Physio (Rhythmical Excitation of the heart) Flashcards

1
Q

Abnormal Paceakers –“Ectopic” Pacemaker

A

pacemaker elsewhere than the sinus node; called an “ectopic” pace maker

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2
Q

What Causes?

  • abnormal sequence of contraction
  • can cause feeble heart pumping
A

ectopic pacemaker

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3
Q

causes?

  • blockage of transmission
  • from sinus not to other parts of the heart
A

shift

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4
Q

Syndrome

-delayed pickup of heartbeat

A

Stokes Adams syndrome

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5
Q

Supplied by:

  • sympathetic nerves
  • parasympathetic nerves
A

the Heart

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6
Q

-the Vagi

A

Parasympathetic

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7
Q

Distributed to:

  • SA nodes
  • AV nodes
A

Vagi

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8
Q
  • distributed to all parts of the heart

- stron representation to ventricular muscle

A

sympathetic nerves

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9
Q
  • causes release of acetylcholine

hormone

A

stimulation of. parasympathetic nerves

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10
Q

2 major effects this are:

  • decreases rate of rhythm of the sinus node
  • decreases the excitability of the AV junctional fibers between the atrial musculature & AV node
A

acetylcholine

hormone

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11
Q

what causes vagi to:

  • stop completely
    (rhythmical excitation by sinus node)

-block completely transmission of cardiac impulse
(atria into ventricles through the A-V node)

A

strong stimulation

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12
Q
  • ventricles stop beating for 5 to 20 seconds
  • develops a rhythm of its own
  • causes ventricular contraction at a rate of 15 to 40 beats/minute
A

ventricles stop beating; CALLED ventricular escape

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13
Q
  • released at vagal nerve endings

- increases (greatly) permeability of membranes to K+

A

acetylcholine

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14
Q
  • increased negativity inside the fibers

- makes tissues much less excitable

A

hyperpolarization

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15
Q

-state of hyperpolarization

-decreases “resting” membrane potential of sinus nodal fibers
more negative than usual

A

sinus node

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16
Q

caused by

  • sodium leakages
  • calcium leakages
  • requires much longer reach the threshold potential for excitation
A

initial rise of sinus nodal membrane potential

17
Q
  • caused by vagal stimulations

- makes it difficult for small atrial fibers to excite nodal fibers

A

hyperpolarization

18
Q

-moderate decrease

rate of rhythmicity

A

delays conduction

19
Q

-large decrease

rate of rhythmicity

A

blocks conduction

entirely

20
Q
  • opposite effects on heart, compared to vagal stimulation

- increases overall activity of the heart

A

sympathetic stimulation

21
Q
  • increases rate of sinus nodal discharge
  • increases rate of conduction/level of excitability in all options of the heart

-increases force of contraction of all cardiac musculature
(atrial & ventricular)

A

effects of sympathetic stimulation on cardiac rhythm & conduction

22
Q
  • triple frequency of heartbeat

- increase the strength of heart contraction (2x)

A

maximal stimulation

23
Q

-norepinephrine released
(hormone)

  • at sympathetic nerve endings
  • increases permeability of the fiber membrane to Na and Ca ions
A

stimulation of sympathetic nerves

24
Q
  • in Sinus node

- causes increase of the heart rate

A

increase of Na-Ca permeability

25
Q
  • in AV node & AV bundle

- conducting fiber bundles are easily excited?

A

increased sodium-calcium permeability