Poultry & Aviary Birds Flashcards

1
Q

Passerines v. non-passerines

A

*Passerines: Songbirds, perching birds– real definition: structure of the foot- 3 toes direct forward free of webbing and one toe directed backwards

* Non- passerines: Waterbirds with webbing or parrots with two toes directed forwards and two directed backwards

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Galliformes

A

turkey, grouse, chicken, quail, partridge, pheasant

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Columbiformes

A

Pigeons and doves

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Psittaformes

A

Parrots (psittacines)- 393 species- mostly tropical and subtropical

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Falconiformes

A

species of diurnal birds of prey

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Anseriformes

A

Ducks and geese

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Raptors

A

Same as falconiformes?? Kites, kestrels, hawks, falcons, eagles

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Ratites

A

Ostrich, emu, rhea, cassowary

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Praecocial vs. Altricial birds

A

Praecocial birds are those whose young can feed themselves as soon as they are hatched e.g. domestic fowl, ducks, ostrich

* Altricial birds- young require intensive feeding by parents for some time after hatching e.g. pigeons, budgeries

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

With renal disease in birds, what other system might be affected due to proximity?

A

Air sac– or vice versa. Kidneys are in close association with abdominal air sac- ventral surface of the kidney forms part of the dorsal wall air sac

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

What is the avian blood supply to the kindey? What is unique about bird blood supply with IM injection to the leg?

A

3 renal arteries plus the renal portal vein– valves associated with external iliac vein and vena cava

** IM injection into the leg may be delivered directly to the kidney and possibly excreted before entering systemic circulation therefore fail to achieve therapeutic concentration in systemic circulation; nephrotoxic substances may result in severe renal toxicity since direct dose

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

How is uric acid excreted in birds? How is this different to mammals?

A

* Uric acid excreted via tubular secretion independent of GFR– excreted with faeces (no loop of henle)

* mammals- urea excreted in urine

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

What are the functions of the avian kidney?

A

* Excretion of uric acid, control of electrolytes, fluids

* Water resorption by renal tubules, etc.- uric acid precipitation in lower aspect of ureter

* Urinary water may be resorbed by cloaca/ rectum/ colon etc. by antiperistaltic movement of renal excreta into rectum/ colon etc.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

When does polyuria occur in birds?

A

stress, prior to egg laying, nephrosis e.g. heavy metal poisoning, diabetes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

What is the normal color of urates? What does it mean if they are yellow/orange/brown? Green? Sick?

A

* chalky white to cream colour

* Yellow/orange/brown: B-complex vitamin injections

* Green: liver disease resulting in failure of excretion of biliverdin via bile results in excretion via the kidney with green colouration

* Sick: cream-pale yellow– depressed, sick, anorexic

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

What is the most likely ddx with haematuria, haemoglobinuria, porphyrinuria in birds?

A

* heavy metal poisoning

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

What is visceral gout?

A

Deposition of urates on epicardium and peritoneum especially over liver capsule, associated with acute renal failure

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

What is articular gout?

A

Deposition of urates on joints and synovial sheaths, usually associated with chronic renal failure but sometimes without renal disease (e.g. idiopathic, genetics, high protein diet)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

What is renal gout?

A

Tophus formation (tophi), foci of acute inflammation associated with uric acid crystals in interstitial tissues of kidney, renal tubule degeneration/rupture- crystals into interstitial tissue- tophi

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

Causes of pre renal disease?

A

Dehydration, anaemia (E. nectarix, E. tenella coccidioisis)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

Causes of post renal disease?

A

Obstruction/ partial obstruction of ureters such as urolithiasis or impaction of cloaca and/or pelvic canal with faecoliths (constipation), uroliths, FBs, egg impacted, distended oviduct, cloacitis, prolapsed cloaca

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

What are common congential conditions of the kidneys in birds?

A

* absence of one or more lobes, renal cysts, and inherited metabolic abnormalities e.g. reduced uric acid clearance

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

What does toxic nephropathy in birds normal result in? What are some causes of toxic nephropathy?

A

** Acute or chronic nephrosis

* Heavy metal toxicity, mycotoxins (ochratoxicosis), medications (aminoglycosides, sulphonamides), excess dietary calcium (young chickens), hypervitaminosis D (also through hypercalcaemia)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

Common metabolic nephropathies in birds

A

* Associated with fatty liver – kidney syndrome or renal amyloidosis (amyloid deposition in kidney similar to that in liver) or urolithiasis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
What is urolithiasis? Consequences? Usually affects? Cause?
\* Obstruction of one or both ureters by uroliths \* Often leads to atrophy of kidney, subsequent hypertrophy of unaffected kidney, and renal and/or visceral gout if both kidneys compromised \* Usually seen in adult laying hens but also occasionally in younger birds \* Cause is unknown but Infectious Bronchitis Virus is suspect, Excess dietary Ca and mycotoxicosis
26
What are viral infectious diseases of the kidneys in birds?
\* Infectious bronchitis virus- some strains are more nephrotropic than others \* Infectious bursal disease \* Avian nephritis virus (astrovirus- enterovirus)-- associated with nephrosis and visceral gout, lesions include degeneration/ necrosis of tubular epithelium, heterophils, and lymphocyte infiltration, interstitial nephritis and fibrosis \* Polyomaviruses in budgies- causes inclusion bodies (e.g. proteins typically represent sites of viral multiplication) in glomeruli and/or epithelial cells lining convoluted tubules
27
Bacterial infections that impact the kidneys in birds
\* Septicaemic diseases that localize in the kidneys \* Ascending infections of the ureter- pyelonephritis- cage layer hens (trauma to cloaca due to vent pecking) \* extension from abdominal air sac disease- fungal nephritins
28
Protozoan infections that impact avian kidneys
\* renal cocciodiosis in geese and little penguins \* Short-tailed shearwater (mutton birds)-- limey disease- causes focal pale areas in kidneys due to urate retention
29
Helminth infections in avian kidneys
\* Flukes within ureter such as Renicola spp or within renal veins e.g. Schistosome (Austrobilhazia spp etc) causing granulomatous inflammation of ureter (ureteritis), possibly leading to obstruction
30
What is the most commonly seen companion bird with what type of neoplasia? What can occur?
Adenocarcinoma middle aged to older budgerigars (3-4 years old or more)--\> pressure on sciatic nerve plexus--\> paresis/ paralysis
31
What is unique about the female avian repro system?
Most species left ovary and left oviduct are active, right ovary and oviduct are vestigial
32
What does stimulation of ovarian development depend on?
\* Environmental factors \* Exposure to light- increasing photoperiod, minimum photoperiod below which ovary is inactive \* Moisture, nest availability, etc. \* Blood Ca level-- higher during active egg production, reduction below level will result in ovarian inactivity
33
When does ovarian regression occur?
\* Seasonal during winter \* Or adverse conditions such as nutritional stress or disease stress
34
What can ovarian regression lead to?
\* rupture of the yolk follicle--\> "yolk peritonitis" \* relatively bland condition unless yolk is contaminated with infectious agent OR if free yolk grow microorganisms over time \* if so can cause peritonitis \*\* worst case yolk embolism leading to brain infarction
35
What are ectopic ovules?
\* Ovules released from ovary that fail to be inducted into oviduct \* can be free within the peritoneal cavity without problem.... or can be caseous adherent to peritoneum causing peritonitis-- more severe inflammation
36
Ectopic eggs?
\* shelled or non-shelled occasionally found free within the peritoneal cavity implying rupture in oviduct-- may be associated with obstruction of oviduct \* Egg peritonitis can result from severe salpingitis
37
Oophoritis in birds?
\* Usually caused by bacterial infection of ovary, localization in ovary from septicaemia e.g. from Salmonella pullorum, S. enteritidis, Pasteurella multocida
38
Common ovarian tumours of birds?
\* Ovarian adenocarcinomas- metastasize, invade peritoneal lymphatics--\> blockage--\> ascites--\> distended abdomen. \* Oviductal adenocarcinoma \* Lymphosarcomatous neoplasms or oviductal adenocarcinomas that metastasized to ovary \* Marek's disease \* Lymphoid leucosis
39
Hypoplasia/ Segmental aplasia of the oviduct
\* often caused by Infectious Bronchitis Virus (IBV)
40
Degeneration of oviduct? causes?
\* effects epithelium of oviduct and result in abnormalities of albumen, internal egg quality, eggshell thickness/ quality, pigmentation or lack of \* Causes: nutrition e.g. inadequate Ca, Toxic effects e.g. chlorinated hydrocarbon insecticides, IBV
41
Egg Drop Syndrome? Causes? Transmission? Signs?
\* Caused by EDS viruses, haemagglutinating adenovirus, IBV, Newcastle disease virus, heat stress (common in AUS), acute toxicities e.g. sulphonamides \* Viruses commonly associated with waterfowl \* Transmission: vertical, lateral between flocks, spread from ducks/geese (domestic or wild) or via drinking water contamination by such birds \* Signs: loss of egg shell strength/ pigmentation, slightly depressed 1-2 days, protective antibody but flock without previous antibody sudden fall in production
42
What are feathers purpose?
\* Display, insulation, waterproofing, protection, sensory function, flight
43
What is important about feather tracts?
Injections or procedures, must stay away from them
44
Bird molting
\* Natural - cyclic (annual or biennial) - continuous \*\* all dependent on species (e.g. Penguins lose them all at once and stay in the nest for a long period of time) \* Induced-- when they regrow their feathers which takes a month-- can use the same beds
45
PBFD transmission?
Transmission: vertical and horizontal \* Treatment: destroy the flock \* Diagnosis: histopath (biopsy with feather follicle in formalin) or PCR (PICK UP THE NEWER STUMPY FEATHERS)
46
Treatment of mites in birds
Ivermectin on the skin-- be careful not to overdo it as it can be fatal + remedicate in 2-3 weeks time (could also use Malathion wash/ dip)
47
Significance of mites in birds
\* dermatitis, anaemia, transmission of blood-borne pathogens
48
Backyard chicken presents with severe scabs and nodules on faces... what do you do?
Euthanasia and necropsy \* Bacterial colonies seen with histopath, as well as inclusion bodies in the cytoplasm \*\* Treatment: vaccination + antibiotics
49
50
Possible aetiology?
\* normal/ seasonal loss of feathers \* congenital defect \* nutritional \* feather picking \* infectious
51
Unique characteristics of bird skin
\* Skin is thinner \* Dermis is looser \* No sweat glands \* Not as vascularized \* No sensory organs
52
\* Dermis is extensive \* Epidermis is very thin
53
Highly cornified
54
55
Contour feather \* Vane of the feather \* Barbs and barbules important for flight
56
57
Considerations when clipping feathers
58
Clubbed down- a condition in chickens suffering from a nutritional deficiency of riboflavin: the erupting feathers do not rupture the feather sheaths properly, causing the feathers to have a coiled structure.
59
Feather cysts- subcutaneous accumulations composed of keratin \* Congenital defect
60
Causes of feather picking
\* Pruritis \* Systemic disease \* Boredom (95% of the time) \* Psychosis - sexual frustration - change of environment \* Overcrowding (especially chickens) \* Light intensity \* Nutritional (protein, sodium, (multi-vitamin))
61
Inclusion bodies in the inclusion layers in the feather follicles + inflammatory cells \* Circovirus (Psittacine Beak and Feather Disease) \* Cockatoos and Princess Parrots are very susceptible \* skin is dark
62
PBFD
63
64
\* Unable to perch or fly DDX PBFD
65
Budgerigar Fledgling Disease
66
Budgerigar Fledging Disease
67
Budgerigar Fledging Disease inclusion bodies, large and pale
68
Diagnosis of Budgerigar Fledgling Disease Diagnosis? Transmission?
Viraemic-- so secretions Control: - hygiene, quarantine and testing, isolation, vaccination (N. America, not in AUS)
69
What is this? Signs and symptoms?
70
Pox virus- though you can see viral dermatitis in other viral diseases
71
What will you see with Poxvirus?
Viral nodules
72
Pox virus
73
Pox virus-- with secondary bacterial infection Tx with antibiotics as well Clavulonic Acid to kill staph as well
74
Inclusion body- pox virus
75
Types of Poxvirus, which is not in Australia?
Parrot Pox not in Australia (at least rare)
76
Transmission of pox virus? Treatment?
Sheds from infected skin (nodules are full of virus- inclusion bodies) \* BUT Cannot penetrate intact skin therefore you need some physical trauma therefore disease happens in the warmer part of the year because of insects \* Minimum cross species transmission except pigeon pox--\> chickens \*\* Treatment: antibiotics and vaccination
77
Control of Pox Virus
78
Vaccination methods in birds
79
Staph bacterial infection
80
Blue Wing Disease (smell) Circovirus + Clostridium + Staph Septicaemic, die quickly
81
Mites- parasitic dermatitis- often Knemido- coptic
82
Mites- tasselfoot
83
Mites- scaly face + often causes overgrowth of the beak
84
Mites from a bird
85
Scrape close to the dermis until you see oozing-- the mites are close to the dermis \*\* oil first and then the scraping
86
Dermanyssus
87
Northern Fowl Mite (Ornithonyssus sylvarium)
88
Argas persicus
89
Lice, lay a lot of eggs-- not normally very harmful however
90
Treatment in birds of lice and mites
91
Signs of an ill bird
• Recommended formularies and dose rates • Pediatrics and aviculture 1.Handling/clinical exam/class of bird/wing clip and nail clip 1. ASSESSMENT OF THE SICK BIRD RECOGNITION OF SIGNS OF ILLNESS It often appears that birds die suddenly from their illnesses, however, in many cases this is not true. Usually it is a failure of recognition that there is a problem until it is advanced. Birds have a strong natural instinct to disguise illness (most species of birds are flight, fright prey species). In the wild, it is safer for a bird to look the same as the rest of the flock. Showing signs of illness (e.g. weakness) will make them a target for predation. However there are plenty of signs that can be seen early in the disease process for the vigilant owner or the experienced vet. This is a good reason for regular examinations. What do we look for?? A bird MAY be ailing if:- (a) The feathers are fluffed up, the head is drawn into the body or tucked under a wing. This is the classical sick bird look and the bird may have been sick for a considerable amount of time before this stage is reached. (b)It is inactive; i.e. not climbing or flying around the aviary or cage and not preening itself, or changes it's usual perching position. (c) It is not calling or responding to its mates calls. (d) The eyes have a tendency to close and do not open to a full round eye (looks sleepy). e. Movements of the head are slow. f. Appearance is not symmetrical, or posture is unusual. (g) It frequents the ground in the aviary or the bottom of the cage. (h) Breathing movements are noticeable or audible. Bobbing of the tail is a guide to respiration but varies from bird to bird. (i) The vent is soiled, (dirty bottom) ! 3 (j) If there is any discharge from the nostrils at all . (k) If a singing bird stops singing or has a change in voice. (1) It is not regularly eating or not passing normal droppings regularly over the day. (m) Excessive stretching of wings, and legs or shivering. (n) The body weight of the bird is declining. A sleeping bird may have some of these signs but a sleeping bird - a. Is found mostly in the late morning, noon or early afternoon. b. Will often be perching on one leg. c. Will wake to a slight sound and maybe stretch a wing and/or leg. Adduct it's shoulders and fly off, afterwards assuming a sleek appearance
92
What can you do for a stressed bird?
Put it in a warm, dark spot in the clinic
93
Important things to remember about bird handling
Does not use gloves unless handling birds of prey Like to get the birds out of the cage themselves Using towels and tea towels Do not hold chest tight because you'll compress air sacs needed to push air into the lungs Basic Handling Tips Useful equipment – towels (suitable size and thickness, I find tea towels really useful to use especially with small birds, upto Galah size). Avoid using gloves, they will significantly reduce your ability to properly hold and examine the bird and will result in a higher incidence of deaths as a result of reduced digital sensitivity. Nets can be useful if you have a wild escapee. Important things to remember - Parrot beaks hurt so have control of the head first. - Birds of prey can cause incredible damage with their feet. - pigeons need support of the body. - Chickens/fowl, control the wings and support the body - Swans are really good at pecking at eyes, so control the head. - Rough handling of a tame bird can cause significant trust issues with future handling not only at the vets but also with the owner. ! 4 -If you are seeing experienced breeders it may be better to get them to handle their birds, most pet owners however don’t know how to handle their birds and they often end up flying around the room for 10 minutes before you can get hold of them. It’s a good idea to explain to an owner how you are going to handle the bird and how this minimizes the stress to the bird. -Try to cause minimal stress, often birds need time to settle so get history and do observations first. Remove excess cage furniture to make things easier for you and the bird. -Have everything you need ready and handle the bird for as little time as possible to reduce stress especially if it is a sick bird
94
95
History taking with birds
• History taking - It is important to have a working knowledge of commonly kept species. This is not only useful in gain client trust but can be helpful in offering possible differentials of disease and treatment plans. Some species are more prone to certain diseases and some have specific husbandry requirements. - Sometimes it is helpful to have clients fill out questionnaires before their initial consultation. This can tell you a lot of basic information with out taking up to much consultation time. - Often a bird is stressed from the car ride into the clinic and may also be stressed from the waiting room, so consultation should always start with history taking and observation before there is any attempt to touch the bird. - Check list for history taking includes • age, not all owners know the age of their birds, the age can be confirmed if the bird has a ring • details of why the bird was bought in • information about diet – ask about every aspect of the diet. • how the bird is kept, is it an aviary bird? Has it been hand reared and how? Is it a breeding bird? • what is the birds daily routine? • what environment factors may be significant. • Is the bird in contact with other birds • Previous medical history – previous tests etc may be helpful. • Detailed history of husbandry, some diseases can be corrected by simply improving the husbandry. In the past 10 years there has been a great increase in knowledge of how to keep birds. It may make a considerable difference to the recovery to improve husbandry – cage location, sleep at night, available perches, toys to play with, branches to chew, access to UV light, what is the substrate at the bottom of the cage? • Client history forms – (see appendix) • Examination of cage and environment - Always encourage owners to bring in the cage as is (uncleaned) with the bird. This could provide essential clues as to why the bird is sick and what is causing the problem as well as clues as to the owners ! 5 understanding on husbandry care of the bird. Birds often become sick due to being kept in an inappropriate environment, very rarely will a client come in who you can’t teach better husbandry care for their pet.
96
Clinical exam of birds
\* Observing bird in the cage to look for abnormalities in behaviour, movement, body shape, respiratory depth and rate, birds response and reaction to the environment \*Give them some time to relax and adjust to the consult room \* beware of handling sick birds-- towel \* dim the light, make sure door and windows are closed
97
Put it down list
98
equipment in every consult room
99
Minimize stress when transporting a bird especially when ill
100
Nursing a sick bird
\* Body temperature for small finches/ canaries have a normal body temperature above 41 degrees C, normal heart rates of more than 500 beats/ minute, and breath normally 80-120 times/ minutes. \* Avoidance of stress is vital for recovery \* Warmth-- 30-35 degrees C for smaller birds, larger ones e.g. Cockatoos slightly lower temperatures (hot hair blower fan will seldom be satisfactory du to air movement-- cardboard box and a covered or red globe (100W)-- with a thermometer to ensure correct temp. Thermostat is better. Over 6-7 days the temperature may be reduced gradually. \* Darkness \* top quality food and water- seed, grass, cuttle fish, thistle, seeding grassed.. place near sick bird e.g. remove perches and put food or water on the floor \*Isolation- esp separated and separate food and water containers \* Monitor droppings-- keep a written record-- number of droppings, consistency \* Remove grit from bird's cage-- depraved appetite end up engorging themselves on grit resulting in impact of the gizzard and proventriculus. Grit, unlike minerals do not dissolve in the GI tract \* Exercise- playing and free flying should be avoided until full recovery-- avoid bathing
101
Used for crop feeding
102
Keeping the beak open for example, throat swab for Chlamydia
103
Unique anatomy and physiology
• Unique bird anatomy. - respiratory system, no diaphragm, air sac system (9 air sacs in most birds), tubular lung tissueattached to ribs and rigid tracheal rings. - integument – thin skin (10 cell layers) covered with feathers used for flight, temperature regulation, protection and display. - large keel bone resulting in a small abdominal area. - Skeleton – medulla-cortex ratio, pneumatic bones and no teeth. - Digestive system – crop, proventriculus and gizzard (with back and forward movement of food) and cloaca - Urogential system - Internal gonads and egg layers with 1 developed uterine horn. Kidneys attached to the backbone and pelvis, main waste product is uric acid and no bladder. - The anatomical and physiological difference between birds and mammals holds the key to understanding disease processors, how one might expect a disease to present itself and the best way to approach and diagnosis and treatments. A full knowledge of the differences can also help us realize that we can’t treat birds like little cats or dogs avian medicine is a specialty of its own.
104
\* All 10 on both sides to keep balance so they dont' injure themselves if they do fly (if you clipped one side) Just primary flight feathers
105
Pre-anaesethetic assessment bird
106
Bird anaesthetics
\* Isoflurance- rapid induction and recovery- safe and almost entirely excreted by the resp system. Induction at 3-5% via a face mask and a high flow rate 1-2 L/min... maint at 1.5-3%. \* O2 flow rates about 150-200 ml/kg/min \* injectable unfavorable compared to inhalation anaesthetics especially in small species but may be needed in large species e.g. Ostrich... ducks and geese and other diving birds have a breath holding mechanism which can make inhalation anaesthetics tricky
107
Bird premeds
108
Bird intubation
109
Radiology positioning
\* Manual restraint with protection \* taping conscious bird to film or cassette. Elastoplast is effective but will damage feathers more on removal and may reduce detail. \* Do not tape across the chest. \* can also tape a bird to a Clear plastic sheet placed over the film-- but may not be possible without anaesthetic \*\* Specific bird restraints made with plastic flexiboard and special guillotine neck restraint \* If high GA risk or a piece of info like presence of an egg or pellet can place the bird in a cone of cardboard, paper roll, or piece of stocking (which the bird will often stay still in the dark environment of the bag) OR standing on a perch if the bird will stay still
110
111
What is unique about the avian skeleton?
112
Unique anatomy fact how kidney swelling relates to lameness in birds?
Sciatic nerve runs through the kidneys-- disease causing swelling in the kidneys puts pressure on the sciatic nerve which can cause lameness
113
Determining alignment?
114
115
Hour glass appearance-- all fit in imaginary parallel lines between the shoulder and hip-- anythign outside of that swelling internally e.g. liver or tumour, etc \* air sacs, birds are full of air-- look for haziness or changes-- air sac disease
116
117
Loops of the trachea
118
Ovaries and follicles
119
huge testicles
120
Budgie with an egg
121
Poor nutrition, poor diets, low Ca
122
Distance across largest part of the heart, distance across largest part of the chest If heart is \> 60% of the distance across the chest, you are likely to have heart disease
123
Metal poisoning-- but have to consider is it just stones and grit or actually metal poisoning?
124
Use of contrast radiography in avian medicine
More for highlighting internal changes-- really small birds because hard to differentiate between organs-- where the liver starts and finishes.
125
126
Contrast radiography confirming a hernia in a duck because there are many reasons a mass could be there
127
Ultrasound use in avian medicine?
U/S not that useful in birds because of their air sacs-- e.g. RG and see ground glass only-- then U/S could be helpful to determine is it fluid? Solid?
128
Sites for blood samples in birds
129
Taking blood samples from birds. Which is the only good jugular in a bird?
Right jugular
130
131
General CBC and biochem in a bird? Liver? Kidney?
Basic: TP, Uric acid, AST, glucose, haematology, PCV, WCC and blood film Liver- AST, GLDH, LDH, ALT (not helpful in birds), bile acids, CK (helps to distinguish if AST is liver or not) Kidney- Uric acid, urea- dehydration, crea (not helpful in avian medicine) GI- Amylase and Lipase, electrolyte changes, CK, AST and LDH Bloood glucose
132
Low TP, what does this mean?
Bird is not going to survive
133
How much of a bird's blood can you take?
e.g. 40-50 g budgie Can take maximum of 10% of circulating blood volume. That is 1% of their body weight... but in a sick bird you dont' want to take that full amount.
134
Green poo? Seeds?
Biliverdin = Psittacosis (not always but commonly) \*\* Seeds on top of poo or mixed in the poo- are the seeds landing on top of the poo or is there possible maldigestion
135
Excess urine-- reabsorb water really quickly in health some are designed to live in the desert
136
Rotation of tibiotarsus- what causes this? Is it congenital? Two birds come from different genetic pools, so unlikely Nutrition- too normal of a diet so unlikely TWISTED LEG SYNDROME from textbooks-- outward or inward deviation of the leg bilateral or unilateral, can occur in any species of bird. Sometimes causes by angular deformities. Dyschondroplasia?? Formation of avascular aplasia in the bone ends-- couldn't find evidence
137
What is this? Causes?
Dyschondroplasia?? Formation of avascular aplasia in the bone ends-- couldn't find evidence
138
What is this? Causes?
139
Diseases of the Musculoskeletal System
140
Rickets- Failure of mineralisation Causes: - Vit D def - Ca def - Phosphorous def Ca/P imbalance
141
Growing period nutrition recommendations in chickens
142
Osteoporosis "cage layer fatigue" Signs: \* fractures and callus formations, lameness, paralysis, poor egg shell, deviated sternum Causes: \* Low dietary Ca: laying ration, growing ration... \* Low dietary vit D \* Low dietary Phosphorous \* Lack of exercise
143
Fracture causes in birds
e.g. captive eagle fed only meat
144
Osteomyelitis
145
Other than bacteria, what else can cause arthritis/ tenosynovitis?
\* Viral \* Gout- renal failure, idiopathic--\> dietary?
146
Diseases of tendons in birds? Causes?
\* Tenosynovitis \* Ruptured tendon syndrome Causes: - tenosynovitis - tendon structure - nutritional factors - spontaneous - Management-- increasing feeding space, decreasing density
147
Unique properties of bones in birds?
Medullary bone-- in a chicken, the femur does this job \* Some don't have marrow- helps make them lighter (pneumatic bones)
148
149
Difference between resp tract of birds vs. mammals, signs of conjunc. rhinitis, treatment of acute vs. chronic sinusitis, code of antibiotic use in poultry
150
Anatomy of the avian respiratory system
151
Diseases of the avian respiratory system
152
153
Signs and forms of sinusitis
154
Acute sinusitis
155
Organisms in avian mycoplasmosis
156
Chronic respiratory disease
157
Sinusitis treatment in birds vs. mammals
Birds do not have to drill into any bone
158
Other signs/ lesions of chronic respiratroy disease
159
Diagnostic options of chronic respiratory disease
160
Treatment of chronic respiratory disease
161
Prevention and control of chronic respiratory disease
162
Fowl Coryza
163
Prevention and Control of Fowl Coryza
164
Treatment of Fowl Coryza
165
Australian Veterinary Poultry Association Code of Practice for use of Antibiotics in the Poultry Industry
166
Australian Veterinary Poultry Association Code of Practice for use of Antibiotics in the Poultry Industry Veterinary care and supervision of recipient stock
167
Australian Veterinary Poultry Association Code of Practice for use of Antibiotics in the Poultry Industry Off Label Use of Antibiotics
168
Currently registered antibiotics for laying hens in Australia
169
What is PUBCRIS?
170
Clinical signs in diseases of the trachea
171
Diseases of the trachea DDX
172
Infectious bronchitis
173
IB forms
174
IBV
175
Transmission of IBV? Diagnosis?
RT- PCR Serology- ELISA
176
Prevention of IBV
Biosecurity Vaccination-- the right strain
177
Infectious Laryngotracheitis (ILT)
Gallinaceous birds- includes turkey, grouse, chicken, New World quail and Old World quail, ptarmigan, partridge, pheasant, junglefowl and the Cracidae
178
Diagnosis of ILT
179
Prevention of ILT
180
Prevalence of ILT in Australia
181
ILT Strain ID
182
Newcastle Disease
183
NDV history in Australia? Forms of the disease?
\* Velogenic (classical ND) \* Mesogenic \* Lentogenic
184
Velogenic ND
185
186
Mesogenic ND
187
Lentogenic ND
188
NDV in other species (aside from chickens)
189
Aetiology of NDV
190
Transmission of NDV
191
Diagnosis of NDV
192
Control of NDV
193
Prevention of NDV
194
3 possibilities?
195
196
Resp mycoses predisposing factors? Diagnosis? Treatment?
197
Dealing with resp mycoses
198
Diseases of the air sacs
199
Disease of the air sacs
200
Clinical signs of Chlamydiosis (Psittacosis, Ornithosis)
201
Chlamydiosis
202
Important points about Chlamydiosis (Psittacosis)
203
Recommended prevention Chlamydiosis
204
Treatment Chlamydiosis
1 mL (20 drops) per 100 mL of drinking water
205
Prevention and control Chlamydiosis
206
Diseases of the air sacs
207
Three main behaviors in non-breeding season of birds
Normal behaviour - Most species of parrot are only 1 or 2 generations from wild state except for a few domesticated species (eg budgie, cockatiel, lovebird and ringnecks) which means many birds maintain their wild tendencies but lack the stimuli and training for full social development. - Natural bird behaviour can be divided up into three to four key areas. • In the non breeding season a bird spend their time in social interaction, foraging and maintenance behaviour • During breeding season they continue the 3 key behaviours but they are heavily influenced by reproductive behaviours. During breeding season birds that may normally flock may divide in pairs, will remain close to the nest site becoming aggressive of intruders and spending much time in sexual interaction with their mate. Most of the features in a captivity life style is more conducive to a breeding arrangement than normal social template.
208
Biggest tip on keeping birds
209
Social behaviour in parrots
210
Foraging and maintenance behaviours in parrots
211
What is training?
212
Behaviours to avoid
213
Feather plucking DDX and diagnostic algorithm
214
Reproduction and captivity
215
Choosing the right bird
216
Common nutritional diseases of birds
217
Natural supplements of birds
218
Dominance over birds
219
general tips on how to best keep birds
Avoid sexual behaviours such as nose rubbing excessive petting and grooming, allowing the bird to groom the owner and do sexual acts on the owner. Randy Lorries will hump anything during the breeding season, they especially love the little soft toys owners seem to like to give them (so thought needs to be given to the types of toys people buy).
220
Common behavioural problems
221
Feather plucking treatment
222
Examples of unique nutrition for different birds
223
the all-seed diet
224
Veggies, fruit and green food for birds
225
Nuts and grit for birds
226
Pelleted and extruded diets for birds?
227
Synthetic homemade diets and vitamin and supplements for birds
228
Foods to avoid with birds
229
Typical diet for a psittacine
230
Typical diet for a Lorikeet or Budgie
231
Typical diets for finches & canaries, pigeons, gallinaceous birds & waterfowl, magpies & ravens & kookaburras?
232
Obesity in birds
\* Lipomas will often regress over time or respond to increased exercise and dietary modification
233
Vitamin A Deficiency
234
Calcium, Phosphorous, Vit D3, and metabolic bone disease in birds
235
Vitamin E deficiency
236
Vit K, Thiamine (B1) Deficiency, Goiter in birds?
237
Pre-anesthetic assessment
238
Inhalation anesthetics for birds? Injectable anesthetics?
239
Premeds in birds
240
Intubation of birds
241
Depth of anesthetic and monitoring
242
Post-op recovery
243
Equipment and suture material with birds
\* Fine monofilament nylon or PDS \* Haemoclips for BVs \* Electrocaurtery essential for minimal haemorrhage and trauma \* Heat during surgery (heat pad)
244
Skin preparation in birds
Minimal feather pluck-- never flight feathers, avoid excessive wetting and using a lot of alcoholic tincture
245
Common surgeries of birds
\* Wounds and injuries: thin skin-- decision on primary v. secondary healing needs to be considered; e-collars may be necessary but can do more harm than good \* Lumps and bumps- lipomas are common-- nutritional manipulation before surgery; xanthomas-- lumps where there has been trauma or haemorrhage, intradermal deposits of cholesterol clefts with an associated inflammatory reaction, can be highly vascular and invasive (similar to lipomas) \* Treatment of crop burns- hand fed parrots fed hot feeds-- signs are delayed crop emptying or a wet skin patch over the crop that can lead to necrosis and fistula formation-- surgery should be delayed until the extent of necrosis is IDed (4-5 days)-- double inversion suture pattern 4/0-6/0 \* Laparotomy- impossible without opening up caudal air sacs, will have an effect on heat loss and anesthetic absorption-- left lateral approach for gonads, left kidney, oviduct, C-section, proventriculotomy and yolk sacculectomy \* Surgical sexing and endoscopy-- intestines must be empty as can be difficult to see internal organs with full intestines in laproscopy. Parrots weighing more than 200 gm require fasting for 12 hours-- ill birds should be starved for lesser periods.
246
Euthanasia of aviary birds
247
When should a bird be seen asap?
248
When should a bird be seen within 24 hours?
249
Shock tx
250
Fluid treatment in birds
251
Oral fluids in birds
252
Subcutaneous fluids
253
IV Fluids
254
Intraosseous fluids
255
Intraosseous fluids
256
257
258
Oxygen
259
Common avian emergencies
260
Egg binding
261
heavy metal poisoning