Physical Med Flashcards

1
Q

drowning

Nonfatal drowning –

A

someone gets immersed, has respiratory impairment, but doesn’t die

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2
Q

drowning

Water rescue –

A

someone gets immersed and then removed without evidence of respiratory impairment

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3
Q

drowning

Fatal drowning –

A

someone gets immersed, has respiratory impairment, and dies

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4
Q

Drowning

When to Discharge

A

if no increased work of breathing, no decreased sats, little-to-no rales

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5
Q

drowning

The mammalian dive reflex –

A

metabolism slows when immersed in cold water – more pronounced in children vs adults

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6
Q

Drowning

resuscitation

A

Aggressively resuscitate nonfatal/fatal drowning (<60 min immersed for adults, longer for kids)

Shock from water rescue or nonfatal drowning is not typical – look for other causes
Hemorrhagic
Neurogenic

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7
Q

Scuba- disorders of descent

Barotitis

A

Barotitis (pressure inflaming the ear)
Mild: pain
Mod/severe: TM rupture / vertigo / N&V / SN hearing loss

One of the squeeze syndromes – this can happen to sinuses, facemask, suit, lungs – gas in the area shrinks under pressure as you go down (Boyle’s Law)

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8
Q

Scuba- disorder of descent

Barotitis Tx

A

Treat with decongestants, pressure equalization (Valsalva), analgesics, vertigo medications (meclizine, benzodiazepines)
Inflamed, intact TM? Consider droppered lido 2% into EAC

Mod/severe: refer to ENT

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9
Q

Scuba- disorder of descent

Nitrogen narcosis

patho, Sx, Tx

A

Pathophys: neurotoxic levels of nitrogen in blood
Sx: AMS & poor coordination at depth
Tx: controlled ascent – decreases nitrogen in blood

~ at depth, partial pressure of nitrogen in lungs goes up, pushing more nitrogen from your lungs in to your bloodstream (vicious tag-team of Dalton’s Law & Henry’s Law)

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10
Q

disorder of ascent - Scuba

Decompression sickness

type 1 and 2, Tx

A

Dissolved nitrogen forms bubbles in blood, tissue during too-rapid ascent – delayed onset – 98% w/in 24h, 100% w/in 48h

Type I – skin, lymphatic, MSK
“the bends” or “caisson disease” – joint pain, rashes, itching

Type II – more serious – other organs than above (like CNS)
gradual neuro sx – paresthesias, numbness, weakness, AMS (not ALOC)

Prevention – follow dive table
Tx – hyperbaric chamber

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11
Q

ascent disorder

A

Decompression sickness
Type I – skin rash called “cutis marmorata”

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12
Q

scuba disorder of ascent

Pulmonary barotrauma

A

Rapid ascent with closed glottis
Pneumothorax, hemothorax, pneumomediastinum
Gas in the area expands as pressure
drops (Boyle’s Law strikes back!)

Prevention – exhale as you ascend

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13
Q

ascent disorder

Gastrointestinal barotrauma

A

Pathophys – diving after carbonated beverages, gas-generating foods
Rarely – intestinal perforation, diaphragm rupture
Usually – abdominal cramps, eructation, flatulence

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14
Q

Scuba- disorder of ascent

Arterial air/gas embolism

A

Sudden air in arterial circulation immediately upon surfacing or within 10 minutes
Altered LOC and not just AMS, seizures

Prevention – follow dive table
Tx – hyperbaric chamber

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15
Q

Altitude illness

general

A

Due to relative hypoxia / increased sympathetic activity / capillary leak (brain and lungs)
Can occur in anyone; being fit is not protective

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16
Q

Acute Mountain Sickness

general

A

Usually > 8k feet (Vail is 11k)
Starts early – often day 1 at altitude

HA, nausea, fatigue, insomnia
Worsened with sedatives, alcohol

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17
Q

Acute mountain sickness

Tx
Preventative

A

May be prevented by prophylactic acetazolamide
Causes a metabolic acidosis which generates a respiratory alkalosis – increased ventilation increases oxygenation
Usually self-limited (1-2 days); can treat with NSAIDs, steroids, oxygen, descent

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18
Q

HACE / HAPE

General

A

HACE = high altitude cerebral edema
Severe, life-threatening; increased ICP
Ataxia, vomiting, confusion, seizures, coma
**Descent ASAP **(+/- steroids, hyperbarics)

HAPE = high altitude pulmonary edema
Most lethal of the altitude illnesses
Usually second night at altitude
Shortness of breath, fever, rales, pink sputum, hypoxia; normal heart size on CXR
Descent ASAP

Usually have symptoms of Acute Mountain Sickness before HACE/HAPE

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19
Q

Hypothermia

general

A

Core temperature < 35oC (95F)
Can occur even in non-freezing temperatures
Only use accurate thermometers; rectal, bladder, esophageal
Risks include extremes of age, altered sensorium, burns, trauma; may also see in sepsis, hypoglycemia
Findings – altered mental status, bradycardia

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20
Q

Hypothermia

A

Bradycardia with an idioventricular or junctional escape rhythm
Prominent J waves in the anterior leads = Osbourne waves
(named after the fact that they occur on the “J” point of the EKG)

21
Q

Hypothermia

Tx

A

Rewarming should start ASAP
External rewarming
Passive – remove wet clothes, give warm blanket
Active – radiant heaters, hot water bottles, warming blankets
Watch for core temperature afterdrop with passive rewarming (cold blood from the periphery goes into the central circulation)
Active core rewarming
Warm humidified oxygen, warmed IV fluids, warm lavage (NG tube, foley)

22
Q

Frostbite

general

degrees, Tx

A

Think of it as burns caused by cold (local tissue freezing)

First degree – superficial
Second degree – full thickness, clear blisters
Third degree – hemorrhagic blisters (blood = deeper)
Fourth degree – to bone

Rapid rewarming with warm circulating water
Do not allow refreezing
Do not debride bloody blisters

23
Q

Types of Heat Illness

Heat edema
Do not treat with

A

swelling of feet/hands, transient, do not treat with diuretics

24
Q

Types of Heat Illness

Heat cramps

A

after exertion in hot climates / usually after cooling / resolves without treatment

25
# Types of Heat Illness Heat syncope
– pooling of blood in extremities / treat by lying patient down in cool place
26
# Types of heat illness Heat exhaustion
– “summer flu” / nausea, vomiting, due to salt and water depletion / treat by moving to cooler environment, PO hydration with fluids with electrolytes/sugar
27
# Heat Stroke general
A true emergency – must cool ASAP Multiorgan failure / brain, liver and endothelium at highest risk, but all systems affected Core temperature usually ≥ 40oC (104F) Two types, classic and exertional
28
# Heat stroke S/Sx
**Altered mental status,** tachycardia, tachypnea, may be hypotensive May see acidosis, rhabdomyolysis, ARDS, renal failure, hyperkalemia
29
# Heat Stroke Classic
Epidemic (with heat wave) **Non-exertional** Elderly with chronic disease, very young, chronic illnesses Dry skin Rare to see acute tubular necrosis (ATN), rhabdomyolysis
30
# Heat Stroke Exertional
Isolated Exertional Healthy, active Profuse sweating May have diarrhea DIC, ATN, rhabdomyolysis common
31
# Heat stroke Tx
Cooling is crucial – change “ABCs” to “C-ABCs” Resuscitate as usual, but cool simultaneously Methods of cooling External -- strip patient / tepid mist plus fans / wet sheets plus fans; *ice packs to groin, axillae, neck* Internal -- iced water gastric lavage / iced water bladder lavage May need chlorpromazine to control shivering Goal is to decrease temperature by 0.1-0.2oC per minute to 38.5oC (101.3F), then stop; watch for overshoot (hypothermia)
32
# Burns 1st degree
(superficial) -- epidermis only, painful, red and dry, hypersensitive. Heals in 3-7 days.
33
# burns 2nd degree
Superficial partial thickness --to dermis, painful, red and wet, thin-walled blisters, hypersensitive. Heals in 7-14 days without scarring (called “second degree”)
34
# Burns 3rd degree
Deep partial thickness: To dermis, painful, red and white (mottled), thick-walled blisters, slight decrease in sensation. Heals in weeks-months with scarring and pigment changes.
35
# burns 4th degree
Full thickness: To deeper tissues, painless, white and brown and dry. No sensation. Needs grafting.
36
# Burns Body Surface Area
Body surface area (BSA) Patient’s palm = 1% of patient’s BSA Adults – rule of 9’s Children – Lund/Browder chart
37
# Minor burns Tx
Minor burns Cool ASAP Treat pain with NSAIDs, opiates Leave blisters intact if unruptured and not tense Debride if blisters broken, tense, across joints Update tetanus Can manage ”open” (topical antimicrobials) or “closed” (occlusive dressings)
38
# Major burns Tx
Major burns Cool > watch for hypothermia Assess airway > look for singed nasal hairs, oral burns; intubate early for respiratory distress, concern for airway edema Enclosed space? Think carbon monoxide, cyanide toxicity. **Fluid resuscitation crucial** – Parkland formula 4 mL x kg x %BSA per day of Ringers lactate Half of volume given over first 8 hours Watch urine output – goal is > 1ml/kg/hr
39
# electrical injuries general
Tissue damage caused by electrical current Injuries common (falls) AC 3X worse than DC at same voltage Degree of injury determined by Type of current Duration of contact Tissue in path of current Biggest risk of death is either trauma or cardiac arrhythmia – treat them as a trauma patient Mouth burns in children usually from biting electric cord; risk of arterial bleeding at 7-10 days after burn
40
# electrical injuries Tx
Examine carefully – look for entrance and exit sites Resuscitation and ABCs Household current exposure generally requires no specific treatment, testing or monitoring If more than minor electric shock, consider labs – CMP, CBC, CK (risk of rhabdomyolysis), coag studies if seriously injured Monitor urine output / admit to monitored bed
41
# mammalian bites human, dog, cat
Human bite is the worst “mammal” bite “Fight bite” – wound over MCP joint High risk; may need to go to OR Dog bites < infection rate, > crush injury rate Cat bites Puncture wounds > risk of infection
42
# animal bite Tx
1st line: amoxicillin/clavulanate 2nd line: doxycycline OR TMP/SMX PLUS clindamycin OR metronidazole
43
# Rabies general
Rare in US; most cases imported in developing countries (often dog bites) Bites at risk -- fox, skunk, raccoon, bats (most common risk animal) / rodents, rabbits and squirrels do not pose risk A bat in an occupied room – assume bite
44
# rabies Tx
Early post-exposure prophylaxis is key Passive human rabies immune globulin (HRIG); give as much as possible around and into wound, remainder give IM Active human diploid cell vaccine (HDCV) – 4 doses (days 0-3-7-14)
45
# Bites and stings general, Sx, Tx
Bees, wasps and ants Usually only cause local symptoms – burning, pain, swelling Treatment usually ice to area, OTC meds (NSAIDs, acetaminophen, nonsedating antihistamine) About 10-15% of those stung will have unusually large areas of swelling lasting up to a week with a sting
46
# black widow spider bite general Sx, Tx
Black with red hourglass, aggressive Immediate pain N/V, cramps, rigid abdomen (appy mimic) Treat with ice, opioids/benzos, antivenom (?)
47
# brown recluse general
Brown with “violin,” reclusive Delayed pain, “volcano” lesion Systemic symptoms including hemolysis, necrosis Treat with analgesics, debridement if needed
48
# Rattlesnake bite
Envenomation – amount of venom in bite / up to one quarter are “dry bites” Local effects – swelling, ecchymosis, pain Systemic effect – DIC, capillary leak Males, teens-twenties, intoxicated – highest risk Ooze at site of bite suggests envenomation Do not I&D, tourniquet or try to extract venom If ecchymosis, blisters, systemic effects – treat with antivenom – look up doses, side effects