Perio- ABGD Flashcards

(103 cards)

1
Q

what are the peiodontal disease and condition classification categories

A

perio health, gingival disease and conditions
periodontitis
Other conditions affecting the peridontium

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2
Q

What are the two categories of perio health?

A
  1. health on an intact periodontium
  2. health on a reduced periodontium - 2a: stable perio, 2b: non-perio
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3
Q

What are the 4 levels of perio health

A
  1. pristine
  2. well maintained clinical perio health with a structurally sound/intact perio
  3. perio disease stability with a reduced periodontium
  4. perio disease remission /control with reduced periodontium
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4
Q

What indicates perio health?

A

minimal to no BOP
Shallow pockets or deep “healthy” pockets
Minimal to no radiographic bone loss
Physiologic tooth mobility <0.2mm (unless associated with occlusal trauma with widened PDL)

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5
Q

Which drugs can cause Drug-Induced gingival enlargement.

A
  • Anti-epileptic drugs (phenytoin, sodium valproate)
  • Calcium channel blockers (nifedipine, amlodipine, verapamil, diltiazem, felodipine)
  • Immunoregulating drugs (cyclosporine, high dose oral contraceptives)
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6
Q

What is a genetic/developmental gingival disease that is non-biofilm induced?

A

hereditary gingival fibromatosis

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7
Q

What type of non-biofilm gingival disease are bacterial?

A

necrotizing perio disease, acute streptococcal gingivitis, Neisseria gonorrhoeae , orofacial TB

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8
Q

What type of non-biofilm gingival disease are viral?

A

coxsackie, HSC/herpetic gingivostomatitis, HPV, VZV, molluscum contagiosum

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9
Q

What type of non-biofilm gingival disease are fungal?

A

candidiasis, histoplamosis, aspergillosis

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10
Q

What type of non-biofilm gingival disease are hypersensitivity reactions??

A

contact allergy
plasma cell gingivitis
erythema multiforme

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11
Q

What type of non-biofilm gingival disease are autoimmune disease?

A

pemphigus vulgaris
pemphigoid
lichen planus
lupus erythematosis

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12
Q

What type of non-biofilm gingival disease are granulomatous inflammatory conditions?

A

orofacial granulomatosis, Crohn’s disease, sarcoidosis

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13
Q

What are the 3 sub categories of peiodontitis?

A

Necrotizing perio disease
periodontitis
perio as a manifestation of systemic diseases

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14
Q

What are the three subcategories of necrotizing periodontal disease and how do you tlel them apart?

A
  1. Necrotizing gingivitis: necrosis/ulcer of papilla, bleeding, pain, halitosis, pseudo membranes, lymphadenopathy, fever, sialorrhea
  2. Necrotizing perio: same but with rapid bone loss
  3. Necrotizing Stomatitis: soft tissue necrosis beyond the gingiva, bone denudation, osteitis, severely compromised.
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15
Q

What are the primary flora involved in necrotizing periodontal diseases?

A

Treponema, Selenomona, Fusobacterium, Prevotella intermedia (TSFPi)

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16
Q

How do you define a periodontitis patient?

A

Interdental CAL at >/= 2 non-adjacent teeth
B/L CAL >/= 3mm with PD >3mm detectable at >2 teeth

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17
Q

What are the 3 broad diagnostic categories for perio?

A

Stage, Grade, Extent and Distribution

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18
Q

What are the stages of perio?

A

severity and complexity of management
I: initial
II: moderate
III: severe with potential for addtl tooth loss
IV: severe with potential for loss of dentition

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19
Q

What are the extent and distribution categories?

A

Localized <30% of teeth
Generalized
Molar-Incisor distribution

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20
Q

What are the grades of perio

A

evidence or risk of progression and anticipated response
A: slow
B Moderate
C Rapid

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21
Q

What are the steps to determine perio dx?

A
  1. initial overview- screen the xray PD tooth loss and put them into 1-2 or 3-4 stage
  2. Fine tune the state: look closly at the max CAL/BL determine if BL is H or V, perio tooth loss, complexity including furcation grade, PDs, occlusion, need for extensive multidisiplinary tx. Start at B then judge from there.
  3. Determine grade: hx of perio, risk factors and the ability to control them, response to tx, compliance with recs, and their systemic health factors
  4. diagnose- stage, extended/disp, and grade
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22
Q

In the category of periodontitis as a manifestation of systemic disease(under perio), what diseases are associated with immunologic disorders?

A

down syndrome
leukocyte adhesion deficiency
papillon-Lefevre syndrome
Haim-Munk syndrome
severe neutropenia
primary immunodeficiency diseases such as agammaglobulinemia, hyperimmunoglobulin E G,
Cohen syndrome

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23
Q

In the category of periodontitis as a manifestation of systemic disease, what diseases are associated with those that effet the oral mucosa and gingival tissue?

A

epidermolysis bullosa, plaminohen deficieny

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24
Q

What diseases affect CT (and contribute to perio)

A

Ehler-danlos syndrome, angioedema, and systemic lupus erythematosis

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25
What metabolic diseases and endocrine disorders have manifestations as periodontal disease
glycogen storage disease, gaucher disease, hypophosphatasia, hypophospatemic rickets, Hajdu-cheney syndrome DM obesity Osteoporosis
26
Under the section of periodontal disease and conditions, what 5 things fall under "other conditions affecting the periodontium?"
1. systemic diseases/conditions affecting the periodontal supporting tissues 2. endo perio lesions and abscesses 3. mucogingival deformities and conditions 4. traumatic occlusal forces 5. Tooth and prosthesis related factors
27
Which pathogens are most associated with a periodontal abscess?
Porphyromonas gingivalis, Prevotella intermedia, Prevotella melaninogenica, Fusobacterium nucleatum, Tannerella forsythia, Treponema spp., Campylobacter spp., Capnocytophaga spp., Aggregatibacter actinomycetemcomitans
28
Common pathogens in an endo perio lesion?
P. gingivalis, T. forsythia, Parvimonas micra, Fusobacteriumspp., Prevotellaspp., and Treponemaspp.
29
How to grade EPL?
Grade 1: narrow deep PF in one surface 2: wide and deep in one surface 3. deep >1 surface
30
What are the three gingival phenotype?
Thin scalloped: Slender triangular crown, subtle cervical convexity, interproximal contacts close to incisal edge, narrow zone of KT, clear thin delicate gingiva, thin alveolar bone Thick Flat: Square shaped crowns, pronounced cervical convexity, large interproximal contact located more apically, broad zone of KT, thick fibrotic gingiva, thick alveolar bone Thick Scalloped: Thick fibrotic gingiva, slender teeth, narrow zone of KT, pronounced gingival scalloping
31
What are some pre-disposing factors for recession?
Thin phenotype absence of attached gingiva reduced thickness of alveolar bone incorrect toothbrushing? maybe? intracrevicular restorations Ortho
32
What are the Cairo Recession classifications
Recession Type 1 (RT1): no loss of interprox attachement, interprox CEJ not detectable clinically RT2: Recession with loss of interprox attachement buccal CAL
33
For Cairo RT1, what is the anticipated/predicable root coverage
100%
34
For Cairo RT2, what is the anticipated/predicable root coverage
100% CAN be achieved but not always
35
For Cairo RT3, what is the anticipated/predicable root coverage
100% root coverage cannot be achieved
36
What are the Pini-Parato NCCL Classifications?
Clasa A or B=CEJ, Step = + or - CEJ detectible = A CEJ not detecable = B Step with or without: +/-
37
What are some indicators of occlusal trauma?
Fremitus Mobility Occlusal discrepancies Wear facets Tooth migration Fractured tooth Thermal sensitivity Discomfort/pain on chewing Widened PDL space Root resorption Cemental tear
38
What is? Occlusal force exceeds reparative capacity of periodontal attachment apparatusocclusal trauma, excessive tooth wear or loss
Excessive O Force
39
What is?Injury resulting in tissue changes to PDL, supporting alveolar bone and cementum Occurs in an intact periodontium or in a reduced periodontium
Occlusal Trauma
40
What is Injury resulting in tissue changes on a tooth/teeth with a normal, healthy periodontium Normal CAL, normal bone levels and excessive occlusal force
Primary Occlusal Trauma
41
What is? Injury resulting in tissue changes on a tooth/teeth with a reduced periodontium CAL, bone loss and normal/excessive occlusal force
Secondary O trauma
42
Do excessive O forces initate plaque induced peio disease or loss of attachement?
NO
43
At what roughness threshold is there greated plaque accumulation and increased inflammation?
0.2 micrometer
44
What metals in prosthesis are associated with hypersensitivity reactions that appear similar to gingivitis?
Ni and Pd
45
Overhangs at what size increase crestal bone loss, increased BOP and PDs?
>0.2mm
46
What are some examples of peri implant soft tissue deficiencies?
Tooth loss Lack of buccal bone Periodontitis Papilla height Systemic diseases Keratinized tissue Migration of teeth and life-long skeletal changes
47
What are some explained of implant hard tissue deficiencies?
Tooth loss Defects in healthy situations Trauma from tooth extraction Malpositioning of implants Periodontitis Peri-implantitis Endodontic infections Mechanical overload Root fractures Soft-tissue thickness General trauma Systemic diseases Bone height in posterior maxilla Systemic diseases
48
Describe the Seibert Classifications
Class 1: B-L Loss Class 2: A-C loss Class 3: combo of 1 and 2.
49
What are the categories in Peri implant disease and conditions?
Peri-Implant Health Peri-Implant mucositis Peri-implant Implantitis Peri- Implant Soft and Hard tissue deficiencies
50
Describe the glickman grades
Grade I: pocket into flute, bone intact II: into furcation III: through and through but with soft tissue coverage IV: no soft tissue, through and through
51
Is the absence of BOP a sign of stability? What about suppuration?
Yes- to BOP No- to Suppuration
52
What cells are in suppuration?
PMNs
53
Describe the Hamp, Nyman Lindhe Furcation classification?
Degree I: Horizontal loss <3mm Degree II: >3mm loss but not the entire widt Degree III: through and through
54
What are the four broad approaches to the management of diseased sites?
Reflective Resective Additive Combination
55
What medication is Arestin?
Minocycline
56
What are the 2007 prognosis categories?
favorable, quesitonable, unfavorable, and hopeless
57
With osseous resection, what side would you approach from and why?
P and L: avoids B furcation exposure avoid shallow vestibule more thicker bone more keratinized tissue (max) increased embrasure size natural cleansing of from the tongue
58
What are 5 causes of a gummy smile and how to tell them apart?
Altered Passive eruption: CEJs Dental Alveolar extrusion: Wear Vertical Maxillary Excess: disproporitionate face height: look at Glabella to base of nose, BON to chin. Short Upper lip: <20mm Hyperactive upper lip: lip mobility is 6-8mm (botox)
59
What options do you have for furcation management?
Increase access, elimination of furcation, and non-surgical scrp
60
What procedures increase access for furcation?
Tunneling, Odontoplasty/barreling, gingivectomy/flap, osteoplasty/ostectomy,
61
What procedures help to eliminate the furcation?
coronally positioned flap, obilteration guided tissue regeneration osseous graft Root resection and bicuspidization
62
Are autographs osteogenic, osteoinductive, or osteoconductive?
all three
63
Are allografts osteogenic, osteoinductive, or osteoconductive?
Freeze dried Bone Allograft- OC Demind FDBA - OC and OI
64
Are xenografts osteogenic, osteoinductive, or osteoconductive? What are some examples?
Osteoconductive Osteograft N300,700, - HA BioOss BioOss Collagen (bovine bone, + porcine collagen)
65
describe the miller classifications of recession
1: normal periodontium, recession does not extend past MGJ 2. normal periodontium, recession pas MGJ 3. moderate interprox attachment loss and recession past MGJ 4. advanced/severe interprox loss with recession past MGJ
66
What are some reasons to graft a mucogingiva defect?
progressive recession esthetics, sensitivity deepen the vestibule relieve the fremum, pre-prosth pre-ortho
67
what is the amount of attached gingival tissue that is considered adequate?
2+mm
68
Miller 1-2: is it predicable for grafting? What about 3-4?
1-2: yes 100% root coverage 3-4: not predicable
69
What kind of mucosal grafting techniques are there?
Free soft tissue autografts Pedicle grafts CT grafts combination grafts GTR
70
What are some techniques for CT grafting?
Raetzke Pouch Langer-Langer Coronally Advanced Flap Lateral Pedicle Graft Tunnel Technique Alloderm
71
What are the 4 bone types?
1-4
72
At what temp will bone necrose?
47*C, (116* F). for 1 min.
73
What is peri-mucositis?
an inflammatory lesion of the soft tissues surrounding an endosseous implant in the absence of loss of supporting bone or continuing marginal bone loss. caused by plaque accumulation smoking lack of compliance with maintenance implant prosthesis design is over contoured and submarginal restorative margins radiation more evidence is needed for potential risk factors like diabetes, lack of KM, and excess cement.
74
Describe Peri-implant Implantitis
Clinical signs of inflammation (may include erythema, edema, BOP, suppuration) with loss of supporting bone and/or progressive bone loss How can it present itself? -bone loss pattern is circumferential or as a facial dehiscence with semicircular bony defect What are risk factors? -history of periodontitis, smoking, diabetes, poor OH/lack of maintenance
74
How can periapical peri implantitis occur?
direct correlation between periapical peri implantitis and adjacent endo related infection
75
What are the three red complex bacteria associated with perio disease and describe them
TANNERALLA FORSYTHIA- bacillus, gram neg, non-motile, anaerobic TREPONEMA DENTICOLA- spirochete, gram neg, motile, anaerobic PORPHYROMONAS GINGIVALIS- bacillus, gram neg, non-motile, anaerobic ALL anaerobic, gram neg. BSB, NMN
76
What is the new term for biological width? Why is it important for periodontal health? What does it consist of and its average measurements?
Supracrestal attached tissues It is vital in the preservation of periodontal health by acting as a natural barrier around the teeth to protect the alveolar bone from disease and infection. 2.04mm: total 0.97: JE 1.07":CT
77
Explain the concepts of repair and regeneration?
Repair: Healing of a wound tissue that does not fully restore the architecture or the function of part of the long junctional epithelium Regeneration: Reproduction or reconstitution of a lost or injured part- Bone, cementum, PDL, CT, are reformed in a region that has been deprived of its attachment apparatus.
78
In term of regeneration, what does "new attachment" and "reattachment" mean and what is the difference?
new attachment: union of ET and/or CT with a previously disease rt surface Reattachement: reunion of ET and/or CT with rt surfaces and bone after incision or injury One is diseased and other is not Think flap reflection for healthy teeth for gingivectomy= reattachment.
79
What is guided tissue regeneration? What is used to achieve regeneration?
Attempt to regenerate lost perio structure through differential tissue responses Bone graft and barrier are used to facilitate GTR
80
Why is a barrier used in GTR?
retard apical migration of ET and exclude CT from the root or existing bone surface in the belief that the interfere with regeneration.
81
Whta are examples of a reduced periodontium situation?
stable perio patient or non-perio with bone loss/ recession from abrasion
82
Broadly, what are the Perio 2017 classifications?
Major Categories: 1. Periodontal Health, Gingival Diseases, and Conditions (3) 2. Periodontitis (3) *Significant changes in labeling of Periodontitis with Staging, grading, and extent/distribution 3. Peri-implant diseases and conditions (4) 4. Other Conditions affecting the Periodontium (5)
83
What are the categories wihtin perio health, gingival diseases and conditions?
periodontal health and gingival health gingivitis: dental biofilm induced gingival disease: non-dental biofilm induced.
84
Name the categories within periodontitis
necrotizing perio disease periodontitis perio as a manifestation of systemic disease
85
name the categories under peri implant diseases and conditions?
perio implant health peri implant mucosistis peri implantitis peri implant soft and hard tissue deficiencies
86
Name the categories within other conditions affecting the periodontium
systemic diseases or conditions affecting the perio tissues periodontal abscesses and endo/perio lesions mucogingival deformities and conditions traumatic occlusal forces tooth and protheses related factors
87
According to the new perio classification- describe a state of health
pristine periodontium well maintained and intact stable (even if reduced) excellent OH lack of palque and calc no CAL shallow PDs (<3mm) with no BOP <--- best predicotr of health
88
How would you describe the etiology of perio disease
multifactorial- bacterial plaque in a susceptible host subgingival microorganisms host immune-inflammatory response local factors ( pockets, restorations, anatomy, crowding) systemic factors (immune, disease, genetics) Environmental (smoking, meds, stress, nutrition)
89
What are some meds that cause gingival enlargement?
cyclosporin- immunoregulating drugs Calcium Channel Blockers- Nifedipine, verapamil, dilitazem, amlodipine Anti-epiletics----Phenytoin, sodium valproate
90
Name some system factors that can lead to gingival disease
Sex steriod hormones hyperglycemia leukemia smoking malnutrition large subg restors hyposalivation
91
What are the features of a bioflim induced gingival disease?
Inflammation confined to the free and attached gingiva BOP and gingival erythemia/edema high bac load stable attachement levels tenderness with probing halitosis prereq to perio, reversible
92
Name examples of non-biofim induced disease
gingival fibromatosis bacterial, viral, fungal- necrotizing, acute strep gingiva etc hypersensitivity rxns autoimmune disease granulomatosus inflammatory conditions reactive- epulides - fibrous epulis, pyogenic granuloma, Perf GCG Neoplasm (leukoplakia, SCC) endocrine (vitamin) Traumatic lesion ging pigmentation- smokers melanosis, amalgam tattoo
93
What are the 3 types of necrotizing periodontal diseases?
necro ging- ulcer of papillae, ging bleeding, pain, fever, pseudomembranes, lymphadenopathy, necro perio- + rapid bone loss Necro stomatitis - necrosis beyond the ging, bone denudation through mucosa, osteitis, boney sequestrum, severely compromised
94
What is the difference between necro perio and necro gingi?
rapid bone loss with perio
95
What are the pathogens in necrotizing perio?
Treponema selenomonas fusobacterium prevotella intermedia PTSF like PTSD(but F)
96
Name some systemic risk factors for necrotizing perio disease?
compromised host HIV/AIDs <200 with a viral laod Malnutrition Physiologic stress lack of sleep inadquete OH, hx of NPD, ging tobacco/etoh use young, white, winter
97
What dies the statement mean " a perio pt is a perio pt for life"?
never leave the risk level of perio. always at a higher risk than someone who never had
98
With the 2017 guidelines, what is the definition of someone with perio
Interdental CAL at >2 non-adjacent teeth B or L CAL >3mm with pocketing >3mm detectable at >2 teeth
99
Name an example of CAL that would not be considered for CAL periodontitis?
TB abrasion trauma loss dental caries on cercial aspect of tooth presence of CAL on distal 2nd molars due to 3rd molars endo lesion draining through marginal gingiva VRF
100
DOes staging and grading help you come to a dx?
No. dx is determined first. THEN stage and grade to clarify extent, severity and complexity inaddition to rate of disease progression and predicted response to therapy and impact to systemic health
101
Can you stage/grade by quad?
No. Whole mouth only
102