Part 21 Flashcards
Most common type of distributive shock
-Sepsis (sepsis is the next step of SIRS -systemic inflammatory response phenomenon)
Nonhemorrhagic hypovolemic shock is most often due to ___ losses
Gastrointestinal
Third space losses
A type of nonhemorrhagic hypovolemic shock where fluid in a potential space sees filling due to capillary leakage (think peritoneum or pleura) and this depletes intravascular volume overall
Massive transfusion protocol
1:1:1 of packed RBC (first choice O neg, Op pos and type specific are alternatives), plasma, and platelets
Neurogenic shock pathophysiology
Dilation of the arterial and some degree of the venous side of the systemic circulation
Examples of obstructive shock (5)
(this is the least common type)
- PE
- tension pneumothorax
- pericardial tamponade
- constrictive pericarditis
- abdominal compartment syndrome
Swan ganz catheterization of the right heart to obtain pulmonary capillary wedge pressure gives a measure of ___
left atrium
Cardiogenic shock diagnosis (2)
- pumonary artery catheterization
- echocardiogram of left ventricle
SIRS criteria (4)
- temp >38 or <36C
- HR >90bpm
- RR >20 or PaCO2 <32 (respiratory alkalosis)
- WBC count >12000 or <4000 or >10% immature bands
qSOFA criteria for sepsis (3)
- RR >22
- SBP <100
- altered mental status (due to suspected infection)
Sepsis treatment options (3)
- administer broad spectrum antibiotics piperacillin, ampicillin
- steroids that mute the immune response (leukotriene storm)
- initiate pressors as needed (norepi)
Goal directed therapy for sepsis (4)
Meet the following criteria:
- Central venous o2 sat of > or =70
- central venous pressure > or =8-12
- mean arterial pressure > or = 65
- urine output > or = .5cc/kg/hr
Neurogenic shock etiologies (4)
- spinal anesthesia
- vagal stimulation
- cord transection
- NOT head trauma
Why don’t you use antihistamines for anaphylaxis?
Because most mast cells have already degranulated at the point of anaphylaxis presence
Conduction requires ___ while convection is dependent on ___
physical contact, wind velocity air and water vapor molecules moving around the body
Thermoregulation mechanism of action
- increased body temp
- thermostat in hypothalamus activated by temp receptors
- skin blood vessels dilate so warm blood allows heat to radiate from the skin surface
- sweat glands activated increasing evaporative cooling
- when body temp decreases
- thermostat in hypothalamus activated by temp receptors
- skeletal muscle activated to begin shivering to generate heat
- skin blood vessels constrict diverting blood from skin to deeper tissues reducing heat loss from skin surface
- body temp increases
Heat rash (prickly heat) definition and treatment
Skin irritation due to blocked sweat ducts trapping sweat beneath the skin, typically found on the neck, chest, groin, in skin folds, might be papular, pustular, or vesicular, may sting or be pruritic but typically self limiting and not a problem, can be prevented by wearing loose fitting clothing, avoiding extreme heat, etc
Heat edema definition and treatment
Dependent edema from vasodilatory pooling, too much sodium can aggravate fluid retention, greater risk in rapid transiton from cold to warm climate, treatment is NOT diuretics but rather just elevate extremities and put in a cool environment
Heat exhaustion signs and symptoms (4)
- early identification critical to prevent progression to heat stroke
- fatigue/malaise/weakness
- N/V/D
- in tact mental status
Heat exhaustion treatment options (3)
- move to cool area, ice packs, remove excess clothing, spray with lukewarm water, trendelenberg, etc
- gradual rehydration oral and or IV
- monitor for progression to heat stroke (patients should respond if all they have is indeed heat exhaustion)
Heat stroke signs and symptoms (4)
- Elevated core temp >40.5C
- Hot dry skin
- CNS symptoms and lack of intact mental status
- NVD
Heat stroke complications (2)
- high core body temp leads to multisystem damage (DIC, hepatocellular necrosis, acute kidney injury)
- cerebral hypoperfusion leads to mental status changes and can cause cereberal ischemia
Heat stroke diagnostic studies (5)
- CT of head to rule out edema
- CXR
- CBC, CMP, PT/PTT
- EKG
- urinalysis (rhabdo concerns)
Heat stroke treatment options (5)
- Rapid cooling measures within 30 min
- ice water immersion most effective
- antipyretics ineffective and may be harmful
- IV hydration
- admit to hospital
2 types of cold injuries
- Freezing (frostbite)
- nonfreezing (chilbain or trench foot)
Frostnip definition and treatment
Mildest form of peripheral cold injury, superficial nonfreezing cold injury secondary to vasoconstriction, sees pale skin with associated numbness and paresthesias but skin is still pliable***, occurs in the apical structures (ears, nose, hands, feet), treated by simple warming techniques
Chilblains (pernio) definition and treatment
More severe than frostnip caused by exposure to nonfreezing temps and damp air, onset 1-5 hours of cold exposures but can be longer, develops over hours and subsides slowly over weeks, develop red to violet raised lesions (papules and nodules) most often on unprotected extremities such as fingers and toes, seen most often in middle aged women or those with chronic conditions such as raynauds, treated with local heat, gentle massage, nifedipine, corticosteroids, minimize cold exposure to prevent recurrent lesions or secondary infection
Immersion (trench) foot definition and treatment
Occurs when feet are wet but not freezing for prolonged periods of time, most common in homeless and military starting with numbness and tingling pain with pruritis progressing to leg cramps and complete numbness, managed with gentle rewarming, careful washing and air drying, slight limb elevation, possible bed rest, prevented by keeping feet dry at least 8 hours a day
Frostbite definition and its 4 degrees of severity
Skin and deeper structure freeze resulting in tissue injuries, water crystals disrupt cell membranes and tissue structures resulting in ischemia and death of cells, has 4 degrees of severity (hyperemia and edema, accompanied by blisters, has hemorrhagic fluid in the blisters, and comprete necrosis with gangrene)