Parkison Disease Flashcards

(38 cards)

1
Q

What is parkison disease?

A

Chronic progressive neurodegenerative disorder characterized by slowness in the initiation and execution of movement ( bradykinesia ), increase muscle tone ( rigidity ), tremor at rest and gait changes

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2
Q

From the PowerPoint what does Parkinson’s disease mean?

A

Generalized slowness- loss of dopamine
( a neurotransmitter in the brain needed to initiate movement )

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3
Q

Tremor at rest usually is the ___
What happens to the hand writing?

A

First sign
Large & trails off

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4
Q

Slowness in initiation of movement is called?
What happens to the arm?
What happens to swallowing?
What happens to blinking, facial expression, posture?

A

Bradykinesia

Arms swinging
Decreasing swallowing saliva
Decrease everything

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5
Q

Why do patient have an increase muscular tone ?

A

Because they have jerking quality
( cog wheel rigidity )

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6
Q

Parkison is more common in who?

A

Men

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7
Q

Is there a specific test to Parkinson’s disease? And if yes/ nowhat is it?

A

No
Only based on a history and physical assessment
And positive response to anti parkison medication

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8
Q

Parkison effects how many people
And increases with?

A

160/10000
Age

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9
Q

Clinical manifestations
Pd is what and how does it occur?

A

Gradual with ongoing progrsssion

1 limb or 1 side of the body may be effect first

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10
Q

What are the 4 common manifestations?

A

Tremors
Rigidity
Bradykinesia
Postural instability

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11
Q

Tremor is often the first __
It’s more prominent at ___
It worsen by ___
Hand tremor is called as ___

Why is it called that ^
How does this effect hand writing?

A

Sign
Rest
Stress
Pill rolling

Because of the thumb and forefinger appear to move in a Dorsey fashion

Makes it large and trails off

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12
Q

What is rigidity?

What is the jerky quality called?

A

Increased resistance to passive motion when limbs are moved through their range of motion

Cogwheel Ridgity

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13
Q

Does rigidity make the patient feel tired ? Why?

A

Because the constant uncontrolled movement

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14
Q

What is bradykinesia?
What is this due from?
What will a normal patient have vs PD

A

Slowness if movement
Decreased impulses from the basal ganglia

Involuntary movement
( blinking normally )
Vs PD won’t
Swinging arms, blank facial expression

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15
Q

What are the physical attributes of PD (6)

A

Blank facial expression
Slow monotonous slurred speech
Forward tilt to posture
Tremor
Short,shuffling gait
Drooling

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16
Q

What is festination mean?

A

Shuffling gait

17
Q

What is postural instability? (2)

What test do we do for this?

A

Propulsion ( going forward )
Retropulsion ( going backward)

Pull Test
- examiner stands back and gives a tug and patient falls back

18
Q

What are the complications of PD? (10)

A

Dysphagia ( swallowing )
Malnutrition
Aspiration
Immobility problems
Orthostatic hypotension
Falls
Dyskinesias( spontaneous involuntary movement )
Weakness
Neuropsychiatric proeblems
Dementia

19
Q

What’s an anagram to help me remember the 10 complications of PD?

A

Danny
Mails
Annie
Ice
Over
Favorite
Dyes
Water
Neurons
Dimes

20
Q

Does PD have a cure? So we focus on?

A

No
Symptom management

21
Q

Why do PD patients have an aspiration risk?

A

Slurred speech
Drooling

22
Q

What do PD patients have a fall risk?

A

Tremors & postural

23
Q

How to reduce risk of aspiration? (5)

A

Assess for gag reflex
OOB to chair for meals
Full fowlers position
Speech consult
Medications

24
Q

How do you reduce falls for PD patients? (7)

A

Out of bed with assistance
Position change slowly
Walker
Call bell
Soled shoes
Physical therapy
Medications

25
Staff must be educated on parkison diseases when giving medications What is the point of medications? What is “on and off time” What can medications cause? What is it ^ Examples?
Correct imbalance of dopamine On medication = patients can initiate movement Off medication = patient decrease movements Dyskinesia Uncontrolled movements Face hands, arms
26
Anti parkison drugs will either do what two things?
Release or supply dopamine Black the effect of the overactive cholinergic neurons in the striatum
27
You must be very careful when giving PD drugs why?
Because it can cause paradoxical intoxication
28
What are the 4 PD drugs ?
Hytrin Sinemet Lexapro Celebrex
29
What does Hytrin & sinemet cause? Function of Hytrin ? Function of sinemet?
Orthostatic hypotension Control hypertension & decrease BPH Control PD symptoms
30
What does Celebrex (NSAID) may cause? (Negative effect ) Function?
Decrease effectiveness of Hytrin ( hypertension ) Decrease inflammation & pain of degenerative joint disease
31
What does lexapro do?
Help with depression
32
The patient with PD will have slurred and speech problems so we will give them which therapy?
Speech therapy
33
How can we help improve communication with PD patients? (4)
Be patient Do not finish sentences Assist family Don’t be loud
34
What has a negative impact on levodopa ( sinemet ) drugs? How so? Patients want to have more during when And less during when?
Proteins It impairs levadopa absorption More during bed time Less during day time
35
Patients should be on what diet? To prevent?
High fiber Constipation
36
Pd patients should what types of bites ? Should they have adequate hydration? To prevent ^
Small Yes Orthostatic hypotension/constipation
37
Why can’t we give coffee?
Aggravate symptoms But may help with apathy/drowsiness
38
Home care notes Promote exercise Encourage loud speaking Big movements Chairs with arms Raise back legs Know side effects