Pediatrics: cardiopulmonary Flashcards

1
Q

Pulmonary and systemic pressures in
1. utero
2. before birth
3. after birth

A
  1. In utero: increased pulmonary pressure
  2. due to lungs being a fluid filled system, the lungs are a higher pressure system than the sysematic circulation
  3. increased systemic pressure now that the lungs are filled with air; the lungs are a lower pressure system than the systemic circulation. The blood will flow the path of least resistance
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2
Q

congestive heart failure in children

A
  • heart is unable to provide adequate cardiac output to meet the cirulcatory metabolic requirements of the body
  • initially failure may right or left sided
  • if left untreated the entire heart will fail
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3
Q

Causes of CHF in peds

A
  • heart muscle dysfunction
  • strucutral abnormalities
  • pulmonary abnormalities
  • systematic disease examples: juvenile idiopathic arthritis, sjorgren syndrome
  • infectons example: myocarditis or kawasaki disease
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4
Q

What are other examples of causes of cardiovascular disease in pediatrics

A
  • obstructive lesions in heart
  • dysrhythmias
  • chemotherapy drugs
  • sepsis
  • respiratory failure
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5
Q

what are symptoms seen with CHD in pediatrics

A
  • failure to gain weight in infancy
  • right sided venous congestion
  • hepatomegaly
  • left sided pulmonary venous congestion
  • tachypnea
  • central cyanosis
  • fatigue
  • sweating
  • dyspnea or breathing abnormalities/sounds
  • altered level of concciousness
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6
Q

Congestive heart failure management in children

A
  • control fluid status diuretics such as lasix or spironolactone
  • limit PO intake (intially)
  • fluid sodium restrictions daily
  • (BID) weight and maintain nutritional status
  • nutrition avoids fluid retention
  • address underlying disorder
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7
Q

Pediatrics: cardio pulmonary rehab

components/factors to think about

A
  • aerobic
  • resistance
  • flexibility
  • divide as equally as possible
  • correct determination of the dosage and intensity of exercise for optimal benefits
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8
Q

describe pediatric cardiac rehab

what types of interventions

A
  • aerobic exercise are endless (fun for kid)
  • resistance and flexibility training 2-3 times per week and incorporate all major muscle groups
  • resistance training machines are safer than free weights
  • fixed pattern of movement and easer to learn
  • all stretches should be performed to the point of mild discomfort and sustained for 30-60 seconds
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8
Q

what is the most common cardiomyopathy

A
  • dilated cardiomyopathy
  • left ventricle is enlagred and weakened
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9
Q

rehab guideline for cardiopulmonary in peds: initial treatment

A
  • 24 visits spanning 12 weeks of training; three visits per week would be optimal, however two is acceptable
  • the execise session: last approximately one to 1.5 hours
  • should consist of aerobic exercise and resistance training with warm up and cool down periods
  • blood pressure and heart rate should be monitored
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10
Q

Pediatric cardiac rehab phases

A
  • baseline assessment
  • intensive phase: 2x per week with aerobic, resistance and flexibility
  • completion assessment
  • maintainance phase: home based with follow up appointments as often as deemed necessary
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11
Q

cardiac problems affect on development in peds

A
  • evidence shows that children have more developmental problems
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12
Q

long term outcomes in children with congential heart disease

A
  • compared to children without CHD, children with CHD were 3 times more likely to report worse health in the last year
  • 3 times more likely to have missed more than 10 days of school or day care
  • more likely to need help with or to have had difficulty with crawling, walking, or running, or to have needed special equipment for these activities
  • as children with CHD got older they were more likely to report a learning disabiility
  • ADHD or ADD or an intellectual disability
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13
Q

Congential heart defects: etiology

A
  • genetic factors
  • abnormalities
  • teratogens: drugs
  • maternal infections
  • environmental exposures
  • prematurity
  • advanced maternal age
  • pregnancy complications
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14
Q

types of congential heart defectts

A
  • patent ductus arteriosus
  • arterial septal defects
  • ventricular septal defect
  • atrioventricular canal defect
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15
Q

Ventricular septal defects

A
  • most common
  • most small/close spontaneously (usually by age 2 years; uncommon after age 4)
  • symptoms of congestive heart failure may occur especially if significant size
  • child has failure to thrive/fatigue, respiratory, pulmonary hypertension
  • murmur (turbulent flow through abnormal or obstructive opening)

murmurs are hard to hear in peds

16
Q

VSD contributing factors are

A
  • a congential cardiovascular defect in a parent or sibling
  • maternal diabetes
  • maternal alcohol consumption for muscular VSD
17
Q

4 types of VSD

A
  • perimembranous
  • muscular VSD
  • conal VSD
  • inlet VSD
18
Q

inlet VSD

A
  • occurs in the lower right ventricle and adjacent to the tricuspid valve
  • typically occurs in patients with down syndrome
19
Q

patent ductus arteriosus

A
  • from week 6 of fetal life until birth the ductus is responsbile for most of the right ventricular outflow
  • normally functional closure of the ductus arteriosus occurs by about 15 hours of life in healthy full term infants
  • in full-term infants PDA accounts for 5-10%
  • in preterm infants incidence of PDA up to 60%
20
Q

atrial septal defects

symptoms

A
  • most children with isolated ASDs are asymptomatic
  • if patients are untreated however symptoms can occur in adulthood
  • patients who have large arterial shunts can experience symptoms related to excess pulmonary blood flow and right sided heart failure
  • in infants, children and young adults up to approximately age 20 symptoms may include: heart murmur, frequent respiratory infections , slow weight gain
21
Q

atrial septal defects: is associated with

What types of characteristics

A
  • fetal alcohol syndrome
  • cigarette smoking particularly in the first trimester
  • advanced maternal age >35
  • certain antidepressant use
  • diabetes

maternal characteristics and behaviors:

22
Q

Tetralogy of fallot

causes? when is it dx? what happens?

A
  • caused by genetics and environmental impacts of mother during pregnancy
  • usually diagnosed after birth
  • episode of turning blue during crying or feeding (tet spell)
  • baby may have bluish-looking skin or heart murmur
  • however it is not uncommon for a heart murmur to be absent righ at birth (several people listen)
23
Q

4 defects with tetralogy of fallot

A
  • right ventricular hypertrophy
  • overriding aorta
  • ventricular septal defects
  • pulmonic stenosis
24
Q

Tetralogy of fallot symptoms

A
  • cyanosis,
  • systolic murmur
  • metabolic acidosis
  • poor growth
  • clubbing
  • severe hypoxia
  • surgical treatment: palliative shunts
25
Q

tet spells hypercyanotic

A
  • occurs in first year of life
  • may be preceded by feeding, crying or defecation, dehydration, fever, increased stressed
  • characterized by hypoexmia, blue extremities circumoral cyanosis
  • darker colored skin
  • increased hemoglobin and hematocrit counts
  • requires immediate attention and treatment to prevent brain damage and death
26
Q

hypoplastic left heart syndrome

A
  • strucutres of the left side of the heart are underdeveloped
  • mitral and aortic valves closed or small
  • left ventricle non funcitonal
  • 4th most common congential heart defect
27
Q

Hypoplastic left heart syndrome: signs

A
  • low levels of oxygen in the blood (detected by pulse oximetry new born screening)
  • problems breathing
  • pounding heart
  • weak pulse
  • ashen or bluish skin color
27
Q

treatments for hypoplastic left heart syndrome

A
  • medication
  • nutrition
  • surgery