Patho-cardio Flashcards

1
Q

most specific blood test for MI

A

troponin (increases substantially within 4-6 hours and peaks 10-24 hours

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2
Q

initial compensation for congestive heart failure to keep up with cardiac output

A

cardiac hypertrophy

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3
Q

CHF “forward effect”

A

less blood reaches organs

  • decreased cell function leads to lethargy and fatigue
  • mild acidosis from the build up of lactic acid = increased respiration
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4
Q

CHF “back up congestion”

A

blood sits in ventricle b/c less blood being pumped out by affected ventricle than the amount of blood coming into the ventricle

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5
Q

when blood accumulates in pulmonary circulation (pulmonary edema)

A

left-sided CHF

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6
Q

when blood accumulates in systemic circulation (peripheral edema)

A

right-sided CHF

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7
Q

“forward effects” signs and symptoms (SAME FOR BOTH SIDES)

A

general hypoxia, dyspnea, fatigue, dizziness, exercise intolerance, cold intolerance

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8
Q

“back-up”effects signs and symptoms (LEFT SIDE)

A

cough, dyspnea, orthopnea (fluid accumulates in lungs while laying down), Paroxysmal nocturnal dyspnea

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9
Q

compensation mechanisms CHF

A

tachycardia, vasoconstriction, daytime oliguria

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10
Q

“back up” effects signs and symptoms (RIGHT SIDE)

A

increased blood volume in systemic circulation (edema), jugular vein distention, cerebral edema (flushed face, headache), ascites (distention of tummy)

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11
Q

high blood pressure is defined as having ____ mmHG

A

140/90 mmHg

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12
Q

pre-hypertension

A

bp between 120/80 and 139/89

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13
Q

localized dilation and weakening of arterial wall from defect in tunica media

A

aortic aneurysm

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14
Q

develops when there is a tear in the tunica intima of the wall and blood continues to dissect tissues

A

dissecting aneurysm

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15
Q

circumferential dilation along a section of an artery

A

fusiform aortic aneurysm

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16
Q

Defective heart valves are infected by normal flora (Streptococcus viridans; mouth)

A

subacute infective endocarditis

17
Q

Normal heart valves are infected by highly virulent organism (Staphylococcus aureus)

A

acute infective endocarditis

18
Q

May compress the heart (cardiac tamponade) → impair expansion and filling → decreasing cardiac output

A

pericardial effusion

19
Q

slows conduction of heart impulses and HR but the contractions are stronger

A

digoxin

20
Q

prevents SNS stimulation of the heart

A

beta blockers

21
Q

Ca++ channel blockers

A

decrease cardiac contractility and vasodilates smooth muscle

22
Q

ACE inhibitor

A

prevents vasoconstriction to decrease heart workload and decrease blood pressure

23
Q

What converts angiotensin 1 to angiotensin II?

A

ACE

24
Q

Where are lipids synthesized?

A

liver

25
Q

inhibit cholesterol production in liver

A

statins

26
Q

what might you have if you feel relief when you lean forward or sit upright to breathe?

A

pericarditis