Orthopaedics in Children Flashcards

1
Q
A
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2
Q

common orthopaedic presentations in children

A

fractures - salter harris classification
osteochondritis - perthes
SCFE
Scoliosis

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3
Q

common musculoskeletal and sports presentation

A

apophyseal inflammation and avulsion
hip complaints e.g FAIS

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4
Q

What outcome measure may be used to examine the child’s musculoskeletal system

A

pediatric gait, arms, legs, spine screen
screening questions - ask about pain or stiffness across joints muscle back, ask about difficulty with functional tasks e.g going up/down stairs or dressing yourself
gait assessment - do they walk in tip toes/heels

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5
Q

what should be assessed before you approach the child for an orthopaedics physio session

A
  • Review notes
  • PEWS*
  • Talk to Nursing Staff
  • Has the child had pain relief
  • Has the child had any anxiety/fear
  • Is there a parent present
  • Communication – is the child likely to be impulsive
  • Are there any attachments IVs etc
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6
Q

what physical differences are between between children and adults

A

articular cartilage thicker in children
junction b/w epiphyseal plate and metaphysis is vulnerable to shear forces and disruption in children
relatively weak apophyses
metaphysis of long bones in kids resilient and elastic - prone to greenstick fracture
growth temporarily impacts co-ordination

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7
Q

classification salter harris fracture

A

fracture involving physis/growth plate

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8
Q

describe a type 1 salter harris fracture

A

slipped - complete physeal break comprised of 2 components
2 components are slightly displaced

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9
Q

describe a type 2 salter harris fracture

A

physeal fracture extends to Metaphysis = produce chip fracture

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10
Q

describe a type 3 salter harris fracture

A

a physeal fracture that extends to the articular epiphysis

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11
Q

describe a type 4 salter harris fracture

A

a complete physeal fracture that includes metaphysis and ephiphysis

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12
Q

describe a type 4 salter harris fracture

A

a complete compression physeal fracture of the growth plate

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13
Q

what are the clinical implications of a type 1 salter harris fracture

A

Most common (75%), uncomplicated and 2-3 week healing time

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14
Q

what are the clinical implications of a type 2 salter harris fracture

A
  • Triangular fragment can be reduced normally and cast applied
  • Sometimes open reduction needed
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15
Q

what are the clinical implications of a type 3 salter harris fracture

A
  • Mostly seen in older children when growth plates have begun to close
  • Most require ORIF
  • Can form a pyseal bar leading to assymetric growth
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16
Q

what are the clinical implications of a type 4 salter harris fracture

A
  • Fracture through all elements – ORIF often
  • If growth hs ceassed then ok
  • If not, careful monitoring needed to ensure no physeal bar/grwoth disturbance
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17
Q

what are the clinical implications of a type 5 salter harris fracture

A
  • Often missed, spotted when growth has be impacted.
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18
Q

SCFE what is it?

A

Slipped upper femoral epiphysis/ Slipped capital femoral epiphysis
damage to the growth plate of the femur
resulting in the head of the femur to slip and become misaligned with the remaining femur

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19
Q

what is a risk factor of slipped capital femoral epiphysis

A

obesity

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20
Q

explain the epidemiology of slipped capital femoral epiphysis

A

Bilateral in ~ 20% of cases
* Affects boys more than girls
* Affects boys later (10-17) than girls (8-15

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21
Q

what are the presentations of slipped capital femoral epiphysis

A
  • Antalgic gait
  • Pain in groin thigh or knee
  • If you flex the hip it will externally rotate and abduct
  • Advise to restrict weight bearing
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22
Q

potential complications of slipped capital femoral epiphysis

A

Osteonecrosis (risk up to 50% in untreated SUFE)
Chondrolysis (risk 7%)
Osteoarthritis
Femoral acetabular impingement - severe

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23
Q

what is osteochondritis

A

joint disorder in which a segment of bone and cartilage starts to separate from the rest of the bone after repeated stress or trauma.

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24
Q

what is perthes disease

A
  • Idiopathic avascular necrosis of the head of the femur
  • Ostechondrosis
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25
Q

what are risk factors of perthes disease

A

transient synovitis*, paternal smoking/passive smoking in pregnancy,
thrombophilia,
low birthweight,
family history

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26
Q

where in the body does osteochondritis dessicans typically occur

A

medial femoral condyle
capitellum
talar dome

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27
Q

what is the typical presentation of perthes disease

A
  • Pain and limp
  • Pain in groin, thigh or knee
  • Pain increases with hip movement
  • Decreased hip medial rotation and abduction
  • Thigh muscle atrophy
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28
Q

what are the possible consequences of perthes disease not being appropriately addressed

A

can cause leg length discrepancy
may need THR long term

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29
Q

what long surgical interventions available for perthes disease

A
  • Pain relief with anti-inflammatories
  • Observation
  • Protected weight bearing
  • Splinting/casting - Petrie
  • 4-6 weeks
  • Bed rest with traction
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30
Q

what surgical interventions are available for perthes disease

A

Femoral Osteotomy with plates and screws to fix alignment
* May require pelvic osteotomy if child is under 8

31
Q

what is the aetiology of scoliosis

A

idiopathic
neuromuscular - CP, Muscular dystrophy
Secondary - leg length discrepancy

32
Q

what is the typical presentation of idiopathic scoliosis

A

strong family hx
present around early adolescence (Adolescent Idiopathic Scoliosis - AIS)
Uneven shoulder - symmetric scapular prominence
uneven pelvis
rib prominence

33
Q

What tests are conducted during an objective of a patient with suspected idiopathic scoliosis

A

leg length - measure in supine
adams forward bend test - may still be impacted by LLD
feet together, knees straight, bend forward, free arms
+ive if asymmetry in ribs is present
cobb angle

34
Q

how is scoliosis classified using the cobb angle

A

angle opposite apex
and between the end vertebrae
0-10 degree ignore unclassified
10-20 - mild monitor and reassure
20 and young child - intervene
30-40 in skeletally mature - unlikely to progress

35
Q

conservative management of scoliosis

A

bracing - no evidence in fully grown
some evidence if more than 40 degrees lateral curvature
high risk of progression

36
Q

what is the schroth method

A

a conservative intervention for scoliosis
provide exercises based on unique curvature of spine
address anatomical planes - sagittal, frontal, transverse
3 components
muscular symmetry
rotational angular breathing
awareness of posture
methods include - self elongation and postural correction to specific curve patterns

37
Q

what are the indications for surgical intervention patients with scoliosis

A

curve above 40 degrees and non responding

38
Q

describe the surgical intervention for idiopathic scoliosis

A
  • Intervertebral fusion using bony grafting
  • Held in place with rods until fused
39
Q

describe the discharge plan for children post op scoliosis correction surgery

A

day 1 walk w/o brace
day 4 - d/c
4/52 - return to school
6-9/12 - return to sport - no contact may be advised

40
Q

what are the apophyses

A

secondary ossification centre
Fuses with bone in second decade
Site of insertion for tendons and ligaments

41
Q

common apophysis locations of lower limb

A

iliac crest
ASIS
AIIS
Ischial tuberosity
pubic symphysis
lesser and greater trochanter
inferior patellar pole
tibial tuberosity
calcaneal tuberosity
5th metatarsal base

42
Q

apophysitis

A

Vulnerable to ‘overload’ in early adolescence
inflamamtion of apohysis

43
Q

severs disease

A

inflammation in TA insertion

44
Q

Sinding Larsen Johansson Syndrome

A

Distal Patella inflammation

45
Q

Osgood Schlatters Disease

A

Tibial Tubercle inflammation

46
Q

Iselin disease

A

base of 5th meta-tarsal

47
Q

what is the typical presentation of apophysitis

A

subjective - pain, participation limiting, can be from overloading, higher suspicion of red flag indicators
objective - observe swelling,
palpation - isolated pain in apophysis
biomechanics - hop, single leg squat, landing, cutting,
ROM, MMT

48
Q

Intervention for apophysitis

A
  • Reduce training load
  • Focus on movement control/quality, S+C etc
  • Graded return to load in a controlled manner
  • Persistent pain, exostosis and pseudoarthrosis can occur if not
    managed well
49
Q

what is apophyseal avulsion fracture

A

sudden forceful contraction causes avulsion of apophysis to which tendon attaches
typically in pelvis

50
Q

what is the typical presentation of an apophyseal avulsion fracture

A
  • May describe pop, pain and sudden loss of function
51
Q

what is the intervention of an apophyseal avulsion fracture

A

Generally managed with immobilisation and graded return to load

52
Q

clinical signs of FAIS

A
  • Restricted flexion (+ adduction and internal rotation) - FADIR
  • CAM defomity may begin to develop in adolescence due to plastic
    nature of femoral neck
53
Q

What possible physio interventions are available for FAIS

A

Current drive is to use graded exposure, loading/strengthening approaches
+/- biomechanical intervention

54
Q

what surgical interventions are available for FAIS

A
  • Excise CAM/Acetabular osteophytes & overgrowth - Femoral or pelvis osteotomy
  • Repair any labral damage
  • Outcomes not great
55
Q

types of FAIS

A

Cam impingement - overgrowth of neck of femur
pincer impingement - overgrowth of socket of pelvis

56
Q

describe epidemiology of pars defect fracture/aponylolysis

A
  • 6-7 % incidence in adolescent
    athletes
  • 47%+ cause of LBP in adolescents
57
Q

in what athletes is pars defects fracture/spondylolysis common

A

Repeated extension in LSP – javelin, fast bowler, tennis

58
Q

how can spondylolysis turn into spondylolisthesis

A
  • Bilateral spondylolysis may result in anterior translation of vertebral segment
59
Q

what is the management of undisplaced fracture of spondylolysis

A
  • Rest
  • Graded exposure
  • Biomechanical re-ed
60
Q

what is the possible intervention of pars defect displacement

A

possible surgical intervention

61
Q

there types of musculoskeletal tumors

A
  • Rhabdomyosarcoma
  • Ewings Sarcoma
  • Osteosarcoma
62
Q

what is rhabdomyosarcoma

A

cancer of the muscle or connective tissue e.g. tendon or cartilages

63
Q

describe epidemiology of rhabdomyosarcoma

A

50% in head and neck, 25% in genital area, 15% in extremities

64
Q

describe signs of rhabdomyosarcoma

A
  • A lump or swelling that keeps getting bigger or does not go away. It may be painful.
  • Crossed-eyes or bulging of the eye.
  • Headache.
  • Trouble urinating or having bowel movements.
  • Blood in the urine.
  • Bleeding in the nose, throat, vagina, or rectum.
65
Q

Ewings sarcoma

A

cancer of bones and tissue around bones

66
Q

what are the common locations for Ewings sarcoma

A

bones of the legs,
arms,
feet,
hands,
chest,
pelvis,
spine,
skull.

67
Q

signs for ewings sarcoma

A
  • A lump (which may feel soft and warm) in the arms, legs, chest, or pelvis.
  • Pain and/or swelling near the lump.
  • Fever for no known reason.
  • A bone that breaks for no known reason
68
Q

treatment ewing’s sarcoma

A

excision of tumour + chemo and radiation
* May necessitate amputation of limb

69
Q

osteosarcoma

A

most common type of bone cancer
common in adolescents

70
Q

where does osteosarcoma typically develop in the body

A

Commonly forms in the
ends of the long bones of
the body often near
the knee

71
Q

signs of osteosarcoma

A
  • Swelling over a bone or bony part of the body.
  • Pain in a bone or joint.
  • A bone that breaks for no known reason
72
Q

subtypes of juvenile idiopathic arthritis

A

oligoarthritis - < 4 joints
polyarthritis > 5 joints
Psoriatic arthritis - joints and rash
enthesitis - tendon insertion
undifferentiated - not fitting with subtype

73
Q

what pharmaceutical interventions are available for juvenile idiopathic arthritis

A
  • Pain control meds
  • Non-steroidal anti-inflammatories
  • Disease Modifying Anti-rheumatic drugs (DMARDs)
  • Biologic Therapies
  • tanercept, adalimumab, infliximab, tocilizumab, abatacept, anakinra
  • Intravenous infusion