Pintrest Study Helpers Flashcards
Area of infarction
O2 deprived
Damage irreversible
Causes “Q” wave on EKG
3 areas of damage after a myocardial infarction
Area of Infarction
Area of injury
Area of ischemia
Area of injury
Next to infarct. Tissue is viable as long as circulation remains adequate. Increasing O2 may save this area from necrosis. Causes S-T Segment Elevation on EKG
Area of ischemia
Viability may not be damaged as long as MI doesn’t extend and collateral circulation is able to compensate. Causes depressed ST Segment.
Preload
Volume of blood in ventricles at end of diastole (end diastolic pressure)
Increased in: hypovolemia, regulation of cardiac valves
Heart failure
After load
Resistance left ventricle must overcome to circulate blood
Increased in: hypertension and vasoconstriction
Increase afterload= increased cardiac workload
Myocardial Infarction
- pain: sudden onset, substernal, crushing, tightness, severe, unrelieved by nitro, and may radiate to: back, neck, jaw, shoulder and arms
- dyspnea
- syncope ( decreased bp)
- nausea and vomit
- extreme weakness
- diaphoresis
- denial in common
- increased heart rate
- dietary restrictions: lower sodium, lower cholesterol and lower caffeine
Treating congestive heart failure
Unload fast
U-upright position N-nitrates L-Lasix O-oxygen A-ace inhibitors D-digoxin
F-fluids (decrease)
A-afterload (decrease)
S-sodium retention
T-test (digoxin level, ABGs, Potassium levels)
Left sided failure
- Paroxysmal nocturnal dyspnea
- Elevated pulmonary wedge pressure
- Pulmonary congestion: cough, crackles, wheezes, blood-tinged sputum, tachypnea
- Restlessness, confusion, orthopnea, tachycardia, exertional dyspnea, fatigue, and cyanosis
Right sided failure (Cor Pulmonale)
- fatigue
- increased peripheral venous pressure
- ascites
- enlarged liver and spleen
- may be secondary to chronic pulmonary problems
- distended jugular veins
- anorexia & complaints of GI distress
- weight gain
- dependent edema
PQRST waves
P (ok here I come)
R (I got to get from the atrium to ventricles)
QRS (making it happen… Ventricles contract)
ST (I think I’ll take a little rest)
T ( ready to go again)
Heart murmurs causes: SPAMS
S-stenosis of a valve P-partial obstruction A-aneurysms M-mitral regurgitation S-septal defect
Types of heart murmurs
Systolic: crescendo: increase during systole. Decrescendo: decrease during systole
Diastolic: indicates pathological disease