periodontal pocket and pattern of bone loss Flashcards

1
Q

deepening of the gingival sulcus may occur by what 3 ways?

A

coronal movement of gingival margin, apical displacement of gingival attachment, or combination of both.

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2
Q

define periodontal pocket

A

bottom of sulcus to gingival margin

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3
Q

periodontal pocket is classified as…..

A

gingival pocket or periodontal pocket( infrabony pocket and suprabony pocket)

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4
Q

whats a gingival pocket?

A

formed by enlargement without destruction of underlying periodontal tissues. Sulcus is deepened because increased bulk of gingiva

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5
Q

whats a periodontal pocket?

A

formed via apical migration of attachment apparatus, Includes destruction of supporting perio tissues, either suprabony of infrabony

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6
Q

suprabony pocket

A

aka supracrestal or supraalveolar pockets, bottom of pocket is above alveolar crest

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7
Q

what are some clinical features of periodontal disease?

A

mobility, diastemea, localized pain, perio probing depth, BOP, bluish red, thickened marginal gingiva

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8
Q

what kind of probe shoud you use on an implant?

A

a plastic one

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9
Q

how many sites are there to check while probing each tooth?

A

6

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10
Q

while probing what motions should you use.

A

use little bouncing motions. Its called walking stroke.

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11
Q

why walk the probe?

A

to make sure to record the deepest reading per site.

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12
Q

T/F, probe should be parallel to the long axis of the tooth while walking ?

A

ture

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13
Q

while probing the interproximals you should use an angle. what is this angle?

A

no specific angle , each site is a little different.

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14
Q

perio probing should be ___ of force?

A

.75 N, BOARD ? IF YOU USE THE SAME AMOUNT OF FORCE ON HEALTHY AND DISEASED POCKETS, THE PROBE PENETRATES MORE ON DISEASED POCKETS

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15
Q

first 4 steps of pathogenesis of diseased pockets.

A
  1. bacterial plaque
  2. hosts immuno-inflammatory response triggered
  3. release of cytokines by PMNs, fibroblasts, monocytes and others

4.degeneration of surrounding connective tissue apical to junctional epitheliuim

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16
Q

next 5 steps of pathogenesis of diseased pockets

A
  1. apical cells of JE proliferate along root
  2. coronal end of JE detaches from rooth surface
  3. apical shift of junctional epithelium
  4. pocket depth increases
17
Q

soft tissue

A

Vasoproliferation, vasodilation and engorged blood vessels, length of JE reduced, leukocytes increase and edema occurs

18
Q

what happens to the tooth morphology during perio disease

A

calculus covers cementum, plaque attachement, zone of unattached plaqu, zone of attachement of the junctional epithelium to the tooth, zone of semidestroyed connective tissue fibers.

19
Q

Horizontal bone loss

A

most common, bone reduces in height, bone margin remains perpendicular to tooth surface
( horizontal bone loss results up to 50% loss of interdental septal bone)

20
Q

what 2 problems must you remember with someone who had horizontal bone loss?

A

mobility and functional stability.

21
Q

vertical/angular bone defects

A

occur in oblique direction, leave a hollowed-out trough in the bone alongside the root, base of defect is apical to surrounding bone, mostly seen accompanying infrabony pockets.

22
Q

infrabony defects

A

these are classified on the basis of remaining osseuos walls, ( either 1,2,3 walled defects,

23
Q

which infrabony defect most frequently occurs?

A

three- walled.

24
Q

when being flapped and bone grafted, which infrabony defect has the most success rate?

A

3, ( the higher number of walls the better the success)

25
Q

when a 3 walled defect wraps around the tooth , what is it reffered to as?

A

cercumferential defect.