Porth Chapter 61 Flashcards

1
Q

pigmentary skin disorders

A

Melanocytes:

  1. Absence of melanin production, as in​: Vitiligo, Albinism​
  2. Increase in melanin: Mongolian spots, Melasma
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2
Q

rashes

A

Temporary eruptions of the skin​; Childhood diseases, heat, diaper irritation, or drug-induced reactions

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3
Q

lesions

A

A traumatic or pathologic loss of normal tissue continuity, structure, or function

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4
Q

types of rashes

A
  1. Blanched (white)​
  2. Erythematous (reddened)​
  3. Hemorrhagic or purpuric (containing blood)​
  4. Pigmented
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5
Q

ringworm / fungal infections

A

Superficial fungal infections: Tinea corporis, Tinea faciale, Tinea capitis, Tinea pedis, Tinea unguium, Tinea versicolor, Tinea incognito

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6
Q

primary bacterial infections

A

superficial

  1. Impetigo—appears as a small vesicle or pustule or as a large bulla on the face or elsewhere ​
  2. Ecthyma—ulcerative form of impetigo
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7
Q

secondary bacterial infections

A

deep cutaneous infections; 1. Infected ulcer ​

2. Cellulitis

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8
Q

Human papillomavirus

A

Verrucae (warts) are common benign papillomas.

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9
Q

Herpes simplex virus

A

Type 1: The organism is spread by respiratory droplets or by direct contact with infected saliva.​
Type 2: Genital herpes

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10
Q

Herpes zoster/shingles

A

Localized vesicular eruption distributed over a dermatomal segment of the skin

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11
Q

noninflammatory acne

A

Comedones; Plugs of material that accumulate in sebaceous glands that open to the skin surface

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12
Q

inflammatory acne

A

Papules, pustules, nodules, and, in severe cases, cysts; Believed to develop from the escape of sebum into the dermis and the irritating effects of the fatty acids contained in the sebum

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13
Q

Acne vulgaris

A

Chronic inflammatory disease of the pilosebaceous unit

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14
Q

Acne conglobata

A

Comedones or cysts have multiple openings, large abscesses, and interconnecting sinuses; Discharge is odoriferous, serous or mucoid, and purulent; Affected persons have anemia with elevated white blood cell counts, sedimentation rates, and neutrophil counts

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15
Q

Rosacea

A

Chronic inflammatory process accompanied by vascular instability with leakage of fluid and inflammatory mediators into the dermis​; Accompanied by gastrointestinal symptoms​

Types​:

  1. Erythematotelangiectatic ​
  2. Papulopustular ​
  3. Ocular ​
  4. Phymatous
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16
Q

Contact dermatitis

A

Allergic contact dermatitis results from a cell-mediated, type IV hypersensitivity response.​

Irritant contact dermatitis is caused by chemicals that irritate the skin.

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17
Q

Atopic dermatitis

A

Inflammatory skin disorder that is characterized by poorly defined erythema, edema, vesicles, and weeping at the acute stage ​

Lichenification in the chronic stag

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18
Q

Nummular eczema

A

Coin-shaped papulovesicular patches involving the arms and legs ​

Lichenification and secondary bacterial infections are common.

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19
Q

Drug-Induced Skin Eruptions

A

Systemic drugs cause generalized skin lesions.​

Topical drugs are usually responsible for localized contact dermatitis types of rashes.​

Bullous skin lesions ​

Erythema multiforme minor​

Stevens-Johnson syndrome​

Toxic epidermal necrolysis​

Skin detachment

20
Q

Papulosquamous Dermatoses

A

Skin disorders characterized by scaling papules and plaques ​

Psoriasis ​

Pityriasis rosea​

Lichens planus

21
Q

Arthropod Infestations

A

Scabies​

Pediculosis​

Ticks​

Rocky mountain spotted fever​

Lyme disease

22
Q

uvc rays

A

Short (100 to 289 nm); do not pass through the earth’s atmosphere​

23
Q

uvb rays

A

290 to 320 nm; responsible for nearly all the skin effects of sunlight​

24
Q

uva rays

A

321 to 400 nm; can pass through window glass, are more commonly referred to as sun tanning rays​

25
Acute Effects of Ultraviolet Rays of Sunlight
Short-lived and reversible​ Erythema​ Pigmentation​ Injury to Langerhans cells and keratinocytes
26
Chronic Effects of Ultraviolet Rays of Sunlight
Directly damaging skin cells​ Accelerating the effect of aging on skin​ Producing changes that predispose to development of skin cancer
27
sunburn
Excessive exposure of the epidermal and dermal layers of the skin to ultraviolet radiation​ Resulting in an erythematous inflammatory reaction​ Inflammation, blistering, weakness, chills, fever, malaise, and pain often accompany severe forms of sunburn
28
Measures for Protection from the Sun
Avoidance of sun exposure​ Use of protective clothing​ Use of sunscreens​ Chemical agents absorb UVR.​ Physical agents reflect UVR.​ Biological agents reduce inflammation.​ Broad-spectrum suntan lotions protect against both UVA and UVB.
29
Photosensitivity Occurring with Drugs
Definition: exaggerated response to ultraviolet light when the drug is taken in combination with sun exposure​ Examples​: Some anti-infective agents​ Antihistamines​ Antipsychotic agents ​ Diuretics ​ Hypoglycemic agents ​ Nonsteroidal anti-inflammatory drugs
30
First-degree burns
(superficial partial-thickness burns) involve only the outer layers of the epidermis.​
31
Second-degree partial-thickness burns
involve the epidermis and various degrees of the dermis.​
32
Second-degree full-thickness burns
involve the entire epidermis and dermis.​
33
Third-degree full-thickness burns
extend into the subcutaneous tissue and may involve muscle and bone.​
34
Estimation of Total Body Surface Area Involved in Burn
Rule of nines ​ Multiple of 9%​ Perineum 1% ​ Arm 9% ​ Leg 18% ​ Anterior trunk 18% ​ Posterior trunk 18%​ Head 9% Lund and Browder chart ​ American Burn Association classification of burns
35
Systemic Complications of Burn Tissue
Magnitude of the response is proportional to the extent of injury. ​ Hemodynamic instability​ Impaired respiratory function​ Hypermetabolic response​ Major organ dysfunction​ Sepsis
36
Treatment of Burns
Immediately submerge area to cool tissue​ Emergency care​ Resuscitation ​ Stabilization ​ Maintaining cardiac and respiratory function​ Intermediate and long-term treatment is dependent on the extent of the injury.​ Protection from desiccation​ Escharotomy/fasciotomy
37
Pressure Ulcers (Bedsores)
Ischemic lesions of the skin and underlying structures caused by unrelieved pressure ​ Four factors contribute to the development of pressure ulcers. ​ Pressure ​ Shear forces​ Friction ​ Moisture
38
Prevention of Pressure Ulcers (Bedsores)
(1) Identifying at-risk persons who need preventative measures and the specific factors placing them at risk​ (2) Maintaining and improving tissue tolerance to prevent injury​ (3) Protecting against the adverse effects of external mechanical forces (i.e., pressure, friction, and shear) ​ (4) Reducing the incidence of pressure ulcers through educational programs
39
The Development of Skin Cancer
Nevi (moles)—benign tumors of the skin that predispose individual to cancer​ Nevocellular nevi ​ Dysplastic nevus​ Great tendency to transform
40
Malignant melanoma 
Rapidly progressing malignant tumor of the melanocytes
41
Basal cell carcinoma​
Neoplasm of the nonkeratinizing cells of the basal layer of the epidermis; most common skin cancer in white-skinned people
42
Squamous cell carcinomas​
Second most frequently occurring malignant tumors of the outer epidermis​
43
Four Types of Melanomas
Superficial spreading​ Nodular​ Lentigo maligna​ Acral lentiginous
44
Skin Disorders of Infancy
Mongolian spots​ Hemangiomas​ Port-wine stains ​ Nevi Vascular and pigmented birthmarks​ Diaper rash​ Prickly heat​ Cradle cap
45
Skin Manifestations of Common Infectious Disease
Roseola infantum​ Rubella ​ Rubeola (measles)​ Varicella​ Scarlet fever
46
Normal aging changes
Diminution in subcutaneous tissue​ Thinning of epidermal and dermal layers​ Decrease in the number of melanocytes, Langerhans cells, and Merkel cells​ Decrease and thickening of blood vessels
47
Skin Lesions Common among the Elderly 
Skin tags​ Keratoses​ Lentigines​ Vascular lesions