic event.
- 3 types of re-experiencing symptoms:
- An intrusive memory is unwanted, usually vivid, and causes intense fear or horror.
- A flashbacks is an episode where the person believes and acts for a moment as though they are back at the time of the event, living through it again. People with flashbacks lose touch with reality, usually for a few seconds or minutes.
- Frightening dreams
- In adults, the frightening dreams must be of the event or of aspects related to the event.
- In children, re-experiencing may involve frightening dreams without clear content, night terrors or trauma specific re-enactments in repetitive play or drawings.
Remembering things to the point that it affects you to the point of interfering with everyday life e.g. have to leave a shop as distressed
What are avoidance symptoms?
- These include purposely avoiding situations, activities, thoughts or memories that remind the person of the traumatic event.
- a person may not want to get in a car after a road accident
- a rape survivor may all the time try to avoid thinking of the rape.
- the person may wish not to talk about the event with the health-care provider
- Paradox: trying very hard not to think of something, makes one think more it.
- Exercise (plenum): try not to think of a white elephant for 1 minute.
What are symptoms related to a sense of heightened current threat?
• Affected persons may feel constantly in danger,
• 2 types of symptoms related to a sense of heightened current threat
• Hypervigilance: exaggerated concern and alertness to danger
○ Eg the person is much more watchful in public than others, unnecessarily selecting “safer” places to sit.
• Exaggerated startle response: being easily startled or jumpy - reacting with excessive fear to unexpected sudden movements or loud noises.
○ Eg person reacts much more strongly than others and takes considerable time to calm down.
What are features associated with PTSD?
- All ages
- Anxiety, depression, anger
- Numbing, insomnia
- Medically unexplained complaints
- In adolescents and adults
- Alcohol and drug use problems
- In adolescents
- Risk-taking behaviour.
- In children
- Regressive behaviours, such as bedwetting, clinging and temper tantrums.
This slide covers associated features with PTSD. These are not PTSD symptoms (according to current draft ICD 11 proposals) but they often co occur with PTSD.
Explain that age groups differ in associated features. Knowing the features associated with PTSD helps in suspecting PTSD.
What is the importance of communication in individuals following trauma?
COMMUNICATION SKILLS
• People who have traumatic stress may avoid communicating about traumatic events because talking about it may trigger re-experiencing symptoms.
• Important to ensure confidentiality when discussing traumatic and private issues.
• E.g. people often have great difficulty talking about sexual violence and torture.
• Never pressure the person to talk about the issue.
• It is very important to listen if the person wants to talk about the issue.
Emphasize the importance of talking about the traumatic experience at the pace with which the patient is comfortable. People may be very reluctant to talk about what happened. Talking about the stressor may make the person very emotional. It may take some time to build the trust necessary for the person to talk to someone about the stressor. People may take weeks or months before they are ready to share
What symptoms apply to DEP and STR presentations?
- Low energy is common in depression and grief.
- Sleep problems are common in depression and traumatic stress.
- Anxious or irritable mood is a common associated features of depression and traumatic stress.
- Medically unexplained somatic symptoms are common associated features of depression, grief and traumatic stress.
- Difficulties in carrying out work (whether at school, home or the office) are common in depression and traumatic stress.
Assessment of individuals following traumatic events…
- Significant symptoms of acute stress
- PTSD
- Grief
- Prolonged grief disorder
- Concurrent conditions
What is management of acute stress symptoms?
- Significant symptoms of acute stress
- PTSD
- Grief
- Prolonged grief disorder
- Concurrent conditions
Relaxation, breathing exercises, doing things you like to do, talking about what has happened to you if its something you want to do. The exception might be in the case of rape for legal implications.
Don’t force people to talk if they don’t want to.
Attend to peoples basic needs if they are in distress whatever that might be.
Help people to connect with family or other people that could help them.
Safe places to stay.
Dot give medication generally for stress disorders although there might be a pressure to do this.
Managing stress disorders like insomnia by breathing exercises, avoid coffee, avoid tv, routine etc.
There are specific techniques for dealing with bedwetting.
Cycle education is explaining to the person that this is a stress response and that external factors can make your heart go faster as a response, this is not a heart attack. Explaining why stress might cause you to get more cold etc.
What is PFA?
Psychological first aid
• Psychological first aid is a humane, supportive response to a fellow human being who is suffering and who may need support
• All health workers should be able to provide very basic psychological first aid.
What is STR psychological first aid?
- Key actions:
- listen to the person without pressuring them to talk.
- provide practical care and support without asking intrusive questions.
- assess needs and concerns.
- help the person to address immediate, basic physical needs (e.g. shelter for the night).
- help connect to services, family, social supports and accurate information.
- As far as possible, protect people from further harm.
Addressing current psychological stressors (STR)
- Sometimes the trauma is ongoing (eg domestic abuse) or can lead to a whole new range of stressors (eg refugee camp life).
- Ask about current psychosocial stressors and as far as possible, use problem-solving techniques to help the person reduce major psychosocial stressors or relationship difficulties,
- Assess and manage any situation of abuse (e.g. domestic violence) and neglect (e.g. of children or older people).
- As appropriate, Identify supportive family members and involve them as much as possible.
Explain that providing assistance with current psychosocial stressors may help to relieve some of the symptoms.
Explain that the health care worker should involve community services and resources as appropriate (eg with the person’s consent). It may be necessary and appropriate to contact legal and community resources (eg social services, community protection networks) to address any abuse (eg with the person’s consent).
Ask the group for some other examples of ways that the healthcare worker might help with psychosocial stressors.
Problem solving in 6 steps (following trauma)
Identify and define the problem Analyse the problem Identify possible solutions Select and plan the solution Implement the solution Evaluate the solution
Not all problems can be ‘solved’
How can we strengthen positive coping methods and social supports?
- Encourage the person to seek the support of trusted family members, friends or people in the community.
- Build on the person’s strengths and abilities.
- Ask what is going well.?
- What are some methods to cope with hardship that have worked in the past?
- Encourage resumption of social activities and normal routines as far as possible
- school attendance, family gatherings, outings with friends, visiting neighbours, social activities at work sites, sports, community activities.
- Alert that use of alcohol and drugs does not help recovery and can lead to new problems.
Insomnia as a symptom of acute stress (within 1 month of the event)
- Apply general management strategies for symptoms of acute stress. In addition:
- Rule out or manage external causes (e.g., noise) and physical causes (e.g. physical pain).
- Ask for the person’s explanation of why insomnia may be present.
- In adolescents and adults, consider relaxation techniques and advice about sleep hygiene (regular bed times, avoiding coffee and alcohol).
- Explain insomnia is a common problem after experiencing extreme stressors.
- If the problem persists after one month, re-assess for and treat any concurrent mental or physical disorder.
Explain that we DO NOT prescribe any psychotropics for symptoms of acute stress.
The only exception to the rule is for insomnia and only in exceptional circumstances as outline in the next slide.
Pharmacological management of insomnia
In exceptional circumstances only
- In exceptional cases in adults when psychologically oriented interventions are not feasible, short-term treatment (3-7 days) with benzodiazepines (e.g., diazepam 2-5 mg/day, lorazepam 0.5-2mg/day) may be considered for insomnia that severely interferes with daily functioning.
- Precautions:
- Risk of dependence. Only prescribe benzodiazepines for insomnia for a very short time and in exceptional cases.
- In the elderly, use lower doses (e.g., half of adult doses).
- Do not prescribe benzodiazepines for insomnia during pregnancy and breastfeeding or in children and adolescents.
Note that the suggested duration is short and the suggested dose is low, and that these should prescribed in exceptional cases.
The named medicines are on the WHO Model List of Essential Medicines.
Bed-wetting as a symptom of acute stress in children
- Apply general management strategies for symptoms of acute stress. In addition:
- Obtain history of bedwetting (to confirm whether the problem started only after the event). Rule out and manage possible physical causes.
- Manage carers’ mental disorders and psychosocial stressors.
- Educate carers. Explain that they should not punish the child for bedwetting. It may make the symptoms worse.
- Consider training parents in the use of simple behavioral interventions (e.g., rewarding avoidance of excessive fluid intake before sleep, rewarding toileting before sleep). The rewards can be extra play time, stars on a chart, etc.
- If the problem persists after one month, re-assess for and treat any concurrent mental or physical disorder.
Bed-wetting is one of the classic features associated with conditions specifically related to stress.
If the problem persists after one month, re-assess for and treat any concurrent mental or physical disorder. CONSULT A SPECIALIST if there is no concurrent mental disorder or if there is no response to treatment of a concurrent mental disorder.
Management of PTSD
- Assess for and address current stressors
- Psycho-education for PTSD
- Stress management
- Referral for CBT or EMDR
- DEP 3 Anti-depressants
- Strengthen coping and social supports.
INT referral for CBT or EMDR
- Cognitive-behavioural therapy (CBT) and eye movement desensitization and reprocessing (EMDR) are 2 psychotherapeutic techniques that have good evidence for PTSD.
- Other therapies (whether psychotherapeutic or pharmacological) do not have such evidence basis for PTSD.
- Refer for CBT or EMDR, if competent (trained and supervised) CBT or EMDR therapists are available. Are they available here?
CBT and EMDR should be considered when the person is within a safe environment, i.e. there are no ongoing traumatic events and the person is not at imminent risk of further exposure to traumatic events. Expert opinion is divided about their appropriate use in unsafe environments.
Unfortunately, there is no time to discuss in detail what is involved in CBT and EMDR. Dedicated training and supervision is needed to learn these interventions.
DEP 3 antidepressants
- Research shows that antidepressants in most people with PTSD only have a small effect (they tend to work on average a bit better than placebo).
- In adults, only consider antidepressants when CBT, EMDR, or stress management prove ineffective or are unavailable.
- In children and adolescents, NEVER offer antidepressants to manage PTSD.
It is assumed that the workshop participants have previously learned how to prescribe anti-depressants (through training of the mhGAP –IG Depression module)
Frequency of follow up after traumatic events
- For symptoms of acute stress (i.e within 1 month of event)
- Follow up is needed after 1 month in case person is not improving.
- At follow up, assess for a range of conditions, incl. PTSD.
- For PTSD
- Follow is also needed after 2-4 weeks to see whether management is working.
- Long-term follow up at regular intervals may be necessary.
- For grief
- Follow-up is needed 6 month after loss to assess for prolonged grief disorder.
- Follow up may be done in different ways (e.g. in person at the clinic, by phone, or through community health worker).
What to do at follow up after traumatic events
- Monitor improvement by asking the person and family
- Ask about and possibly address ongoing psychosocial stressors
- Monitor adherence, response and side effects of medications, if prescribed
- Provide more psychoeducation
What is the definition of intelligence?
A hypothetical mental ability that enables people to direct their thinking, adapt to their circumstances and learn from their experiences.
What is the theory of hereditary genius?
Francis Galton
Theory of Hereditary Genius (1869)
• Variation in ability within the population
• Variations are inherited
• Nature vs nurture
• First to use questionnaire assessments
Wha si the theory of typical intelligence of age?
Alfred Binet and Theordore Simon • Identified typical intelligence for age • Intelligence scale (1905, 1908, 1911) • Developed age norms • 'mental age'
This was done after France opened schools to all children, so many weren’t able to read or write etc, so Binet and Simon were asked to develop assessment tools to be able to assess a child’s intelligence without them having to read or write so that they could put the children into classes. Try and see what you would expect a child to be able to achieve at each age group. Also enabled them to identify children that might be struggling and might need extra help - a way of supporting children in education and also developing an efficient education system.
What is general intelligence?
Charles Spearman (1904) notion of underlying general intelligence (g)
• Two factor theory of intelligence
• Special factors/abilities (s)
Performance on all tests comprise of g + s
What is crystallised intelligence?
Raymond Catell (1971) • Crystallised intelligence (gs) - retain facts, develop experience and skills, a solid factual type of knowledge, eg vocabulary • Fluid intelligence (gf) - identify patterns, solve complex problems, see order in chaos
How did intelligence used to be explained?
Spearman used s factors to explain individual variability/variation. This was a very dominant theory and still is. It has developed over the years.
Catell supported the idea of an underlying level of g, and developed this further. He thought there were two types of intelligence - crystallised and fluid.
In 1960s cognitive intelligence started to be researched more widely - eg memory, perception, processing, problem solving. A shift in ways of assessing intelligence.
What is the theory of multiple intelligences?
Modern theorie - Howard Gardner (1993) Multiple intelligences - potentials rather than a general underlying intelligence 1. Linguistic 2. Musical 3. Logical/mathematical 4. Spatial 5. Bodily-kinesthetic 6. Intrapersonal 7. Interpersonal
No longer bound to these seven - there have been more forms of intelligence which have been added eg moral intelligence, environmental intelligence
….ski - social inteligence - look up
Peargy??? - developmental intelligence
We have a number of different potentials which give us access to different ways of thinking. The 7 given are distinctive and don’t necessarily have overlap.
Evidence provided for this is people who have brain damage are sometimes very impaired in some areas but completely fine in other eg maths vs language. Also in people who may be classified as retarded in one way but may have huge musical or artistic ability, or in individuals who have savant (?) syndrome but are hugely talented.
People withbodily/kinestheticintelligence are skilled at using theirbodyto convey feelings and ideas. They have good hand-eye coordination and are very aware of their bodies. Their fine and gross motor skills are more advanced than the average person’s.
The wordintrapersonalmeans “within the self”—so, “intrapersonal intelligence” is another term for self-awareness or introspection. People who have highintrapersonal intelligenceare aware of their emotions, motivations, beliefs, and goals.
Interpersonal intelligenceis the ability to understand and interact effectively with others. It involves effective verbal and nonverbal communication, the ability to note distinctions among others, sensitivity to the moods and temperaments of others, and the ability to entertainmultipleperspectives.
About intelligence tests
- Valid and reliable psychological tests
- Performance on intellectual tasks: correlates with performance on other tasks - is stable over time
- Consistency between tests infers an underlying level of general intelligence - g factor
Different tests using different components. Find that people that score well in one test, tend to do well in others too.
Some of the most reliable tests in psychology.
Intelligence grows with age, but at around age 11 you can get a good idea of how ones intelligence is likely to sit for the rest of life.
What is IQ?
Intelligence Quotent - Index of intelligence derived from scoring intelligence tests (Stern 1912)
Originally calculated as:
(mental age/chronological age)x100
In modern times, calculated used percentile rankings which are the converted to equivalent IQ scores and projected onto a normal distribution curve
IQ is a scoring system, rather than a particular measure. Less broad than intelligence as a whole.
Was originally calculated by taking m
What are intelligence (IQ) tests typically subdivided into?
Typically subdivided into:
- Performance tasks: ie picture completion (fill in missing part of picture), block design (recreate pattern with blocks without instructions), matrix reasoning (number sin a grid or symbols and have to say which one is missing to complete the pattern), object assembly (like flat pack furniture, without instructions) - Verbal tasks: ie verbal reasoning, comprehension, arithmetic, digit span (measure of memory), letter-number sequencing
About the bell shaped IQ curve…
95% of population lie wit +/- 2SDs of the mean (ie IQ 70 to 130)
68% (2/3) of the population have an IQ within 1SD of the mean. Average is considered to be within +/- 1SD of the mean (85-115)
What are the scores of intellectual ability?
Very superior: >130 Superior: 120-129 High average: 110-119 Average: 90-109 Low average: 80-89 Borderline: 70-79 ---------------------------------------- *learning disability: <70 * Mild: 55-70 Moderate: 40-54 Severe: 25-39 Profound: <25
About intellectual ability/disability
Often concerned with people who are in the borderline range, as access to services and support then people get this if have an IQ score of less than 70. Others very borderline may not be able to have these services.
Tests often carried out by educational psychologists as there is a lot riding on them.
Double edge sword - good because can get help needed, but also can be a stigma attached to having a learning disability.
Tests get less robust at the very top end and very bottom end. Those with IQ of 130+ are generally considered to be gifted.
What are some common IQ tests?
Wechsler scales Raven's progressive matrices Cattle's culture fair IQ tests National adult reading test (NART) Dementia rating scale Bayley scales of infant and toddler development
What are the Wechsler scales?
WAIS (those who cant sit through the whole thing, eg someone who is ill), WASI (adult), WISC (children between 6-10), WPPSI (preschool and primary 2.5-7) - sit in a room with an examiner and go through these tests, age dependent, use diff version depending on the age of the person, 11 types of tests, 6 verbal and 5 performance
What are Reaven’s progressive matrices?
non-verbal, series of progressively more difficult matrices looking for patterns and symbols, timed, can have many people doing it at onc
What are Cattle’s culture fair IQ tests?
Tries to account for different culture and reduces the confounding factors
What is the national adult reading test (NART)?
strong correlations between reading and intelligence, person given a list of words to read one at a time, tricky words, asked to read and pronounce those words correctly, useful as provides reliable estimate for peoples intelligence premorbidly, reading and pronouncing is one of the skills that stays longest with illness, can’t use if damage to language area of brain or in dyslexia
What is the dementia rating scale?
way of monitoring progress of people with confusion, and monitoring fluctuations in their IQ
What are the bayley scales of infant and toddler development?
in young children, basic tests, looking at developmental milestones, seeing how a child’s cognitive ability is developing
What are the clinical uses of IQ tests?
• Diagnosing and quantifying the extent of learning disability
• Diagnosing and characterising specific learning difficulties
- Examples: dyslexia, ADHD, dyscalculia, dysgraphia
• Assessing intellectual impairment following trauma
- Examples: head injury, stroke, poisoning, oxygen deprivation - suffocation, drowning, choking
• Assessing intellectual impairment associated with medical problems or disease processes, and monitoring these.
- Examples: alzheimers disease, dementias, multiple sclerosis, psychosis
• Assessing intellectual capabilities in genetic or developmental disorders
- Examples: downs syndrome
• Other uses: assessing the suitability for particular occupations or educational opportunities
Learning disabilities usually diagnosed in early life by an educational psychologist. Difficult understanding and retaining skills. If diagnosed this is a permanent thing. Often caused by things before or during birth. Sometimes something that happens during the first few years of life. Often the causes are unknown. Affects 1/50 people in the UK. Helps identify where people are struggling and helping them to develop ways to dealing with this.
What are some extraneous influences on IQ test scores?
- Familiarity with the particular concepts and materials used (doesn’t increase your IQ, just speed of processing the questions)
- Motivation to do well
- Distraction
- The way the test is administered
- Sensory problems
- Cultural issues - language, difference in world view
The people who devise these tests say they are testing innate ability but it is clear from results that coaching and practice does make a difference.
What are internal; influences on IQ test scores?
- Emotional state - anxiety, depression, bereavement
- Physical illness
- Mental illness - psychosis, schizophrenias
What are the gender differences in IQ scores?
- Test scoring designed to reduce any sex differences
- Mean 100 - artefact of test construction
- Males better at performance (visual spatial), females better at verbal tasks
- Greater variation in males scores- more spread out - more males at extreme ends. Females scores tend to cluster around the mean.
What are the changes in IQ one the lifespan?
Intelligence scores rise quickly and significantly up to the age of 11. IQ peaks around the age of 20-25.
Older people sometimes have a slight decline. See some decrease in fluid intelligence to do with problem solving etc, but this is probably related to processing time. Don’t tend to see any decrease in crystallised intelligence.
How does an intelligence test predict stability in educational performance?
- Research conducted by Smith et al 2001
- 24000 students aged 11-12 years sat the Cognitive Abilities Test (CAT)
- Four years later their national examinations (GCSEs) were recorded
- Correlation between CAT and total GCSE performance score was 0.74
What was found in the Scottish Mental Survey?
- June 1st 1932 all children born in 1921 in scotland simultaneously sat an IQ test (n=87,497) age 11 years
- Re-tested at around age 80
- Stability in scoring
- Rank scores at age 11 and 80 years very similar - low, medium, high
Lower IQ scores at age 11 were associated with:
- Mortality - shorter life span
- More likely to develop lung cancer
- More likely to experience psychiatric illness and/or dementia
Wanted to know what peoples cognitive abilities were before they developed cardiovascular disease and how this affected them after their diagnosis. See whether there is any relation between disease process and cognitive abilities.
Test results got left unused and then were found later in life. The children were then followed up in their 80s and retested. Found that peoples intelligence rankings tended to stay similar throughout their lives. Also found some relationships with health - those with higher scores were likely to live longer and les likely to develop physical and mental illness.
Nature vs nurture - intelligence
- Hereditability - how much of a trait’s variation in a population can be attributed to genes
- Heritability for intelligence in UK ranges between 40 to 70%
- “The more successful we are at equalising environments, the more genes account for the differences between us.” - Prof Robert Plomin (2007)
Non genetic factors - socioeconomic class, nutrition, poverty
The more equal the environment, open and standard access to education, the more our diets improve etc, the more we standardise these factors then the differences seen between individuals in intelligence must then be due to genes.
What IQ data was found from twin studies?
An example of an American twins study. Tend to show similar things - we see that people who have very similar identical genetic profiles with the same environment you see a very high correlation in IQ at about 9 which is as high as it will get.
These are people with same environmental influences growing up and same genetics.
For identical twins reared apart we see correlation of around 7, and this is still very high. There is a difference that might be put down to environment but there is still a strong correlation that may be put to genetics.
Non-identical twins also still have a high correlation.
Impact of genetics diminishes are people become less genetically similar.
Weaknesses of twin studies: studies looking at twins reared apart have probably been adopted so may have shared same environment at one point, the adoption environment itself is very regulated so those who adopt are likely to have similarities as it is a standardised process (socio-economic. Educational background, families who really want them).
Twin studies also used to have fairly small sample groups but this is less true now as they have been running for quite a long time.
What are some environmental influences on intelligence?
- Socio economic status - education (years and type), nutrition, access to resources
- Poor childhood nutrition - introduction of breakfast clubs, free school dinners etc
- Exposure to environmental toxins
- Exposure to certain drugs in utero (alcohol, cocaine)
- Lack of exposure to an intellectually stimulating environment in childhood
- Neurological injury/disease (zika, cytomegalovirus)
- Genetic disorders affecting brain development (due to environmental toxins - Chernobyl, Fukushima)
How does socioeconomic status affect intelligence?
- IQ scores of 7-year-old twins. Many living near or below the poverty level
- Impoverished families - 60% of the variance in IQ was accounted for by the shared environment, and the contribution of genes was close to zero
- Affluent families - the result was almost exactly the reverse
Good evidence not support the idea that there are strong environmental influences on intelligence
What is the effect of prolonged childhood malnutrition on intelligence?
Pre-schoolers in 2 Guatamalan villages (where undernourishment is common) were given ad lib access to a protein dietary supplement for several years then years later, many of these children scored significantly higher on school related achievement tests than comparable controls.
Another study:
Study included 241 children. Examines effect of dietary patterns in infancy on cognitive function. Duet was assessed at 6 months and 12 months during weaning period.
At age 4 years, children who’s diet in infancy was characterised by high consumption of fruit, vegetables and home prepared foods has higher full-scale IQ, verbal IQ and better memory performance.
Southampton women’s survey - Gale CR et al (2009)
How does exposure to environmental toxins effect intelligence?
Study:
Blood lead levels of children growing up near a smelting plant were substantially and negatively correlated with intelligence tests throughout childhood.
Those with higher levels of lead in blood at birth had Iqs up to 7.2 points lower than comparisons.
McMichael et al 1988
Baghurst et al 1992
Exposure to certain drugs in utero and their effect on intelligence
Study:
Mothers who confsumer even small does of alcohol dialy (the equivalent of 3 table spoons) during pregnancy had children who scored 5 points lower than contorls at the age of 4
Streissguth et al 1989
Study:
2year olds whose mothers used cocaine in pregnancy scored 6 points lower than unexposed peers on IQ tests. They were also twice as likely as peers to have IQ <70
Singer et al 2004 JAMA 291 (20): 2448-2456
What is the flynn effect?
Intelligence has been increasing with time
Tests have changed with intelligence over time. Things that might explain the rise in intelligence scores include technology, access to resources, opportunities, internet
What is higher childhood IQ associated with?
- Fewer adult hospital admissions for injuries/assault
- More likely to become vegetarian (???)
- Less likely to some
- Less likely to become obese
- Lower morbidity and mortality
PTSD, severe depression and schizophrenia are less prevalent in higher IQ bands
What is lower IQ associated with?
- Lower socio-economic status
- Increased change of hospitalisation due to violent assault
- Incarceration
- Early death
What is the definition of emotional intelligence?
An ability to accurately recognise the meanings of emotions and their relationships, to reason and problem solve on the basis of them. Emotional intelligence is involve din the capacity to perceive emotion, assimilate emotion related feelings, understand the information of those emotions and manage them.
What are the criteria for true intelligence?
- Conceptual - mental performance rather than preferred ways of behaving
- Correlational - describe a set of closely related abilities
- Develop with age and experience
Mayer and Salovey 1997
About emotional intelligence
- Term first used in 1960s
- 1983 Howard Gardener published theory: Multiple intelligences - inter/intra personal intelligence
- 1985 Wayne Payne’s referred to EI in his PhD thesis - a study of emotion: developing emotional intelligence
- 1995 Reuven Bar-On - introduces the concept of an ‘EQ” (‘Emotional Quotient”) to measure ‘emotional and social competence’
- 1989 Stanley Greenspan put forward a model to describe EI
- 1989 Peter Salovey and John Mayer published their model
- 1995 Daniel Goleman publishes book: emotional intelligence - why is can matter more than IQ
What is the are the concepts of emotional intelligence (EI)?
2 distinct unrelated conceptualisation of EI:
ABILITY EI
- Ability to monitor and distinguish emotions and feelings of self and others - Operationalised using maximum performance tests, such as IQ tests
TRAIT EI
- Based on lower level personality - Operationalised using self report personality questionnaires
What are the 3 models of emotional intelligence?
- Ability model
- Mixed model
- Trait model
What is the ability model of emotional intelligence?
Peter Salovey and John Mayer (1990, 1995) - Yale David Caruso
The capacity to reason about emotions and emotional information, and of emotions to enhance thinking.
The ability to accurately perceive emotions, to access and generate emotions so as to assist thought, to understand emotions and emotional knowledge, and to reflectively regulate emotions so as to promote emotional and intellectual growth.
Measure: a series of tasks designed to assess ability to perceive, identify, understand and work with emotion.
MEIS (Multifactor Emotional Intelligence Scale), (MSCEIT) Mayer-Salovey-Caruse Emotional Intelligence Test
What are the four branches of emotional intelligence?
- Perceiving emotions: understanding nonverbal signals and facial expressions
- Reasoning with emotions: using emotions to promote thinking and reactivity. Emotions help prioritise what we pay attention and react to
- Understanding emotions: interpret meaning from emotions
- Managing emotions effectively: regulating emotions, responding appropriately and responding to the emotions of others
What is social and emotional learning (SEL)?
Evidence for SEL school programmes such as RULER and PATHS:
- Better academic performance - Improves school climate - Increases students emotional intelligence and social skills - Decreases anxiety and depression - Students are less likely to bully other students - Students have better leadership skills and attentions - Teacher shave better relationship wit students, less burn out, better relationships with admin, more positive about teaching
What are criticisms of the ability model of emotional intelligence?
- Problems with concept - is this just a convenient way of re-describing a collection of traits measured in other ways?
- Ability EI measures might be measuring personality in addition to general intelligence
- Problems with measurement and validity
- Self report - socially desirable responding (SDR) faking goof
- Tests knowledge of emotions but not necessarily the ability to perform tasks that are related to that knowledge
- May only measure conformity - base don method of consensus-based assessment
What are the social and emotional learning (SEL) 5 competencies?
- Recognising emotions in self an others
- Understanding the causes and consequences of emotions
- Labelling emotions appropriately
- Regulating emotions effectively
What is the mixed model of emotional intelligence?
1985 Reuven Bar-On - uses term EQ in doctoral thesis
Measures 5 factors - the ability to:
1. Be aware of emotions, understand and express them 2. Understand how others feel and interact with them 3. To manage and control emotions 4. To manage change, adapt to and solve personal/interpersonal problems 5. To generate positive affect to enhance self-motivation, in order to facilitate emotionally and socially intelligent behaviour.
Self report measures:
1997 published Bar-On Emotional Quotient Inventory (EQ-i)
2012 EQ-I 2.0 released - recommended for use in UK schools
Combines abilities such as perceiving, assimilating, understanding and managing emotions but also motivation, non-ability dispositions and traits, global and personal and social functioning self regard, independence, problem solving, reality testing.
1995 Daniel Goleman published book: Emotional Intelligence
Popularised EI - leadership and performance
Measures:
- 1999 Emotional Competency Inventory (ECI) - 2007 Emotional and Social Competency Inventory (ESCI)
What are Goleman’s 5 competencies in the mixed model of emotional intelligence?
- Self-awareness - knowing own emotions, strengths, weaknesses, drives, values and goals. Recognises impact on others while using gut feelings to guide decisions.
- Self-regulation - controlling/redirecting disruptive emotions and impulses, and adapting to chaning circumstances
- Social skills - managing relationships to move people in the desired direction
- Empathy - considering other people’s feelings
- Motivation - being driven to achieve for the sake of achievement
What is the trait model of emotional intelligence?
- Konstantinos V Petrides (2007) UCL
- Defined as an individuals self-perceptions of their emotional abilities. The purpose of EI encompasses behavioural dispositions and self-perceived abilities.
- Equates to trait emotional self- efficacy - same construct
- A distinct compound construct and a constellation of emotional self-perceptions located at the lower levels of personality hierarchies. Trait EI should be investigated within a personality structure.
- Concerns emotion-related dispositions and self-perceptions, it is best measured via self-report
- Measured using Trait Emotional Intelligence Questionnaire (TEIQue)
What are higher trait emotional intelligence scores associated with?
• lower risk for mental disorders, such as depression and anxiety.
• higher extraversion, social competence and enhanced emotion regulation abilities
• using problem-focused rather than avoidance coping strategies
• appraising situations as challenging rather than threatening
• a recurring resilience in response to life stressors
• being more creative
• Thinking and acting in ways that encourage positive and discourage negative emotional experiences
(Mikolajczak, Bodarwé et al. 2010; Petrides, Pita et al. 2007; Mikolajczak, Luminet et al. 2007: Mikolajczak, Petrides et al. 2009)
What is memory?
The ability to store and retrieve information over time
About neuronal architecture and memory…
- Architecture of human brain - we know that a computer stores information on a hard drive which contains previously a spinning disk, magnetic information, now a days there’s no moving parts.
- By learning something new your brain structure is being changed as new information is given and you form new axonal and neuronal connections which store the information.
- Neurones die over the course of life time, die all the time, memories not stored in one neurone for this reason otherwise they would be lost. Memory is instead stored in multiple neurones in a distributed process - neuronal network.
- Advantages of this is that you can’t lose all the information and its difficult to screw it up.
- You cant eradicate a memory by attacking a memory as you don’t know which ones its distributed within. Lose memories in illnesses such as dementia because so many have been lost, can lose up to 80% before you even notice the change. Whole sections can die and you would only notice that you’re slowing down. When someone presents with dementia they will have already lost over 50%, often whole sections.
- Neural architecture acts the same as the internet, there’s always multiple routes to the same neurone, so if one is blocked then it’ll take another one to get the information.
- Brain stores information at different levels of readiness. In Alzheimer’s and dementia lose the useless information first, and last the things you need all the time such as how to go to the toilet and the name of your partner. Your brain prioritises information based on utility and relevance to you.
What are the types of memory?
Iconic memory short term memory working memory immediate term memory long term memory episodic memory (autobiographical) semantic memory declarative memory procedural memory prospective memory
What is iconic memory?
this is the after image on retina if visual, tactile feeling on skin if you have been touched by something for long enough, echoic if you still hear a sound after it has stopped. These stores are very momentary, 250ms.
What is short term memory?
online storage for things that are happening now. Phone numbers originally 7 digits as can only remember 7-8 things for about 30 seconds (unless constantly sub-vocally rehearsed). 7 pieces of information +/- 2 (standard deviation) is the average limits of human memory. Short term memory is the old fashioned way of saying working memory
What is working memory?
three components: phonological loop (auditory info), visuo-spatial sketchpad (visual), and something resembling consciousness and attention. As there is a consciousness part this means conscious awareness is very limited, but unconscious awareness is vast (eg hearing name across the room even though working memory is full
What is immediate term memory?
a new form. This is the bit between working and long term. Stored for up to 2 days. Eg location of car in carpark
What is long term memory?
potentially forever eg Paris is the capital of France. Divided into separate sorts of stores (episodic/autobiographical which is events which have happened to you, totally separate to semantic memory which is factual memory eg paris - these two are functionally separate, can lose one and not the other
What is episodic (autobiographical) memory?
events which have happened to you
What is semantic memory?
factual memory
What is declarative memory?
things you know you know, when something is on the tip of your tongue
What is procedural memory?
memory for doing things such as riding a bike, driving, swimming
What is prospective memory?
ability to remember to do things in the future, e.g. important tasks you need to do late in the day/week
What is expectation in relation to memory?
Expectation is a memory phenomenon when you’re doing something and your brain fills in the information its expecting, eg hearing mum shout you or jumping when you see something outside at night. Also means that if you see someone out of context when you’re not expecting it you struggle to remember who they are or where you know them from.
There is evidence that human memory is rapidly declining due to technology, in particular google.
What are the critical concepts in neuropsychology in relation to memory?
- Dissociation and double dissociation
- Help to identify neural substrates of various brain functions
- Dissociation = when you know localisation of brain damage or lesion and find that one function is knocked out by other related functions are preserved eg individual loses ability to name category of animals but can name all other objects
- Double dissociation = lesion in specific brain area impairs function x but not y then demonstrate that lesion of separate brain area impairs y but not x
- Patternson and Plaut “the gold standard was always a double dissociation” 2009
What are the three key processes in memory?
- Encoding - processes occurring during initial encounter with to be remembered stimulus = stage 1
- Storage - storage in the memory system = stage 2
- Retrieval - recovering stored information form the memory system = stage 3
Forgetting can result from failure of any of these stages, rather than a process in itself.
About encoding and storing memories…
Encounter with initial stimulus you’re trying to remember eg a lecture, you’re trying to record it by paying attention in order to hopefully remember it.
The deeper the level you encode something to the level you remember it the better you remember it. To deepen the level of encoding you pay more attention, write notes, etc. asking questions makes you encode to a deeper level because you think about it I a deeper way as you have to search your memory for answers which activates similar structures which mean when you are told the answer you can hook it onto something. Making things personally relevant also means that you can encode things deeper.
Storage is transferred between types of memory. One of the best ways to store memory is by asking questions, doing practice questions, making it relevant to you and making notes (rehearsal not shown to have much effect). Consolidation is the transfer from short term to long term.
In retrieval the memory is reconstructed more than recollected, you are not accessing the exact same thing but just the jist of it. We reconstruct things, which we know by telling stories about things that happened - they change slightly. This tells us that human memory is not very reliable - scientific evidence from eye witness testimonies which are very inaccurate in at least 50% of cases.
What we think of as forgetting can result from failure at any of these stages.
About memory structure
A basic model is the multi-store model of memory (Atkinson and Shriffin, 1974)
sensory inputs > sensory store >(encoding)> short term memory (retrieval) long term memory
When gets to long term memory, long term memory has capacity which is almost infinite if you can always lay down memories.
Have done studies to try and test the capacity of this, by showing around 10,000 faces you’ve never seen before, and then the next week they show you them and ask if you’ve seen them before, and they keep increasing the number over months and they haven’t found the capacity of limitation.
What is sensory memory?
- Auditory [echoic] memory - holds information for approximately 3 seconds
- Visual [iconic] memory - holds information for approximately 250ms
Have all the other modalities too, but there is very little research on them because it is very rare. 50% of brain is vision so 90% research on vision, 10% on auditory, very little on olfaction. Smells are weirdly one of the most infallible sources of memory however.
About short term memory
• STM - now termed Working Memory (Baddeley and Hitch, 1974)
• This consists of three components:
- Central executive: this resembles what we would call ‘attention’
- Phonological loop: holding information in a speech-based form
- Visuo-spatial sketchpad: specialised for holding cisual and spatial information
Older form, more historical but still know it.
Central executive - this is almost a representation of consciousness/attention. Attention is a way of deepening the level of encoding. The brain pays most attention to information that is relevant to you. This biases us in terms of attention, means that what we see in the world is not what’s there but is what is interesting to us. If you change who you are as a person, you have trouble remembering things you learnt as your old self - this is called state dependent memory. Best example is locational memory - what you learn in this class, if you took the exam in the same room as the lesson then you would get about 5% more on the paper.
About working memory…
• WM is a much more dynamic store than a unitary STM
• Working memory has a limited capacity (LTM is theoretically without limit, in both capacity and duration of storage)
- It is usually found that we can only store around 7 ± 2 pieces of information in memory (Miller, 1956)
- Be beware of chunking - grouping bits of information to make remembering easier
Chunking is how you group bits of information, for example not remembering each number in a phone number, but putting parts of the number together.
What is double dissociation in memory?
- Are short term memory and long term memory distinct?
- If they are we would presumably see patients with impaired STM and preserved LTM? If we also saw the reverse pattern preserved STM and impaired LTM this would be an example of a double dissociation
- Researchers and clinicians have found this pattern to be supported
- Eg. KF = patient with impaired STM but fine LTM (Shallice and Warrington, 1970)
No short term memory means no really attention span
Inference in memory recall
Inference in WM
- Traces only remain in WM with repeated rehearsal (ie attending to the information) - They are therefore prone to interference - Retroactive interference: new information interferes with the retention of old information in WM (trying to remember your old phone number, having used your new number for a few months; finding it difficult to revert back to manual gear changes having recently been shown how to use an automatic) - Proactive interference: old information interferes with the recall of new information (mistakenly giving your old phone number instead of your new one; attempting to change gears manually in a newly bought automatic)
An example of interference is making the person think about something completely different when trying to remember something else - when new information interferes with old (retroactive) and when old information interferes with new information (proactive)
What is the primary-recency effect?
Primacy-Recency or the Serial Position Effect
When presented information in serial order, we remember more form the beginning and end of the list, at the expense of intermediate items.
- 4000-5000 studies on this effect, never really not found.
- We are always presented with information in a serial order as its over time.
- Means that the first 20 mins of a lecture you’ll remember a lot, last 20 mins you’ll remember a lot but in the middle you’ll remember less. The same happens with films, books, experiences, etc. this is a really strong effect.
What are the levels of processing (Craik and Lockhart 1972)?
Craik and Lockhart - 1972
- Depth o encoding determines later memory for information - Processing at encoding can be shallow or deep. The greater the processing the meaning of stimuli at encoding the deeper the level of processing and greater likelihood of later storage and retrieval - Importantly Craik and Lockhart disagree that rehearsal will improve LTM as it just repeats previous operation. BUT rehearsal has beneficial effects but they are small and nor for every type of memory eg prospective memory may not benefit
About long-term memory
Craik and Lockhart - 1972
- Depth o encoding determines later memory for information - Processing at encoding can be shallow or deep. The greater the processing the meaning of stimuli at encoding the deeper the level of processing and greater likelihood of later storage and retrieval - Importantly Craik and Lockhart disagree that rehearsal will improve LTM as it just repeats previous operation. BUT rehearsal has beneficial effects but they are small and nor for every type of memory eg prospective memory may not benefit
What is consolidation?
Involves structural change: the pattern of neural pathways are changed
- Long term potentiation: strengthening of synapses, believed to be the cellular foundation for memory - Research into how to induce LTP useful for dementia treatment - rats given NMDA receptor antagonists display memory deficits. Mice bred with enhanced NMDA function show greater LTP and better memory
Requires metabolic activity for minutes/hours after the stimulus has been presented.
Believed that the hippocampus plays a vital role in consolidation.
Human memory has got significantly worse over the last 20 years due to this process being weakened by the advent of google. Hippocampi are getting smaller as we are externalising memory to smart phones etc.
What is retrieval (memory)?
- Cue required (eg prompt, reminder, question)
- Original stimulus reconstructed using information from cue
- The more information available, the easier the retrieval
- A good ‘filing system’ also aids retrieval - eg mnemonics (memory aids, Method of Loci), make something personally relevant to you and you’re more likely to remember it
- Retrieval can be affected by interference - proactive (new phone number replaces old number), retroactive (old routine home blocks new route home)
- Tip of the tongue phenomenon
We know that memory isn’t just recalling what you laid down - it is reconstructive.
Retrieval even from long term memory can be interfered with by the same processes as in short term memory.
What are factors affecting retrieval?
• Levels of processing
- The more something is elaborated at the time of encoding, the easier it will be to retrieve
• Organisation
- Of concepts in memory (also, associations between concepts stored in LTM may facilitate recall)
• Context
- Being in the same place/emotional state as the time of encoding facilitates recall - State Dependent Memory
Organisation of concepts in memory affects how you retrieve them. The hardest thing you could be asked to remember is information that you have nothing similar to in the past (similar to if you don’t have visual stimuli from birth you cant see even when given sight back later in life). This is why there is some evidence that if your critical period during childhood is more varied and exciting you will do better in life as - has been seen in rat studies where brains bigger, denser, more neuronal connectivity. This tells us that you can make your brain better than biology gave you through environmental use.
State dependent information also important for mood - if taught something drunk you wont remember it sober, but drunk you will - also works with drugs.
What is state/emotion dependent recall?
Organisation of concepts in memory affects how you retrieve them. The hardest thing you could be asked to remember is information that you have nothing similar to in the past (similar to if you don’t have visual stimuli from birth you cant see even when given sight back later in life). This is why there is some evidence that if your critical period during childhood is more varied and exciting you will do better in life as - has been seen in rat studies where brains bigger, denser, more neuronal connectivity. This tells us that you can make your brain better than biology gave you through environmental use.
State dependent information also important for mood - if taught something drunk you wont remember it sober, but drunk you will - also works with drugs.
Works with biological environment with chemicals in blood stream, mood, mental state and locational environment.
What is declarative and non-declarative knowledge?
Declarative knowledge (sometimes known as explicit memory):
- Memory for facts, for information that can be conveyed by statements - Involved conscious recollection of events/facts - Encompasses semantic and episodic memories - Eg your birthday
Non-declarative:
- Generally doesn’t involve conscious recollection eg procedural memory - Implicit memory - Priming
Implicit memory: one of the two main types oflong-term human memory. It is acquired and used unconsciously, and can affect thoughts and behaviours. One of its most common forms isprocedural memory, which helps people performing certain tasks without conscious awareness of these previous experiences. Can work out if implicitly racist using this by looking at speed of processing these things,
Priming: where one concept being active in your mind leads to another concept being active eg bread = butter, doctor = nurse. One concept primes another. You self-prime yourself by your interests, if your really love football you will prime yourself to notice all things football in the environment.
About procedural memory
Procedural knowledge
- Memory for how to do things/skills
- Often in the absence of conscious recollection
Eg how do you tie your shoelaces, driving, riding a bike, playing chess
This form of memory is also dissociated from the others. Can lose all other forms of memory but still be able to do things using this form of memory.
Chess is interesting because they can see it and don’t think they know how to play as don’t think they ever have before, but if you tell them to guess then they can do it perfectly.
This is an unconscious form of memory and it can’t access conscious.
About semantic and episodic memory
This form of memory is also dissociated from the others. Can lose all other forms of memory but still be able to do things using this form of memory.
Chess is interesting because they can see it and don’t think they know how to play as don’t think they ever have before, but if you tell them to guess then they can do it perfectly.
This is an unconscious form of memory and it can’t access conscious.
About episodic/autobiographical memory
- Most episodic memory decline over time however decline is not uniform over all memories eg
- Behrick et al 1975 - memories of student days showed remarkable preservation - reminiscence bump = surprising perseverance of memories occurring between ages of 10 and 30. why?
Most episodic memories decline over time but not uniform over all memories.
Memory across human lifespan interesting as if get someone at 80 to recount the whole life they get reminiscence bump and get a lot of information between the age of 10 and 30. partly primacy effect as before 10 structures for laying down memories aren’t properly developed, and because the time in this period is an important transitional time, and should make this time a good part of your life as will remember it forever, this is also because during this time you’re exposed to a lot of new experiences. These are emotionally significant events. If they are new experiences then there cant be proactive interference as you have never done it before. Also have a bias towards positive rather than negative memories. Brain has some process where it rewrites your own history as you age - selectively forgets negative things and then you remember your past better than it actually was.
What is the reminiscence bump?
- Stability of early adulthood memories is remarkable
- Why
- Majority of first time experiences. Novelty is memorable because of the lack of proactive interference from previous learnings
- More evidence for reminiscence bump for positive than negative memories (Bertsen and Ruben, 2002)
Facebook and social media exacerbates this as we mostly tend to post positive things, biasing ourselves. Facebook is like a selectively positive diary, if compared with actual diaries we see that this fully positive life is fake.
What are flashbulb memories?
- Flashbulb memory = detailed and extremely vivid memories regarding circumstances surrounding very shocking event eg September 11th
- Only occur with shocking and emotionally significant events. Evolutionarily adaptive?
- Emotional arousal during encoding enhances retention
- Also frequently rehearsed and talked about
Memories where something happens with such great significance that you can’t ever forget it - eg 9/11
Happens with things that are usually negative and very different and are encoded very rapidly because there is emotion associated with (amygdala, limbic system) and this encodes it in a different way with memory and emotion. This is evolutionary for survival, useful to remember this information so as to avoid it as well. Can have personal versions of this as well not just famous things for all people, eg first real breakup. This is also partly how trauma memory works.
You never forget flashbulb memories, and it is one instance memories.
What is recognition?
- Sensing that a stimulus has been previously encountered, sense of familiarity
- Generally easier than recall, as information is contained in the cue
Recognition can be direct from a set stimuli or from a mental search of LTM store
- Generally easier than recall, as information is contained in the cue
What is recall?
- Reconstruction of a stimulus using the information available from the cue and information from LTM
- Checked by the process of recognition
- Greater ‘cognitive demand’ than recognition
Recall can be direct from the cue or arise after problem-solving strategies have been employed
About recognition and recall being inaccurate…
Memory is not entirely ‘stable’ and recall and recognition can be inaccurate
- Eye witness reports (Loftus 1979) - Participants watch a film of an accident, one includes a STOP sign, the other a YIELD sign. Using specific working, you can change people's memory: - "How big was the sign by the road?" the participant wont remember the fact that they saw a yield sign - "how fast were the cars going when they smashed into each other?" participants will recall the cars were going faster than they actually were
Loftus looked at actual eye witness testimonies. Showed a video then asked about speeds varying use of ‘smashed’ and ‘hit’ and found when used the world smashed people said on average 10mph faster than when said hit each other.
Police interviews are now recorded, 2-3 people present, lawyer present and wording is now thoroughly checked. This study influenced this.
About forgetting…
The main causes of forgetting from either working memory or long-term memory:
Passage of time:
- Rapid decay of information from WM - Cell death leads to loss of LTM (can be accelerated eg dementia)
Interference:
- Interference to storage in WM eg pro/retroactive - Connections are remolded over time in LTM, influenced by pro- and retroactive interference of information
What is the relationship between age and memory?
Phenomena
- Infantile amnesia = inability to remember events from early childhood - Generally people can not remember anything before 2 years. First memories usually occur between 2 and 5 years. Probably because brain regions responsible for episodic memory are nor sufficiently developed - Reminiscence bump = preserved memories for 10 to 30 years (many first time occurrences)
Normative aging:
- Speed of processing declines (perfect 1994) - Memory declines and forgetfulness increases but shouldn't interfere with ongoing functioning
Everyone has infantile amnesia
30-60 age period kind of gets lost, as opposed to the 10-30 gap
About memory repression
- Freud 1915 – suggested threatening information is often blocked from conscious awareness
- Most evidence based on adult patients that often spontaneously retrieve memories of being abused as children that they report no previous memory of.
- Clancy et al 2000 – Experiment
- Butter, food, eat, sandwich, rye, jam, milk, flour, jelly, dough, crust, slice, wine, loaf, toast
- Nurse, sick, lawyer, medicine, health, hospital, dentist, physician, ill, patient, office, stethoscope, surgeon, clinic, cure
- Bread? Doctor?
- Clancy et al 2000 compared women with recovered memories of childhood sexual abuse, with women who believed they were abused but could not recall it and women who had always remembered abuse and controls.
- Also issue of prompting – subsequent studies have shown that adult patients that admit to reporting false recovered memories had clinicians that categorically suggested they had been abused as children
- This finding relates to the literature of eyewitness testimony and its unreliability.
Block things out so that we don’t have to keep reliving it. The evidence is very mixed on whether this exists at all.
Confabulation is when you remember things in a different way but that is plausible. Remember things that weren’t there for example.
Something worse about the peoples memories in the results to make them unsure if something happened or not.
Suggestibility factor - eg from therapists etc suggestion of abuse
About dissociating function in WM and LTM
- The case of HM (Milner, 1978)
- Had surgery to cure severe epilepsy
- After operation had near normal memory for events prior to surgery, and STM normal (approx 15 s)
- But severe deficit for new memory of facts or of events occurring after surgery (encoding from STM to LTM).
- Since operation he forgets the events of his life as they occur
- Able to learn motor skills but could not remember which skills he had learned (Short term declarative)
- Hand buzzer example
Can remember anything that happened from then on - semantic and episodic. However could learn chess which is procedural but has to guess the moves having been taught after the accident.
Every time the doctor would go in he would say hello doctor who are you and he would say I’ve never seen you before in my life and come back in 30 seconds later would say the same thing again. Doctor then used electric shock hand shake thingy after meeting about 100 times and then went out and came back and then he still says he’s never seen him before and doesn’t recognise him BUT refuses to shake hands, and said he just didn’t feel like it so he had partial awareness.
About dissociating function in procedural and declarative memory
• Temporal Lobe Amnesia (e.g. Korsakoffs)
• Patients are able to learn skills
○ Procedural
• Patients are unable to learn new facts
○ Declarative
• E.g. a patient may show improved performance on a simple task over time, but each time they will show no recognition or memory of ever having performed the task before (despite obvious improvements)
Irreversible when gets to Korsakoffs
Able to learn new skills but not new facts - so declarative and semantic mainly damaged. Can have functioning episodic memory though
About dissociating function in semantic and episodic memory
• The case of KC (Tulving, 1989)
• Damage to specific brain regions, (left frontal-parietal and right parieto-occipital lobes) following a traffic accident
• Can no longer retrieve any personal memory of his past (autobiographical memory) but general knowledge remains good
• Plays chess well but does not remember where he learned to play
• Can learn semantic knowledge when care is taken but cannot acquire episodic knowledge
○ E.g. will claim never to have met people met recently, but will be more friendly towards them than people he really hasn’t met
Amnesia
• Amnesia can be brought about by:
• Damage
○ Head injury, stroke
• Chronic alcohol abuse
○ e.g. Korsakoff’s syndrome, affecting storage and retrieval processes
• Disease
○ e.g. Alzheimer’s: extensive damage to cortex, progressive deterioration from some impaired memory skills (esp. episodic memory) à more general cognitive impairment
• Reversible brain disease
○ Tumours, hydrocephalus, subdural haematoma, deficiencies in B1, B6, B12, endocrine disease, syphilis
• Psychogenic Memory Loss
○ Abuse, war
• Anterograde amnesia (as in patient HM)
• Inability to store new information
• Inability to transfer information from STM to LTM
• Retrograde amnesia (rarer)
• Inability to recall information prior to trauma
• Often following closed head injury, can be relatively short duration
Most depictions of amnesia in films is someone hits their head and they lose the ability to remember who they are from the past - this is very rare and is called retrograde amnesia. The much more common form is anterograde amnesia which is where you can’t make new memories.
Also need to clinically separate into semantic and episodic to see which system is damaged.
Semantic is rarer.
Trauma could be so great that some kind of regressive mechanism is acting - people forget things because it is too traumatic.
Could also just be lying.
Common denominator of PML is that you never lose a nice memory, always traumatic things.
Following a stroke you gradually get function back, get memories back, but if a whole brain region is killed that wont recover. However we know that another area can learn and adapt to what you’ve lost, so areas around the dead area can take on some of the function. Get a slow, gradual recovery.
About HM and the hippocampal region
- HM had both hoppocampi surgically removed yet retained information from the past.
- Therefore suggests this is not the actual repository
- Memories are stored in various regions of the cortex
Biology of memory storage
Frontal lobe - stores certain semantic and episodic memories
Prefrontal cortex - involved in storage of STM
Motor cortex - procedural memory
Temporal lobe - formation and storage of LT semantic and episodic and also new STM
Amygdala - emotions, emotional response, emotional memories
Hippocampus - new LT semantic and episodic memories, not the repository but where theyre formed - amygdala on the end of it, close biologically as get bang for buck as it stores the memories, both lighting up saying to store quickly, perhaps one time learning
Cerebellum - storage of procedural memories and also for balance. If drink enough for long enough you permanently damage it and you get a permanent alcoholic gate
Green one - occipital lobe but this is all visual, but it does have to be interrelated to all the rest as most of what you learn comes in the visual format. Encoding it visually as well as audition.
We don’t know where all of this stuff is integrated in the brain. Looks more like the whole brain integrates it. Couldn’t just be one integrated region because this would be dangerous as if that area got damaged then there would be major consequences. The brain can make up the differences and fill in gaps of hearing and also vision.
What is hippocampal function
- Memories are stored in a distributed fashion throughout the cortex
- Therefore something has to integrate these into coherent memories
- That something is probably the hippocampal region (Schacter, 1996)
Not only the hippocampus because can remove it and there’s still function - likely to be the regions around the hippocampus more than others.
About temporary amnesia
• Can result from head injuries, psychological states or drugs
• Concussion often associated with memory loss
• Cinema experience
• Usually retrograde amnesia for events immediately preceding the cause
• Fugue states (Schacter et al. 1982)
• Occur with extreme psychological trauma often loss of previous experience and identity
• Drugs
Date rape drugs such as rohypnol can cause amnesic effects for events occurring while under the influence
Rohypnol, alcohol
Fugue states - when you have a big psychological trauma, do something horrific (eg kill someone) and then forget what you’ve done and are found wandering. Unaware of what you’re doing.
About ageing and memory
• Memory across the lifespan is unequally distributed as we have seen (Rubin, Wetzler & Nebes, 1986).
• If we ask an older adult to recall their lives the following general pattern would emerge
• Infantile amnesia – failure to recall anything from first 3 years often first 5 years
• Reminiscence Bump – better memory for period 10 to 30 but strongest for positive information
• Alternative theory
• Conway & Pleydell Pearce (2000)
• Knowledge base
○ Lifetime periods – vast swathes of time defined in terms of ongoing situations
○ General Events – often repeated but related to each other
○ Specific knowledge of events
• Working self
○ Concerned with goal attainment frames knowledge in terms of progress or lack of progress towards important goals
As opposed to being because of new experiences, might be because of the Conway and Pleydell Pearce theory - the things you remember are discrete normative events, so average life would look like birth, school, university, work, tv, marriage, children, divorce??, death. This theory argues that these are the structures that we hang memories on.
Working self - anything related to you is better remembered, cognitive bias to things you’re interested in, also cognitive bias if set a goal for your life then you have a cognitive bias to anything related to that goal. This means that you notice some things more that you didn’t notice before or wouldn’t usually.
- At advanced age memory starts to decline
- Speed of processing slows with age (Perfect, 1994)
- Generally normative age related decline should not interfere with daily functioning
- Where it does its more likely to indicate a pathological process
- Working memory declines more
Average life expectancy is 80 (M) and 81 (F), and 80 is where we class as starting to see advanced old age.
Older adults used to refer to 60-80 and after 80 were referred to as either advanced old or old-old, but have now had to create new terminology as people are living longer now.
About amnesia research
• Brain damage studies are broadly supportive of the 3 types of autobiographical knowledge
○ Generally event specific knowledge is more compromised than general events or lifetime periods
○ Confabulation on basis of plausibility is common
General event autobiographical knowledge is every time you’ve been to uni - repeatedly go, similar to dating etc
About dementia
- Affects roughly 800,000 people in the UK
- Increases with age
- Is a syndrome associated with multiple losses
- Memory loss
- Speed of processing
- Language ability
- Understanding
- Emotional ability
- Alzheimers is most common form representing the majority of cases
- 10% of individuals develop dementia at some point
- Cure is impossible because of neurodegeneration but slowing is possible
Massive cause of memory loss.
800,000 is going up, significantly higher than schizophrenia rates, going up because living longer and lifestyle factors such as alcohol use, using your brain/lack of use.
Not just memory loss - also speed of processing, language, comprehending, emotional, personality change.
An almost certainty if live long enough is the lens of the eye wears out and get cataracts, prostate cancer, also memory loss is another certainty.
Rest of us (90% not with dementia) wont escape memory loss.
Memory loss with dementia is problematic because cure is impossible because it is caused by neuronal death. Drugs can slow it but not stop it. Slowing it is a problem though because by the time someone presents with dementia they can have lost 50% of the neurones already.
Looking currently at transplanting parts of brains, just the regions which are damaged not the whole brain. Could be taken from elsewhere or grown.
Other treatment possibilities include encouraging neurone regeneration/regrowth. They are looking at this by using certain animal species and looking at how they regenerate eg starfish.
What is attention?
- The human information processing system is limited in terms of allocation of resources to sensory and perceptual information
- The term attention s sued to refer to this allocation of processing resources
- Attention acts as a filter (Broadbent) or an attenuator (Triesman), to prevent this limited set of resources from being overloaded
- It can act as a ‘bender’ of object features and as a ‘binder’ of related information from different sensory modalities.
What is the special problem of auditory attention?
Auditory system has a special problem: it requires processes that permit a listener to attend to the specific set of sounds without being confused by the overlap of other, irrelevant noises.
Auditory system is able to separate different, superimposed sounds on the basis of their different source directions.
Uncertainty remains over the fate of the unattended material.
Ears are positioned in a way that we cant direct them to avoid certain stimuli, unlike the eyes where you can just look away.
If the way we hear so much auditory stimuli would be like seeing loads of sentences superimposed on each other if you cold see it.
We have two ears which focus on long wavelengths, so we can focus on some stimuli over others.
What is the dichotic/shadowing procedure?
(Broadbent, 1952, 1954; Treisman, 1960)
- Subjects wear a set of headphones and made to hear two messages at the same time, one entering each ear, and asked to shadow (repeat back the words from one message only)
- A typical shadowing task:
· Left ear: and then John turned rapidly toward
· Right ear: a series of words - ran,house,ox,cat
Ask to repeat back the left ear material
Shadowing procedure was the first wave of research. Subjects wear headphones, hear one in each ear and told to only repeat the words from one ear only.
Typically one is asked to repeat the prose not the random works.
What was found from the dichotic listening/shadowing procedure?
- Unattended material appears not to be processed in a shadowing tasks; only most recent unattended material is available, while still preserved in the echoic memory
- The listener is normally unable to report significant details concerning the unattended information: can only tell whether the unattended message is a human voice or a noise, or if human, whether male or female and the language used by the voice
- These results suggest parallel acquisition of all available information, followed by serial processing to determine meaning for one attended message
Found people couldn’t really repeat the unattended words except possibly the last 3 words. Beyond that it would fade away incredibly quickly, only in very short term memory.
However, were able to register sensory aspects of it, such as pitch, language, male or female voice etc. Just cant get semantic value from it.
What is broad bent’s 1958 filter theory?
The basic claims of the model:
• Sensory channels have an unlimited capacity
• There is a bottleneck allowing only one piece of information into working memory at a time
• A selective filter allows in information from only one channel at a time based on the physical characteristics of the message (ear, pitch, etc)
• Information from unattended channel is completely blocked
Take information in via sensory organs and then there’s a gate at which point we decide which message we are going to receive. After that choice is made only one stream of information will go through. Then goes onto short term memory.Biggest mistake is that the filter could only be done on superficial sensory aspects (Eg pitch)
What are the problems with the bottleneck theory?
- Although there is little conscious awareness of unattended material, it may receive more processing than the above results imply
- Sometimes selected messages are processed on the basis of their semantic content rather than their physical characteristics (eg cocktail party phenomenon)
- Words presented to the unattended ear can produce priming and physiological effects (eg ‘ignored’ shock words give rise to galvanic skin responses; Corteen and Wood, 1972)
- Trying to ‘shadow’ one ear will follow the message to the other ear (Treisman, 1960)
Wasn’t true. Found that although there wasn’t much awareness of the unattenuated information, it was actually processed at a further level than initially thought.
Eg cocktail party phenomenon - shows it must be in some way
Can measure physiological aspects eg simple word association task in one ear and critical words every 5 or 6 words are followed by an electric shock and this is repeated a lot of times. After that dichotic listening task where have to shadow one ear, but in the other year every time the signal word is said their skin responds and this suggests there was some processing to the stimuli in the first part.
What is evidence for parallel processing (Treisman)?
• Treisman instructed subjects to shadow a particular ear into which was played a meaningful message
• The nonshadowed ear received a random string of words
• At some point in delivery, the meaningful message switched ears and the random words were switched into the nonshadowed ear
• left ear: In the picnic basket she had peanut butter book, leaf, roof, sample, always
• right ear: cat, large, day, apple, friend, every, select, sandwiches and chocolate brownies
• Although instructed to shadow a certain ear, many ignored this and followed the meaningful message instead (temporary sensitisation?)
Results imply that processing takes place in parallel, to the extent that meaning is extracted even from unattended material
Start sentence in one ear and then switch to the other and people will follow it seamlessly. Treisman said this is because you are primed to hear the next words in the sentence, as stimuli around us prime us. When you hear your name you have a very low threshold to recognise your name, very few processing resources needed for it as it is naturally primed. Similar with the sentences.
Some semantic value in the other information.
What is visual attention?
- Attention can be directed selectively towards different areas of the visual field, without the need to re-focus
- Visual attention linked to specific objects rather than to general regions of space
- Unlike hearing, seeing is typically extended over space (and not time), although seeing does require some finite time to capture and analyse information – this process has been the focus of much research
Similar message but a little more complex. With visual attention we are changing the focus. In auditory it’s the info available to us but we choose to ignore it, in visual its on information that’s there are we really want to process, but might not have time to do so because of its brief duration.
We an read a sentence, take visual imagery of a split second as opposed to auditory which takes longer to understand a sentence.
What are the ways of testing iconic memory (visual)?
• Presented an array of letters for 50 ms
X M R J
C N K P
V F L B
• Whole-report method: recall as much as possible from array
• People recall 3-6 letters; report that the letters “fade away” before they can report them all
• Part-report method: only certain elements from array
• Employs a tone (high, medium, or low) to cue subjects to report a particular row (top, middle, or bottom)
• Recall a higher proportion of letters: labelled the ‘partial report superiority effect’
All material captured in parallel, some selected for further, serial processing on the basis of position or colour
How long is long enough to process something?
1/20 of a second was shown letters and asked to repeat as many as possible. Found that generally get between 3-6 letters and say saw all the other letters but they faded away while repeating the ones did remember.
Part report method - straight after the letters shown a tone is sounded. Only have to repeat one row and found people pretty good at this suggesting that have taken on the whole thing but only need to say some so can manage it.
What was the backwards masking technique?
• Backwards masking procedure (Evett & Humphreys, 1981; Pecher et al., 2002)
• the mask is presented after the target, usually appearing in the order of 10-50ms after target first appeared
• time between onset of target display and onset of mask is called the stimulus onset asynchrony (SOA)
Experiments using this technique have provided persuasive evidence (e.g., significant priming effects) that meaning can be extracted from material of which participant is unaware
Seemed too simple. Became a more complex system.
Backwards masking - generally where a stimulus is shown and then something immediately replaces it. Can be done now digitally to the point where people don’t know what the initial stimulus was or can do it so fast they don’t even know there was an initial stimulus.
Normally around 40ms after.
There is persuasive evidence that even though people cant necessarily say the initial stimulus, it has subsequently affected their perception of something else.
What evidence was found from backwards masking studies?
- Participants say they cannot see masked words but often do better than chance when forced to guess whether or not one had actually been presented (Cheesman et al, 1984)
- Evett & Humphrey (1981) used a perceptual identification task - stimulus sequences containing 2 words, both of which masked (SOA = 40ms) – when second word related to first, it was more likely to be reported accurately
- Pecher et al. (2002)
- perceptual identification study using a potential prime (lion) followed by hard-to-see masked target which was related (tiger) or unrelated (dice)
- manipulated priming word duration (short vs long) and proportion of related target words (10% vs 90%)
If people say they didn’t see it but then get a 50/50 choice of words they do better than chance.
Experiments where they use words such as lion and the second will be tiger or dice, where it is related people are much better at identifying the words. Cant report the first word but much more able to get the second word if they’ve been primed for it, even though cant recall seeing lion. Both words shown very briefly.
They looked at the priming word duration. Where you have a 1 second prime you can prepare much easier for the second word and see much higher percentages well above chance (50%). People then are good at picking up on what’s going on and see how it works and get a massive priming advantage.
One is called a strategic prime, and one is called a hope for the best prime.
The 40ms is enough to get a small priming advantage.
Some of the time people are taking the semantic value of the words without being able to consciously report it, some of the time.
What results were found be Pecher et al?
2002
At 1 sec, benefit of related prime when targets more likely to be related to preceding prime (participants spot connection)
At short duration, priming advantages are far more modest – do not produce large increase in priming effect
Participants presumably unable to guess in brief condition but did produce a small priming effect – must have received sufficient analysis to activate their meaning
Automatic priming effects are caused by spreading activation between nodes at level of representation.
What is the test of of RSVP (rapid serial visual presentation) technique?
The sequence of stimuli, shown in the same location on a computer screen, in which the participant has to identify a white letter, then decide whether an X was also present
Number of stimuli occurring rapidly one after the other. Done so guessing wont help.
How spaced to they need to be before they are frequently seen
Typical results, showing the likelihood of detecting the X, when presented in the first and subsequent positions following the white target - the attentional blink
(a) Target 2 is seen more easily when Target 1 is made easier to see by removing the following item
(b) Target 2 is also seen easily when items following it are omitted
Found that if follows straight after people are pretty good, thought that they just put them together as they came so closely.
If have a lag of 2/3/4 then get pretty bad at seeing it. 6/7 later then people have recovered from first letter and start seeing it more frequently again.
Why is the lag of 2/3/4 so bad? Found that in effect after target one, have a bit of a gap then can remove the intentional blink then people are much better. If put at the very end irrespective if straight after or not then people are much better.
What was the Giesbrecht and Di Lollo model (1998)?
Two-stage model of visual processing to account for RSVP findings
• Stage 1: a range of info about target characteristics is captured in parallel (identity, size, colour, position)
• Stage 2: serial processes act upon information preparing it for awareness and report
• While Stage 2 is engaged, later info cannot be processed so has to remain at Stage 1
• Disruption to Stage 1 (masking) increases processing difficulty, so info from T2 is kept waiting longer
• If T2 masked by following stimulus, then run risk of overwriting it
• Damaging to episodic information; semantics info may be able to survive (revealed through priming effects, EEG)
Individual differences in RSVP: personality traits important to distinguish blinkers from non-blinkers (Morrison et al., 2016)?
First trying to get target one which you capture, taking in colour and identity of the letter which takes resources. People are good at doing this. However while doing this don’t have control over resource allocation to do something else to onto stage 2. serial processes are needed, preparing it for conscious awareness, and while still doing that you cant really process other information coming later. Disruption to this stage increases the difficulty.
If target 2 is then masked you have lost the change of carrying on doing it. But if its not masked, eg comes at the end, then you can do it. It is the damage to episodic information that is bad. Lose the ability to consciously report on the event, that is what attention is, the ability to take on the semantics of what’s in front of you and being able to report on it.
What is the visual awareness and attention model?
- Inability to report detail from brief, masked visual displays is linked to need to assemble various information components
- The visual information is captured in parallel, but assembly is a serial process
- Episodic detail (e.g. colour, position) is vulnerable to the passage of time, or to ‘overwriting’ by a mask
- Semantic information (i.e. identity/meaning) is relatively enduring, but does not reach conscious awareness unless bound to the episodic information(Coltheart, 1980)
- Attention, in this context, is the process of binding the information about an item’s identity to its particular episodic characteristics
- ‘Unbound’ semantic activation can be detected by priming and electrophysiological techniques
Many inputs are unconscious. This comes at a later stage, not the same as consciousness. There is a difference between consciousness and being consciously aware (being able to report).
Blinkers and non-blinkers in the RSVP task. People who are particularly prone to blinkers is more likely to be in people who: lack of sleep, depression and autism - but not anxiety (don’t really know why though).
What is subliminal messaging?
- Registration of sensory input without ‘conscious awareness’
- Limen is another word for Threshold - so subliminal means “below the threshold”
- Controversy began in 1957 when hidden messages such as “Eat Popcorn” were placed in films
- James Vicary claimed to have flashed the words “eat popcorn” and “Drink Coca-Cola” on a movie screen for 1/200th of a second, every 5 seconds during the movie Picnic
- He claimed popcorn sales increased 58% and Coke sales 18%
- Vicary’s experiment was never successfully replicated
- He later acknowledged the study was a fraud (Advertising Age ,1962)
Even though cant consciously report something can take some semantic value from the stimulus.
Hard to accept that there are things there that we aren’t aware of but are affecting our actions.
What are the types of subliminals?
- Embedded images: pictures or words that are hidden or flashed quickly (in 100ths of a second)
- Sub-audible messages: sounds or words that are too faint to be heard, or are played at extremely high frequencies
- Electronically altered signals: backward masking and other voice alterations
Can messages lead to priming, the activation of various mental constructs unbeknownst to individuals via perception of external stimuli, which not only alters beliefs or perception, but instead reaches the domain of action?
People are very interested in very quick images being flashed up.
Backmasking different to backwards masking. Beatles first to do it. Its where you play something backwards, record that, and then put it in the album.
Question is can this lead to priming?
What is public belief in the power of subliminals?
- 75% of Americans believe that subliminal messages are omnipresent in advertising, and that they work (Rogers & Seiler, 1994)
- Why?
- Vicary’s purported movie theater experiment in 1957
- Wilson Brian Keys claims of planted images in advertising
- Claims of subliminals in Disney movies and other media
- Media spoofs: In a Simpson’s episode, Homer receives a subliminal self-help tape which increases vocabulary instead of weight loss. He begins talking like Shakespeare
People do believe in this - they believe they occur and are very powerful despite advertising companies saying they don’t bother etc.
There is a belief that musicians etc are backmasking and putting in messages.
What is evidence for the power of subliminals?
• Meta-analysis assessed behavioural impact of and psychological processes associated with presenting words - revealed a small behavioural priming effect which was robust across methodological procedures (Weingarten et al., 2016)
Priming words, put a criteria for where awareness was there the study didn’t count. Controlled studies that may not extend outside the lab, but found some effects.
Betting where on fruit machines had the same 20 spins, for those who they displayed $$$ signs very briefly they tended to bet more.
What is the Stroop Effect?
• The effect was first demonstrated by J. R. Stroop (1935), who found that people required an average of 110 seconds to name the ink color of 100 words that were incongruent color names (eg, blue ink used in writing the word red). In contrast, people required an average of only 63 seconds to name the ink color of 100 solid color squares
• Since the original experiment, more than 400 additional studies have examined variations of the Stroop effect (e.g., MacLeod, 1991; Richards et al., 1992)
Older adult find the Stroop task to be even more difficult than do younger adults (Hartley, 1993)
People have to name the ink colour of words as quickly as possible.
• The effect was first demonstrated by J. R. Stroop (1935), who found that people required an average of 110 seconds to name the ink color of 100 words that were incongruent color names (eg, blue ink used in writing the word red). In contrast, people required an average of only 63 seconds to name the ink color of 100 solid color squares
• Since the original experiment, more than 400 additional studies have examined variations of the Stroop effect (e.g., MacLeod, 1991; Richards et al., 1992)
Older adult find the Stroop task to be even more difficult than do younger adults (Hartley, 1993)
How do we explain the Stroop Effect?
- Most promising account is provided by a parallel distributed processing approach (e.g., Cohen et al., 1990): Stroop task activates two pathways at the same time. One pathway is activated by the task naming the ink color, and the other pathway is activated by the task of reading the word. Interference occurs when two competing pathways are active at the same time. As a result, task performance suffers.
- Usually find significant effect in reverse Stroop (identify word; ignore colour) although weaker; account for that with parallel processing, but with faster accumulation of evidence for word recognition
Two pathways interfering. There seems to be interference. Slight slowing.
What is modified (emotional) Stroop?
• Participants must name the colour of ink of ordinary words, or threat words related to the source of their anxiety
• High levels of anxiety impair goal-directed attentional system (Attentional Control Theory)
• Foa et al. (1991): people with PTSD colour name words related to source of trauma - Slowed more to trauma-related words
- Those who coped better showed less interference
People have started to look at people with clinical conditions, and how their attention doesn’t seem completely normal. This was done with stroop in people with fibromyalgia for example, people with pain do they slow down reading the colour of words which are very relevant to them and their condition. Lose some of ability to direct attention to certain stimuli and becomes stimuli driven rather than self-driven.
Has been seen in PTSD as well, words related to the source of the trauma. Much more interfering effect on the behaviour.
Healthy people seen to tend to avoid negative words and are quicker at naming the colours for these words.
The Amygdala initiates fast (automatic/unconscious?) “affective” responses through efferent pathways to the hypothalamus and other lower brain regions.
The Amygdala also relays info to the frontal lobes where deliberate response processing may be influenced unconsciously ( cognitive bias?)
What is the dot-probe task?
Idea of the dot-probe task (or Visual Probe Task; MacLeod et al., 1986) is to measure how strongly your attention is drawn toward and held by specific types of stimuli. Participants’ task is to identify location of dot(s) as quickly as they can.
Focus on fixation point and press up and down of where the stimulus appears. 2 words appear very briefly. Depressed people go to the negative word so quicker when the stimulus is in that direction.
(Distinguishing depression from dementia in later life: a pilot study employing the emotional stroop task.)
How is short term memory traditionally measured?
• Memory-span procedures:
• participant presented a sequence of items, required to repeat them back; start with 1 item, increasing number of items by 1 until participant make mistakes
• point at which the participant is able to recall all items correctly 50% of the time is designated as her/his memory span
Reading span task: originally developed by Daneman and Carpenter (1980). Task requires participants to read a series of sentences aloud and recall the final words
What are the various views on short term working memory?
- John Locke (1690) explicitly distinguished between temporary workspace for the “idea in view” and more permanent “storehouse of ideas”
- William James (1905) coined the expression “primary memory” as distinct from the storehouse of “secondary memory“
- Atkinson & Shiffrin (1968) referred to short-term memory as combination of storage and control processes
- Craik & Lockhart (1972) emphasised processing rather than structure in memory; nature or level of initial processing determines recall. Thus, memory was seen as a byproduct of cognitive processing rather than as a separate entity.
- working memory as multiple components: Baddeley (1992) refers to a brain system that provides temporary storage and manipulation of the information necessary for such complex cognitive tasks as language comprehension, learning and reasoning
What is the ‘Brown-Peterson” paradigm of short term memory (1959)
• participants had to recall trigrams (strings of three letters) at intervals of 3, 6, 9, 12, 15, or 18 seconds after the presentation of the last letter
after oral presentation of each trigram, participants asked to count backward by threes from a three-digit number spoken immediately after the trigram
What factors affect memory span?
• auditory presentation leads to larger memory span estimates than visual presentation
• rhythmic presentation is better than non-rhythmic presentation
recoding or chunking information; George Miller showed in his classic paper (1956) that memory span is determined by the number of ‘chunks’ or integrated items you need to recall, not the number of items presented
What is the model memory model?
Atkinson and Shiffrin (1968)
1. Short-Term Store Information maintained in an active state Verbal rehearsal (capacity = 7 ± 2)** Phonemic encoding 2. Long-Term Store More permanent record of experience Semantic encoding Cue-dependent Information is transferred from STS to LTS**
Goes through organs, once attended to goes into short term store.
Critical aspect of this model is the implication that the short term store is an anti chamber for memory proper. Must go through short term store in order to go into the long term model. Can only get into long term is stays in short term long enough and is rehearsed - this is critical aspect of their model.
Sensory stores decay rapidly. Short term store is forgetting via displacement. Capacity assumed to be very limited but unlimited in long term store and eventually becomes permanent.
What is evidence of the short term memory model?
• coding differences in long-term & short-term storage (Baddeley, 1964)
• brain-damaged (amnesic) patients:
1. had severe difficulty in learning new information but could recall information that they had learned prior to their injury
2. short-term buffer appeared to be intact but their long-term storage was grossly impaired (Teuber et al., 1968)
• different components of the serial-position curve in verbal free recall:
1. Immediate vs. Delayed Recall (decreased recency effect)
2. Speeded Lists (decreased primacy effect)
3. Incidental Learning (decreased primacy effect)
Had 3 lines of evidence supporting their models. Long term memory semantic coding.
Neuropsychology of brain damaged patients both ways. One incredibly short memory span (1 word eg) but long term memory still accessible as can remember things before the accident for example.
Others could never get things into long term memory - normal digit span in short term memory while rehearsing, but cant keep it once stop.
Their short-term buffer also appeared to be unimpaired in that they could store sequences of digits and maintain the sequence by verbal rehearsal. However, as soon as rehearsal stopped or was prevented, the sequence was forgotten. Short-term buffer also appeared to be unimpaired in that they could store sequences of digits and maintain the sequence by verbal rehearsal
What is recall performance?
Postman and Phillip (1965)
- Immediate vs. Delayed Recall
No distractor task after last item à Strong recency effect
15-s distractor task after last item à No recency effect - Final Free Recall
Recall all words from all lists
Plot # recalled from each serial position
“Negative recency” effect
Found in general task with immediate recall people good at first and last few words. First as rehearsed long enough having commanded attention from the start, and last few because recency effect still in short term store haven’t been displaced yet. If delay the recall, give distractive task for 30 seconds, get rid of their recently effect of those in short term store but still got those in the long term memory.
What were the three outcomes of free recall performance (Craik 1970)?
- Overt Rehearsal
Subjects asked to rehearse out loud
First few items receive the most rehearsal - Incidental Learning
Subjects unaware of impending memory test
No primacy effect - Speeded Lists
Less opportunity to rehearse
Primacy reduced, recency unaffected
What are the failures of the modal model?
• under this model, multiple working memory tasks should limit working memory capacity
• not the case; tasks having to do with lexical information do not limit the capacity to perform visualization tasks
• numerous other studies have shown that normal adult subjects can simultaneously carry out two tasks, each of which would apparently place heavy demands on a single flexible working memory system
• several studies have shown that estimates of the difficulty of performing tasks are on their own poor predictors of how readily those tasks can be performed concurrently
• K.F.: Suffered injury to the left parieto-occipital regions and showed severe limitations in verbal STM (digit span of 2)
• but K.F. could get information into LTM and retrieve info. How?
• information had to be presented visually; auditory information was not encoded
• thus, likely there are must be “multiple-components” to WM
• K.F. had an alternative route into long-term memory
(a) that did not involve working memory and/or
(b) there are several working memory subsystems, not all of which are damaged in patients such as K.F.
Too much focus on cognitive structures and not enough on processing information. People get good at certain types of tasks. Impossible with one short term store - either got to capacity or didn’t, cant use different modalities according to this model.
Can have incredibly localised lesions - eg oliver sachs
Seemed that there must be multiple components to the working memory and the model was far too simple.
Second, I shall argue that working memory is better thought of as a system that operates after access to long-term memory has taken place, rather than acting as a means of transport for sensory input to long-term memory. On this view, working memory is seen as a workspace rather than a gateway, and sensory input reaches working memory via long-term memory, not the other way around.
Suffered injury to the left parieto-occipital regions and showed severe limitations in verbal STM (digit span of 2)
K.F. could get information into LTM. How?
Information had to be presented visually Auditory information was not encoded Thus, there must be “multiple-components” to working memory
Despite having a digit span of just two items, K. F.
appeared to have normal long-term learning and retrieval.
How can we assess working memory?
Dual-task methodology
Participants perform 2 tasks at once with two assumptions
• if tasks use same component, they cannot be performed successfully together
• if two tasks use different components, it should be possible to perform them as well together as separately
Common results
• visual tasks interfere with visual retention (eg, football/driving)
• verbal tasks interfere with verbal retention (eg, articulatory suppression)
• demanding verbal/visual tasks interfere with reasoning (tap executive function)
Implications:
• multiple short-term memory systems
• articulatory loop vs. visuo-spatial scratchpad (VSSP)
About Baddeley’s working memory model
The original model of Baddeley & Hitch was composed of three main components; the central executive which acts as supervisory system and controls the flow of information from and to its slave systems: the phonological loop and the visuo-spatial sketchpad. The phonological loop stores verbal content, whereas the visuo-spatial sketchpad caters to visuo-spatial data. Both the slave systems only function as short-term storage centers. In 2000 Baddeley added a third slave system to his model, the episodic buffer.
What is the phonological loop?
- responsible for speech coding
- consists of two components: Phonological store and articulatory control process
Idea that the distinction between speech inputs and non speech inputs. Serial recall of verbal information. Get preferential putting away to short term store. This is about a 2 second store of memory and only way can keep it there is by subvocally rehearsing the information.
Assumed somehow important in language learning particularly developmentally. Sequential information is good - not good for seeing array of objects, just visual or speech input
How does the phonological loop work?
- lexical information is coded and stored in the phonological store for about 2 seconds
- the stored information is refreshed by means of sub-vocal rehearsal
- the sub-vocal rehearsal is the articulatory loop
- assumed to have developed on the basis of processes initially evolved for speech perception and production
- suited to retention of sequential information (memory span)
Determined jointly by durability of memory trace and time required to refresh trace by subvocal rehearsal
What are the limits of the phonological loop?
- capacity (items): Seven plus or minus 2?
- not necessarily
- Miller’s magic number does seem to hold (even with chunking), however…
- rehearsal rate is the main determinant of capacity
- capacity (time): about 2 seconds
Some people are better at chunking information. Large literature, is it decay or interference that limit ability to short term shore. Rehearsal rates is what keeps it going, quicker can get through the list of words, the better you can recall. If the words all sound the same people don’t tend to recall them as easily as get mixed up and the phonemics are too similar - letters or words. Hard words take time - both to articulate and store.
Preventing people from rehearsing, eg saying ‘the’ over and over again while visually presenting the words. Stops linking as cant rehearse any more and this disrupts the memory of works enormously.
What is evidence for the phonological loop?
- the Phonological Similarity Effect (GPVT harder than RHXK)
- the Word-Length Effect (More monosyllabic than polysyllable words remembered)
- Articulatory Suppression (Overt or covert inner speech disrupts lexical memory)
- the Unattended Speech Effect (Lexical-based sounds disrupts lexical based tasks)
Speech inputs preferentially access the store and can disrupt the memory
What is the visuospatial sketchpad (VSSP)?
- maintaining and manipulating visuo-spatial information
- spatial orientation
- solution of visuo-spatial problems
- interface between visual and spatial information (from senses or LTM)
- bind visual information with motor/haptic/tactile information
Memory with spatial component, about remembering something that we visually see.
• Information can enter the sketchpad visually or through the generation of a visual image
• access to this store by visual information is obligatory
• the information in this store may be visual or spatial or both
• Not ideal for serial recall
What is evidence for the visual-spatial sketchpad?
- sketchpad disrupted by requiring participants to tap repeatedly a specified pattern of keys or locations, a procedure that impairs the use of visuospatial imagery (Baddeley & Lieberman, 1980)
- unattended patterns or visual noise may disrupt the visual component of the system (Logie, 1986)
- Corsi span (test in which task is to reproduce visuo-spatial sequences using set of 9 identical cubes mounted at locations on board) and auditory digit span impaired independently in patients with different lesions
Found that when people have to remember images of items, if have to follow a specified order they do much worse as disrupts ability to spatially remember sequences.
People who had parietal lobe problems, they tend to be good at auditory digit span but very bad at spatial memory.
Participants were given a visual tracking task: track a moving line with a pointer at the same they were given one of two tasks:
- To describe the angle of the letter F (which system did this task involve?)
- To perform a verbal task (which system did this task involve?)
They performed better in the second task Why?
What did Logie (1995) say about the visuo-spatial sketchpad?
• Logie (1995) argues that VSSP can be subdivided:
1. visual cache: stores info about visual form and colour
2. inner scribe: deals with spatial and movement information (and basic mechanism for rehearsal)
• stroke victim LH performed much better on memory tasks involving spatial processing than on tasks involving the visual aspects of imagery (eg, judging animal sizes)
Stroke victims are able to dissociate these two
What is the central executive?
- the ‘black box’
- drives the system; allocates the resources
- timesharing in dual task studies
- modality free: can manage information in any sense modality
- has no storage capacity
- limited capacity so cannot attend to many things at once
- temporary activation of long-term memory
Baddeley (1996) assumed four main functions of the central executive:
- the coordination of simultaneous tasks and task switching
- the control of encoding and retrieval strategies of temporarily stored information (also when retrieved from the long-term store)
- the selection of attention and inhibitory processes
- the retrieval and manipulation of long-term stored information
Model proposed as the governor - source of intentional control. Some temporary work space which is our consciousness that governs everything.
Like prefrontal cortex functioning - problem solving, manipulating. Modality free, not a storage model. But can easily load this part of the system.
Pulling information out of long term memory that might be useful. Selecting what to attend to and what not to be distracted by.
The central executive plays an important role in controlling attention. Our discussion of the central executive will begin with a discussion of the interplay of attention and memory
Takes decisions on how subsystems should be used
What was a study done on the central executive?
Baddeley (1996)
• asked participants to think of random digits that bore no connection to each other (by tapping in numbers on a keyboard) Either carried out on its own, or with one of the following tasks:
1. Reciting the alphabet
2. Counting from 1
3. Alternating between letters and numbers e.g. A1 b2 c3
• generated number stream was much less random in condition 3 – Baddeley said they were competing for the same central executive resources
One good way of loading central executive - ask someone to keep producing random numbers and after a while it gets very hard and to not get redundancy which is pairs which keep going together.
Verbally alternating between words and numbers puts more stress on and fail at one of the tasks.
Similarly when load extended memory span start putting pressure of central executive, higher order of brain gets pressure. Redundancy goes up when trying to generate random numbers and trying to do two tasks at once.
What is evidence of the central executive?
Randomness of digit generation (greater redundancy means reduced randomness) as function of concurrent digit memory load (Baddeley, 1996)
What are the neural structures of working memory?
- executive part of Working Memory involves the prefrontal lobe
- verbal part - such as rehearsing words or numbers silently - involves speech areas of the cortex (especially dominant hemisphere; eg Broca & Wernicke’s areas)
- visual part — such as visual imagery to think about how to walk from one place to another — seems to involve visual regions, including the occipital lobe
But across neuroimaging studies, STM/WM tasks often activate areas of the brain that also are involved in LTM (Jonides et al., 2008)
Pre frontal cortex basis for a lot of executive function. Verbal part really hard - is a functional loop, Brocas and Wernickes.
Some argue that long term memory activated by short term memory.
What are the problems with the 3-component model?
• Articulatory suppression
○ saying ‘the’ repetitively (occupying the phonological loop) does not have a devastating effect on recall of visually presented numbers
○ recall drops from 7 to 5 digits
○ one might expect recall to drop dramatically because Phonological loop is occupied and VSSP is not very good at storing this type of information
• patients with grossly impaired short-term phonological memory with auditory span of only one digit can typically recall about 4 digits with visual presentation
• prose recall of a patient (PV) with word-span of 1 word is 5 words. This is less than the span of 15 words, but much more than 1 word
○ are sentences stored in PV’s LTM? Implausible because PV has normal LTM
What is the binding problem of working memory?
- Binding problem
- information that is processed independently by separate cognitive processes must be bound together because our experience of the world (and our memory of it) is coherent
- people can also retrieve information about an episode when given part of an episode (e.g., given a spatial cue, state what object was stored there)
- episodic buffer is one way in which the binding problem can be solved
What are the properties of the episodic buffer?
- Episodic buffer
- integrates info across modalities and from different sources
- limited storage capacity
- buffer retrieval through conscious awareness
- originally thought to be capable of manipulating information; but now thought to be a passive structure on which bindings achieved elsewhere can be displayed
What is the episodic buffer of working memory?
• accounts for finding that amnesics can retain relatively large amounts of complex information briefly (e.g., sentence span)
• allows executive processes to carry out further manipulation
• attention useful for maintaining:
○ visual bindings (eg, objects’ colour & shape)
○ verbal bindings (eg,words into chunks)
but binding process itself largely unaffected by attentional load
• although concurrent tasks (including those with executive components) impair overall memory performance, they do not interfere with the binding process itself (for a review see Baddeley, 2012)
What are the ongoing controversies of working memory?
Linking long-term and working memory
• limited links with LTM in early multicomponent models
• Ericsson & Kintsch (1995): people utilize previously developed structures in LTM as a means of boosting WM performance
• number of approaches describe working memory as activated LTM (e.g., Cowan’s Embedded Process Model; Cowan, 2005)
• phonological loop likely to depend on phonological/lexical representations within LTM as well as procedurally based language habits for rehearsal
• Baddeley (2012): argues these studies clear example of way in which WM and LTM interact rather than a separate LTM system
What are outstanding issues of working memory?
- Episodic buffer: how to measure capacity? What extent is the buffer limited by number of and similarity between chunks? How is rehearsal maintained?
- Baddeley assumes that the buffer provides access to conscious awareness; does this mean that we are not directly aware of the other subsystems?
- are there separate subsystems for smell and taste?
- VSSP: visual and spatial aspects appear to be clearly separable but linked within the sketchpad; is this true of haptic, tactile, and kinesthetic memory?
- to what extent is the phonological loop used for remembering nonverbal material such as music or environmental?
About working memory and fluid intelligence…
• Fluid intelligence: “Gf reasoning abilities consist of strategies, heuristics, and automatized systems that must be used in dealing with novel problems, educing relations, and solving inductive, deductive, and conjunctive reasoning tasks” (Horn & Hofer, 1992)
commonly stated that approximately 50% of the true score variance between WM Capacity and Gf is shared
WM training programs; claims of profound beneficial effects on children’s academic and intellectual attainment
WM training produces limited benefits in terms of specific gains on short-term and working memory tasks that are similar to training programs, but advantages for academic-based reading and arithmetic outcomes less clear (Au et al., 2015; Redick et al., 2015)
Can we train working memory?
• Performance task on WM task predicts performance on other cognitive tasks
e.g. performance on reading span task correlates well with reading comprehension
Working memory refers to a brain system that provides temporary storage and manipulation of the information necessary for such complex cognitive tasks as language comprehension, learning and reasoning.
(Baddeley, 1992)
Associated with reading (Gathercole & Pickering, 2000) and mathematic (Geary et al., 2004) ability
10-15% of all students have working memory deficits
(Alloway et al., 2009)
Children with poor working memory make poor academic progress
Of 300 children with poor working memory (Gathercole & Alloway, 2008):
83% scored poorly on either reading or maths tests
the vast majority of these scored poorly in both areas
What is the definition of personality disorder?
- ICD-10 (WHO, 1992)
- Enduring and deeply ingrained ways of behaving, thinking, feeling and relating
- Deviate significantly from the norm
- Sufficient to cause significant personal and social distress and disruption
- Usually present since adolescence or childhood and persists throughout most of adult life.
Effects every domain of function - really pervasive.
A lot of people have bizarre personality traits but they find their place in society, jobs etc - with PD it must cause some of personal distress of hindrance. If weird but fine then don’t have PD.
Also needs to be clear that the disorder was present at some point during adolescence. Been going on for a while.
How do ICD and DSM cluster personality disorders into groups?
Cluster A = odd
Cluster B = difficult
Cluster C = anxious
Might consider cluster first then work back to the specific PDs after that during diagnosis.
What are the problems with classification of personality disorder?
- Most patients meet criteria for >1 PD
- Extreme heterogeneity within PD diagnoses
- Arbitrary diagnostic thresholds
- Poor coverage (PD NOS the most common)
- Poor convergent validity
- Longitudinal course more like Axis 1 than previously realised
- Problems communicating about dimensions to other clinicians.
What does the DSM 5 say about personality disorder?
- PPDWG Proposal
- Hybrid categorical dimensional model
- Evaluation of impairments in personality functioning
- Six specific patterns of traits: borderline; obsessive-compulsive; avoidant; schizotypal; antisocial; narcissistic
- “PD – Trait specified” if fail to meet criteria for specific PD
- Rejected at last minute by the APA Board of Trustees
- Placed in Section III, “Emerging Measures and Models”
- Retains DSM-IV diagnostic criteria
- Single axis system
Tried to think about a combination of a dimensional approach with some categories, but it was rubbish
What was the McLean study of adult development?
• McLean Study of Adult Development Zanarini et al. (2006)
• Prospective study of borderline PD
• 70% meet remission criteria at 8 years
• ~6% of remissions experience recurrence within 8 years
• Different symptoms resolve at different rates
○ Impulsivity resolves most quickly, followed by interpersonal, cognitive and then affective symptoms.
This contradicts the idea that it is a diagnosis for life. Behavioural symptoms seem to resolve early on, but emotional issues are more enduring.
What is the prevalence of personality disorder?
- Community
- 10-13% (2% antisocial PD) De Girolama and Dotto (2000)
- Primary care
- 10-30% (Cluster C most common) Moran et al. (1999)
- Inpatients
- 36% Pilgrim and Mann (1990)
- Drug and alcohol services
- 78% of alcohol inpatients De Jong et al. (1993)
- Prison
- 60-70% Singleton et al. (1998)
What sit eh border of personality disorder?
- Mortality and accidents
- Mental illness
- Poor treatment outcome
- Increased service utilisation
- Antisocial behaviour
- Deliberate self-harm
- Suicide
Outcome of any other disorders is made worse by PD - not go to appointments, into take medications properly, self harm, etc.
Tend to use services more, don’t go to monthly appointments but go to A+E, police etc - use all the services they shouldn’t be
Self harm more common in some forms of PD than others, in particular borderline PD.
About personality disorder and violence…
• Personality disorder in high risk populations
• Mean 3.6 in high secure hospital/prison sample.
• Violence in epidemiological samples
• Cluster B: x10 criminal convictions; x8 time in prison.
• Any PD + substance dependence – 52% violent in previous 5 years.
• ~1:2 ASPD not violent in previous 5 years.
• Violent vs. non-violent offenders
• ñASPD in violent offenders (23.6% vs. 14.6%)
• ASPD corr. with violent convictions in violent offenders
• Cohort studies
• Cluster A in adolescence associated with burglary and threatening behaviour.
• Cluster B (excl. ASPD) associated with any violent act, including arson, vandalism, fights, robbery.
• Role of alcohol in mediating relationship.
See Duggan and Howard (2009) in McMurran and Howard (eds.) for review.
What specific personality disorders have a link with violence?
- Four dimensions operate as clinical risk factors for violence:
- Impulse control
- Affect regulation
- Narcissism
- Paranoid cognitive personality style
- ASPD and BPD selectively co-occur
- Co-occurrence increases with security level.
- Cluster C PDs may be protective
- Particularly obsessive-compulsive personality disorder
Poor impulse control, regulating emotions, rules are for other people/exploiticism/only few people understand you (narcissism), hypervigilance, thinking people will attack you so you pre-emptively attack etc.
Antisocial and borderline PD tend to co-occur, diagnostic system not very discriminative and people often have more than one kind. Increases risk of being admitted to high security hospitals.
Always look for functional impairment, don’t exclude other types etc.
What is the assessment of personality disorder?
- Interview-based measures
- International Personality Disorder Examination (IPDE)
- Structured Clinical Interview for DSM-IV Axis II Personality Disorder (SCID-II)
- Self-report measures
- Millon Clinical Multiaxial Inventory (MCMI)
- Minnesota Multiphasic Personality Inventory Personality Disorder Scales (MMPI-PD)
Prompting the accurate answers, eg asking if other people think they are a certain way in which they might say yes, whereas if you asked if they thought they were they would completely disagree as they don’t see it.
Self-report measures important as in time constraints at appointments they can have sent back response before hand so got an idea beforehand. Not as systematic as interviews but still a useful tool.
What is borderline PD?
- Frantic efforts to avoid abandonment
- Unstable and intense interpersonal relationships
- Identity disturbance
- Impulsivity
- Affective instability
- Chronic feelings of emptiness
- Difficulty controlling anger
- Transient psychotic or dissociative symptoms
- Recurrent self-harm and/ or suicidal behaviour
Anxiety of abandonment in relationships - can manifest through appointments also, don’t leave on time, don’t come, etc. relationships tend to be quite unstable and intense.
Anger is disproportionate.
If asking about self-harm try and figure out the meaning of it for them, could be a way of managing emotions, could be because they feel chronically empty and want to feel something, may be attempts to manipulate relationships such as the doctor seeing they need to carry on looking after them etc.
How do we measure PD outcomes?
- Methodological problems
- Lengthy evaluation period required
- Treatment rejection
- Multifaceted; comorbidity
- Lack of consensus on outcome measures
- Outcome measures
- Symptoms; personality
- Quality of life; social functioning
- Behaviour; recidivism
- Service use
Some people are treatment rejecting - don’t think they’ve got a problem, actively reject, schizo don’t interact with other or seek out treatment.
Lots of different parts to the diagnosis so might get some areas really improving and others seeing no change.
Meta-analyses, only really possible is using the same kinds of outcome and that doesn’t work in PD research, looking at all sort of different outcomes only recently have they begun looking at the same things to get the results
Quality of life, relationships improving, working and jobs, other community integration measures.
Behavioural includes self harm, accidents, reoffending behaviour.
The best studies have looked at one outcome in at least 4 or 5 of these areas. Tricky but manageable and improving.
Where is the evidence for PD?
- ‘What works’ literature
- Behaviour focused
- Ignores PD
- Mental health literature
- Non-offenders
- Usually borderline PD
What works literature is the prison literature form offending behaviour programmes looking at how effective they are. They are relevant because the vast majority of people engaging in them have PD and the focus is on reducing behavioural issues.
Mental health literature less on prisoners and focusses on general borderline PD.
What is the ‘what works’ literature on PD?
• Behaviour focused – ignores PD diagnosis
• ~ 10% reduction in reoffending overall
• Evidence for CBT, relapse prevention and multisystemic therapy
• Evidence for DTCs if stay >18 months and high risk
• Drop outs do very badly
• Low risk offenders do worse in treatment
See Craig et al. (2013) for review
Focussing purely on behavioural.
Most people in prison have PD - exceptionally high prevalence
Underwhelming results - 10% reduction - might expect a lot more.
Therapeutic communities - residential treatment programmes. Daily community meetings, real effort in giving residents responsibility and making them accountable of their behaviour. There are 3 or 4 now which are based in prisons, previously just in general community.
Those who drop out often do worse than those who don’t have any treatment at all, this is why it is important to consider what treatment methods might be best for each individual.
Can be destabilising, if someone is low risk might have less of an impact.
What is the mental health literature on PD?
- Non-offenders; usually borderline PD
- Evidence supports the following manualised treatment models:
- Mentalisation-based treatment
- Transference-focused psychotherapy
- Schema-focused therapy
- Dialectical Behaviour therapy
- Cognitive therapy
- Cognitive analytic therapy
- Systems training for predictability and problem solving
- Evidence from cohort studies for therapeutic community treatment
Whole range of model based on a rang elf things
What is mentalisation based treatment (MBT) for PD?
MBT - mentalisation based treatment
• Borderline PD (?antisocial PD)
• Enhancing mentalisation will promote emotion regulation and more adaptive cognitive functioning
• RCT MBT vs. treatment as usual (5 year follow-up):
• Suicide attempts (23% vs. 74%)
• Reduced A&E visits, hospitalisation and OPD
• Reduced medication use(0.02 vs. 1.9 using 3+)
• BPD diagnosis (13% vs. 87%)
• Global functioning (45% vs. 10% GAF >60)
• Vocational status (3.2 vs. 1.2 years employed/in education)
Bateman and Fonagy (2009)
Mentalisation is relatively new therapy model based on attachment theory formed by psychodynamic thinking. Try and understand own and other peoples minds through what they’re thinking/feeling/their intentions/what’s going on in their mind.
Evidence that its really effective in BPD, trail in antisocial PD
About trying to get people to identify when they’ve stopped mentalising and need to start improving this.
Outcomes are quite pervasive - reduced suicide attempts, medication use, symptoms, improved function, more likely to be in employment.
Been followed up for 8 years now - works and persists.
Quite often know when not mentalising as something comes unexpectedly from an interaction - haven’t kept and open mind and curiosity about their intent and how they might feel.
What are definitions of metallisation?
- The mental process by which an individual implicitly and explicitly interprets the actions of himself and others as meaningful on the basis of intentional mental states such as personal desires, needs feelings, belief, and reasons.
- To see ourselves from the outside and others from the inside
- Understanding misunderstanding
- Having mind in mind
- Introspection for subjective self-construction – know yourself as others know you but also know you subjective self (your experience)