Physiology final- chapters 22 and 23 Flashcards

1
Q

anabolism

A

to build it up (synthesis). fed state/absorptive state.

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2
Q

catabolism

A

to break it down. (“lysis”) fasted state or post-absorptive.

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3
Q

satiety

A

says your full, you’ve had enough. meal to meal. short term.

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4
Q

neuropeptide Y (NPY)

A

made in the hypothalamus. paired with opposite MSH. important in turning on appetite.
leads to orexin

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5
Q

orexin

A

stimulates hunger from NPY’s, paired with corticotropin releasing hormone

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6
Q

Corticotropin releasing hormone

A

reduces appetite, from MSH. opposite of orexin.

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7
Q

melanocyte stimulating hormone (MSH)

A

turns off appetite. made in hypothalamus. paired with NPY.

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8
Q

CCK

A

says i’m full. comes from duodenum.

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9
Q

Grehlin

A

in stomach and increases NPY. opposite of leptin.

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10
Q

Leptin

A

mirrors body fat levels. high fat levels=high leptin levels. decreases NPY. opposite of grehlin.

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11
Q

Direct calorimetry

A

measures energy content of food

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12
Q

indirect calorimetry

A

measures oxygen consumption. how much energy you’re extending.

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13
Q

respiratory quotient

A

CO2 produced in exercise vs. oxygen consumed.

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14
Q

energy production- carbohydrates

A

absorbed primarily as glucose. fed state- stored as glycogen in liver. fasted state- glycogen broken down into glucose

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15
Q

BMI

A

doesn’t measure body composition. (need percentiles for children).

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16
Q

Insulin

A

only hormone that lowers blood sugar. anabolic, promotes fat synthesis. Paired with glucagon. made in beta cells. dominates fed state. from the pancreas.

17
Q

Glucagon

A

raises blood sugar. paired with insulin. catabolic. dominates fasted state. from alpha cells in the pancreas

18
Q

Type 1 diabetes

A

autoimmune. common in children. acute loss of beta cell function and lacks insulin production. virus attacks beta cells.

19
Q

Type 2 diabetes

A

increasing demand for insulin to exhaust beta cells. leads to obesity or opposite.

20
Q

hyperglycemia

A

high glucose amounts. leads to hyperosmolarity, polyuria (pee a lot and drink a lot), dehydration, altered brain function, etc.

21
Q

Growth hormone

A

made in the anterior pituitary. anabolic

22
Q

growth hormone releasing hormone

A

from the hypothalamus. stimulates growth hormone. opposite somatomedin.

23
Q

somatomedins

A

inhibits (prevents) growth hormone. opposite growth hormone releasing hormone.

24
Q

Insulin like growth factors (IGFs)

A

made in the liver. growth hormone raises blood sugar. anabolic.

25
Q

Osteoblasts

A

make bone- produce enzymes and osteoid. takes calcium out of bone bank by making hydrochloric acid.

26
Q

Osteoclasts

A

break down bone- control reabsorption

27
Q

parathyroid hormone (PTH)

A

released when there is a decrease in calcium. opposite of calcitonin.

28
Q

Three ways PTH increases plasma calcium

A
  1. mobilizes calcium from bone 2. enhances renal reabsorption of calcium 3. Indirectly increases intestinal absorption of calcium.
29
Q

Calcitonin

A

puts calcium back into bone. opposite of PTH.

30
Q

Calcitriol- vitamin D3

A

absorbs more calcium from intestine. produced by C cells in thyroid. stimulated in skin and activated in liver and kidney

31
Q

What happens when we have low calcium levels?

A

PTH increases. takes calcium from bone, kidney and gut (intestines)