Practice ECG (KWH AED examples) Flashcards

1
Q
A

Bradycardia
Atrial rate: 76
Ventricular rate: 38
2nd degree of heart block

2:1 heart block

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2
Q

What should patient receive?

A

Rate: 48 bpm
ST elevation in II, III, aVF, reciprocal changes in lead I, aVL, V1-V6

Inferior STEMI

Need to r/o aortic dissection before giving nitrates

AD dissecting to RCA can lead to this picture

For general STEMI

Very short: PCI
Very long: still PCI, but delayed

3rd degree heart block (look at lead II) = right coronary artery is blocked, proximal to take-off of AV nodal branch (no blood going to AV node)

V1 has alternating QRS complex: RBBB = RSR’

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3
Q

What treatment?

A

Wenkebach heart block = Mobitz I

PR interval gets gradually longer until a beat is dropped

Bradycardia

Wenkebach is usually benign = does not need pacemaker

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4
Q

What treatment?

A

Complete heart block (complete AV dissociation)

Atrial rate:

Ventricular rate:

Mobitz II and type 3 heart block NEED pacemaker

In complete heart block
1. QRS complex should be regular (If it is irregular, some PR waves may be conducted to the ventricles)

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5
Q

Is narrow or wide QRS safer?

A

Narrow: Junctional (MORE STABLE, safer)
- Usually escape is coming from site of blockage
-
Wide: Ventricular

May need pacing: place wire into ventricles

If patient has junctional escape: permanent pacemaker

If there is wide QRS, put in temporary wire pacing ASAP = fatal

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6
Q

Patient above develops this

A

Tachycardia = Torsade de Pointes (quite organised, V fib is disorganised)

QT interval is refractory period. There is a window inside the refractory period that is prone to attack. = Long QT syndrome

In patients with bradycardia, the refractory period is prolonged.

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7
Q
A
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8
Q
A

RCA has AV nodal branch.

If blockage is proximal to AV nodal branch, RCA occlusion can cause complexe heart block

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9
Q
A

Inferior MI: aspirin, clopidogrel, LMWH, thrombolytics

If patient has aortic dissection, patient will die

Be careful of inferior MI pattern

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10
Q

1st degree HB

A
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11
Q

c

A

Look at V1: You can see the P waves

PR relationship

Sometimes WRS comes shorter

Bigeminy: once every 2 beats = extra heartbeat between every normal one

Supraventricular/atrial: QRS morphology is narrow = frequent premature atrial ectopics

Benign = only treat is patient is symptomatic, or very frequent

All the PR complex looks weird, but they’re weird in unison (they all look the same)

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12
Q
A

Atrial flutter, 5:1 block

Negative saw tooth pattern

Postitive over aVF

R-sided

Usually involve R atrium only, in counter-clockwise pattern

Shortest distance that flutter wave should pass. Put line across CS = Tachycardia will stop

Go into heart via IVC, then go into right atrium, to the tricuspid valve.

Put line across tricuspid valve to IVC = isthmus is the most narrow part, ablate across the isthmus. Put some heat energy and damage the muscle = does not allow electrical activity to pass through

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13
Q
A

150 bpm for ventricular rate
Rhythm is regular
QRS = 160 m/s
Wide complex tachycardia

Give adenosine:
1. Terminate the tachycardia (once you block the node, tachycardia either stops, or something is unmasked)

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14
Q
A

First column: tachycardia
Second column: effect of ATP

Increased frequency of P wave

After giving ATP, there is some P flutter wave abnormality (morphology is different but mechanism is simmilar)

Something above AV node

Atrium itself has tachycardia. Even if you block AV node, it won’t affect tachycardia.
AV node–indepent tachycardia
AVRT and AVNRT = AV node dependent tachycardia

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15
Q
A

Delta wave
PR interval shortened
Wide QRS (due to prsence of delta wave)
Wolff-Parkinson-White

Delta = pre-excitation, excitation of ventricles via accessory pathway

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16
Q

What are 2 drugs that you cannot give to WPW?

A

Beta blocker: Atenolol, metopolol
CCB: Verapamil

17
Q
A